Cutaneous Mycoses of skin Flashcards

(80 cards)

1
Q

Otomycoses caused by

A

aspergillus
penicillium
mucor
rhizopus

(opportunistic molds that cause outer ear infections

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2
Q

epi otomycoses

A

world wide
soil
humans and animals are NOT reservoirs

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3
Q

transmission otomycoses

A

coming in contact with contaminated fomites

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4
Q

path otomycoses

A

accumulation of large masses of fungi or bacteria in external ear canal

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5
Q

s/s otomycoses

A

chronic ear infection itching pain inflammation and loss of hearing

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6
Q

dx otomycoses

A

fragments of mycelium with or without septa

conidiophores and sporangiospores may be seen

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7
Q

tx otomycoses

A

ketoconazole

wax hook

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8
Q

tinea faciei caused by

A

trycophyton mentagrophytes
trycophyton rubrum
infection on glabrous skin of face (hair less)

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9
Q

epi of tinea facial

A

worldwide

p2p p2m fomite and aerial

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10
Q

pathogenesis of tinea facei

A

by trycophyton

hypae growing release enzymes producing edema and leukocytic infiltrations

mold allergins cause granuloma formations

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11
Q

tinea facei immunity

A

PARTIAL protective immunity

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12
Q

s/s of tinea facei

A

calin annular or circular lesions with raised margins and papule
pruritus burning and erythema are present

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13
Q

dx tinea facei

A

hyphae with microconida

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14
Q

tx tinea facie

A

myconazole

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15
Q

tinea barbae

aka

A

trycophyton interdigitale
trycophyton violaceum
aka barbers itch

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16
Q

epi of tinea barbae

transmission

A

worldwide
commonly in rural areas (farmers) with high temp and humidity

p2p m2p fomite and aerial

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17
Q

pathogenesis of tinea barbae

A

trycophyton

mycelia grow into pores of hair shafts causing edema and leukocyte infiltrations

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18
Q

immunity tinea barbae

A

partial

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19
Q

s.s tinea barbae

A

trycophyton

mild superficial- scaly lesions with vesiculopusturlar borders bearded areas

  • deep pustular- follicular pustules on bearded areas of skin
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20
Q

dx tinea barbae

A

trycophyton

microconida on hyphae

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21
Q

tx tinea barbae

A

trycophyton

terbinafide

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22
Q

tinea capitis ectothrix caused by

AKA

A

microsporum
(audouini and canis) `

aka grey patch ring worm

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23
Q

tinea capitis ecto epi

A

by microsporum

ecothrix world wide
tina favosa- africa and europe

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24
Q

tinea Capitis ecto transmisison

A

p2p m2p fomite airborne

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25
tinea capitis ecto immunity
partial
26
tinea capitis ecto s/s
microsporum begin as coalesced papule and develop into legions (weeping) and keloids (scars) allergic reaction -- causing severe itching and alopecia
27
tinea favosa
type of tinea capitis caused by tricophyton schoenleini - mycelia interact with s camp proteins producing dead cell masses in mats of hair-- yellowing brown cup shaped skin crusts
28
tx tinea capitis exto
microsporum tx- griseofulvin
29
dx tinea capitis exto
hyphae with macroconida
30
difference between tinea capitis endo and exo
exo- microsporum | endo- trichophyton (harder to get rid of)
31
tinea capitis endo caused by AKA?
trichophyton (tonsurans and violaceum) aka black dot ring worm*
32
path tinea capitis endo
trichophyton conidia found inside hair shaft if cuticle involved- initial lesions that develop into erythematous circular lesions
33
immunity tinea capitis endo
by trichophyton | partial
34
s/s tinea capitis endo
erythematous scaling circular lesions w black dots (broken off hairs)--- infection can result in hair loss
35
dx tinea capitis endo
conydia and hyphae inside hair shafts
36
tx tinea capitis endo
terbinafine
37
tinea corporis caused by
trichophyton (interdigital and rubrum ) epidermophyton floccosum microsporum canis
38
epi tinea corporis
world wide | transmission (4)
39
path tinea corporis
hyphae or conydia infect horny layer of skin -- spread out in glabrous skin
40
s.s tinea corporis
1. annular - small spreading circular areas of erythema that become scaly in center with red margins BECOME CHRONIC 2. vesicular - Vesicles and pustules behind erythematous border
41
dx tinea corporis
mycelia and conidiospores | microsporum canis cas club shaped macroconidia with rough walls
42
tx tinea corporis
terbinafine
43
tinea unguium caused by
epidermophyton flocosum | trichophyton interdigitale
44
tinea unguium associated with
tinea pedis and magnum infections in males after puberty
45
tinea unguium pathogenesis
unknown incubation period, nail injury predisposes infection infection starts under nail plate of fingers or toes , grow out of nail plate into stratum corneum
46
s/s tinea unguiumm
soft friable keratin, THICKENED NAIL | causes white patches/pits
47
dx tinea unguium
hyphae or macroconida presen t
48
tx tinea unguium
terbinafide most infections become resistant and are chronic
49
sub ungual dermatophytosis
lateral or distal edges of nail are first involved, then develop into nail plate-- brittle friable and thickened
50
tinea manuum caused by
tricophyton interdigitale epidermophyton flocossum microsporum gypseum on palms and fingers
51
epi tinea manum
world wide | associated with males
52
pathogenesis tinea manum
infection begins due to maceration ( moisture due to extended wear like gloves) due to occupational activities infection result in lesions that are exfoliative, erythematous and scaly sheets if stub
53
s.s. tinea manum
diffuse hyperkeratosis of palms and fencers with concentric exfoliation of skin scaly sheets develop on dorsal of hand
54
dx tinea magnum
hyphae macroconida
55
tx tinea magnum
miconazole
56
tinea cruris caused by AKA
trycophyton rubrum epidermophyton floccosum jock itch
57
epidemiology tinea cruris
worldwide p2p areal and fomites 20-30 males commonly infected
58
pathogenesis tinea cruris
hyphae invade groin and perianal areas causing edematous lesions
59
s/s tinea cruris
circular lesions in groin and scrotum areas develops into serpiginous* erythematous lesions erythema and intense itching occur
60
dx tinea cruris
epidermophyton- club shaped macroconidia tricophyton- microconidia
61
tx tinea cruris
terbinafine loose clothes and lsoss of weight lessen chance of infection
62
tinea pedis caused by aka
tricophytoon interdigitale and rubrum epidermophyton floccosum aka athletes foot
63
epi of tinea pedis
most common fungal infection of humans | p2p aerial and fomites
64
pathogenesis of tinea pedis
breakdown in host skin promotes invasion of epidermis, localized to keratinized skin ( between toes) - papulosquamous hyperkeratotic skin is seen
65
immunity tinea pedis*
poor cel mediated- no partial or protective immunity
66
s.s tinea pedis
itching intertriginou dermatitis with peeling maceration and fissuring of skin intertriginous (between 3 4 5 toes) also vesicular form that ulcerates-- cause secondary bacterial infections to develop
67
dx tinea pedis
stained prep of hyphae with micro or macroconida
68
tx tinea pedis
terbinafide
69
onychomycosis caused by
``` arthroderma aspergillus candidda geotrichum scopulariopsis ```
70
epidemiology onychomycosis transmission who gets it?
worldwide in soil transmitted via implantation with spore contaminated fomites nail infection accompanies disease that causes patient to be immunosuppressed*
71
pathogenesis of onychomycoses
damage to nail architecture THAT DOES NOT THICKEN painful with brittleness of nails
72
dx of onychomycoses
various forms of hyper elements- branching non branchin
73
tx of onychomycoses
ittraconazole
74
description of candidiasis
monomorphic opportunistic yeast
75
epi of candidiasis
normal floral human | p2p nosocomial and fomites
76
path of candidiasis farnesol?
gelatinous types of lesions develop -- due to mucoid degeneration of tissue --- causing formation of large emboli farnesol helps turn from invasive to non invasive
77
s/s candidiasis
thrush, etc paronychia- swelling of paronychial folds systenic- fever headache anorexia myalgia hepatosplenomegaly heart problems can be induced due to prolongues antibiotic tx
78
dx candidiasis
chains of oval budding yeast cells with pseudophyphae and blastospores in serum* in cheek its part of normal flora
79
tx candidiasis
fluconazole or amphotericin B
80
immunity of candidiasis
NONE | allergic rxns can develop