Chest Pain Flashcards

(34 cards)

1
Q

6 life threatening causes of CP

A
ACS
PE
Aortic dissection
Esophageal rupture
Tension pneumothorax 
Pericardial tamponade
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2
Q

Common non-life threatening

A
gastrointestinal 
pulmonary (pneumonia or pleurisy)
chest wall syndromes (musculoskeletal pain)
psychiatric
shingles
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3
Q

the main trifecta of

A

a. Myocardial Infarction (cardiac ischemia)
b. Pulmonary Embolism
c. Aortic Dissection

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4
Q

exertion related pain that is consistent

A

Angina! (CAD)

i went out yesterday i usually walk three block before experiencing some discomfort

If fully occlude –>you’ve had a STEMI

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5
Q

main and other RF for Angina and CAD

A
males greater than 45
women >55
trasnfats and cholesterol 
family
DM
HTN
Smoking

a. Stress, depression, insomnia
b. Amphetamine/cocaine use
c. ESRD
d. Connective tissue disease (SLE, RA)
e. Vasculitis
f. HIV/HAART medications
g. Trauma
h. Any condition where O2 demand exceeds supply (GI bleed, sepsis)

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6
Q

for stable angina, how do you target with questions

A

ask about pain
what were you doing yesterday and last week and last year

need to get a progressive HX of sxs

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7
Q

unstable angina differs from stable

A

class III or class IV

at leas than two blocks or one flight of stairs

significant atherosclerosis
keep for stress test or send to cardiology

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8
Q

when ruling out ACS what are you ruling out exactly

A

want to rule out a STEMI and NSTEMI

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9
Q

three patterns of STEMI

A
  1. ST Depressions
  2. T wave inversions
  3. Wellens’ pattern
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10
Q

Definition of a NSTEMI

A

troponin increase in the absence of strict ECG criteria

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11
Q

2 Different causes for NSTEMI

A

2/3rds of the time supply/demand mismatches

can have this occur in sepsis with troponin release

can also be severely anemic and not have adequate oxygen delivery

1/3 occurs with occlusive myocardial infarction

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12
Q

NSTEMI tx

A

balloon, stent, thrombolitic

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13
Q

cardiogenic shock

A

hypotension and hypoprofusion associated with MI

due to MI or in the setting of cardiac dysfunction resulting form smaller events

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14
Q

AMI RF

A

EVERYONE

if you suspect MI get a troponin

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15
Q

common sxs with MI

A

CP does not radiate to legs but will radiate to back neck jaw shoulder and arms

sudden onset

can also see with dyspnea
syncope
nausea
vomitting
extreme weakness
diaphoresis
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16
Q

ATYPICAL sxs of MI seen in this population

A

Women,

diabetics (b/c of neuropathy and visceral nerves have been dulled)

elderly, barriers to communication

(language, dementia, altered mental status/psych)

17
Q

atypical sxs of MI

A
N/V
cold sweats
SOB
fatigue
syncope
cold and clammy
back pain palpitations
18
Q

what two medications can not be used to rule in or rule out cardiac related CP

A

NTG–> will relieve non related CP

GI cocktail–> will relieve MI

19
Q

current STEMI standard criteria

A

any ST segment elevation of over 1mm in all leads other than V2 or V3

20
Q

how to

A

II,III,AVF

I and AVL (lateral)

21
Q

v1,v2,v3,v4 depression

A

inferior wall MI

22
Q

all pts coming in with CP with suspicion of MI

A

i. IV – 2 large PIVs
ii. O2 – Nasal Cannula (could be harmful)
iii. Cardiac monitor – HR/rhythm + BP
iv. At least 2 sets of EKGs/biomarkers

23
Q

b. STEMI Treatment

A

i. Cath lab as soon as possible
ii. Balloon angioplasty or stent
iii. May need bypass surgery if severe or multi-vessel disease
iv. Thrombolytics only if delay in transferring to STEMI center

24
Q

NSTEMI/Unstable Angina Treatment

A

Aspirin - 162mg, NON-enteric coated, chewable
1.Mortality benefit

2.4-5% mortality benefit

Additional anti-platelet agents (e.g. clopidogrel/plavix preferred)

LMWH
small benefit
Nitroglycerin
1. Except in hypotension/R sided MI/recent phosphodiesterase use

analgesia-opiates

25
once admitted
1. High dose statin (Atorvastatin) 2. Beta blockers (after 24 hours) – don’t give in acute phase `3.ACE Inhibitors (when stable) VI. Disposition
26
when would you give BB
Not acute--> associated with cardiogenic shock initially just plavex and ASA send to cath lab maybe second day
27
AD high risk conditions
Marfans syndrome connective tissue disease family history of aortic disease known aortic valve disease recent aortic manipulation
28
high risk pain features of AD
chest, back or abdominal pain described as the followingL abrupt in onset, severe in intensity and ripping/tearing or sharp quality
29
High risk exam features for AD
evidence of a perfusion deficit (pulse deficit, systolic BP differential, focal neurologic deficit- in conjunction with pain ) murmur of aortic insufficiency hypotension or shock state
30
what is the CM of AD
sharp, knife like ripping or tearing pain syncope on exam a pulse deficit new diastolic murmur focal neurological deficit hypotension that may be related to cardiac tamponade, aortic valve regurgitation, acute myocardial infarction
31
whta type of focal neurological deficit would you expect to see in pt with AD
stroke, ALOC, horner syndrome, haorseness, acute paraplegia from spinal cord ischemia
32
most common population with AD
HTN (80%) ▪ most important predisposing factor MC Age 50-60y
33
AD diagnostics
CXR ▪ widening of mediastinum (classic) MRI Angiography → Gold standard CT with contrast ▪ becoming test of choice Transesophageal echo ADD-RS plus D-dimer (low risk with neg D-dimer--> you're good) either one high might need a negative D dimer
34
management of AD
ED management is lowering the HR and BP HR<60 SBP 100-120 opiates for pain control Surgical management ▪ Ascending or ▪ Descending with complications Medical management ▪ Descending if no complications - B-blockers 1st line: Esmolol, Labetolol