CHAPTER 54 VALVULAR EMERGENCIES Flashcards

1
Q

T/F: Any diastolic murmur or new systolic murmur with symptoms at rest is pathologic and warrants emergent echocardiography

A

TRUE

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2
Q

GRADING for murmur

A

fa1nt
2uiet, but heard immediately with steth on chest wall
3oderately loud
L4WD (Loud)
5ff the steth partly
6ff the steth entirely

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3
Q

Mid-diastolic rumble, crescendos into S2

Loud OPENING SNAP S1 , small apical impulse, tapping due to underfilled ventricle

A

MITRAL STENOSIS

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4
Q

Harsh apical systolic murmur starts with S 1 and may end before S2

A

ACUTE MITRAL REGURGITATION

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5
Q

High-pitched apical holosystolic murmur radiating into S2

A

CHRONIC MITRAL REGURGITATION

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6
Q

Mid-systolic CLICK may be followed by a late systolic murmur that crescendos into S2

A

MITRAL VALVE PROLAPSE

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7
Q

Harsh systolic ejection murmur

A

AORTIC STENOSIS

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8
Q

High-pitched blowing diastolic murmur immediately after S2
Wide pulse pressure

A

AORTIC REGURGITATION

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9
Q

You have discovered a NEW systolic cardiac murmur, mid-systolic, grade 2, asymptomatic (no signs of cardiovascular disease, normal ECG and CXR, murmur does not increase in intensity with Valsalva or standing).

What is your next step?

A

No further workup!

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10
Q

You have discovered a NEW systolic cardiac murmur, mid-systolic, grade 3 (or Early or late systolic diastolic murmur, holosystolic) symptomatic (with signs of cardiovascular disease, abnormal ECG and CXR, murmur increases in intensity with Valsalva or standing).

What is your next step?

A

Echocardiography

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11
Q

MCC of Mitral Stenosis

A

Rheumatic heart disease

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12
Q

Typical early radiographic finding of MS

A

straightening of the left heart border = left atrial enlargement

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13
Q

Indication of anticoagulation for MS

A

left atrial diameter is >55 mm
atrial fibrillation,
left atrial thrombus,
history of systemic emboli

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14
Q

Primary treatment for symptomatic MS

A

percutaneous mitral commissurotomy,

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15
Q

MCC of Mitral regurgitation

A

Fibroelastic deficiency syndrome
MVP (younger pxs)

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16
Q

Typical cause of Acute Mitral Regurgitation

A

papillary muscle or chordae tendineae rupture from myocardial infarction or valve leaflet perforation from infective endocarditis

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17
Q

Seen in any patient with new-onset and marked PULMONARY EDEMA, especially in patients with near-normal heart size on chest radiograph or in those who do not respond to conventional therapy

A

Acute mitral regurgitation

18
Q

Systolic billowing of one or both leaflets into the left atrium occurring with or without mitral regurgitation.

19
Q

Maneuvers that decrease preload in MVP

A

Valsalva or standing
–> cause the click to occur earlier in diastole

20
Q

Maneuvers that increase preload in MVP

21
Q

Maneuvers that increase afterload in MVP

A

hand grips

22
Q

Classic triad of aortic stenosis

A

dyspnea
chest pain
syncope

23
Q

Severe stenosis: aortic valve area

A

<1.0 cm2

-asymptomatic

24
Q

Classic physical examination findings for AS

A
  1. late peaking systolic murmur at the right second intercostal space radiating to the carotid
  2. single or paradoxically split S2, an S4
    gallop
  3. diminished carotid pulse with a delayed upstroke = pulsus parvus et tardus
25
In the left lateral decubitus position, listen for a mid-diastolic rumble using the bell of the stethoscope at the cardiac apex
Austin Flint murmur
26
"water hammer pulse"
Peripheral pulse with a quick rise in upstroke due to increased stroke volume followed by collapse from a rapid fall in diastolic pressure
27
“to-and-fro” femoral murmur
Duroziez sign
28
pulsatile head bobbing
de Musset sign
29
capillary pulsations visible at the proximal nail bed while pressure is applied at the tip
Quincke sign
30
What is avoided in acute aortic regurgitation because they block the compensatory tachycardia that is critical in maintaining cardiac output?
Beta-blockers
31
The least likely valve to be affected by acquired disease
PULMONIC valve
32
Rare and is generally accompanied by regurgitation
Tricuspid stenosis
33
Murmur of tricuspid valve regurgitation
soft, blowing, and holosystolic best heard along the lower left sternal border and increases with inspiration
34
Associated with a rumbling crescendo decrescendo diastolic murmur occurring just before S1 best heard along the lower left sternal border, increases with inspiration, and is often preceded by an opening snap
Tricuspid valve stenosis
35
36
Harsh systolic murmur, best heard in the left second intercostal space, which increases with inspiration.
Pulmonic stenosis
37
Most sensitive modality for right-sided valvular heart disease
transesophageal echocardiography
38
More durable with lower failure rates, but have a higher risk for thromboembolic complications --> Lifelong anticoagulation is necessary to reduce the thromboembolic risk
Mechanical valves - require an INR of 2.5 to 3.5 - bileaflet mechanical valves in the aortic position require an INR of 2.0 to 3.0
39
T/F: Emboli are more common from mitral rather than from aortic valves
T
40
Less thrombogenic but are more likely to fail and require repeat surgery. Antiplatelet therapy is recommended for all patients with prosthetic valves
Bioprosthetic valves (porcine, bovine, or human sources)
41
T/F: Acute onset of respiratory distress, pulmonary edema, and cardiogenic shock may be associated with mechanical valve failure, tearing of a bioprosthesis, or a large clot obstructing the valve or preventing closure
T
42
T/F: Patients with severe bleeding complications are best treated with fresh frozen plasma or prothrombin complex concentrate. 3 Avoid parenteral, high-dose vitamin K due to risk of overcorrection
T