Chapter 3. Cerbrovascular Accidents (CVA) Flashcards

(43 cards)

1
Q

Define STROKE

A

Sudden, FOCAL neurologic deficit, resulting from ISCHEMIA (clogged blood vessel) or HEMORRHAGE (ruptured blood vessel)

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2
Q

What are (3) Etiologys of a CVA

A
  1. Thrombosis (solid mass of platelets and/or fibrin that forms locally in a vessel-clogging blood flow)
  2. Embolus (a piece of a thrombus that has broken free and is carried toward the brain by the bloodstream-blood clot, air bubble, piece of fatty deposit
  3. Hemorrhage (ruptured blood vessel in brain)
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3
Q

It is important to know if you have a __ or ___ before giving a drug against CVA

A

-HEMORRHAGE or CLOT

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4
Q

What is the Mechanism of a Stroke

A
  • Atherosclerosis is formation of PLAQUE
  • vessel narrowing
  • stenosis (abnormal narrowing) ulceration of lesions, thrombus, EMBOLI
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5
Q

What percentage of formation of clot is in Cerebral arteries or smaller branches in a THROMBUS?

A

60%

-can still have COME Collateral formation

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6
Q

What is collateral formation

A

Extra PATHWAYS for blood to flow during a Thrombus etc.

-survival technique by the body

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7
Q

WHERE does an embolism come from and WHAT is it

A
  • 16% comes from heart, internal carotid, or carotid sinus

- it is a dislodged thrombus

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8
Q

What is the most common place for a stroke

A

MCA- Middle Cerebral Artery

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9
Q

How do Thrombus form

A

Forms from platelet adhesion and coagulations, leading to ischemia with infarct

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10
Q

What is a Hemorrhage

A

abnormal bleeding due to vessel rupture

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11
Q

Is there a chance to develop new Collaterals when an EMBOLISM is formed

A

No, because the embolism happens so quickly

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12
Q

A Thrombus can lead to ___ and then further on even lead to an __

A
  • ischemia with infarct

- Embolism

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13
Q

WHAT occurs within hours, peaks at 4 days due to tissue necrosis/rupturing of cell membranes and can push against your pons (breathing center), making it extremely hard to breath

A

Cerebral edema (swelling in the brain)

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14
Q

What is the acronym for TIA and what is the possible cause

A
  • Transic Ischemic Attack

- Transient Systemic Arterial hyoptension

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15
Q

What are some symptoms/signs of ANTERIOR Cerebral Artery Infarct

A
  • LEFTside neglect
  • Contralateral hemiparesis (LE)
  • Sensory loss (LE)
  • Incontinence
  • Apraxia (can’t move arm)
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16
Q

What are some symptoms/signs of MIDDLE Cerebral Artery Infarct (most common!!)

A
  • Contralateral hemiplegia (UE)
  • Sensory deficit (UE and face)
  • Homonymous Hemianopsia (loss of half of the field of view in both eyes)
  • If left hemi»aphasia
  • if right hemi» neglect on left side
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17
Q

What are the 2 possibilities for an Internal Carotid Artery Infarct

A
  • Comma

- Death

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18
Q

What is ‘Contralateral Hemianesthesia’ in POSTERIOR Cerebral Artery

A

Contralateral (opp. side of body)

-anesthesia on one side of the body

19
Q

‘Locked in syndrome’ comes from which artery infarct; and what is it?

A

VERTEBRAL Artery Infarct

  • can last for months/weeks/years
  • quadraplegia
  • only has vertical gaze
  • can’t speak, yet full consciousness
20
Q

What are the (5) Artery infarcts?

A
  1. Anterior
  2. Middle
  3. Posterior
  4. Internal
  5. Vertebral
21
Q

What is Ashworth’s Scale for Grading SPASTICITY for 0 and 4

A

0- no increase in tone

4-affected parts RIGID in FLEXION or EXTENSION

22
Q

What is Brunnstrom Synergy Pattern for UPPER extremity flexion

A
  • scapular retraction
  • shoulder external rotation
  • shoulder abduction to 90 degrees
  • elbow flexion
  • forearm supination, wrist and finger flexion
23
Q

What is Brunnstrom Synergy Pattern for LOWER extremity extension

A
  • hip extension
  • adduction and internal rotation
  • knee extension
  • anke plantar flexion and inversion
24
Q

Define Dysarthria

A
Difficulty speaking (but physically)
-may have affected respiration, articulation, possible impaired chewing/swallowing
25
Define Dysphasia/Aphasia
Difficulty speaking- but it affects the BRAINSTEM
26
What are some LEFT hemisphere Motor Programming deficits
- apraxia - difficulty in squencing - difficulty initiating movement - harder to learn task
27
What is a RIGHT hemisphere Motor Programming deficits
-difficulty in sustaining movement/posture
28
What are some COGNITIVE and BEHAVIORAL deficits
- orientation - attention - STM
29
What are 5 deficits with a stroke?
1. Speech and Language 2. Motor Programming 3. Cognitive and behavioral 4. Perceptual 5. Psychological
30
What are 3 Psychological difficulties after a stroke
- frustration - depression - anxiety
31
What is RSD and what does it stand for?
- Reflex Sympathetic Dystrophy - warm, red,glossy skin - stiffness
32
What are (5) early CVA Rehab Goals
- mouth and breathing exercises - maintain ROM - increase trunk control, symmetry - increase functional mobility - initiate self-care
33
What are 3 way to treat spasticity
- dont tuch palm/sole of foot - heat - tapping/stroking antagonist muscle
34
What is an AFO
Ankle Foot Orthotic: mots COMMON lower extremity brace
35
What are 2 important factors in the Acute Phase sessions in Rehab
-Keep it SHORT and FREQUENT
36
What are (4) treatments techniques used in the ACUTE Phase
- TACTILE (frequent contact, diff. materials) - PROPRIOCEPTIVE, KINESTETHIC AND VESTIB STIM (position, keep head in midline, frequent body position changes - VISUAL STIM (decorate room with familiar objects) - AUDITORY STIM (speaking to patient)
37
What are some treatment techniques in the SUBACUTE Phase for agitated patients ?
- human contact - familiar voices (sudden loud voices should be avoided) - excessive sensoy stim should be avoided due to confusion
38
INHIBITION techniques appropriate for SUBACUTE PHASE
- wrap limbs in warms blankets - slow rocking - have patient in "reflex inhibiting postures"- have sternum and lumbar spine allignment
39
Know 2 inhibition techniques for the lower extremity (to break up spasticity)
- hip and knee flexion - ankle dorsiflexion and eversion * exactly opp. of Brunnstrum Synergy Pattern
40
List 2 techniques for general inhibition for stroke patients
- calm colours | - change positions Gradually
41
What are (4) Goals at a LATE Phase of Rehab
1. Increase cognitive skills 2. Improve patients time management skills 3. Improve patients Fine motor coordination 4. Increase the patients speed of performance
42
What is one of the most important factor in the LATE Phase of Rehab
-provide a PURPOSE for each activity
43
What are 5 ways in which to refine motor control
- biofeedback - electrical stim - cutaneous (skin) stim - PNF - Joint mobility