Chapter 6 Flashcards
heart rhythms associated with cardiac arrest are divided into two groups which are they ?
SHOCKABLE - VFIB , pulseless VTach
= first monitor rhythm in 20 percent cardiac arrest
NON- shockable rhythm - MORE COMMON = asystole and PEA
what is the usually the next rhythm documented when resuscitations commence in asystole or PEA
25 PERCENT IS VFIB AND PULSELESS VTACH
WHY SHOULD THE INTERVAL BETWEEN stopping compression and delivering shock be minimised ?
chance for pulse to be palpable immediately after defib - time for ROSC and palpable rhythm takes atleasst 2 min
delay in compression - further myocardium compromise - if no ROSC
if perfusing rhythm restored further chest compression has no indication of increasing the chance of VF reoccurring
chest compression can induce VF in post shock a-systole
time taken for ROSC AND PALPABLE PULSE IS USUALLY how long
maybe longer than 2 mins in 25 percent of successful shocks
what is the current evidence pointing to for the drug sequence during CPR
INSUFFICIENT TO SUPPORT OR REFUTE
ADRENALIE AND AMIODARONE CURRENTLY RECOMMENED BASED LARGELY ON INCREASED SHORT TERM SURVIVAL
when there is shock refectory VF and pVTach what is important to do ?
to check the position and contact of defibrillates pads
considered worthwhile continued shock if patient remains in identifiable vfib or pulseless VT if you have started resuscitations - but consider changing thee pad position incase refectory to ANTERIOIR- POSTERIOr
if a rhythm compatible with pulse i seen during 2 min cpr what should be done ?
do not interrupt cpr to palpate pulse unless sign of ROSC
any doubt about palpable pulse
when can a precordial thump be used ?
because precordial thump has very low success rate for cardioversion of shockable rhythm
only done when :
used without delay whilst awaiting the arrival of defibrillater in MONITORED vf and vtach
how to do precordial thump?
use ulnar egge of a tightly clenched fist
sharp impact on lower half of sternum from height of 20cm
retract fist immediately to create impulse like stimulus
PROTOCOL if a patIent has monitored and witnessed cardiac arrest in CATHETER LAB , CORONARY CARE UNIT ,
CRITICAL CARE AREA
OR MONTORED AFTER CARDIAC SURGERY WHAT SHOULD BE DONE ?
Or if INTIAL RHYTHM VTACH and VFIB - AND PATIENT ALREADY CONNECTED TO MANUAL DEFIB OR IN VERY CLOSE RANGE
confirm cardiac arrest
shout for help
three quick successive shocks
rapid rhythm check and if appropriate pulse and OTHER SIGNS OF ROSC after each defb
start chest compression /CPR for 2 mins if third shock unsuccessful
ALSO IN THIS CASE AMIODARONE IS GIVEN AFTER THREE SHOCK ATTEMPTS
then follow ALS algorithm as if the three shock is just FIRST shock (the three shock stacked is considered to be the first shock in ALS algorithm )
ADRENALINE GIVEN AFTER ANOTHER 2 MORE SHOCK ATTEMPTS if VF and Pvtach persist
In which type of arrest is survival unlikely unless a reversible cause can be found?
PEA and asystole
Why do we avoid interruption in high quality chest compression
High quality chest compression and ventilation are important determinants of outcome.
Chest compression - pause causes coronary perfusion to decrease
What depth and rate are chest compressions done
Depth -5-6 cm
Rate -100-120
Ensure full recoil
Soon as airway secured continue chest compression without pause during ventilation
Reduce fatigue change individual every 2 mins
What type of ventilation preferred
A bag mask or SGA (supraglottic - igel) done if no person skilled tracheal tube insertion (if not skilled time taken for insertion and cannot continue CPR) - once inserted ATTEMPt to deliver continuous chest compressions
However no research shows Tracheal intubation increases survival after cardiac arrest compared with BMV and SGA
So alternatively intubation attempt can be deferred until after ROSC
What rate do you ventilate the lungs
10 breaths per min
Technique of securing ventilation when chest compression
Avoid stopping chest compression during laryngoscopy and intubation. Only brief pause in chest compression -if tube passed between vocal cords - pause should not exceed 5s
After intubation confirm correct tube position with waveform capnography and secure tube adequately
How to recognise ROSC
Clinical signs - breathing efforts
Movement , eye opening
Require verification by Rythm and pulse check
But these can also occur without ROSC as CPR can generate sufficient circulation for consciousness
Pulse check - when ECG rythm compatible
Carotid pulsation during CPR does not necessarily indicate myocardial and cerebral perfusion
Also may not detect pulse with those of low CO
ECG - monitoring heart rythm
End tidal CO2 measured with wave form capnography
Blood sampling and analysis problems
During CPR bloods taken to identify reversible causes
Avoid finger prick
Use sample from VEINS or ARTERIES
Blood gas difficult to interpret during CPR - misleading and best little relationship to tissue acid base state
Analagsis of central venous blood provide better estimation of tissue PH
normal concentration of Pac02
4.7-6.0kPa
what is end tidal Co2
partial pressure of co2 measured at the end of an exhaled breath - it reflexts cardiac output and pulmonary blood flow
during CPR end tiday volume is very low reflecting low cardiac output from chest compressions
what is wave form capnography?
enables continuous real time end tidal c02 to be monitored during CPR - works most reliably on patient with tracheal tube - can also be used in supraglottic airway device
(OR BAG MASK?)
uses of waveform capnography ?
measure quality of cpr
end tidal c02 are associated with compression depth and ventilation rate
INCREASE IN END TIDAL C02 may indicate ROSC
presence of end tidal co2 indicates the tracheal tube is in position
PROGNOSIS DURING cpr - VALUES ARE HIGHER AFTER AN INITIAL APHYXIAL ARREST AND DECLINE OVER TIME
LOW END TIDAL CO2 IS RELATED TO lower ROSC , increased mortality
interpreting wave form capnography ?
inspiration - low co2
start of expiration - rapid rise of c02 (intially no c02 as it comes from anatomical dead space - ie volume of resp tract that does not take part o gas exchange the larynx , trachea , main bronchi
there is high platue stage -slight gradual increase is due to not all alveoli empty at the same rate
end of expiration conc of co2 is maximal - this is end tdal c02
healthy patients - 4.8kPa (4.3-5.5kPa)
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soon after the second defibrillation there is sa sigbnificant increase in end tidal c02 - first indicator of ROSC and OFTEN PRECEEDS OTHER INDICATORS such as pulse
IF ROSC IS SUSPECTED DURING CPR HOLD Adrenaline- Give adrenaline if cardiac arrest is confirmed in the next rhythm check
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failure to achieve an end tidal c02 greater than 1.33kPa after 20 MIN cpr associated with poor outcome
when can CPR be stopped ?
COMBINATION OF CLINICAL AND PHYSIOLOGICAL SIGNS - not just by an increase in end tidal Co2
SHARP RISE IN CO2 end tidal co2
purposeful movemnet
consider breifly stopping chest compression for rhythm analysis and if appropriate pulse check
if pulse palbabale - post resus care
if NO PULSE PRESENT - CONTINUE cpr