Clinical Aspects Of Motor Systems Flashcards

(68 cards)

0
Q

What do you see with issues in muscle strength and Bulk

A

Paralysis
paresis
Atrophy

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1
Q

What do you affect with injury to motor systems

A

Muscle strength and bulk
muscle contraction
muscle tone
muscle stiffness reflexes

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2
Q

What is paralysis

A

The inability to voluntarily produce a muscle contraction

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3
Q

What is paresis

A

Weakness

can produce a muscle contraction but not as strong as it should be

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4
Q

What is hemiplegia

A

Weakness or paralysis on one side or one half of the body

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5
Q

What is paraplegia

A

Weakness or paralysis in the lower extremities

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6
Q

What is tetraplegia or quadriplegia

A

Weakness or paralysis in all four extremities

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7
Q

What is atrophy

A

Decrease in muscle size

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8
Q

What are two kinds of atrophy

A

Disuse atrophy and

neurogenic atrophy

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9
Q

What is the main cause of neurogenic atrophy

A

Lose the nerve to the muscle

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10
Q

Examples of involuntary muscle contractions

A

Spasms
cramps
fasciculations

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11
Q

What might involuntary muscle contractions indicate

A

May or may not indicate pathology

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12
Q

What are fasciculations

A

One motor neuron becomes over excitable and can cause spontaneous contractions almost like a twitch

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13
Q

What are involuntary movements that always indicate a pathological condition

A

Fibrillations

abnormal movements caused by dysfunction in the basal ganglia

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14
Q

What are fibrillations

A

You lose the nerve that goes to the muscle, fibers become more excitable and some start spontaneously contracting
do not see the movement but can pick up through EMG recordings

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15
Q

What are problems in muscle tone

A

Tension in resting muscles

hypotonia and hypertonia

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16
Q

What can cause hypotonia

A

Transection of the ventral root, peripheral nerve, or dorsal root rhizotomy
injury to the cerebellum: often temporary hypotonia

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17
Q

What are the causes of hypertonia

A

Chronic injury to UMN or some basil ganglia

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18
Q

What do you often see in velocity dependent hypertonia

A

Accompanied by hyperreflexia of DTR
clasp knife phenomenon
clonus

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19
Q

What is clasp knife phenomenon

A

Lot of tone when stretching then quick release

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20
Q

What is clonus

A

Rapid dorsiflex and rapid planter and dorsiflex by patient

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21
Q

What is leadpipe rigidity

A

Hi resistance with constant level of increased tone

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22
Q

What is cogwheel rigidity

A

Increased resistance, gives a little, then repeats

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23
Q

What two types of rigidity are seen after severe lesions to the brain

A

Decorticate rigidity

Decerebrate rigidity

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24
Where is the lesion in decorticate rigidity
Superior to the midbrain
25
Where is the lesion decerebrate rigidity
In the midbrain level
26
What do you see in decorticate rigidity
Upper extremity flexion lower extremity extension
27
What do you see in Decerebrate rigidity
Extension upper extremity internal rotation plantarflexion
28
What does decorticate rigidity separate
Separates the cerebral cortex from structures that are caudal to it
29
What does decerebrate rigidity separate
Separates cortex and midbrain from brainstem
30
What happens in spinal or cerebral shock
Descending motor commands are interrupted by injury to upper motor neurons lower motor neurons become temporarily inactivated
31
What does spinal or cerebral shock depend on
The location of the injury
32
What do you see in spinal or cerebral shock
Hypotonia and hyporeflexia that resolves the time
33
What are different causes of disorders of lower motor neurons
Trauma infectious/degenerative/vascular diseases Tumors
34
Where does the injury occur in the nervous system for a disorder of the lower motor neuron
Cell bodies in the spinal cord and brain stem Ventral root spinal and peripheral nerve
35
In an upper motor neuron injury where does the injury occur in the nervous system
Anywhere from where cell bodies are down to where it lower motor neurons are Cell bodies in cerebral cortex axons in the base of the brainstem, cerebral cortex, spinal cord
36
What are abnormal cutaneous reflexes
Babinski sign | muscle spasms in response to normally innoculous stimuli
37
When is the Babinski sign normal
Until six months of age occurring | after injury to the corticospinal tract
38
What causes the Babinski sign
Damage to the lateral corticospinal spinals
39
What happens in paresis
Weakness inability to activate lower motor neurons loss of ability to fractionate movements
40
What is the loss of ability to fractionate movements occur in
Lateral corticospinal tract
41
Myoplastic hyperstiffness post stroke is due to
Loss of sarcomeres increased weak binding of actin and myosin atrophy of muscle fibers
42
What is hyper stiffness
Excessive resistance to muscle stretch
43
What are some changes in muscle fibers after injury
Muscle fiber diameter is decreased | selective atrophy of type two fibers
44
After stroke most patients do not have what
Hyperactive stretch reflexesw
45
Hyperreflexia is seen only in people with
Hemiplegia severe contractures spinal cord injury
46
Evidence suggests that hyperreflexia may result from
Contractures
47
What changes do you see after incomplete spinal cord injury
Contractures hypertonicity predominance of type IIb muscle fibers and a decrease in type one muscle fibers
48
What reflexes do you see after spinal cord injury
Hypertonia Decrease in tonic stretch reflex Clasp knife response clonus
49
Types of upper motor neuron lesions
Spinal cord injury stroke congenital lesions
50
What happens in a complete spinal cord injury
All descending motor control is last below the level of the lesion spinal shock
51
When spinal shock is present at and below the level of lesion what happens
Muscle tone is decreased reflexes are lost voluntary control of movement is lost
52
What happens in spinal shock resolves
Tone is increased monosynaptic reflexes are increased polysynaptic reflexes are decreased voluntary muscle control paresis
53
What happens in a stroke
Occlusion or hemorrhage of a blood vessel in the CNS
54
What does occlusion produce
Lack of blood flow | ischemic stroke
55
What is the most common site for a stroke
Middle Cerebral artery
56
What does an MCA stroke do
Disrupt connections between the cerebral cortex and the brainstem spinal cord and cerebellum damages adult upper motor neuron's
57
What do you see when adult upper motor neuron's are damaged
Paresis | loss of fractionation of movement
58
What also might be damaged in an MCA stroke
Communication systems and sensory systems
59
What does an MCA stroke affect on motor systems
``` Abnormal muscle in activation Corticospinal is decreased lateral reticulospinal is decreased medial reticulospinal is active the vestibulospinal is active ```
60
Standing posture of MCA stroke in the hemiparetic upper extremity
Contracture Weak actin-myosin bonds atrophy of type two muscle fibers
61
MCA stroke standing posture in the lower extremity
Excessive extension in standing and walking
62
What is a congenital disorder of upper motor neuron
Spastic cerebral palsy
63
What do you see in spastic cerebral palsy
Abnormal tonic stretch flexes at rest and while moving Reflex irradiation abnormal cocontraction of antagonist muscles lack of postural preparation prior to movement
64
Signs more common with spinal cord injury
Hyperreflexia of phasic stretch reflexes clonus clasp knife phenomenon
65
Signs common with congenital disorders
Abnormal Co-contraction of antagonistic muscles | Reflex irradiation
66
Upper Motor neuron injury signs and symptoms
``` Paresis fasciculations spasticity hyperreflexia Babinski sign clonus Muscle hyper stiffness Abnormal cutaneous reflexes abnormal timing of muscle activation ```
67
Lower motor neuron signs and symptoms of injury
``` Fibrillations atrophy that is rapid and severe paralysis Loss of reflexes Flaccid paralysis ```