Developmental disorders Flashcards

(44 cards)

1
Q

list aspects that create environment

A

diet, maternal drug abuse, orthnography, social envionment, teaching style, deprivation, trauma

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what % of people are affected by developmental dyslexia?

A

3-6%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

talk about dyslexia prevelance

A

cross-cultures
across lifespan
more males than females.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what does dyslexia inhibit?

A

reading achievement is Substantially below that expected given the person’s
chronological age, measured intelligence and age-appropriate
education

Significantly interferes with academic achievement or
activities of daily living that require reading skills

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

talk about causation of dyslexia

A

is not explained by a sensory deficit.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

talk about memory deficits in dyslexia

A

verbal short term memory deficits- norm memory for visual info poor memory for verbal info.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what deficit do dyslexic patients have regarding naming objects

A

Rapid Automated naming deficits- naming well known objects at speed

Denckla & Rudel 1976

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

talk abut dyslexia and phonological awareness deficits

A
  • Syllable-tapping task: tap out the number of syllables (or beats) that youcan hear in the word (cat=1; banana=3)
  • Onset-rime (crust-cross) & initial-final phoneme (coat-goat) judgment tasks: both names were pronounced by the experimenter, and the subjects were asked to decide whether the names had any sounds in common.
  • Phoneme-tapping task: tap out the number of sounds that they could hear (cat=3)

deficits in all

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

discuss visual deficits in dyslexics

A

Cornelissen et al.

dyslexics have higher motion coherence thresholds than controls

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Describe Buck’s phonological account to dyslexia

A

DIFFICULTY ANALYZING THE SOUND STRUCTURE OF LANGUAGE. this leads to a failure to learn systematic relationship between spellings and sounds. failure to master spelling sound correspondences is a primary source of the word recognition problems.

-innacurate representations not difficulty analysing.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what is a flaw with Buck’s phonological account to dyslexia?

A

a very specific account in that the problems are constrained to the domain of language

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what is there debate about regarding dyslexia?

A

how different phonological functions/ issues are related (e.g. rapid naming vs decoding)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

discuss dyslexia in terms of a temporal processing deficit

A
  • have difficulties percieving short streams of sound
  • difficulty perceiving tones of sound–> problems representing speech at a fine-grained level.
  • correlations between rate of auditory processing and reading errors.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

discuss dyslexia in terms of a magnocellular deficit

A
  • visual difficulties- blurring/ moving/ inversed words and letters.
  • magnocellular stream is atypical in dyslexia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

what does a magnocellular deficit account for

A

behavioural results of impared visual motion and reduced contrast sensitivity at low spatial frequencies

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

How can the magnocellular deficit system be reduced?

A
  • eye movements. letters appear to move (coverning one eye reduces errors)
  • attention and peripheral vision (reducing field of view reduces errors)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

what two explainations are there for dyslexia?

A
  • Phonological processing deficit

- sesorimotor deficit

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

what is included within the sensorimotor deficit explaination of dyslexia?

A

temporal processing, magnocellular visual system, poss also cerebellum.

19
Q

what did Ramus et al. find to be the only deficit found in every dyslexic patient?

A

phonological processing

20
Q

what chromosomes are thought to have an influence on dyslexia?

21
Q

what gives evidence for the heritability of dyslexia?

A
  • family studies

- chromosomes

22
Q

what is the direct biological evidence to the magnocellular theory? and when would these arise

A

Postmortem: magnocellular layers of the LGN were disordered, 30% smaller than average neurones (Galaburda and Livingstone, 1993). These differences arise during the 4th or 5th month of foetal development.

23
Q

what is suggested that is needed for the magnocellular system to develop?

24
Q

what biological evidence suggests the phonological processing system may cause dyslexia?

A

Normal- asymmetry of L/R planum temporale (L is bigger)

Dyslexics- symmetrical or R is bigger

poss link between symmetry of L/R planum temporale and severity of phonological deficits

25
what study suggests that there may be an environmental element to dyslexia?
Paulesu et al. shallowness/ depth of orthography
26
discuss autism prevalence
1 in 100 children born more males present through life at all IQ levels
27
define vaguely autism
a severe disorder of communication, socialization and imagination
28
how do those with autism face difficulty in social relationships?
poor use of non-verbal behaviour e.g. facial expressions. lack of spontaneous seeking to share enjoyment / interest lack of recognition of affect lack of social/emotional reciprocity limited sharing
29
how do those with autism face difficulty in language and communication?
- delay / lack of language - atypical nonverbal communication - repetitive language - one sided, awkward conversations - limited pretend play
30
talk about autism and repetitive behaviour
- preoccupation with restricted interests - inflexible, non-functional routines / rituals - stereotyped, persistent motor mannerisms - narrow, restricted fields of interest
31
what are the two deficit accounts of autism?
- theory of mind | - executive functions
32
what is the processing style account of autism?
-local processing
33
when given a theory of mind experiment (where will sally look for her toy) with 3-4 children who are normal, ASD and down syndrome how many pass?
normal- 85% Down syndrome-86% ASD- 20% Baron-cohen
34
how much later do children with autism develop theory of mind?
5 years later | fail to understand mental states
35
how do autistic children compare to normal children on executive function?
planning and cognitive flexibility is far lower normal- 40% ish autism- 33% ish
36
do autistic children have a strong or weak central coherence?
weak
37
how do autistic children percieve features
features are retained and heavily perceived but at the expense of global configuration and meaning.
38
regarding local and global info when will autistic children perform poorly and well?
good performance where attention to local information is an advantage poor performance on tasks requiring recognition of global meaning or integration of stimuli in context
39
what does Happe argue regarding autism explainations?
shouldnt expect there to be one single one
40
discuss autism and genetics
not only autism itself is heritible but also relatives show increased rated of the broader autism phenotype or isolated traits.
41
what did Baron-Cohen show about autism using fMRI?
when asked to pick emotional state/ if people were male/ female from pics of the eye region. people with autism showed less extensive activation in frontal regions and no activation in the amygdala.
42
what did Casteli find using fMRI on autistic patients?
``` ASD (adult) group showed less activation than typical controls in this ‘mentalising system’: Medial-prefrontal Temporo-parietal Amygdala region ```
43
what has been consistent in findings across studies of autism regarding the brain?
Mentalising- underactivation of social brain- PFC, ST, Amygdala- during social processing tasks Overaction of frontostraital regions (dIPFC, BG- reward areas) during restricted/ repetitive task less lateralised patterns during language tasks fewer long range connections and more shorter ones. decreased anterior> posterior connections
44
is there a coherent accepted model for ASD yet?
no