Chapter 5 Flashcards

(57 cards)

1
Q

Taste aversion learning

A

After becoming sick due to food, the item itself becomes a warning stimulus for sickness

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2
Q

What are the explanations for taste aversion

A

Laws of classical conditioning
Law of effect (probability to repeat a situation based on outcome)

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3
Q

Problems with classical conditioning explanations of taste aversion

A

One trial acquisition (one count food poisining)
Delayed conditioning (doesn’t get sick for 10+hours)
Selectivity in taste aversion learning (wont link with other paired stimuli)

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4
Q

Is extinction more likely to occur in classical conditioning or one shot learning?

A

Classical conditioning
One shot learning is highly resistant

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5
Q

Blocking ( the rescorla-wagner model)

A

Group A: group B
pre-training- none. Pre training- noise = shock
Conditioning -noise + light = shock. Conditioning -noise + light = shock
Testing-light. Testing-light
Response- freezing (high fear). Response - Bar pressing (no fear)

Fear of light is not learnt bc associative strength has been used up by noise shock pairing

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6
Q

Natural selection 3 key features

A

1.all individuals are unique behaviorally and physiologically
2. Traits are heritable
3. Typically competition for important resources

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7
Q

Instinctive drift

A

Refers to the tendency of organisms to revert to instinctual ,unlearned behaviours
Ex. Training pig to drop money = inset chive drift of digging behaviour

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8
Q

Hindbrain

A

Lower brain stem responsible for basic physiological functions such as respiration and heart rate
Houses cerebellum which is involved in locomotion and balance

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9
Q

Midbrain

A

Upper brain stem responsible for waking and sleeping and for controlling general arousal
Nerve fibers associated with movement

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10
Q

Forebrain

A

Largest most complex brain structure
Includes: hypothalumus,thalamus, limbic system, cerebrum, cerebral cortex

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11
Q

Learning can take 4 basic forms

A
  1. Perceptual learning
  2. Stimulus-response learning
  3. Motor learning
  4. Relational learning
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12
Q

Perceptual learning

A

Any info coming in through senses and being processed

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13
Q

Stimulus- response learning

A

Ability to learn to perform a particular behaviour when a particular stimulus is present
Involves establishment of connections between circuits involved in perception and movement

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14
Q

Neural model of classical conditioning

A

Sensation (puff of air to the eye) → Neuron in somatosensory (eyes) natural response is to blink. Therefore synapse is strong

Sensation ( 1000-hz tone) → neurons in auditory system, no natural response of blinking to a tone. Therefore synapse is weak - however, it can be strengthened over time through classical conditioning

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15
Q

Hebb rule

A

If synapse repeatedly becomes active at same time that the post synaptic neutron fires, changes will take place in structure + chemistry of the synapse that strengthen it

If weak and strong neuron are activated together. Weak neuron will eventually become strong

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16
Q

Classical vs instrumental conditioning

A

Classical: reward is delivered independently of behaviour
Instrumental: behaviour causes reward to appear

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17
Q

Neural model for instrumental conditioning

A

Stimulus → neural circuit that detects and processes a particular stimuli (perceptual system) → neural circuit that controls a particular behaviour (motor system) → desired behaviour → reinforcing stimuli

Reinforcement system strengthens connection between perceptual and motor system

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18
Q

Motor learning

A

Changes within motor system

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19
Q

What is motor learning dependant on?

A

Sensory guidance from the environment (perceptual system)

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20
Q

Overview: perceptual, s-r, motor learning

A

Stimulus → perceptual system → s-r learning → motor learning → response

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21
Q

Long-term potentiation

A

A particular neuron changes the way its behaving in terms of its voltage
Potentiation =increase in receptors

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22
Q

Hippocampal formation ( used in memory formation)

A

Series of structures that work together in a circuit (activity in one part of formation effects activity in another part)

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23
Q

How can you measure a charge?

A

Using an electrode

24
Q

How can you give a neuron some charge?

25
How do we measure change/stimulation in hippocampal formation?
Repeatedly stimulating axon in perforant path with electrode and recording voltage from a neuron in dentate gyrus
26
What does hippocampul long term pontentiation simulate?
Hebbs rule
27
Long term depression
Low-frequency stimulation of the synaptic inputs A long-term decrease in the excitability of a nueron to a particular synaptic input Ex. After this class you want study this material (low frequency stimulation) and it will be forgotten
28
Is long term depression hyperpolarization or polarization
Hyper-polarization
29
Perceptual learning
Involves learning to recognize things (perceive) not what to do when they are present
30
Ventral stream
What? Stream Object recognition Continues into the temporal cortex
31
Dorsal stream
Where? Stream Location of objects Continues into parietal cortex
32
What responses can an aversive stimulus produce
Behavioural: freezing Autonomic: increased blood pressure Hormonal: secretion of adrenal
33
Where does the amygdala (emotion control) receive its information from?
Lateral nucleus
34
What is the primary function of the basal nucleus?
Stimulating fear response After info is received from lateral nucleus -→ the basal nucleus stimulates fear
35
After the basal nucleus receives info from the lateral nucleus, where does it go?
Central nucleus
36
What are the two major pathways between the sensory association cortex and the motor association cortex
1. Direct transcortical connections thalamus 2. Connections vid the basal ganglia and thalamus
37
What are involved in our pleasure areas?
Ventral segmental area Nucleus accumbens
38
What two functions must any reinforcement systems perform?
1. Detect presence of reinforcing stimulus 2. Strengthen connections between neutrons that detect the stimulus and ones that produce responses
39
Reinforcement: VTA
Has dopaminergic neutrons: neural chemical that results in a pleasurable feeling
40
Detection in reinforcement
Depends on physiological state of organism. Will the reinforcer dectually be of any pleasure to the organism? Is the reinforcer food and they are already full?
41
Where is the pre-frontal cortex?
Right behind the forehead
42
Where is the hippocampus
Buried deep in the brain
43
When first learning is the hippocampus or prefrontal cortex more active?
Hippocampus (activity is high at first and then decreases)
44
Prefrontal cortex activity in relation to learning
Activity in prefrontal cortex is low as learning first occurs and then it increases over time
45
Episodic memory
Memory of a collection of perceptions of events organized in time identified by a particular context Ex. Memory of having birthday dinner with a friend
46
Semantic memory
Memory of facts and general information Ex. Who the president is
47
Spatial memory
Example: remembering how to get from class to class
48
Where are place cells located
Hippocampus
49
What are place cells
Neutron that becomes active when the animal is in a particular location in the environment
50
Morris Water maze task:
Fats use environmental cues to orient themselves in space There are variable and fixed start positions If released from variable spots :relations/connections are made If released from same position: rats will eventually find the platform through stimulus response learning. At a constant start (stimulus response task): the rats with and without lesions will take the same amount of time In a variable start (relational task): lesioned animals use no relations (take longer)
51
Water maze task lidocaine injection:
They questioned what happens if hippocampus vs. Cerebral cortex were inactivated on day 1 of training vs. 30 days after training Results: Hippocampus : day 1 injection then test = no memory. Day 30 injection then test = good memory Cerebral cortex: day 1 injection then test = good memory. Day 30 injection then test = no memory
52
What is relational learning?
Putting the pieces together. "Relating things" to learn
53
Retrograde amnesia vs. Anterograde amnesia
Retrograde: can't remember events prior to brain damage Anterograde (what h.m had) can't remember events that occur after brain damage
54
Is korsakoft's syndrome a form of retrograde or anterograde amnesia?
Anterograde Can be from chronic alcohol use
55
Milner 3 assumptions about h.m
1. Hippocampus is not the location of long term memory 2. Hippocampus is not the location of short term memory 3. Hippocampus is involved in converting (consolidation) short term memories to long-term
56
Declarative memory (excplict)
Memory that can be verbally expressed (telling a story about a past experience)
57
Nondeclarative memory (implicht)
Does not depend on hippocampul formation Motor memory, stimulus - response memory