Challenging Questions Flashcards

1
Q

What dies the effect of traumatic forces depend on?

A

Magnitude

Duration

Direction

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2
Q

What is the pathological response to traumatic occlusion?

A

Occlusal force is too great meaning that width of PDL and therefore mobility does not stabilise

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3
Q

When is intervention for mobile teeth considered?

A

If becoming progressively worse

If causing discomfort

If interfering with restorative tx

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4
Q

What are treatment options for traumatic occlusion?

A

Occlusal management
Break parafunctional habits
Spint
Address tooth symptoms

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5
Q

What is trigeminal neuralgia?

A

A chronic disorder of trigeminal nerve characterised by sudden/severe onset of sharp, shooting unilateral facial pain

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6
Q

What investigations are done for TN?

A

Blood tests:
-> FBC
-> Blood glucose

Imaging:
-> MRI
-> CT
-> PET

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7
Q

What are the causes of TN?

A

MS
Brain tumour
Aneurysm
AV malformation
Epidermoid, dermoid and arachnoid cysts

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8
Q

How do you detect a debonding bridge?

A
  • Visually- with good illumination/magnification
  • Using floss and probe
  • Movement with pushing
  • Saliva bubbles gathering at margin on pressure
  • Evidence of secondary caries at margin
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9
Q

What are the reasons for a bridge or post/core debonding?

A

Issue with cementation- moisture contamination

Unfavourable occlusion

Bruxism

Trauma

Root fracture

Wing fracture- caries under wing

Angulation and parallelism issue
-> divergent guide paths
-> prep being too minimal

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10
Q

What is a perio abscess?

A

Localised infection of periodontal pocket
-> swelling due to pus accumulation
-> caused by food packing, plaque accumulation, lack of cleansing

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11
Q

How is perio abscess differentiated from PA abscess?

A

Position of swelling

Tooth is vital in perio abscess

Poor periodontal condition in rest of mouth

No radiolucency of perio

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12
Q

How does vertical bone loss occur?

A

Plaque induces inflammation which travels from PDL to bone
-> radius of destruction is <2mm meaning only localised area of bone adjacent to affected tooth is lost (some of the septum is still present)

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13
Q

What patients are at high risk of bleeding?

A

Haemophilia patients

Anticoagulant/Antiplatelet medication

Alcoholics and ALD

Liver disease

Patient with previous history of bleeding

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14
Q

What are the treatment options for unerupted ectopic canine?

A

Leave and monitor

Remove C or create space and wait for eruption

Open exposure

Close exposure- with gold chain

Autotransplantation

XLA and replace with prostheses

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15
Q

What are the causes of ABs being ineffective in perio?

A

Bacterial resistance

Failure to penetrate biofilm without mechanical disruption

AB may not be specific to bacteria

Inadequate concentration and retention of AB at required site

Allergy

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16
Q

What are the RFs for Candida infection?

A

Erythrmatous- ill fitting denture, poor denture hygiene, wearing denture overnight

General:
Anaemia
Diabetes mellitus
HIV
Chemotherapy
Broad spectrum AB use
Inhaler with no spacer or rinsing

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17
Q

What are the indications as per FDS2020 for extraction of M3M?

A

Infection- 1 or more episodes of pericoronitis

Caries- making tooth unrestorable

Periodontal disease

Radicular or dentigerous cyst formation- if XLA will help prevent expansion or recurrence

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18
Q

What extra post-op complications can happen following extraction in the upper?

A

Tuberosity fracture

OAC

Root in antrum

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19
Q

What extra post-op complications can happen following extraction in the lower?

A

Altered/loss of sensation (P/T) to lower lip, cheek and tongue (may affect taste)

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20
Q

What are the primary and secondary HSV 1 and 2 infections?

A

Primary- PHG

Secondary- Herpes Labialis

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21
Q

What are the primary and secondary infections of Varicella Zoster (HHV3)?

A

Primary- chicken pox

Secondary- shingles

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22
Q

What are the oral effects of HHV4 (EBV)?

A

Ulceration

Glandular fever

Hairy leukoplakia

Burkitt’s lymphoma

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23
Q

What conditions can be caused by coxsackie virus?

A

Herpangina

Hand, foot and mouth disease

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24
Q

What are the symptoms of a coxsackie virus infection?

A

Pinhead vesicles of back of throat and soft palate

Sore throat

Sore head

Fever

Lymphadenopathy

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25
How is a coxsackie virus condition treated?
Rest Hydration Soft diet Analgesia
26
What is Fluorosis?
Generalised disorder characterised by hypomineralisation of enamel matrix as a result of excessive fluoride ingestion -> usually in first 8 years of life -> Diffuse opacities -> Brown and yellow pitting
27
What are the biological factors influencing masticatory performance?
Number occluding units Number of functional teeth Maximum bite force Age
28
What is the SDA?
3-5 occlusal units remaining -> Ideally 6 anteriors and 4 premolars
29
What are 5 conclusions form SDA?
Achieves mandibular stability Sufficient functions and aesthetics Achieve occlusal stability Same rate of attritive wear Same rate of bone loss
30
What are the different roles in Decontamination?
Owner- owns practice and LDU (responsible for running LDU) User- designated person that is responsible for day to day running of LDU Operator- person with authority to operate equipment and performs simple maintenance Maintenance engineer- employed to carry out maintenance and repairs when required
31
What is vitapex?
CaOH and iodoform paste
32
What are the factors which help relief crowding from primary to permanent dentition?
Further maxillary and mandibular growth Proclined path of eruption in upper incisors Leeway space Primate space
33
How is composite bonded to dentine?
Dentine conditioner (35% phosphoric acid) -> removes smear layer, opens tubules and decalcifies surface dentine DBA is applied- contains primer and adhesive Primer (bifunctional)- Hydrophilic ends bon to dentine, hydro phobic ends are exposed Adhesive penetrates dentinal surface by molecular entangled bonding to primer tails by hydrophobic interactions -> forms hybrid layer (collagen and resin) Composite can bin d to adhesive surface as both are hydrophobic
34
What are the causes of pain to biting and thermal stimuli following cavity prep? How is this rectified?
Deep prep (place lining) Pulp exposure (pulp cap/RCT) Lack of coolant- reversible pulpitis (use coolant) Undercure of composite- ingress into pulp causing irritation (cure for longer and place increments <2mm) Unfavourable contacts (check with articulating paper and adjust)
35
What are the causes of different discolouration primary teeth?
Pink- bleeding into dentine tubules Yellow- tertiary dentine being laid down Grey/dark- necrosis of pulp
36
What are we looking at when reviewing trauma radiographically?
Apical radiolucency External or internal inflammatory resorption Pulp necrosis Continued root development- height and width
37
What faults can occur when preparing a canal with SS file?
Canal blockage Apical zipping Ledging Debris extrusion Perforation File fracture
38
What are the different sizes of reciproc and their use?
R25- small canals R40- medium canals R50- large canals
39
What are the steps in using reciproc?
To 2/3rds: 3 pecks with R25, irrigate, recapitulate, patency file -> repeat Gain CWL -> Take 10 to WL- if no pre-curve use R25 -> If pre-curve- create glide path with 15 and then use R25 -> If 15 not possible- finish with hand files To length- irrigate, recapitulate, latency Check for apical gauging
40
Why is copper enriched amalgam preferred?
Corrosion resistance Strength Creep resistance Marginal integrity
41
What are the signs and symptoms of Albright's syndrome?
Fibrous dysplasia Precocious puberty Hyperthyroidisim Cushing's Cafe au Late spots
42
What are the features of Paget's?
Increased alkaline phosphatase Disturbed balance of bone formation and resorption Bony swellings Nerve compression Ill-fitting dentures Blindness
43
What are the phases of Paget's?
Osteolytic Mixed Osteosclerotic
44
What are the features of Cherubism?
Autosomal dominant inheritance Fibro-osseus condition Progressive, painless bilateral joint swelling in childhood
45
What are the physical forms of Orofacial pain?
TMD Neuropathic pain- TN Myofascial pain syndrome Neurvascular pain disorders- Migraines
46
What are the psychological forms of Orofacial pain?
Mood and anxiety disorders manifesting as atypical facial pain
47
What is an RPI systems function?
Relieves stress and prevents traumatic torque -> on loading, mesial occlusal rest acts and pivot point and I bar/proximal plate move around this going downwards and mesially disengaging from last standing tooth
48
What are the histological features of Sjogrens?
Lymphocyte infiltrate Loss of acini Atrophy Ductal epithelial hyperplasia
49
What are the signs of salivary malignancy?
Hard Fixed Rapidly growing Cervical lymphadenopathy Loss of weight Facial nerve palsy
50
What are the causes of post core fracture?
Trauma Unfavourable occlusion/bruxism Biocorrosion of metal post Lack of 1.5mm ferrule
51
What are the different types of provisionals?
Custom made temporary- pro temp and putty matrix Prefomed temporary- malleable composite, polycarbonate, transparent crown and composite Bonding fractured tooth or old indirect restoration Immediate denture or over denture
52
What are the medical issues associated with Down's?
Congenital heart defect Hypothyroidism Coeliac Epilepsy
53
What are the EO features of down's?
Oblique palpebral fissures Almond shaped eyes (epicanthic fold) Flat nasal bridge Eyes set wide apart Brushfields spots Small head Short thick neck Atlanto-axial instability Palmar crease Small dysplastic ears
54
What are the IO features of Down's?
Hypodontia Microdontia Enamel defects Delayed eruption Macroglossia CLO High vaulted palate High caries risk and perio risk
55
What is done in terms of prevention for patient with down's?
FS FV F supplements Hygienist Bitewings
56
What are the features of primary teeth to consider when restoring?
Larger pulp Thinner enamel and dentine Shorter distance from occlusal surface to pulpal floor Ribbon shaped canals More curved canals Position of apical foramen changes
57
What is the function of the twin block? (C2D1)
Retroclination of upper incisors Proclination of lower incisors Distal migration of uppers Mesial migration of lowers Restrains maxillary growth Encourgages mandibular growth (postures and muscular forces) -> most of change is dentoalveolar with minor skeletal change
58
What are the effects of supernumeraries on permanent dentition?
Root resorption Mobility Impaction Failed eruption Delayed eruption Ectopic positon Crowding
59
What are the general risk factors for periodontal disease?
Genetics/FH Smoking Pregnancy Diabetes Immunocomprimised Drugs Malnutrition Stress
60
What are the treatment options for furcation involvement?
Palliative Repair Resective tx Extraction Regeneration
61
What are the functions of splints for TMD?
Diagnostic tool Cognitive awareness of parafunction Dentition protection Reduced TMJ loading -> postures condylar head forwards in articular fossa
62
What is bracing in RPDs?
Any rigid components of a denture which provide resistance to lateral movement by contacting afainst vertical anatomical structures (tooth/residual ridge)
63
What is reciprocation?
Any component which acts to prevent displacement of denture by active retention forces -> contacts tooth while clasp flexes over bulbosity
64
What are the different types of impression materials?
Polyether- Impregum (philic) Silicones- PVS (phobic) Compound (phobic) Irreversible hydrocolloid- alginate (philic)
65
What is the dahl effect?
Loclaised placement of appliance and resotration in anterior region -> increases inter-occlusal space posteriorly allowing further eruption -> takes 6 months -> Increases OVD
66
What are the contraindications for the Dahl Effect?
Root resorption Perio RCT teeth Implants Post-ortho Bisphosphonates Fixed conventional bridgework TMD
67
What are the issues with SDA in perio patients?
Remaining teeth could drift distally Increased anterior load Teeth already poor prognosis (not suitable for SDA concept)
68
Reasons for using Nickel Chrominium alloy in RBB?
Similar thermal expansion to procelain Corrosion resistant Can be manufactured in thin cross section and cope with occlusal load Can be sandblasted and etched with current for extra retention
69
What are the issues with subgingival or alveolar preparations?
Limited tooth structure to bond to Limited access and visualisation Challenging mositure cintrol Issues with impression Issues assessing marginal integrity Issues removing excess cement
70
What are the different types of preps for resin bonded bridges?
Light- no prep Moderate- cingulum undercut removal, 0.5mm supragingival chamfer margin Heavy- cingulum rest seat prepared, 0.5mm palatal reduction, 0.5mm supragingival chamfer margin, proximal grooves -> 180 wraparound prep
71
What are the issues with mandibular displacement?
Alters growth and developmetn of jaw musculature Potential TMJ instabilty Attritive tooth wear
72
What may you wabt to find out about if a child patient presents with ulcers?
Medical conditions Nutirent defieciency Diet OH regime Are there lesions elsewgwre Systemic symptoms When they started Exacerbating and releiving factors Trauma/biting
73
Whar are the causes of ROU?
Anaemia Trauma Stress Behcets
74
How does PHG present?
Painful ulcerative lesiosn on mucosa and gingivae -> swelling -> bleeding -> yellow vascular lesions
75
What are the sequalae of dental trauma?
Mobility Root fracture Root resorption Pulp necrosis
76
What may hep you determine the aetiology of discolouration?
Trauma Hx Dental Hx Diet diary MH- porphyria, tetracycine staining etc Special investigations- radiographs, sensibilty testing, percussion notes
77
What are the ADV of CoCr as a denture base?
Corrosion Resistance Rigid High thermal conductivity Mechanical retention provided by clasps One piece casting Strong in thin section
78
What are the histological features of malignancy?
Dysplasia Atrophy Candida infection
79
How is HPV infection sampled?
Oral rinse
80
What are the red flags for parotid malignancy?
Deep fixation Rapid enlargement Facial nevre palsy Cervical Lymphadenopathy
81
What is a benign and malignant tumour of the parotid gland?
B- Pleomorphic adenoma M- Mucoepidermoid Carcinoma
82
What is a benign and malignant tumour of upper lip?
B- Monomorphoc adenoma M- Adenoid cystic carcinoma
83
What is a benign and malignant tumour of soft palate?
B- Pleomorphic adenoma M- Mucoepidermoid carcinoma
84
What are the indications for a URA?
Malocculusion can be corrected by simple tipping Only 1/2 teeth to be moved- baseplate provides adequate anchorage Sufficient space available
85
What is done prior to carrying out microabrasion?
Radiograph SHADE recording Clinical photograph Diagram of defect Percussion note Sensibility testing
86
What is a risk ratio?
Probability of an increased or reduced risk of an outcome occurring in one group compared to another
87
What is an odds ratio?
A measure of association between exposure and outcome
88
How long is a consignment note kept for?
3 years
89
What is contained in a consignment note?
Quantity and contents of wast e Waste origin Transport Waste destination
90
What info is required to send to the lab for a bridge?
Putty wash impression Oclusal registration Material Design Shade
91
How is a metal post/core cemented?
GIC
92
How is a porcelain veneer cemented?
Light cure/dual cure composite luting agent with silane coupling agent
93
How is a fibre post cemented?
Dual cure composite luting cement
94
How is a prescription written?
Date Pt name, DOB, age Pt address Practice address Name of drug Preparation Strength Dose and frequency Total quantity to be supplied Total length of time for prescription Signed- draw lines in space underneath
95
How long does a controlled/uncontrolled drug prescription last?
Controlled- 28 days Uncontrolled- 6 months
96
What other conditions is denture stomatitis associated with?
Angular cheilitis Candida leukoplakia Media rhomboid glossitis
97
What medical conditions are associated with Candida infection?
Asthma- inhaler use Microcytic anaemia Immunocompromising conditions -> Diabetes, HIV, chemo/radio
98
What are the ADV/DIS of oral swab?
ADV: Site specific Avoids perioral contamination DIS: Invasive Uncomfortable Not full mouth representation
99
What are the ADV/DIS of an oral rinse?
ADV- full mouth representation DIS- contamination of sample
100
What instructions are given to the lab on suspected Candida infection?
Please culture sample and assess anti-fungal sensitivity and typing
101
How does secondary infection of HSV occur?
Following primary infection- HSV enters peripheral trigeminal ganglia neurons HSV releases viral DNA into nucleus to establish latency -> HSV1 genomes persist in nucleus Stimuli results in reactivation of latent virus in infected ganglia Virus travels to axonal shaft and tip at periphery where they are released to cause blisters and sores
102
What are the triggers for HSV reactivation?
Stress Fever UV Trauma
103
What conditions may we test for when investigating recurrent aphthous ulcers?
Neutropenia Anaemia Nutrient deficiency Inflammatory markers Thyroid autoimmunity
104
What are the effects of cocaine mixed with adrenaline in LA prep?
Cocaine enhances effect Increased heart rate Increased myocardium oxygen demand Coronal artery vasoconstriction Increased risk of stable angina, MI, arrhythmia
105
How is MRONJ treated?
Analgesia Resect necrotic bone Irrigate and debride (saline) Primary closure CHX Prophylactic AB
106
How is MRONJ prevented?
Preventive regime Avoid extractions Atraumatic extraction technique Avoid trauma
107
What are the uses of URA?
Simple tipping of teeth Habit breaker Correction of oB Retainer Space maintainer
108
What other space maintainers are used in ortho?
Fixed palatal arch Nance button
109
What is substantivity?
Capacity of a chemical agent to continue its therapeutic effect iver prolonged time -> dependant on conc and absorption to oral tissues
110
What are the uses of CHX?
Post extraction ANUG Candidiasis Aphthous ulcers Vesiculobullous conditions Endo irrigant Testing Dam Pericoronitis irrigation
111
What is SDA concept reliant on?
Occlusal stability- stable occlusal contacts of equal intensity in centric occlusion -> not provided by sever class 2 or 3
112
What is mandibular displacement on closure?
Mandible deviates from initial path of closure to progress from first occlusal contact to RCP when closing on retruded arc of closure
113
What is herpes labialis?
Manifestation of reactivation of latent HSV1 -> labial sores, blisters, ulcers
114
When should we biopsy LP?
If symptomatic If patient is smoker Red patches
115
What are the topical/systemic treatments for LP?
Topical- hydrocortisone, betamethasone, beclamethasone Systemic- Prednisolone, hydroxychloroquine
116
Which variants of LP have more malignant potential?
Erosive and gingival variants
117
What is Grinspan's syndrome?
Oral lichen planus Hypertension T2 Diabetes
118
How is LP distinguished from Oral lichenoid reaction?
Both clinical and histopathological confirmation required
119
What are the symptoms of Sjogren's?
Tiredness and fatigue Dry mouth- issues eating/speaking/swallowing/tatse, caries, fungal infections Joint pain and aches Tiredness Swollen salivary glands Dryness of skin and digestive tract Dry and sore eyes
120
What are the signs/symptoms of Behcet's?
Oral ulceration- similar to RAS Genital ulceration Occular inflammation Fatigue Thombosis risk Joint pain Headache
121
What gene is behcets associated with?
HLA-B51
122
What immune cells are involved in hypersensitivity reactions?
T1- IgE T2- IgG, IgM, complement T3- IgG and complement T4- T cell
123
What is erythema multiforme?
Acute immuno-mediated inflammatory mucocutaneous disease -> likely T3/T4 rxn to trigger- infection/drugs
124
What are the symptoms of EM?
Oral ulceration/skin lesions- target Lip crusting Flu-like symptoms prior to oral and skin lesions
125
How is EM treated?
Antiseptic mouthwash- CHX Analgesic mouthwash- benzydamine Topical corticosteroids Systemic corticosteroids- if persistent or recurrent Changing medication AB/AV if infection is cause Azathioprine- very severe
126
Types of EM?
Isolated Recurrent- >6 episodes per year Persistent- continuous with no interruption
127
What is SLE?
Autoimmune multi system condition -> systemic inflammation and tissue damage -> Broad spectrum of manifestations
128
What are the signs and symptoms of SLE?
Fatigue Fever Splenomegaly Butterfly rash Weight loss Arthritis OP Myalgia Lupus nephritis Uveitis/scelritis Pericarditis Pulmonary disease- lupus pleuritis GI issues- IBD, coeliac
129
How is SLE treated?
Prednisolone Hydroxychloroquine Methotrexate Rituximumab (monoclonal antibody)
130
What is the clinical appearance of OSCC?
Ulceration Speckled Exophytic Easily bleeds Doesnt heal Indurated Fixed Uneven/rolled margins
131
What are the symptoms of late stage OSCC?
Ulceration Pain Trismus Decreased tongue mobility Increased tooth mobility