Cardiovascular Flashcards

(112 cards)

1
Q

Arteriosclerosis:

A

is a generic term used for hardening of arteries and arterioles. May
involve any artery in the body and also may affect arterioles. A degenerative arterial
disease where muscle and elastic tissue of the tunica media are replaced by fibrous
tissue. The arterial walls become thickened, hard, and lose elasticity.

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2
Q

Atherosclerosis:

A

s a multifactorial disease of arteries
affected by atheromas. Affects only the aorta and its
major branches. Abnormal masses of lipids, complex
carbohydrates, blood and blood products, fibrous
tissue and calcium deposits in the tunica intima of
arteries not arterioles. Note: arteriosclerosis and
atherosclerosis often co-exist.

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3
Q

Coronary atherosclerosis:

A

degeneration of coronary
arteries with gradual decrease of the lumen (e.g. increased
fibrotic tissue, decreased elastic tissue; thrombus may
develop).

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4
Q

Atherosclerosis/Arteriosclerosis: Sign/Symptom

A

likely no symptoms present until the vessel is
blocked or narrowed enough to alter blood flow or thrombus/embolism formation.
Arteries in the heart may cause angina or heart attack. Symptoms: chest pain, difficulty
breathing, restlessness, dizziness, anxiety.

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5
Q

Ischemic heart disease:

A

insufficient coronary blood flow (a.k.a. heart attack,
myocardial infarct = necrosis of part of the heart due to occlusion of the coronary
arteries)

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6
Q

Coronary thrombosis:

A

occlusion of the coronary artery. Very little anastomosis

and/or collateral circulation exists in cardiac muscle.

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7
Q

Arteries that supply head/brain, may experience_______ if embolism occurs

A

TIA or stroke

weakness, numbness, slurred speech

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8
Q

Arteries in the arms/legs patient may experience pain in arms or legs and ___________ (peripheral vascular disease)

A

intermittent

claudication

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9
Q

Aneurysm

A

abnormal dilation of blood vessels. If vessel walls become weakened,
aneurysms may occur – potential to rupture vessel, excessive bleeding and potential
organ damage or fatal. Sudden severe pain in head or abdomen (brain, aorta)

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10
Q

Hypertension:

A

elevation of systolic and/or diastolic BP, either primary (85% due to
unknown cause) or secondary (15% is secondary to renal disease, adrenal cortical
tumour).

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11
Q

High BP threshold is______ ; “dangerous” is

_______

A

140/90

160/95

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12
Q

Signs and symptoms

HBP

A

 Asymptomatic until complications develop
 Variable depending on organs affected
 Dizziness, light-headedness, HA, fatigue, facial flushing and
personality changes can all occur
 Possible edema in lower extremities

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13
Q

The cardiovascular system consists of the

A

heart and blood vessels: arteries, capillaries,

and veins.

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14
Q

It’s estimated there are approximately_______of blood vessels in the
human body.

A

100,000 km

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15
Q

The cardiovascular “loop” transports

A

 oxygen, nutrients and hormones to each cell

 carbon dioxide and metabolic end products out from each cell

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16
Q

Factors that affect the normal function of the heart or blood vessels potentially
predispose the cells to poor nutritional status. Heart disease including:

A

 Angina
 Hypertension
 Valvular dysfunction

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17
Q

diseases of the vasculature that compromise tissue health such as:

A

 Arteriosclerosis

 Atherosclerosis

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18
Q

The massage therapist treating a patient/client/guest with a cardiovascular
dysfunction has many challenges. Techniques and modalities used should not:

A

 Increase the work of the heart: e.g. by dramatically increasing venous return
 Increase the risk of local tissue damage: e.g. XFF can cause prolonged bleeding in
malnourished tissue
 Interact with the use of medications: e.g. vasodilators and hydrotherapy
 Increase the risk of a secondary systemic complication: e.g. dislodging a thrombus

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19
Q

Cardiovascular Conditions: History:

A

Diet, health habits, medications, recent MD visits, fitness level,
general health, pain at rest, intermittent claudication, ask about
respiratory status

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20
Q

Cardiovascular Conditions: Observation:

A

Edema, varicosities, facial colorations, extremity
coloration (skin color: flushing, cyanosis/grey/white caused by poor
arterial supply or hypoxia). Dyspnea caused by pulmonary edema
resulting in labored or difficult breathing – may be related to cardiac
insufficency

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21
Q

Cardiovascular Conditions: Palpation:

A

Pulses – assess if they are normal, absent, dimished,
bounding (compare bilaterally). Bradycardia (decreased) or tachycardia
(increased), temperature of extremities and core, clamminess and
sweating due to sympathetic/reflexive changes, edema (pitting vs non
pitting)

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22
Q

Cardiovascular Conditions: Movements:

A

may assess intermittent claudication if client can not tell

you

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23
Q

Cardiovascular Conditions: Neurological:

A

TOS

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24
Q

Cardiovascular Conditions:

Referred Pain:

A

MI refers to shoulder, down arm and can along back and
sternum. Trigger points can also refer to these areas (pec mj/mn,
serratus anterior, scalene, levator scapula etc)

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25
Special tests:
Central cardiovascular: blood pressure, respiratory status, Peripheral vascular: capillary refill test, allen’s test, manual compression test, pulses, leg raise for varicose veins, pitted edema test, homan’s test
26
How to find pulses: Use the ___ of your finger, not the____ . Don’t push _____ or you can _____ the vessel.
pad /tip too hard/occlude (Sometimes, using a number of fingers along the course of a vessel, and applying firmer pressure with the distal fingers will allow you to feel the pulse better in the proximal fingers. It also may help to use on hand to feel a pulse that’s easy to find while using the other hand to find the harder one)
27
Compare pulses _______.
bilaterally
28
To record pulses: “____” if strong, “_____” if weak, “_____” if absent.
2/2 1/2 0/2
29
Brachial pulse:
ask pt to flex the elbow to feel the biceps brachii tendon on the cubital fossa. Apply fingertip pressure on the medial side of the tendon. The pulse may be more superficial the more proximal you go. Brachila artery used to be the axillary artery
30
Radial pulse:
on the lateral side of the wrist, find the bony prominence of the styloid process . roll your finger medial from this and the artery runs along the bone. The radial artery is a branch of the brachial artery
31
Ulnar pulse:
on the medial side of the wrist find the pisiform bone. Roll your finger laterally from this and the artery runs along here. Harder to find than the radial artery. This is a branch of the brachial artery.
32
Femoral pulse:
along the inguinal ligament, about halfway from the ASIS to | the pubic tubercle. Used to be called the iliac artery
33
Popliteal pulse:
difficult to find – deep. Slightly medial to the center of the popliteal fossa. Used to be femoral artery
34
Anterior tibial pulse:
feel along the anterior shin lateral to the tibia. It is | easiest to feel halfway down the shin. Branch of the popliteal artery
35
Dosalis pedis pulse :
on the top of the foot about 1/3 of the way to the toes, feel laterally to the extensor hallucis longus tendon. The artery runs along side the tendon. This is a branch of the popliteal artery.
36
Posterior tibial pulse :
on the medial side of the ankle find the medial malleolus. Roll your finger towards the angle of the heel and feel for the pulse.
37
Common carotid pulse
only check one at a time. Find the bottom of the | lateral edge of the thyroid cartilage. Then feel 1-2 cm lateral to that
38
External carotid pulse :
only check one at a time. About 2 cm anterior of the | scm along the fold of the jaw line.
39
Temporal pulse:
find the top part of the ear that is attached to your face | (about eye level) and about 1 cm anterior is the pulse
40
What techniques would be safe to perform on a patient with central cardiovascular disease?
MFR depending on tissue health PROM, Stretches, Joint mobs if indicated Swedish/petrissage techniques modified to short and segmental strokes
41
What are red flags to treatment? Consider that pts may be in denial.
Medications Scarring Ask about surgical history, family history of cardiovascular disease, risk factors (smoking, lifestyle)
42
Diseases affecting the blood vessels usually result in some form of
peripheral vascular | disease (PVD).
43
PVD can affect the
venous or lymphatic circulatory systems.
44
Occlusive diseases of the blood vessels are a common cause of
disability and usually | occur as a result of ATHEROSCLEROSIS.
45
Other causes of arterial occlusion include
trauma, thrombus or embolism, vaculitis or vasomotor disorders (Raynaud’s).
46
THROMBOANGITIS OBLITERANS (BUERGER’S DISEASE):
 Inflammatory lesions of the peripheral blood vessels are accompanied by thrombus formation and vasospasm occluding blood vessels.  A vasculitis, i.e. an inflammatory and thrombotic process, affecting both arteries and vein, primary in the extremities  Inflammatory lesions of the peripheral are accompanied by THROMBUS formation and vasospasm occluding and eventually OBLITERATING (destroying) small and medium vessels of the feet and hands  Cause not known Demographics- Usually found in men younger than 40 who smoke heavily
47
THROMBOANGITIS OBLITERANS (BUERGER’S DISEASE): Symptoms: Treatment Goals:
Symptoms:  Symptoms are episodic and segmental (i.e. come and go intermittently over time and appear in different, asymmetrical anatomic locations)  Intermittent claudication in the arch of the foot or the palm of the hand is often the first symptom  Symptoms include pain at rest, edema, cold sensitivity, rubor (redness of the skin from dilated capillaries under the skin), cyanosis, and thin, shiny, hairless skin (trophic changes) from chronic ischemia Treatment Goals:  Treatment goals include increasing circulation to the hand or foot BUERGER’S DISEASE
48
ARTERIOSCLEROSIS OBLITERANS:
Definition:  Proliferation of the intima causes complete obliteration of the lumen of the artery  A.k.a. peripheral arterial disease  Most common occlusive disease (about 95% of cases) Demographics:  Most often seen in elderly patients Etiology:  Associated with diabetes mellitus  Risk factors include smoking, hypertension, hyperlipidemia, obesity, and diabetes
49
ARTERIOSCLEROSIS OBLITERANS: Symptoms: Treatment Goals:
 Bilateral, progressive INTERMITTENT CLAUDICATION is usually present in muscles  Primary symptom may be a sense of weakness or muscular “tiredness”, both the pain and weakness/fatigue are relieved by rest  Pain at rest indicated more sever involvement; may be relieved by dangling limbs (usually leg) over the edge of the bed to use gravity to encourage circulation (dependent position) *Differential diagnosis: this dependent position would relieve arterial pain but would increase symptoms of a DVT (venous) Treatment:  Preventive skin care is a primary goal in treatment; avoid minor injuries, infections and ulceration  Exercise to increase collateral circulation and improved function  Diabetic neuropathy with diminished sensation of the toes or feet often occurs, predisposing the patient to injury or pressure ulcers that may progress because of poor blood flow and ongoing loss of sensation
50
RAYNAUD’S (DISEASE AND PHENOMENON)
 Intermittent episodes of small artery or arteriole constriction of the extremities causing temporary pallor and cyanosis of the digits (usually fingers)  These episodes occur in response to the cold temperature or strong emotion (anxiety or excitement)  Arterial vasospasm in the skin  constriction  pale cold skin  blood pools in surrounding tissues  bluish/purplish skin  white  red (as vessels relax and blood flows in)  warm red skin (may experience throbbing, swelling and paresthesia) Raynaud’s disease: primary, vasospastic disorder (idiopathic) Raynaud’s phenomenon: secondary to another disease or underlying cause
51
RAYNAUD’S DISEASE:
 80% of people with it are women aged 20-49 years old  Idiopathic; seems to be caused by hypersensitivity of digital arteries to cold, release of serotonin, and congenital predisposition to vasospasm  Accounts for 65% of people affected by Raynaud’s  More “annoying than medically serious”
52
RAYNAUD’S PHENOMENON:
 Predicted that 10-20% of the general population has this; usually women between 15-40 years old  Secondary to other conditions (buerger’s, connective tissue disorders, hidden neoplasms); has been known to be precipitated by use of pharmacological med’s and by exposure to temperature changes (warm to cool/cold), also injuries to hand (repetitive stress e.g. keyboarding, using crutches)  Nicotine constricts small blood vessels; smoking can trigger attacks in people predisposed to this phenomenon
53
“The therapist must be aware that spasms and cramps can be caused by pathologies and medication. In these cases, treatment modifications or referral to a physician may be indicated. For example, calf muscle cramps may occur with arterial disorders, such as
 Acute arterial occlusion due to a thrombus or embolism;  Chronic arteriosclerotic vascular disorder due to arterial narrowing and fibrosing, which is frequently associated with diabetes mellitus and with the aging process;  Thromboangiitis obliterans or Buerger’s disease, an inflammatory reaction of the arteries to nicotine, found in smokers;  And Raynaud’s syndrome, an arterial spasm due to abnormal sympathetic nervous system firing…
54
intermittent claudication or pain and cramping in the calf muscles with exercise. Intermittent claudication is due to_______. Pain and cramping are often noted when the client is walking. They diminish slowly with rest.
ischemia
55
Treatment Goals of Chronic Arterial Disease
1. Improve collateral circulation and increase vasodilation 2. Improve exercise tolerance for ADL’s and decrease the incidence of intermittent claudication 3. Relieve pain at rest 4. Prevent joint contractures and muscle atrophy 5. Prevent skin ulcerations
56
2. Improve exercise tolerance for ADL’s and decrease the incidence of intermittent claudication through.....
 regular, graded aerobic exercise program of walking, cycling, whatever they’re able to do comfortably (consider compliance)  vasodilation by reflex heating
57
3. Relieve pain at rest through.....
 sleep with the legs in a dependent position over the edge of the bed, or with the head of the bed slightly elevated
58
4. Prevent joint contractures and muscle atrophy through......
 active or mild resistance Range Of Motion exercises to the extremities
59
5. Prevent skin ulcerations through....
 educate guest in the proper care and protection of the skin, particularly the feet  proper shoe selection and fit  avoid use of support hose
60
Techniques of Examination - Peripheral arterial system | ARMS –
– inspect both arms from the fingertips to the shoulders. Note:  Size and symmetry  Color and texture of the skin and nail beds  Venous pattern  Edema (pallor or cyanosis of the fingers indicates Raynaud’s phenomenon) (edema and prominent veins occur with venous obstruction)
61
With the pads of your index and middle fingers, palpate the______ on the flexor surface of the wrist laterally. Compare the volume of the pulses on each side.
radial pulse
62
Arterial occlusion in the____ is much less common than in the _____.
arms | legs
63
If arm pulses are | markedly diminished or absent, however, consider
thromboangiitis obliterans (Buerger’s disease), scleroderma, or, possibly, a cervical rib
64
If arterial insufficiency is suspected, palpate also 1) for the ______ , on the flexor surface of the wrist medially, and 2) for the______ , in the groove between the biceps and triceps muscle above the elbow.
ulnar pulse | brachial pulse
65
Since the normal ulnar artery is frequently not palpable, the Allen Test may be useful. It tests the patency of the
ulnar and radial arteries in turn.
66
The Allen Test
Ask the patient to rest his/her hands in their lap. Place your thumbs over their radial arteries and ask them to clench fists tightly. Compress the radial arterial firmly, then ask the patient to open hands into a relaxed position. Observe the colour of the palms. Normally they should turn pink promptly. Repeat occluding the ulnar arteries. Persistence of pallor when one artery (e.g. the radial) is manually compressed indicates occlusion of the other (e.g. the ulnar).
67
The Allen Test – version 2
1) The hand is elevated and the person is asked to make a fist for about 30 seconds. 2) Pressure is applied over the ulnar and the radial arteries so as to occlude both of them. 3) Still elevated, the hand is then opened. It should appear blanched (pallor can be observed at the finger nails). 4) Ulnar pressure is released and the color should return in 7 seconds. Inference: Ulnar artery supply to the hand is sufficient. If colour does not return or returns after 7–10 seconds, then the ulnar artery supply to the hand is not sufficient.
68
Capillary Refill Test:
Check the finger nails for cracked nails and skin integrity first. Then apply pressure on nail bed for 10 seconds. Release and observe the color of the nail bed. The color should return within 7-10 seconds. If the color remains white longer then there is arterial insufficiency
69
Acute arterial occlusion due to
a thrombus or embolism;
70
Chronic arteriosclerotic vascular disorder due to
to arterial narrowing and fibrosing, which is frequently associated with diabetes mellitus and with the aging process
71
Thromboangiitis obliterans or Buerger’s disease, an....
inflammatory reaction of the arteries to nicotine, found in smokers;
72
Raynaud’s syndrome, an
an arterial spasm due to abnormal sympathetic nervous system firing…
73
Edema stimulates pain receptors because of the
tension in the tissues, blocks off | circulation inflow to the tissues and impairs mobility
74
Edema means swelling caused by
fluid in your body's tissues. It usually occurs in the | feet, ankles and legs, but it can involve your entire body.
75
Causes of edema include.....
``` local problems or may be a result of a systemic condition:  Eating too much salt  Sunburn  Histamine reactions  Heart failure  Kidney disease  Liver problems from cirrhosis  Pregnancy  Problems with lymph nodes, especially after mastectomy  Some medicines  Obstruction of lymphatic vessels  Trauma  Standing or walking a lot when the weather is warm ```
76
What is Lymph?
Lymph is the excess clear, watery interstitial fluid that is pumped through the arterial ends and not absorbed by the venous vessels. This fluid is returned to the circulation by the lymphatic system. It contains white blood cells, plasma proteins, fats and debris such as cell fragments, bacteria and viruses.
77
How Does Lymph Move?
Lymphatic vessels have a minor contractile ability that is stimulated by the vessels filling or externally by massage to the vessels. Light massage is used to not compress and close off the vessels. Most of the lymph flow is stimulated by the surrounding skeletal muscles, diaphragm and peristalsis and contraction of the arteries the lymphatic vessels are in contact with. Lymph flow is unidirectional to the heart.
78
Edema History:
causes, general health history, cardiovascular health, kidney or liver diseases, injuries (when was the injury, MOI, is the joint affected), infections, surgery, pregnancy (BP, has md or midwife assessed) how long they have had it, have they seen a doctor, are they cleared for treatment, are they on medication, underlying pathologies, ADLs, do they use any devices to assist with swelling (stockings/bandages etc), are they seeking other treatments?
79
Edema Observation:
compare bilaterally for swelling, postural observation, gait, ROM to assess affected joints, skin quality/tissue integrity, color of tissue, area affected
80
Edema Palpation:
tenderness, heat, coolness, pulses (may not be able to feel due to swelling), density/quality of edema (firm, boggy, taut)
81
Edema Movement:
Decreased ROM for joints affected
82
Edema Neurological:
nerve compression may be a factor with swelling
83
Edema Referred pain:
may be due to nerve compression
84
Edema Special tests:
girth measurement of affected area (test bilaterally if applicable), pitted edema test.
85
Girth Measurement:
Use measuring tape to record measurements and compare bilaterally. Take a few measurements as close to bony landmarks as possible to measure accurately.
86
Pitted Edema test:
Compress the tissue with the pad of your finger and hold for 5-10 seconds. Release and observe the tissue for a remained compression. If the tissue remains pitted or indented, an underlying pathology may be the cause. Compare bilaterally. For the lower extremity: You should do this in the anterior shin, behind the medial mallelous and on the dorsum of the foot.
87
Types of Venous Disorders
1. Acute Thrombophlebitis 2. Chronic Venous Disorders - chronic venous insufficiency - varicose veins
88
Thrombophlebitis
e swelling of a vein due to a blood clot. Clots may be as long as 20 inches along the wall of the vein. This is not a condition that can be treated by RMTs. The risk of dislodging the clot poses serious complications.
89
Risk Factors for Deep Vein Thrombosis
```  Age (over 40 years old and elderly)  Prolonged immobilization  Cardiac failure, stroke or heart disease  Anesthesia and surgery  Trauma, especially in the legs or pelvis  History of thromboembolism  Pregnancy and post partum state  Oral contraceptive pill  Diabetes mellitus  Lung or pancreatic cancer  Clotting disorders  Nursing home residents ```
90
What are signs and symptoms of DVTs?
May have localized redness and warmness to the area. A cord like swelling along the course of the vein. Pain at rest and increased with movement. There may also be no symptoms present.
91
DVT History:
Family history, have they been diagnosed, have they been cleared of a dvt, symptoms they experience, how long have they had them, associated calf cramping/intermittent claudication, history of recent surgery/immobilization, pregnant/post partum, heart disease, health history (DVT risk factors), pain increase with elevation, pain does not decrease in dependent position.
92
DVT Observation:
look edema, redness cord like swelling.
93
DVT Palpation:
pain, heat, swelling
94
DVT Movement:
would not do with suspected DVT.
95
DVT Neurological:
compression from edema possible
96
DVT Referred pain:
intermittent claudication
97
DVT Special tests:
Homan’s test (rule out dvt). Refer to MD for diagnosis if you suspect DVT is present
98
Homan’s test (rule out dvt)
forced dorsiflexion of the ankle exerting traction on the posterior tibial vein, causing pain.While classically described in patients with venous thrombosis of calf veins, patients with herniated intervertebral discs and many other conditions have also been noted to exhibit a positive Homans' sign. (A positive sign is present when there is pain in the calf on forceful and abrupt dorsiflexion of the patient's foot at the ankle while the knee is extended.)
99
Great and Small Saphenous Veins are frequent locations of ______ General massage is ______ over varicosities.
varicosities contraindicated
100
Evaluation of Venous Disorders
1. Girth measurements of extremity 2. Competence of the Greater Saphenous Vein 3. Tests for possible Deep Venous Thrombosis 4. Doppler Ultrasound: venogram; phlebogram
101
Define Varicose Veins
Varicose veins are swollen, twisted, and sometimes painful veins that have filled with an abnormal collection of blood.
102
Causes of Varicose Veins
 Prolonged sitting, standing, sitting with legs crossed, tight garter, girdle or waist band  Obesity, heavy lifting, chronic constipation or pregnancy  Secondary to impaired or blocked blood flow from:  Deep vein thrombosis  Congenital venous malformation  Heart failure, liver dysfunction  Abdominal tumor  Vitamen C deficieny can weaken the collagen structure of the veins
103
Varicose Veins – Symptoms
Twisted, bulging blue lines running down all or part of a leg  Legs that ache or become tired and weak, especially after long periods of sitting or standing  Restless legs or legs that are so uncomfortable that a person has difficulty standing on both feet at once  Burning or itchy skin on the legs  Legs and/or ankles that become swollen and possibly have brownish pigmentation  Leg cramps, especially in calf muscles often occur at night
104
Varicose Veins History:
Family history, have they been diagnosed, have they been cleared of a dvt, have they had recent medical intervention to vv, are they painful/sensitive to touch, symptoms they experience, how long have they had them, associated calf cramping/intermittent claudication, history of recent surgery/immobilization, pregnant/post partum, heart disease, health history (DVT risk factors).
105
Varicose Veins Observation:
look for twisting, bulging, inflammation, edema
106
Varicose Veins Palpation:
pain, heat, swelling
107
Varicose Veins Movement
would not do with suspected DVT.
108
Varicose Veins | Neurological:
compression from edema possible
109
Varicose Veins Referred pain:
intermittent claudication or nerve compression
110
Varicose Veins Special tests:
Homan’s test (rule out dvt) leg elevation or greater saphenous manual compression test
111
Tests for Varicose Veins | Leg Elevation
 Elevate legs  Observe whether “normal” bulging of vein disappears  Varicose veins will not disappear completely because the faulty vein has stretched beyond its normal diameter and length
112
Tests for Varicose Veins | Greater Saphenous Manual Compression Test
 Patient is standing. Place one hand at the superior portion of the greater saphenous vein and the other hand about 20 cm below  Compress the upper portion of the vein with your fingers and palpate with the lower fingers.  A pulse of blood in the lower hand indicates valve insufficiency