Critical Care Flashcards
(99 cards)
What percent will a dog or cat recover when they go into cardiopulmonary arrest
<10% but since RECOVER it has increased
Higher ROSC rates are seen in
1) patients with witnessed CPA
2) IVC in place at time of arrest
3) Palpable pulses were generated during CPR
Patients had higher odds of survival to hospital discharge if **
Peri-anesthetic arrest
Were cats or dogs more likely to survive to hopsital discharge after cardiopulmonary resuscitation
cats, same with those of lower body weight
CPA during _____ time was associated with worse patient outcomes
night
Were more or less people associated with decreased offs of ROSC
more people
What two devices do we hook up to for a cardiopulmonary arresting patient
1) End-tidal CO2 (18+ mmHg)
2) ECG
We do not administer high volumes of IVF for cardiopulmonary arrest except for __________ *
when they are hypovolemic (e.g bled out)
when doing CPR, we dont want too much fluid sitting in the venous side
No fluids during CPR unless
hypovolemic (e.g bled out)
What are the non-shockable rhythms
Asystole
PEA
What can you do for neuroprotection after cardiac arrest
Hypothermia if comatose
Mannitol /HTS if neuro signs
Seizure prophylaxis
What are physical signs of shock
1) Abnormal mucous membrane color
2) Tachycardia (cats = bradycardina <120bpm)
3) Cool distal extremities
4) Abnormal pulse quality
5) Abnormal Capillary refill (CRT)
6) Decreased level of consciousness
What are the types of shock
1) Hypovolemic (blood volume)
2) Distributive (blood vessels)
3) Obstructive (vessel occlusion)
4) Cardiogenic (heart/pump)
5) Metabolic
6) Hypoxemic
What are causes of hypovolemic shock
Acute blood loss
-Trauma
-Coagulopathies
-Surgery
-GI bleeding
-Neoplasia/necrosis
Profound dehydration
-Gastroenteritis
-Heat stroke
-Renal disease
-Diabetes
What are compensatory factors for loss of vascular volume
Once the stretch on the baroreceptors occurs
1) Release of catecholamines, Aldosterone, Cortisol, ADH, activation of renin-angiotensin
2) Increased systemic vascular resistance, heart rate, contractility, splenic contraction, renal water retention
3) Increased venous return cardiac function
What are causes of distributive shock
1) Bacterial (pneumonia, perforated intestines, wounds, GI translocation)
2) Trauma
3) Burns
4) Pancreatitis
5) Anaphylaxis
What are causes of cardiogenic shock
Dogs:
-Valvular disease
-DCM
-Pericardial effusion “obstructive”
-Atrial fibrillation
Cats:
-HCM and FUCM
-Restrictive cardiomyopathy
-DCM
What is the pathophysiology of cardiogenic shick
-Myocardial failure (failure as a pump)
-Cardiac tamponade (inadequate ventricular filling)
-Arrhythmias (ineffective contraction or inadequate filling)
How often should you run PCV/TP for shock patients
prior to fluids and every 30-60 minutes until stable
What are the steps of the primary survey
1) Exsanguination (frank, red bleeding, or distended brusined abdomen)
2) Airway (Upper airway sounds, or visual sweep)
3) Breathing (Increased respiratort effort, bronchovesicular sounds, or absent)
4) Circulation (Shock, mucous membranes, pulse quality)
5) Disability: Level of consciousness, brainstem reflexes, motor activity
6) Exposure/Enviroment: cover open wounds and minimize heat loss
What signs might show that a patient has exsanguination
1) frank, red, pulsatile bleeding
-is there blood present on a hand sweep
2) Distended, bruised abdomen
if evaluation of exsanguination reveals no life-threatening abnormalities, what are the adjunct tools that are not indicated until after the primary survey is completed
POCUS
Blood pressure
PCV/TP
Lactate
What immediate actions can you do for exsanguination
Compression
Packing
Application of a hemostatic clamp
Why should you not immediately reach for POCUS to evaluate for internal bleeding
It can be evaluated further during the circulation step of the primary survey and/or during the secondary survey based on the patient status