Degenerative diseases of the CNS Flashcards

1
Q

How can you optimize the quality of life?

A

By providing AE, task strategies, etc

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2
Q

Probable decline in performance of an occupation.
Inconsistent preformance throughout the day.
Impact on the family is related to role change, caregiving needs, and potential loss.
Limited medical intervention.
Psych issues

A

Fairs that influence progressive neurological patients

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3
Q

Pneumonia, blood clots, or pressure ulcers/bedsores

A

Preventative secondary complications

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4
Q

Damage or lesions in neural pathway above the anterior born cell of the spinal cord

A

Upper motor neuron dysfunction

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5
Q
Muscle weakness
 Incoordination, slow movements
spasticity
Babinski sign
increased deep tendon Reflex (DTR)
Pronator drift
A

Symptoms of UMN deficits

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6
Q

Lesion that affects nerve fibers traveling from the anterior horn of the spinal cord to muscles

A

Lower motor neuron dysfunction

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7
Q
Muscle weakness
Fasciculations (shaking, tremors, jerkiness)
Hypotonia
decreased deep tendon Reflex
absent Babinski
A

Symptoms of LMN deficits

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8
Q

An autoimmune disease that affects the CNS.

The immune system attacks myelin, causing demyelination in multiple areas

A

Multiple sclerosis

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9
Q

Leaves scars known as sclerosis, plaques or lesions

A

Demylenation

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10
Q

The protective coating that goes around the nerve

A

Myelin

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11
Q

Acute attacks with full or partial recovery; 85% of initial diagnosis

A

Relapsing and remitting

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12
Q

Starts with relapsing and remitting course followed by progression at a variable rate; 50% develop in 10 years and 90% within 25 years

A

Secondary progressive

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13
Q

Progressive without remission; 10% of initial diagnosis

A

Primary progressive

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14
Q

Progressive with acute relapses; 5% of initial diagnosis

A

Progressive relapsing

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15
Q

Most common symptom of MS caused by demyelination

A

Fatigue

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16
Q

Avoid fatigue, cooler room it normally better, use heart modalities with caution, levels of Independence may fluctuate throughout the day, gaurd against soft tissue injury, monitor for safety issues, be aware of the combined effects of cognitive and physical dysfunction

A

The OT management of MS

17
Q

Due to disease process

A

Primary fatigue

18
Q

More controllable, due to deconditioning and respiratory muscle weakness and pain

A

Secondary fatigue

19
Q

Control of tremors and movement disorders, cognitive compensation, managing sensory deficits

A

Some OT interventions for MS

20
Q

One method to help with Diplopia

A

Cover one eye

21
Q

A contagious vital disease

A

Poliomyelitis (polio)

22
Q

Bedrest, positioning, and warm packs

A

Medial treatment for polio

23
Q

Impairment occurring years after having polio with satisfactory function in the interim

A

Post-polio syndrome

24
Q

Evaluate how strenght, ROM, and endurance affects ADLS, occupational performance, and psychosocial status. Assessment of psychosocial status is needed.
Overwork of muscles should be avoided.
Manage pain with body mechanics, support of weak muscles, and lifestyle modification

A

OT interventions for treating post polio syndrome

25
Q

Acute inflammatory condition of the spinal nerve roots, peripheral nerve, and in some cases, selected cranial nerve

A

Guillain-Barre syndrome

26
Q

Rehab is initiated when the patient is stable.
Initial focus on PROM, positioning, and splinting to prevent contracture.
Graded activities as the patient progresses.
Always gaurd against fatigue and irritation of the inflamed nerve.
Use AE and energy conservation techniques as needed.
Progress with upright positioning and activities.
Provide psychosocial support

A

OT treatment for Guillain-Barre syndrome