CHAPTER 54 VALVULAR EMERGENCIES Flashcards

1
Q

T/F: Any diastolic murmur or new systolic murmur with symptoms at rest is pathologic and warrants emergent echocardiography

A

TRUE

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2
Q

GRADING for murmur

A

fa1nt
2uiet, but heard immediately with steth on chest wall
3oderately loud
L4WD (Loud)
5ff the steth partly
6ff the steth entirely

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3
Q

Mid-diastolic rumble, crescendos into S2

Loud OPENING SNAP S1 , small apical impulse, tapping due to underfilled ventricle

A

MITRAL STENOSIS

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4
Q

Harsh apical systolic murmur starts with S 1 and may end before S2

A

ACUTE MITRAL REGURGITATION

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5
Q

High-pitched apical holosystolic murmur radiating into S2

A

CHRONIC MITRAL REGURGITATION

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6
Q

Mid-systolic CLICK may be followed by a late systolic murmur that crescendos into S2

A

MITRAL VALVE PROLAPSE

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7
Q

Harsh systolic ejection murmur

A

AORTIC STENOSIS

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8
Q

High-pitched blowing diastolic murmur immediately after S2
Wide pulse pressure

A

AORTIC REGURGITATION

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9
Q

You have discovered a NEW systolic cardiac murmur, mid-systolic, grade 2, asymptomatic (no signs of cardiovascular disease, normal ECG and CXR, murmur does not increase in intensity with Valsalva or standing).

What is your next step?

A

No further workup!

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10
Q

You have discovered a NEW systolic cardiac murmur, mid-systolic, grade 3 (or Early or late systolic diastolic murmur, holosystolic) symptomatic (with signs of cardiovascular disease, abnormal ECG and CXR, murmur increases in intensity with Valsalva or standing).

What is your next step?

A

Echocardiography

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11
Q

MCC of Mitral Stenosis

A

Rheumatic heart disease

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12
Q

Typical early radiographic finding of MS

A

straightening of the left heart border = left atrial enlargement

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13
Q

Indication of anticoagulation for MS

A

left atrial diameter is >55 mm
atrial fibrillation,
left atrial thrombus,
history of systemic emboli

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14
Q

Primary treatment for symptomatic MS

A

percutaneous mitral commissurotomy,

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15
Q

MCC of Mitral regurgitation

A

Fibroelastic deficiency syndrome
MVP (younger pxs)

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16
Q

Typical cause of Acute Mitral Regurgitation

A

papillary muscle or chordae tendineae rupture from myocardial infarction or valve leaflet perforation from infective endocarditis

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17
Q

Seen in any patient with new-onset and marked PULMONARY EDEMA, especially in patients with near-normal heart size on chest radiograph or in those who do not respond to conventional therapy

A

Acute mitral regurgitation

18
Q

Systolic billowing of one or both leaflets into the left atrium occurring with or without mitral regurgitation.

A

MVP

19
Q

Maneuvers that decrease preload in MVP

A

Valsalva or standing
–> cause the click to occur earlier in diastole

20
Q

Maneuvers that increase preload in MVP

A

squatting

21
Q

Maneuvers that increase afterload in MVP

A

hand grips

22
Q

Classic triad of aortic stenosis

A

dyspnea
chest pain
syncope

23
Q

Severe stenosis: aortic valve area

A

<1.0 cm2

-asymptomatic

24
Q

Classic physical examination findings for AS

A
  1. late peaking systolic murmur at the right second intercostal space radiating to the carotid
  2. single or paradoxically split S2, an S4
    gallop
  3. diminished carotid pulse with a delayed upstroke = pulsus parvus et tardus
25
Q

In the left lateral decubitus position, listen for a mid-diastolic rumble using the bell of the stethoscope at the cardiac apex

A

Austin Flint murmur

26
Q

“water hammer pulse”

A

Peripheral pulse with a quick rise in upstroke due to increased stroke volume followed by collapse from a rapid fall in diastolic pressure

27
Q

“to-and-fro” femoral murmur

A

Duroziez sign

28
Q

pulsatile head bobbing

A

de Musset sign

29
Q

capillary pulsations visible at the proximal nail bed while pressure is applied at the tip

A

Quincke sign

30
Q

What is avoided in acute aortic regurgitation because they block the compensatory tachycardia that is critical in maintaining cardiac output?

A

Beta-blockers

31
Q

The least likely valve to be affected by acquired disease

A

PULMONIC valve

32
Q

Rare and is generally accompanied by regurgitation

A

Tricuspid stenosis

33
Q

Murmur of tricuspid valve regurgitation

A

soft, blowing, and holosystolic

best heard along the lower left sternal border and increases with inspiration

34
Q

Associated with a rumbling crescendo decrescendo diastolic murmur occurring just before S1

best heard along the lower left sternal border, increases with inspiration, and is often preceded by an opening snap

A

Tricuspid valve stenosis

35
Q
A
36
Q

Harsh systolic murmur, best heard in the left second intercostal space, which increases with inspiration.

A

Pulmonic stenosis

37
Q

Most sensitive modality for right-sided valvular heart disease

A

transesophageal echocardiography

38
Q

More durable with lower failure rates, but have a higher risk for thromboembolic complications –> Lifelong anticoagulation is necessary to reduce the thromboembolic risk

A

Mechanical valves

  • require an INR of 2.5 to 3.5
  • bileaflet mechanical valves in the aortic position require an INR of 2.0 to 3.0
39
Q

T/F: Emboli are more common from mitral rather than from aortic valves

A

T

40
Q

Less thrombogenic but are more likely to fail and require repeat surgery. Antiplatelet therapy is recommended for all patients with prosthetic valves

A

Bioprosthetic valves (porcine, bovine, or human sources)

41
Q

T/F: Acute onset of respiratory distress, pulmonary edema, and cardiogenic shock may be associated with mechanical valve failure, tearing of a bioprosthesis, or a large clot obstructing the valve or preventing closure

A

T

42
Q

T/F: Patients with severe bleeding complications are best treated with fresh frozen plasma or prothrombin complex concentrate. 3 Avoid parenteral, high-dose vitamin K due to risk of overcorrection

A

T