Critical Care/ICU Monitoring Flashcards

1
Q

What are five indications for critical care continuous EEG?

A

-diagnosis of Non-convulsiveand NCES or paroxysmal events, efficacy of therapy for sz and SE,identification of cerebral ischemia, monitoring of sedation and high-dosesuppressive therapy, assessment if severity of encephalopathy andprognostication

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Once an ICU/critical patient is properly set up for continuous EEG, what do ACNS guidelines recommend that the technologist should do next?

A

remain at bedside for first20 minutes, evaluate EEG patterns requiring urgent interp, examine behavior, ensure acceptable data quality, perform activations/test reactivity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

· What things should be checked by the technologist daily for critical care continuous EEG recording?

A

technical artifacts (at least 2x daily), impedance, electrode stability, patients skin for breakdown, asses reactivity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

How often should a patient’s skin be assessed in critical care EEG?

A

daily with written protocols on how and when to check and document

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Rank by severity from LEAST to MOST sever: (A)Burst-suppression,(B) alpha coma, (C) spindle coma, (D) ECI, (E) high-voltage arrhythmic delta, (F)triphasic, (G) IRDA

A

IRDA, HVAD, triphasic, spindle coma, alpha coma, burst-suppression, ECI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

How often should a cEEG be reviewed by a qualified enchepalographer for important events?

A

at least twice a day

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

The preferred term for referring to seizures without prominent motor activity

A

Nonconvulsive

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

DCI

A

Delayed Cerebral Ischemia, clinical deterioration and/or cerebral infarction due to vasospasm after SAH. Usually causes wide spread EEG changes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

ECMO

A

Extracorporeal membraneoxygenation. Requires ligation of the right common carotid artery and right jugularvein

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

GCS

A

Glascow Coma scale classifies the severity of TBI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Whatare four EEG features that are considered favorable prognostic features?

A

background continuity, spontaneous variability, reactivity to stimulation, presence of normal sleep patterns

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

HIE

A

Hypoxic-ischemic encephalopathy, major cause of morbidity and mortality among term newborns

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

ICH

A

Intracranial hemorrhage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Nonconvulsive Seizure (NCS)

A

far more common than convulsive seizures in children

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Electrographic Status Epilepticus (ESE)

A

electrographic seizure for>/ 10 continuous minutes or for a total duration of >/20% of any60-minute period of recording

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

BIRDs

A

Brief potentially ictal rhythmic discharges. >4 Hz in frequency and >/ 0.5 to <10seconds induration

17
Q

SAH

A

Subarachnoid Hemorrhage

18
Q

TBI

A

Traumatic Brain Injury

19
Q

Therapeutic Hypothermia (TH)

A

associated with improved survival in comatose adults after cardiac arrest

20
Q

SIRPIDs

A

Stimulus-induced rhythmic,periodic, or ictal-appearing discharges

21
Q

What EEG findings are seen in intraventricular and subarachnoid hemorrhage?

A

Diffuse slowing and generalized, frontally predominant rhythmic delta (formerly FIRDA)

22
Q

Rasmussen’s Encephalitis

A

Rare inflammatory neurological disease, mostly children and young adults, typically affects one hemisphere, pharmaco resistant epilepsy, progressive loss of motor/speech skills and hemiparesis with uni-hemispheric brain atrophy

23
Q

Alpha coma

A

8-13Hz, mostly frontal areas. Caused by intoxications, brainstem lesions and hypoxic-ischemic encephalopathy

24
Q

Beta coma

A

generalized 12-16Hzbackground activity, maximally frontal regions. Intoxication or withdrawal and severe hyperthyroidism, and brain lesions

25
Q

Spindle Coma

A

predominant theta and delta background activity w/ super imposed, frequent, paroxysmal spindle-shaped bursts. TBI, ICH, intoxication, post-ictal states, hypoxic-ischemic encephalopathy

26
Q

MELAS

A

Mitochondrialencephalopathy, lactic acidosis and stroke like episodes caused by a genetic mutation

27
Q

CBF

A

Cerebral Blood Flow. Normal CBF is 50-70 mL/100g/min

28
Q

What is RAWOD and what is its significance?

A

Regional attenuation without delta. Clinical Significance, Distinctive pattern that indicates a massive and irreversible stroke in ICA/MCA territory

29
Q

What is the most sensitive neurodiagnostic tool for detecting cerebral ischemia and correlates with its location and degree?

A

EEG. Detects reversible andirreversible cerebral ischemia

30
Q

What are the morphology and frequency changesseen in EEG that correlate with CBF level of 35-70mL/100g/min?

A

Normal EEG, no neuronal injury

31
Q

What are the morphology and frequency changes seen in EEG that correlate with CBF level of 25-35mL/100g/min?

A

loss of fast beta frequencies in EEG. Reversible neuronal injury

32
Q

What are the morphology and frequency changes seen in EEG that correlate with CBF level of 18-25mL/100g/min?

A

slowing background to 5-7Hztheta in the EEG. Potentially reversible neuronal injury

33
Q

What are the morphology and frequency changes seen in EEG that correlate with CBF level of 12-18mL/100g/min?

A

Slowing of background to 1-4Hz delta in EEG. Potentially reversible neuronal injury

34
Q

What are the morphology and frequency changes seen in EEG that correlate with CBF level of <8-10mL/100g/min?

A

-Suppression of all frequencies in EEG. Neuronal death