Chapter 8 Aphasias Flashcards

(41 cards)

1
Q

Argument for classifying aphasias

A

Different areas of the brain control different functions so,
different types of aphasias have different lesion sites and,
different lesion sites produce distinctively different types of symptoms
Different types of aphasias require different forms of Tx

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2
Q

Arguments against classifying aphasias

A

Brain is thought to function as integrated unit in controlling language

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3
Q

Factors used in classifying aphasias

A
Speech fluency
Paraphasias
Repetitions
Language comprehension
Site of lesion
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4
Q

Classification by fluency

A

Non-Fluent and Fluent aphasias

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5
Q

Non Fluent aphasias

A
Speaking is slow, halting, and with effort:
Broca's
Transcortical Motor Aphasia (TMA)
Mixed Transcortical Aphasia (MTA)
Global Aphasia
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6
Q

Fluent Aphasia

A
Speaking is smooth and effortless:
Wernicke's
Transcortical Sensory Aphasia (TSA)
Conduction Aphasia
Anomic Aphasia
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7
Q

Classification by comprehension

A

Receptive Aphasia & Expressive Aphasia

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8
Q

Receptive Aphasia

A

Primary difficulties are in area of comprehension

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9
Q

Expressive Aphasia

A

Primary difficulties are in the area of expression

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10
Q

Classification by site of lesion

A

Damage to the cortical centers for language (served by the middle cerebral artery)
Damage to the association fiber tracts important for language
Damage to the subcortical area
Anomic aphasia does not have a clear localization

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11
Q

Aphasia caused by damage to the cortical area

A

Broca’s, Wernicke’s, and Global

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12
Q

Aphasia caused by damage to the association fiber tract

A

Conduction & Transcortical sensory, motor, and mixed aphasias

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13
Q

Broca’s (motor) Aphasia

A

Damage to lower part of the premotor cortex important for planning and performing expressive language
Damage to the anterior portion of the middle cerebral artery
Often accompanied by right sided hemiplegia or hemiparesis

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14
Q

Broca’s aphasia language characteristics

A
Nonfluent 
Agrammatic
Monotonous speech 
May have misarticulations 
Lacking function words
Impaired repetition
Written language similar to spoken 
Comprehension better than their expressive spoken and written language
Good self-monitoring
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15
Q

Wernicke’s (sensory) aphasia

A

Damage to posterior superior temporal lobe of language dominant hemisphere
Damage to posterior branch of the cerebral middle artery

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16
Q

Wernicke’s aphasia language characteristics

A
Fluent
Impaired auditory comprehension is dominant characteristic
Paraphasias, neologisms, jargon
Empty speech
May have circumlocution
Press of speech (logorrhea)
Handwriting resembles speech
If aphasia is mild, may be aware of errors
Less concern with speech than Broca’s
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17
Q

Global aphasia

A

Damage to trunk of middle cerebral artery which causes massive damage to the entire perisylvian area
Broca’s or Wernicke’s sometimes spared
Severe deficits are demonstrated in all areas of language function

18
Q

Global aphasia language characteristics

A

Language limited to few words, exclamations
stereotypical utterances
overlearned phrases (How are you?). May perseverate on these words,
Severely impaired comprehension
Reading and writing also terrible
Usually attentive, alert socially appropriate – different than dementia
May be able to perform nonverbal tasks OK – picture matching, etc.
Performance is better when dealing with personal information

19
Q

Conduction aphasia

A

Rare (5-10%), Probable sites of lesions: lesions in insula,

upper temporal or lower parietal lobe Damage arcuate fasiculus and /or supramarginal gyrus.

20
Q

Conduction aphasia language characteristics

A

Wernicke’s thought by most to be spared Comprehension somewhat spared
Most dominant feature - great difficulty with repetition
Multisyllabic words more difficult than monosyllabic words
Repetition impairment is disproportionate to other impairments.
Rate, intonation and stress are usually normal
May produce paraphasias.
Difficulties reading out loud because oral reading depends on communication between Wernicke’s and Broca’s.
Word retrieval problems are usually quite evident – resulting in inappropriate pauses and/or empty speech.
Writing is usually legible but may contain spelling errors & letter omissions.
Alert and attentive and aware of errors.
self-correction aren’t typically successful

21
Q

Transcortical aphasias

A

Spares Broca’s, Wernicke’s, and arcuate fasciculus

Defining characteristic is that repetition is preservered

22
Q

Transcortical Motor Aphasia (TMA)

A

Damage to anterior superior frontal lobe and association pathways
Areas affected are supplied by anterior cerebral artery and anterior branch of middle cerebral artery

23
Q

TMA language characteristics

A

Markedly reduced speech output
Good repetition (this differentiates it from Broca’s)
Good auditory comprehension
Disturbed writing
Difficulty initiating speech- inertia
May have right sided hemiparesis or hemiplegia
Attentive and alert

24
Q

Transcortical Sensory Aphasia (TSA)

A

Damage to posterior temporoparietal region sparing Wernicke’s

25
TSA language characteristics
Preserved repetition – unlike Wernicke’s No problem with inertia in initiating speech (unlike TMA) May often have echolalia Lesion isolates Wernickes area from rest of brain, so major deficits in comprehension of spoken & written language occur. Visual cortex is isolated from Wernicke’s causing difficulty in reading. Fluent but empty speech (like Wernicke's) Don’t have press of speech (not like Wernicke's) Difficulty following commands
26
Mixed Transcortical Aphasia (MTA)
Rare, Damage that spares the major language areas but separates them from rest of brain Damage often occurs in watershed area Often occurs following conditions that reduce blood flow through cerebral arteries such as: carbon monoxide poisoning, cerebral hypoxia, etc.
27
(MTA) language characteristics
Similar to global aphasia except with better repetition Parrot like repetition of what others say - like an echolalia Nonfluent - very limited spontaneous output NO functional auditory comprehension Severely impaired writing and reading skills
28
Anomic aphasia
Naming difficult, no clear localization, damage may occur to angular gyrus or inferior temporal gyrus
29
Anomic aphasia language characteristics
Sever word retrieval May have mild auditory comprehension impairment Circumlocution and substitutions Reading and writing is good
30
Subcortical aphasias
Anterior syndrome Posterior syndrome Anterior-posterior syndrome Thalamic aphasia
31
Anterior syndrome
Caused by damage to internal capsule & lenticular process extending into anterior white matter
32
Anterior syndrome language characteristics
``` Hemiplegic Slow dysarthric speech Good comprehension Good repetition Poor oral reading and writing ```
33
Posterior syndrome
Damage to putamen and internal capsule extending into posterior white matter
34
Posterior syndrome language characteristics
``` Hemiplegic Fluent speech without dysarthria Poor comprehension Good single word repetition Impaired reading and writing Poor confrontational naming ```
35
Anterior-posterior syndrome
Damage to internal capsule and putamen with both anterior and posterior extensions of white matter
36
Anterior-posterior language characteristics
Mixture of symptoms consistent with both Broca’s and Wernicke’s aphasia.
37
Thalamic aphasia
Damage to the thalamus
38
Thalamic aphasia language characteristics
``` Hemiplegic Difficulty initiating speech Speech is echolalic and neologistic Auditory comprehension and reading is good Writing is impaired Word-finding problems are common ```
39
Aphasia a with fair to good comprehension
Broca's, conduction, anomic, and transcortical motor
40
Aphasia with poor comprehension
Wernicke's, transcortical sensory, global
41
Aphasias with poor repetition
Broca's (labored), global