Eating Disorders Lecture - part 2 Flashcards

1
Q

Co-morbidity with eating disorders

A
  • More than half of those with AN have another mental illness
  • 88-97% with BN have another mental illness
  • Most common co-morbids include mood and anxiety disorders - depression, OCD, social anxiety
  • Strong association with AN and autism spectrum disorders
  • Substance abuse
  • Can be part of EUPD - method to regulate emotions
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2
Q

Where do eating disorders often originate from? - emotion wise

A
  • Coping mechanism for different emotions - distraction, numbing
  • Can help someone feel protected, more confident and less anxious - get compliments initially
  • Can provide a sense of feeling special
  • May present a barrier to engaging with treatment - feels part of self, people want to look after you more
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3
Q

Causes of eating disorders

A
  • Genetics
  • Biological vulnerability
  • Biological stress
  • Psychological vulnerability
  • Psychological stress
  • Social/cultural
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4
Q

Biological pre-disposing factors for eating disorders

A
  • Genes
  • Neurotransmitters - dopamine, serotonin, noradrenaline
  • Hormones
  • Physical illness/allergies eg always had to look at labels, T1DM
  • FH depression, anxiety, addiction
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5
Q

Psychological predisposing factors to eating disorder

A
  • Low self esteem
  • Feelings of ineffectiveness or lack of control
  • Self identity
  • History of depression/anxiety/mood intolerance
  • Personality traits - perfectionism, obsessional
  • Interpersonal style - struggle to recognise cues and emotional states of others
  • Emotional processing - difficulty recognising own emotional state, expressing emotions and difficulty handling stress
  • Thinking style - cognitively rigid, all or nothing thinking, can’t see bigger picture
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6
Q

Social/environmental pre-disposing factors of eating disorders

A
  • History of bullying - esp about weight
  • Trauma - all forms abuse
  • Stressful life events - grief, loss
  • Difficult interpersonal relationships - conflict within family, high expectations, stressful family circumstances, overprotection
  • Taking part in competitive sports - dancing, gymnastics
  • Growing up in household that places value on appearance and dieting

But families not to blame

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7
Q

Precipitating factors of ED

A

Biological:
* Puberty
* Physical illness/trauma
* Dieting and weight loss

Psychological
* Low mood/anxiety
* Sense of lack of control in other areas of life

Social/environmental
* Interpersonal problems - loneliness
* Transitions - taking on new role and new expectations eg moving to uni, new job
* Grief/loss
* Social media/diet culture

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8
Q

Perpetuating factors of eating disorders - biological

A
  • Effects of starvation - energy, euphoria
  • Reduced sex drive - can be positive in terms of avoidance eg previous sexual abuse etc
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9
Q

Psychological perpetuating factors for ED

A
  • Feelings of control, protection and safety
  • Sense of identity
  • Numbing emotions
  • Sense achievement
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10
Q

Perpetuating factors ED social/environmental

A
  • Reinforcement from others - positive comments
  • Eliciting care from others
  • Ability to avoid transitions, events and responsibilities
  • Social media/the media/diet culture
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11
Q

What happened with Minnesota study?

A
  • Semi starved healthy men
  • Initially preoccupied with food
  • Then became agitated and found it difficult to rest
  • More tired and withdrawn as weight dropped
  • Very focused on their bodies - were not previously
  • Some became worried about weight gain
  • Many binged once able to eat, struggled to put weight on

Something with starvation changes brain and the way it functions - rigid

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12
Q

Psychological effects of starvation

A
  • Brain structure and self regulatory system (in forebrain) changes
  • Anxiety and intense negative emotions increase (lose neurones)
  • Both negative and positive emotions are numbed
  • Coping ability reduces
  • Thinking –> rigid, habits and routines become more rigid
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13
Q

Functioning of self regulation system during starvation

A

Decreased effect:
* Social situations thoughts
* Emotional regulation
* Decision making
* Flexibility
* Abilty to plan

Increased effect:
* Compulsive behaviours
* Avoidance
* Anxiety
* Sensitivity

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14
Q

People without ED vs people with them reaction to hunger

A

Without ED:
* Hunger –> irritable –> seek food
* Satiety –> sense of pleasure and reward (enjoyable)

With ED:
* Hunger –> sense of calm
* Eating/satiety –> anxiety and guilt

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15
Q

Assessment of ED - 3 aspects

A
  • Psychiatric assessment
  • Medical
  • Risk - psych and physical
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16
Q

Eating disorder history

A
  • History of ED
  • Current pattern eating
  • Mechanisms of weight control
  • Attitudes to weight and shape
  • Current mood symptoms
  • Current anxiety symptoms
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17
Q

Aspects of psych assessment of ED

A
  • What’s happening?
  • When did it begin?
  • Symptoms?
  • Why unwell?
  • Feelings about illness?
  • What help do they want/need?
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18
Q

History of eating disorder - assessment

A
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19
Q

Current pattern of eating history - ED assessment

A
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20
Q

Mechanisms of weight control history - ED

A

Where do they get pills from? - part of Risk

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21
Q

Attitudes to weight and shape - ED assessment

A
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22
Q

Physical symptoms to check for in ED assessment

A
  • Refeeding syndrome can cause oedema
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23
Q

Driving rules and BMI

A
  • Advise not to drive if BMI under 15
24
Q

Risk assessment - ED assessment

A
  • Current/past deliberate self harm
  • Current and past suicidal thoughts inc plans and intent
  • Risk to others inc children
  • Risk from others
  • Other risks - eg hypoglycaemia and falls etc
  • Driving -
25
Physical examination - BMI and traffic light system grading of physical risk
* Red - BMI under 13 or rapid weight loss of more than 1kg per week in undernourished patient * Amber - BMI 13-14.9 or recent weight loss 0.5-1kg per week in undernourushed patient * Green - BMI more than 15 or recent weight loss of less than 0.5kg per week or fluctuating weight
26
General exam signs of ED
* Cachexia * Signs of dehydration * Lanugo hair * Russells sign * Salivary gland enlargement
27
HR for adults with ED
* Red - less than 40bpm * Amber - 40-50 * Green - more than 50
28
BP physical examination risk of ED
* Red - standing BP under 90, associated with recurrent syncope, postural drop over 20mmHg or increase in HR over 30bpm * Amber - standing BP more than 90, occassional syncope, postural drop 15mmHg or increase in HR up to 30 * Green - normal standing BP, normal CV orthostatic changes, normal rhytym
29
Temperature physicla signs ED risk
* Red - under 35.5 or under 35 axillary * Amber - under 36 * Green - above 36
30
Hydration status physical examination ED risk
* Red - fluid refusal, severe dehydration 10% * Amber - severe fluid restriction, moderate dehydration (5-10%) * Green - minimal fluid restriction, no more than mild dehydration (<5%)
31
Testing muscle power SUSS test
* Red - unable to sit up from lying flat or to get up from squat at all or only by using upper limbs for help * Amber - unable to sit up or standd from squat without noticable difficulty * Green - able to sit from lying flat and stand from squat with no difficulty
32
Inv for ED
Bedside: * Full obs - as above * ECG * Blood glucose Bloods: * FBC * U&E * LFT * Bone profile * TFT * Mg
33
What happens to thyroid in biological adaptation to low weight?
* Adjusts free T4 to reduce its metabolic requirments - sick euthyroid syndrome * = reduced resting metbolic rate, reduced temp and bradycardia * Do not treat with thyroxine - make worse
34
Anorexia nervosa effects on body - medical complications
35
Medical complications of bulimia
| Often advise to use mouthwash after vomitting to reduce risk of acid dam
36
Managing medical emergencies in ED
* Use traffic light system to assess risk * If need help - ask * Use MEED guidelines - medical emergencies in eating disorders
37
Cause of refeeding syndrome (starvation and refeeding) - biochemical
38
Biochemical changes you get with refeeding syndrome
* Hypophosphataemia * Hypomagnesaemia * Hypokalaemia * Vitamin deficiency * Fluid retention --> oedema
39
Refeeding syndrome consequences
* Sodium retention and extracellular fluid expansion + thiamine deficiency --> congestive cardiac failure * Cardiac arrythmias from deficiencies of K+, phosphate and Mg * Neuro - delirium, neuropathy, seizures * Respiratory failure - poor ventilation function * Rhabdomyolysis - low phosphate causes muscle weakness and myalgia * Thrombocytopenia and impaired blood clotting - low phosphate
40
Management of refeeding syndrome
* Specialist management - with dietician * Close blood monitoring - daily * Oral supplements and IVs may be needed
41
Management of anorexia nervosa - general points
* Psychoeducation about disorder * Monitor weight, MH, physical health and risk factors * Involve family/carers * Aim to keep healthy weight/BMI * Offer dietary advice as part of MDT - encourage multivitamins, meal planning and regular eating. Not more than 4hrs without eating etc
42
Psychotherapy for adults with anorexia
* Individual CBT-ED * Individual maudsley anorexia nervosa treatment for adults - MANTRA * Individual speciliast supportive clinical management - SSCM * Individual docal psychodynamic therapy (FPT)
43
Psychotherapy for children with anorexia
* Anorexia family focused therapy (FT-AN) - first line * Individual CBT-ED * Individual adolescent focused psychotherapy for anorexia nervosa (AFP-AN) - 2nd line
44
Management bulimia nervosa - adults
* Guided self help in milder cases * Otherwise - 20 sessions individual CBT-ED
45
Treatment for bulimia nervosa - children and young people
* Bulimia nervosa focused family therapy - FT-BN * Individual CBT-ED (second line)
46
Treatment for binge eating disorder
* Stabilise eating and stop bingeing - not aimed at losing weight * Guided self help programme - 1st line * If ineffective after 4 weeks - group CBT-ED * If not available, consider individual CBT | Same in children
47
Treatment of OSFED
* Treatment for eating disorder it most closely resembles
48
Aim of therapy for ED
* Shift motivational position - hard work, so need motivation * Restore healthy weight * Help cope with life and express selves without resort to dietary restriction * Get lifes back on track
49
Why is anorexia so difficult to treat?
* Egosyntonic - feels right * Frightening - to gain weight, buy larger clothes * Ambivalence - want to get better but also don't want to. Sometimes seen as seperate unhelpful part of person
50
When do we admit those with ED?
* Last resort usually * Wants change but not progressing as outpatient * OR immediate danger * OR no adequate treatment locally We can admit someone and detain them under MHA and feed against wishes if really necessary - always better to avoid this if possible
51
Capacity and use of MHA in eating disorders
* If they are at medical risk and refuse - need to be assessed for capacity and treated under appropriate criteria (common law in emergency if lack capacity or MHA) * Impairment is often in their ability to rationally balance consequences of treatment that results in weight gain against their extreme fear of gaining weight * If unsure - speak with MH liason team, ED consultant specialist
52
Outcomes of bulimia
50-70% recover completely Rest have relapse and remitting course
53
Outcomes anorexia nervosa
* Around half fully reover * 33% improve * 20% remain chronically unwell * Best chance of recovery is within 3 years of onset and when younger, first episode * Outcome poor if disease lasts 10yrs or longer * Average duration is 8yrs
54
Mortality rate AN
* Higher than any other MH disorder * 4 in 5 related to physical causes * 1 in 5 related to suicide
55
What predicts good outcome from ED?
* Motivation to change * Short duration of illness * Lower level of severity * Onset during adolescence - brain neuroplasticity * Good family function/support * Lack of co-morbid conditions
56
Charities help for ED
* First steps * Beat eating disorders
57