CNS class 3 (SCI, stroke, transfer techniques) Flashcards

(67 cards)

1
Q

SCI define

A

“Occurs from a direct injury to the spinal cord or indirectly from damage to the surrounding bones, tissues, or blood vessels. These events cause paralysis or a complete or total loss of the ability to move or feel sensation in part or most of the body. “
Salvo, Susan; Mosby’s pathology 5th edition

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2
Q

SCI classification

A

direct vs indirect

primary vs secndary

complete vs incomplete

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3
Q

direct vs indirect SCI

A

Direct: direct trauma on the spinal cord

Indirect: damage to tissues and bones surrounding the spinal cord

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4
Q

primary vs secondary SCI

A

Primary: immediate damage caused directly from trauma

Secondary: delayed damaged caused by complications after the injury

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5
Q

complete vs incomplete sci

A

Complete: full lesion of spinal cord → total motor and sensory loss below lesion

Incomplete: partial lesion of spinal cord → partial loss of sensory and motor function

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6
Q

sci etiology

A

trauma vs non-trauma

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7
Q

trauma sci

A

Trauma: more common than non traumatic (can be direct & indirect)

Motor vehicle accidents: most common (41% in Canada) - 97% of times, patients did not wear a seatbelt

Diving: often leads to quadriplegia

Contact sports: American football & rugby (6% in Canada, 17% UK)

Violent trauma: gunshot/stab wounds (incidence increasing)

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8
Q

non trauma sci

A

Spinal hematoma, infection, radiation, neoplasm

Vascular complication: cardiac arrest, aortic aneurysm, surgery

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9
Q

spinal cord injuries (incomplete)

A

central cord syndrome

Brown-Sequard syndrome

Anteiror cord syndrome

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10
Q

central cord syndrome

A

What: Damage to centre with periphery unaffected

Most common incomplete injury

Cause: Hyperextension or arthritic changes to c-spine

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11
Q

central cord syndrome SSx

A

Upper limbs: motor and sensory abilities affected, mm weakness, flaccidity

Lower limbs: less affected

Bowel and bladder control normal or partially affected

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12
Q

Brown-Sequard syndrome

A

What: Damage to one side of the spinal cord

Cause: stabbing/gunshot wound

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13
Q

Brown-Sequard Syndrome SSx

A

Ipsilateral impairment: motor function, proprioception, sensation (vibration, 2-point discrimination).

NORMAL: pain and temperature perception

____

Contralateral impairment: loss of pain and temperature perception.

NORMAL: motor function

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14
Q

Anterior cord syndrome

A

What: damage to anterior spinal artery/anterior spinal cord (corticospinal & spinothalamic tract injury)

Cause: Hyperflexion injury

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15
Q

anterior cord syndrome SSx

A

bilateral loss of motor function, perception (pain, temperature, crude touch)

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16
Q

vulnerable regions of spine

A

Most vulnerable part of the spine is C4-C6, where the spinal canal loses stability in favor of mobility.

T12-L1 also commonly injured.

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17
Q

mm function vs level/severity of SCI

A

C1-C3- No function maintained from the neck down. Need ventilator to breathe

C4-C5- Diaphragm, which allows breathing

C6-C7- Some arm and chest muscles (feeding, dressing, propelling wheelchair)

T1-T3- Intact arm function

T4-T9- Control of trunk above the umbilicus

T10-L1- Most thigh muscles, allows walking with long leg braces

L1-L2- Most leg muscles, allows walking with short leg braces

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18
Q

-plegia etymology & types

A

plegia = strike

.. types in following cards

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19
Q

monoplegia

A

Monoplegia- paralysis of one limb

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20
Q

diplegia

A

Diplegia- paralysis of both upper OR lower limbs

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21
Q

paraplegia

A

Paraplegia- paralysis of both lower limbs

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22
Q

paraparesis

A

Paraparesis- muscle weakness in legs

paresis = from paralysis

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23
Q

hemiplegia

A

Hemiplegia- paralysis upper limb, trunk and lower limb unilaterally

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24
Q

quadriplegia

A

Quadriplegia- paralysis of all four limbs

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25
quadriparesis
Quadriparesis- muscle weakness in all limbs
26
autonomic dysreflexia
What is is? an acute exaggerated sympathetic response. People at risk generally are those with a lesion at or above T6 How? by a painful or uncomfortable stimulus in the abdomen or pelvic area eg: distention of a full bladder ... ---> muscles spasms, an extensive stretch placed on the muscle ---> a kink in the catheter bag ---> the presence of infection such as decubitus ulcers
27
what happens during AD
stimulus sends nerve impulses to the spinal cord - they travel upward until they are blocked by the lesion at the level of injury the impulses cannot reach the brain, a reflex is activated that increases activity of the sympathetic portion of ANS. This results in spasms and a narrowing of the blood vessels, which causes a rise in the blood pressure.
28
AD SSx
severe hypertension (300/160), bradycardia sudden pounding headache Vasospasms, piloerector response and skin pallor occur Flushed skin and sweating
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AD -- what to do?
It is considered a medical emergency and is potentially life threatening call for paramedic services
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MASSAGE & SCI
Most clients are wheelchair-bound, adapt massage to clients need and abilities Clients are generally inactive so have a larger risk to bone density and blood clots in the legs. Avoid deep pressure over bones and Forceful PROM because of risk of fractures Avoid vigorous massage techniques because of risk for clots ---> ALSO B/C DEEP TECHNIQUE IS CI'ed ON ATROPHIED MM Assess contractures Assess for potential decubitus ulcers
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stroke happens when
when blood flow to the brain is blocked or there is sudden bleeding in the brain
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hemiplegia
a non-progressive condition of paralysis on one side of the body
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stroke, result
insult/injury/event The range of consequences is very wide; it may cause death; it may leave a permanent state of dysfunction, it may be partially recovered from, it may be fully recovered from
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hemiplegia -- sometimes via
It sometimes results from a stroke It may be caused by many things: genetics, stroke, spinal cord injury, other brain injury…
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side of paralysis vs side of brain lesion
Paralysis occurs on the opposite side of the brain lesion
36
severity of effects (stroke) duration/severity
Effects depend on the location and how severe the damage to the brain They can be temporary or permanent, almost imperceptible to severely disabling with profound spasticity and extreme sensory or perceptual loss
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(FYI) LEFT brain damage result
right side paralysis speech and memory deficits cautious and slow behaviour
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(FYI) RIGHT brain damage result
left side paralysis perceptual and memory deficits quick and impulsive behaviour
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hemiplegia SSx post-stroke
Immediately post stroke: muscles on the affected side will be weak or flaccid - acute phase gradually, flaccid paralysis leads to spastic paralysis (some flaccidity can remain) ---> lesions interfere with the brain's control over spinal cord activity = uninhibited alpha motor neuron firing
40
flexor vs extensor pattern of paralysis most common pattern UE? most common pattern LE?
This type of paralysis can become a flexor or extensor pattern based on the uninhibited dominant reflexes ---> Most common pattern: flexor pattern in upper limbs combined with extensor pattern in the lower limb
41
other SSx hemiplegia
Altered posture due to spasticity Altered gait - circumducted Hemiplegic shoulder - GH ADd, IR, scapular retraction Seizures, edema, pain, compensatory changes Sensory deficit Shoulder-hand syndrome - decreased ROM of GH & hand, followed with throbbing pain & edema Neglect of affected side Behavioral & emotional changes Visual impairment, speech difficulties Cognitive impairment
42
more observations/clinical manifestations/SSx (hemiplegia)
Spasticity pattern on the affected side Circumducted gait, balance and weight shifting may be impaired Ambulatory aids - canes, walkers Muscle bulk differences between affected and unaffected sides Functional abilities will vary depending on severity of stroke and spasticity or flaccidity Postural asymmetries Neglect of the affected side
43
stroke occurs when
A stroke occurs when blood supply to any part of the brain is interrupted, leading to the impairment of brain cells. The level of impairment depends on the location and degree of damage done.
44
3 main types of strokes:
ischemic stroke hemorrhagic stroke transient ischemic attack (TIA)
45
TIA lasts how long?
less than 24 hours "A transient ischemic attack, commonly known as a mini-stroke, is a temporary stroke with noticeable symptoms that end within 24 hours." "Transient ischemic attacks usually last a few minutes. Most symptoms disappear within an hour. Rarely, symptoms may last up to 24 hours."
46
rare causes of stroke
In rare cases, an underlying condition such as a tumor, an infection, or brain swelling due to an injury or illness can cause a stroke.
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stroke risk factors (within control)
Unhealthy weight - hypertension (high BP) high cholesterol, diabetes, heart disease Physical inactivity Excessive alcohol & drug abuse Smoking Stress Birth control/ hormones replacement therapy (HRT)
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stroke risk factors
Gender Age Family history Ethnicity History of stroke or TIA
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main modifiable risk factors (stroke)
Blood pressure (hypertension is associated with hemorrhagic strokes) Atherosclerosis (atherosclerosis is associated with ischemic strokes)
50
stroke FAST signs
Face – facial numbness or weakness, especially on one side Arm – arm numbness or weakness, especially on one side Speech – slurred speech or difficulty speaking or understanding Time – time is important; call EMS/9-1-1 immediately
51
warning signs (stroke)
Sudden numbness or weakness in the face, arm or leg (especially on one side of the body). Sudden confusion or trouble speaking or understanding speech. Sudden vision problems in one or both eyes. Sudden difficulty walking or dizziness, or problems with balance & coordination. Severe headache with no known cause.
52
Brunnstrom stages of recovery (stroke)
1. Flaccidity (immediately after the onset) No "voluntary" movements on the affected side can be initiated 2. Spasticity appears Basic synergy patterns appear Minimal voluntary movements may be present 3. Patient gains voluntary control over synergies Increase in spasticity 4. Some movement patterns out of synergy are mastered (synergy patterns still predominate) Decrease in spasticity 5. If progress continues, more complex movement combinations are learned as the basic synergies lose their dominance over motor acts Further decrease in spasticity 6. Disappearance of spasticity Individual joint movements become possible and coordination approaches normal 7. Normal function is restored
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Stage 1 - Flaccidity
PROM Passive stretching (gentle) Position to support upper limb, especially shoulder (subluxations common in this stage)
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Stage 2 – Spasticity develops and abnormal synergies may be present
Continue with PROM (very slow, gentle to not provoke spasticity) Massage for sensory stimulation
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Stage 3 – Spasticity increases
Continue with above techniques Increased emphasis on stress reduction Massage to relax spastic/synergistic areas May be using splints
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Stage 4 – Spasticity decreases
Continue with above techniques Added emphasis on using the recovering arm as much as possible with home care and ADLs PNF patterning (PROM, AROM)
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Stage 5 – Spasticity continues to decrease (minimal)
Continue with above Begin strengthening routine (as opposed to ROM-based exercises) PNF patterning (RROM)
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Stage 6 – Spasticity disappears and coordination reappears
Continue with above and focus on fine motor skills (e.g. hand and finger exercises)
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stroke hx q sample
goals energy levels ADLs? P? where? hm? when? rehab presentation? sensory? motor? when? stress/mental health meds
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stroke ax sample
palpation MMT ROM (goniometer) postural
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stroke goals sample
decrease SNS ---> inh ROOD, DB, heat, slow GSM decrease spasticity ---> GTO, inh ROOD increase strength of weak mm (E.g. extensors) ---> neural tapping, stim ROOD, isometric/RROM maintain jt health JM, PROM, circumduction increase ROM slow stretch, PROM
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Synergy
A whole series of muscles are recruited when just a few are needed. For example, when trying to reach forward, the shoulder abducts and elevates, and the wrist flexes
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synergy patterns (stroke)
5 patterns 1 (AKA Flexor synergy (spastic?) pattern) = IR, add (GH) = flexion (HU) = supination (RU) = flexion (RC) 2 = same as 1, except wrist = extension 3 = same as 1, except forearm/wrist are neutral 4 = same as 1, except forearm = pronation 5 (AKA extensor synergy (spastic?) pattern) = IR, retroversion (GH) = extension (HU) = pronation (RU) = flexion (RC) ---> Looks like waiter's tip
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E.g. Ax
Palpation / postural ROM Sensory testing
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E.g. goal
Promote relaxation Decrease pain Addressing postural changes/ muscle imbalances Decrease edema Decrease spasticity Maintain joint health Full body integration
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Tx/goals rationale
Relaxation and stress management because stress makes spasticity worse PROM and stretching are important to prevent neglect of the affected limb, to maintain sensory mapping of the area, and to prevent contracture formation Trying to lower the tone of spastic or overused muscles is important to decrease contracture formation Decreasing edema is important for tissue health Trying to stimulate muscles outside of synergy or opposite to spasticity PROM and joint play to maintain joint health Full body integration for overall wellbeing
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