Chapter 6 Flashcards

1
Q

Consciousness What is it?

A

Moment to moment awareness of ourselves and environment

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2
Q

Components of consciousness

A
Subjective & private
Others cannot directly know our reality (and  vice versa)
Dynamic
	Ever-changing
Self-reflective
	Mind is aware of its own consciousness
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3
Q

How do we decide if computers are conscious?

A

Ask a bunch of people and computers and question and have to know which was computer answers and which was human answers?

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4
Q

How do we Measuring Consciousness

A

How do we operationally define inner states?
1) Self-reports
Direct but not verifiable
2) Physiological
E.g., EEG ( not what your conscious of but how conscious states vary )
Are objective but cannot indicate what person is experiencing subjectively
3) Behavioural
Performance on tasks (e.g., rouge test - are you self aware? look in a mirror do you know its you?)
Need to infer state of mind (cant observe state mind have to observe it through environment and behavior but then you hit mind body problem)

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5
Q

Rouge test?

A

When you look in a mirror you know you are looking at yourself

but where did that realization come from? … only primates can do,

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6
Q

What are the Psychodynamic view – 3 Levels

A

1) Conscious (high tract)
Mental events we are currently aware of
(reading this slide)
2) Preconscious
Outside of current awareness; easily recalled
(what you had for supper last night)
3) Unconscious (low tract)
Not brought into conscious awareness under ordinary circumstances
(unacceptable urges; traumatic memories- tied to anxiety, pain)

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7
Q

Cognitive View: Controlled vs Automatic Processing

A

1) Controlled (short term memory)
Effortful - Voluntary use of attention, conscious effort
Difficult or new tasks
Slower but more flexible than automatic
2) Automatic (can get in the way when we experience new things)
Little or no conscious processing- Routine, well-learned
Fast but can inhibit finding ‘new’ solutions
Facilitates ‘divided attention
“cant hit the ball and think at the same time”

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8
Q

Explain Divided Attention

A

–How many things do you do at once? can only do one high tract activity at once but can do many low tract activities at once (walking and chewing gum)

–We can ‘divide’ attention
Otherwise every action would require full attention

–Is adaptive but has limits
Difficult if tasks require similar resources
Impact other actions - e.g., using cell phone & driving

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9
Q

Describe Emotional Unconscious

A
  • –Unconscious processes influence emotions and motivations
  • –Are influenced by events we are unaware of
  • –Mood can be affected by positive or negative words subliminally presented

subliminal advertising- inserted pictures of popcorn and coke into the frames of movies and sales increased! – was a lie no one could replicate it

can you brain wash? (americans saying they hated america)
CIA experiments results proved that you cant brainwash people

you want people to do something: put it in their face, obvious and conscious! not subliminal

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10
Q

Modular Mind

A

Mind consists of separate but interacting information processing modules

  • -Sensation, perception, memory, problem solving, emotion
  • -Process information in parallel

Experience of consciousness is integrated output of modules

at any time massive amounts of info is being processed, are awareness is the integration of the parallel bits

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11
Q

Circadian Rhythms

A

circa - around
dian- day

circadian rythms are regulated by the brains suprachiasmatic nuclei (SCN) located in hypothalamus –> function like a biological clock

Rhythmic daily cycles (wakefulness, sleep)
phase relationships: between daily cycles

Why are there these cycles? endogenously–> internal clock? or exdongenous–> cues? driven?
Helotrophic plants .. follow sun across the sky.. if put in dark do they still follow the cycle.. and they did .. problem? didn’t time the cycle.. if still 24 hours it could be something else! Need to know the link of the cycle
–> new experiment the cycle was timed after dark for a certain time the period wasnt 24 hours anymore, slightly longer or slightly shorter. hard to explain exdogenous. Suggests there is a clock.
—> blinded monkey peroid slightly longer then 24 hours if consistant predict when monkey wakes up on christmas years away. Longer but consistant! Endrogenous!

Why is time efficient? can predict! early bird gets the worm. why birds make sound before sun comes up.. they anticipate

example: Bees anticipated when breakfast would be ready—> advantageous

animal that doesn’t have rhythmic behavior- cats

Circannual- hibernation, change hair coulor
humans; Putting on weight end of summer and fall

circalunar (29 days) - development of fetus

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12
Q

How are circadian rhythms monitored?

A

—Circadian rhythms regulated by suprachiasmatic nuclei (SCN)
—SCN neurons link to pineal gland, which secretes melatonin
—Neurons in SCN active during daytime
Inhibits melatonin secretion
Raises body temperature and alertness
—Inactive at night
Allows melatonin secretion to increase
Melatonin promotes relaxation and sleepiness
Temperatures drop

hands of clock- rhythmic

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13
Q

Disruptions of circadian rhythms

A

Jet Lag
Shiftwork
Changing to Daylight Savings
Seasonal Affect Disorder (SAD)

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14
Q

Jet leg

A

ex. bees were fed at certain times in the day then move them from parid to new york, Do bees get up paris time? thats a clock mechanism endogenous or new york time—> jet leg!! exdodenous responding to light

our clock resets by days, disadvantage to hockey teams like vancouver canucks always playing east.. didn’t do so well, far more jet legged. Have people to help reset the clock faster in athletes

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15
Q

Shift work

A

Can you sleep during the day,

go forwards

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16
Q

daylight savings

A

worse day to be on road – right after time change

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17
Q

seasonal affect disorder

A

Daylight, prevalence to places further north

SCN only exposed to light 2 points in the day sunset and sunset, SAD sufferers are particularly sensitive to light

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18
Q

Stages of Sleep

A

BARC

Cycle through stages roughly every 90 minutes

1) Brain activity, other physiological responses change
2) Beta waves occur when awake and alert (15-30 cps)
3) Alpha waves occur when relaxed and drowsy (8-12 cps)

every 90 minutes we hit a gate where we are more suspecatable to sleep

when you fall asleep, alpha waves

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19
Q

describe Stages 1-4 of Sleep

A
Stage 1 (falling asleep at the wheel)
	Light sleep
	Theta waves (3.5-7.5 cps)
	Lasts few minutes
	May experience ‘body jerks’

Stage 2 (cease to respond to environmental stimuli)
Sleep deepens - muscles more relaxed - harder to awaken
Sleep spindles (1-2 second
bursts of rapid brain activity) - way to differentiate between stage 1 and stage 2

Stage 3
Sleep deepens aka slow wave sleep
Regular appearance of delta waves (0.5-2 cps)
high altitude low frequency

Stage 4 (hard to wake up and completely disoriented)
Sleep deepens aka slow wave sleep
Delta waves dominate pattern
Stage 4 and Stage 3 together - called “slow- wave sleep”

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20
Q

What happens after stage 4 of sleep?

A

After stage 4 period, go ‘back through earlier stages
Stage 3, then Stage 2, but not another Stage 1
Instead, a new stage appears

REM sleep
Rapid eye movements under eye lid (hence the name!)
High arousal
Frequent dreaming
EEG looked like you were awake, however ——-trunk and limb “paralyzed” – paradox
—when wake people up form this stage of sleep: likely to report they were dreaming

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21
Q

What happens during REM stage of sleep?

A
Physiological Changes
	Heart-rate increases
	Breathing more rapid and irregular
	Brain-wave activity increases
	‘REM sleep paralysis’
		Difficult for voluntary muscles to contract

Sleep cycle changes
Stage 4 & then Stage 3 no longer occur
REM periods become longer

–Get your best sleep in the early hours of your -

–sleep spend more time in REM later on in your sleep period

— REM get crazy dreams

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22
Q

Sleep wave patterns as you sleep

A

Changing brain activity
REM resembles Stage 1
Waves become slower and larger

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23
Q

What are the brain structures involved in sleep

A
  • -brain uses more sugar and oxygen over night then in day.
  • -Reticular activated system: continuations sensory input, during sleep it cuts it off, but brain is still active however can not feel it.

Falling Asleep
Regulated by basal forebrain & regions of brainstem

REM Sleep

- Regulated by brainstem (reticular formation)
- Limbic system (feeding fighting and sexual behaviour)  activity increases
- Association areas near visual cortex active 
- Motor cortex active but signals blocked
- Decreased activity in prefrontal cortex (lower areas)

when brain is woken up still has a bit of disconnect from sensory input and is trying to make sense of what is going on

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24
Q

Why Do We Sleep?

A
Restoration Model (common belief)
	Sleep recharges bodies
	Sleep allows recovery from mental & physical fatigue

—> Evidence is mildly supporting
Sleep about 10 minutes longer on days we exercise
Adenosine (cellular waste product) get adenosine from ATP when used by-product is adenosine, when levels of adenosine feel tired, may play role in decreasing alertness

caffeine is adenosine antagonist, relieves you of fatigue

lecture answers: We sleep because its adaptive, we arnt adaptive to be awake at night. If active at night we would be prone to accidents and easy for predators that can see very well. We wouldnt be good at hunting , expend more calories then we could take in–> in efficient better to conserve calories –example: hybernation, let our body temperatures drop to conserve calories
some species even freeze at night!

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25
Q

Why do we sleep cont.

A

1) Evolutionary/circadian models
Is adaptive
Evolved sleep-wake pattern that increased chances of survival
Mechanism for conserving energy (metabolic rate 10-25% slower during sleep)

2) Restoration & evolutionary theories
Contribute to Two-factor model of sleep

amount you sleep is dependent on your niche, cats have high calorie meals they sleep a lot, cattle don’t sleep much because they eat low calorie grass

REM wake up quickly to raise the alarm. being paralyzed would be good to keep you from falling out o f tree, if group is at different stages one is bound to notice predator

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26
Q

Sleep Disorders

A

1) Insomnia
Chronic difficulty in falling asleep, staying asleep, or experiencing restful sleep
Most common sleep disorder (10 - 40% of population)

don’t wanna use melatonin because it hasn’t been tested because it isn’t a drug could have side effects

drugs suppress REM sleep

best way to eliminate insomnia Address sleep hygiene , dont make sleep place work place, watch caffeine intake — 5 hours, prepare for sleep: dont watch tv before sleep

2) Pseudoinsomniacs (never feel rested)
Complain of insomnia - but sleep normally
Individuals truly believe they have insomnia
Research in sleep labs show most sleep normally
National Sleep Foundation
Canadian Sleep Society

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27
Q

Insomnia

A

Multiple causes
Biological, psychological, environmental
Several treatments
Stimulus control
Based on learning principles
Associate stimuli in sleep environment only with sleep (dont sleep with screen in room)

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28
Q

Narcolepsy

A

Extreme daytime sleepiness & sudden, uncontrollable sleep attacks!!

Treated with high doses of stimulus

Cataplexy (sudden loss of muscle tone)
Cause is unknown
Genetic? (selectively bred in dogs)

29
Q

REM-Sleep Behaviour Disorder

A

Loss of muscle tone that causes REM-sleep paralysis is absent

Sleeper may “act out” dreams: kick, punch, move wildly

Many RBD patients have injured self or sleep-partner

30
Q

Sleep Apnea

A

more common in males and linked to obesity, but can be other factors

Repeated cycle in which breathing stops and restarts during sleep
About 1-5% of population
Lasts 20-40 seconds up to minute or two
Severe cases = 400-500 times/night

Most common cause
obstruction of upper airway

Not getting any sleep

31
Q

Sleepwalking

A

1) Typically occurs during Stage 3 or Stage 4
More common among children (10-30%)

2) Causes
Heredity, stress, alcohol, illness, medications

3) Treatment
Psychotherapy, hypnosis, drugs, behavioural (waking before sleepwalking), wait to outgrow it

4) Myth
Waking a sleepwalker is dangerous– isnt bad for their brain, can be dangerous for sleepwalker if they run into something ect.

ex. Guy that slept walked into car and drove to mother and law and killed her

32
Q

What does SCN do?

A

SCN is linked to tiny pineal gland which secretes melatonin a hormone that has a relazing effect on the body. SCN neurons become active during daytime and reduce the pineal glands release of melatonin, raising your body temperature and hightening awareness. At night time SCN neurons are inactive, allowing melatonin levels to increase and promote relaxation and sleepiness

33
Q

free running circadian rhythem

A

is desynchronized (out of sync) with the 24-hour day-night cycle

34
Q

RMD (REM-sleep behavior disorder)

A

in which the loss of muscle tone that causes normal REM sleep paralysisn is absent (punch, kill partner ect)

35
Q

Differentiate between Nightmares and Night terrors

A

NIghtmares:
“Bad” dreams
Everyone has them
Occur more often during REM sleep

NIght terrors:

  • Frightening dreams that arouse sleeper to near-panic state
  • Sleeper may flee room yet not remember event in morning; typically occur during stages 3 & 4 (not paralyzed)
  • Most common during childhood
36
Q

When do we dream?

A

occur during any sleep stage
Hypnagogic state: transition from waking to early stage 2
Most occur during REM
REM-sleep dreams tend to be more vivid and storylike
higher chances of dreaming in later hours then in early hours of sleep due to ciracadian sleep cycle

37
Q

Hynagogic state

A

transition from waking to early stage 2

38
Q

Are dreams pleasant experiences? Blissful?

A
  • Is a stereotype
  • Negative/unpleasant content is common
  • Content is affected by cultural background, life experiences, current concerns
  • 50% of content may reflect experiences of recent day
39
Q

Why do we Dream- Freud’s Psychoanalytic Theory

A

Wish fulfillment
Gratification of unconscious desires / needs (sexual & aggressive urges)

Two important concepts
Manifest content
“Surface” story of dream (what its about)
Latent content
Disguised psychological meaning of dream (what its really about)

40
Q

Activation-synthesis theory

A

Activation = random neural activity but because we are asleep non of this neural activity matches external sensory events , but our cerebral cortex continues to perform its job of interpretation..
Synthesis = brain making ‘sense’ of this

41
Q

Problem-solving models

A

Help us find solutions to personal problems and concerns because dreams are not constrained by reality

42
Q

Cognitive-process dream theories

A

-Takes mental ability to dream thats why young children typically dont dream much.
-Dreams & waking thoughts produced by same systems in brain
involve processes (e.g., rapid shifts of content)
“all of a sudden i was…“that are more similar than we typically realize

shifts are normally informed why sensory information but because u are sleeping limited input of sensory info

43
Q

Integration

A

-During REM - various ‘modules’ (of brain) are stimulated

Perceptual module - Cognitive module - Emotional module - Motor module

44
Q

Modules interact

A

Brain attempts to find ‘best fit’ to activity
top down process, down up process is disabled
-example see something in the distance best guess what it is until it get closer

45
Q

Drugs and altered consciousness -Hallucinogenics

A

been used for millennia

common, TSG

46
Q

Psychoactive drugs pervasive part of society

A

-have to be Lipid soluble
- have to be non polar to get through the barrier
Modify brain chemistry - cross blood-brain barrier
Facilitate or inhibit synaptic transmission

THC takes a month to get out of body

47
Q

Agonists

A

increase neurotransmitter activity ( may enhance the production, storage, or release of neurotransmitter ect)
(direct .. fit the receptor) (indirect agonist.. effect neaurotransmitter…use second messengers before being able to affect their final destination)

48
Q

Antagonists

A

(can block indirect—> slow activity in the system drug used for hypertension or direct—> drugs to block domamine receptors help skitofrenia)
inhibit or decrease neurotransmitter activity

49
Q

Tolerance

A

Decrease in responsivity to drug (need larger doses)
Body attempts to maintain homeostasis
3 ways develop tolerance
1) metabolic (get exposed to a little) Enzymes that break down alcohol also break down testosterone
2) Behavioral (learn to manage impairment)
3) Nervous system (up -more sensitive ex. block domamine when people come off drugs they go crazy or down- less sensitive ex. anabolic steroids)

50
Q

Compensatory Responses

A

Physiological reactions opposite to that of drug
Brain is adjusting to body imbalances. (example: alcohol increases heart rate compensatory responses decrease heart rate)

51
Q

Withdrawal

A

Compensatory responses after drug use is discontinued
-rebound syndrome
For example in the absence of alcohol’s sedating and relaxing effects the chronic drinker may experiance increased heart rate, anxiety and hypertension

52
Q

Learning tolerance and overdose- Environment is a powerful influence

A

Classical conditioning
Environment becomes associated with drug
Physical setting triggers compensatory responses

53
Q

Learning tolerance and overdose- Conditioned Drug Responses

A

Tolerance for drug influenced by familiarity of drug setting
In unfamiliar setting ‘overdose’ reaction can occur even when typical amount of drug is used
- couldn’t find drugs until they finally find it so they run into the closest wash room and shoot up.
- get offered drinks in unfamiliar setting it will hit you way faster, very small amount can make you very impaired

54
Q

Myths about substance abuse

A
  • Drug tolerance always leads to significant withdrawal (tolerance to ephadamine and cocaine but its not withdrawal) only happens with opiates and alcohol related drugs
  • If drug does not produce tolerance or withdrawal, cannot become dependent
  • Physiological dependence major cause of addiction (NOT TRUE)
55
Q

Depressants

A

-Decrease nervous system activity
Low doses : stimulants

Moderate doses:
	Reduce  anxiety; produce euphoria
High doses:
	Slow vital life processes
	Can be fatal
  • reduce behavioral output
  • effects like alcohol
56
Q

Alcohol

A

-Nervous system depressant
1) Initial upper phase then brain centres become depressed
2) Increases activity of GABA (main inhibitory neuro-transmitter) - decreases brain activity
3) Decreases activity of glutamate (major excitatory neurotransmitter)
Combination creates ‘high’ & then ‘down’ phase

  • neural slow down depresses the action of inhibitory control centres in the cerebral cortex
  • at higher doses the brains control centres become increasingly disrupted
    as dose increases is massed by depressant affect, stimulant effect gets un masked as stimulant goes down (hard to sleep once you get home)
    -build tolerance to setitive effects of alcohol
    -stimulant effects the user is seeking, angry young men to manage themselves their low tolerance to alcohol as they grow tolerant to depressant they pushed by stimulant
57
Q

Blood Alcohol Level (BAL)

A

-females- less body fat for mass, more stays in blood
-Measure of alcohol concentration in blood
-Elevated BAL linked to risky & harmful behaviours
-Alcohol Myopia - shortsightedness in thinking
.08 for most ppl ugly impaired

58
Q

Barbiturates (sleeping pills) & Tranquilizers (anti anxiety drugs)

A

Depress nervous system:
E.g., sleeping pills & anti-anxiety drugs
Increase activity of GABA (inhibitoru)
Highly addictive:
Several months needed to lose physiological dependency

  • dead line doesn’t move however tolerance line does move.
  • mix with alcohol and it multiples not adds
59
Q

Stimulants

A

Increase neural firing & arouse nervous system

Increase blood pressure, heart rate, respiration, alertness

60
Q

Stimulants: Amphetamines

A
  • “speed uppers”
  • reduce appetite and fatigue to decrease the need for sleep
  • Increase dopamine & norepinephrine
  • Heavy use can produce amphetamine psychosis
  • far outside drug abuse, used for military , keep air crews up and active
61
Q

Stimulants: Ecstasy (MDMA)

A

-derivative of amphetamines, causes the release of serotonin and blocks its re uptake
-Feelings of pleasure, elation, empathy, warmth
Interferes with serotonin reuptake
- stimulant and hallucinates
- might not be buying ecstasy, cant thermoregulate.. get heat stroke cant deal with heat

62
Q

Cocaine

A

Blocks reuptake of norepinephrine, dopamine

Fever, convulsions, hallucinations, delusions

63
Q

Crack (smoke-able cocaine)

A

Chemically converted form

Effects are faster, more intense

64
Q

Opiates:

A

Opium = product of poppy plant
Morphine, codeine, heroin derived from opium = opiates
Bind to receptors activated by endorphins
Pain relief
Mood changes, euphoria (excitment and hapiness)
Highly addictive & traumatic withdrawal
2% of Americans have used heroin
25% of them become addicted (fabrication) 1/8

  • heroin- suppression of cough
  • Opiate- heroin = cooked up morphine difference isn’t what they do, difference is their lipid solubility, heroin is more lipid soluble. they are the same drug. Can use less heroin
  • drugs that have one effect will always have the other effect as well
65
Q

Hallucinogens

A

Produce hallucinations

Many derived from natural sources

66
Q

LSD & phencyclidine - synthetic

A
Distort or intensify sensory experience
		Blur boundaries between reality & fantasy
		LSD has inhibiting effect on serotonin
		dreamlike perceptions & hallucinations
-make stupid decisions lasts 8-12 hours
-tolerance to LSD grows rapidly
- used in many brain washing 
- disassociate anesthetics
- PCP chedomine - anasthetic does not compromise vital function , rape drugs they produce wax dumbie that wont remember anything - viteman K
67
Q

Marijuana

A

Most widely used illegal drug in Canada
THC may increase GABA, dopamine activity
Myths
Unmotivated & apathetic = amotivational syndrome
Start using more dangerous drugs
No significant dangers with use

basement dwellers are more likely to use marijuane and eat doritos.

68
Q

Determinants of Drug effects

A
DRUG:
-what drug, how much and how did you take it?
SET:
Genes
Physical and social setting 
Culture
Beliefs and expectations
Personality factors
SETTING:
Where and why you are taking the drug, rare for ppl who wernt previous drug users to abuse pain drugs. 

Effects Depend on More than Chemical Structure