Case Pres Flashcards

1
Q

What is asthma?

A

Narrowing of the airways resulting in reversible airflow obstruction

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2
Q

Incidence of asthma?

A

5-10% all children

2-5% adults

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3
Q

How does asthma attack occur on cellular level?

A

Allergen inhalation followed by mast cell degranulaiton
- histamine and leukotrienes deposited into airway wall

Or allergen phagocytosed into antigen presenting cell
- presents antigen to CD4 T cell which interacts with B cell to produce inflammatory response

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4
Q

how mat airways narrow?

A

Bronchial smooth muscle constriction

Bronchial smooth muscle oedema

Excessive muscles secretions into the airway lumen

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5
Q

Symptoms of asthma?

A

Cough

Wheeze
- expiratory noise

Shortness of breath

Diurnal variation
- worse at times during day

Difficulty breathing out

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6
Q

What can trigger asthma?

A

Unknown

Allergens

Stress

Environmental stimul
- dust
- smoke
- chemicals

Cold air

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7
Q

what are the core asthma drugs?

A

Intermittent short acting beta adrenergic agonists
- relieve symptoms

Inhaled corticosteroids - low dose
- prevent issues

Inhaled corticosteroid - high dose

Regular long acting beta adrenergic agonist

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8
Q

How do beta adrenergic agonists work?

A

Engage with beta - 2 adrenergic receptors on walls of bronchial smooth muscle

  • reducing bronchoconstriction
  • reduce resting bronchial tone
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9
Q

Best tx for asthma?

A

Inhaled corticosteroid
- deal with all effects

  • if SABA used more than 3 times per week, use a preventer inhaler daily
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10
Q

How might one give smoking cessation?

A

3 A’s

Ask - patient about their smoking habit and record this

Advise - on health risks and benefits of smoking

Act - on patients response, point them towards services that aid with quitting smoking

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11
Q

How would i treat an asthma medical emergency?

A

A - struggling to speak
B - increased rate with wheeze
C - increased
D - alert
E - tripods

Use of salbutamol inhaler - 100mcg/puff
- repeat until attack stops

Or 10 puffs into spacer device
- if pt cannot use inhaler

15L/oxygen/minute

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12
Q

Fx of marijuana on dental treatment?

A

Generally
- poor OH, higher risk caries and periodontal disease

Prolonged tachycardia, la with vasoconstrictor

High cariogenic diet due to the effect cannabis has on the desire to eat - commonly the munchies

Increased risk of oral cancer, oral candida and infection

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13
Q

What is asymptomatic apical periodontitis

A

Inflammation at the apex of the root of a tooth that is of pulpal origin, confirms pulp necrosis.

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14
Q

Why use MCC?

A

Not in aesthetic zone

Cheap

Excellent aesthetics

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15
Q

Benefits of using a MCC?

A

Alloy limits strain the ceramic experiences

Bonded via metal oxide on surface
- alloy is more ductile so it results in less strain on the porcelain
- porcelain is hard, rigid and has excellent aesthetics

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16
Q

Prep for MCC?

A

Axial palatally - 0.5mm chamfer

Buccal - 1.3mm shoulder

Non functional cusp - 1.3mm

Functional cusp - 1.8mm

6-8 degree tapered walls

17
Q

How cement MCC, how does it work?

A

GIC cement

  • acid in GIC reacts with metal oxide on alloy to form salt
  • salt further reacts with acid to form bond to the alloy
  • very weak bond

GIC bond to tooth
- ion exchange with the calcium in enamel and dentine
- hydrogen bonding with collagen in dentine

18
Q

Properties of GIC?

A

Pros
- fluoride release
- low modulus - cervical restorations
- no contraction on setting
- bonds to tooth surface

Cons
- aesthetics not ideal
- moisture sensitivity
- low bond strength
- worse tensile and compressive strength than composite

19
Q

What did i mean by extreme canal curvature?

A

AAE - case complexity form

  • moderate canal curvature 10-30 degrees