ECMO Flashcards

1
Q

VV EMCO and indications

A

Veno-venous ECMO

  • drained from IVC returned to SVC
  • does not bypass heart
  • hemodynamics not completely dependent on ECMO
  • oxygenating and ventilating
  • “Pulmonary ECMO”- PNA, ARDS, massive aspiration, rib fractures, chest trauma, primary graft dysfunction after lung transplant, COVID
  • PE?
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2
Q

VA EMCO and indications

A
  • veno arterial ECMO
  • Venous drain and arterial return
  • completely bypassing the heart and lungs, replaces cardiac function
  • hemodynamics dependent on ECMO flow
  • USES: cardiac arrest (known to cardiology?), EPS/Cath lab, end stage cardiomyopathy/ possible VAD/transplant candidate (acute decompensation, failure to wean from bypass, ?massive PE?
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3
Q

Contraindications to ECMO

A
  • severe peripheral vascular disease, aortic dissection, severe aortic valve insufficiency
  • malignancy, severe brain injury
  • advanced age >75
  • cardiac arrest of prolonged duration
  • mechanical ventilation >7 days
  • pulmonary HTN (VV EMCO only)
  • Obesity >125kg, BMI>40, very difficult to cannulate and keep clean
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4
Q

Sweep

A

minute ventilation of ECMO
- Total amount of gas pushed through oxygenator in one minute L/min
sleep flow is increased or decreased based on CO2 (jusy like we increase or decrease the RR on the vent based on CO2)
-

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5
Q

Peripheral VA ECMO

A
  • the classic VA ECMO cannulation is RIGHT femoral VENOUS drain and LEFT femoral arterial return (better to have the cannulas in different spots
  • Right venous drain cannula is very long 30-40inches the tip sitting in the IVC maybe in the right atrium
  • sometimes may need a second venous drain because this is full hemodynamic support and need high flow — right IJ is “Y”ed into the venous cannula SVC completely decompressing the heart
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6
Q

North/ south syndrome

A
  • seen in VA ECMO
  • As the heart starts to recover, the LV will start to eject and that blood will push out again the ECMO retrograde flow
  • the area where the antegrade and the retrograde flow meets is called the “mixing cloud”
  • The strong the heart gets the farther it will perfuse, if the lungs are normal, the LV output will adequately oxygenate/ventilate areas that it is perfusing
  • if the lungs are compromised, the areas perfused by the LV may become hypoxic— north south syndrome
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7
Q

VA central ECMO

A

usually patients who cannot come off bypass

  • all antagrade so no risk of north south syndrome
  • right atria venous drain
  • ascending aorta arterial return
  • skin may be closed but sternum cannot be wired unless the cannula are tunneled– risk for unstable sternum
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8
Q

VA ECMO flow goals for full support

A
  • ECMO flow= cardiac output
  • “ECMO index”= 1.8-2.4l/min or better
  • Take patients BSA and x2 for CO/ flow
  • Increase RPMs util goal flow is achieved
  • Indications for full support
    • little to no native cardiac functio
    • myocardial stunning– post cardiotomy/ STEMI
    • goal is to rest the heart
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9
Q

ECMO flow goals– low flow less than 3L/min

A
  • allows for LV ejection
  • decreases risk of clot formation
  • unloads LV and reduces or prevents pulmonary congestion/edema
  • lower flows may allow us to diuresis the patient and improve pulmonary congestion
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10
Q

Dx of north south syndrome

A

draw a right radial and post ECMO oxygenator ABG and compare- a different in PaO2 indicates N/S syndrome especially if right radial ABG is hypoxic

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11
Q

North. South syndrome treatment

A
  • increase ECMO flow?? (careful.. it may worsen the problem by increasing LV distention and pulmonary congestion)
  • Maximize vent settings
  • impella to unload LV (will decrease pulm congestion)
  • LV vent spliced into ECMO inflow lone
  • Consider decannulation or switching to VAV or VV ECMO
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12
Q

VV ECMO peripheral cannulation

A
  • Right femoral vein tip of IVC drainage
  • Right IJ SVC return
  • maintains pulsality
  • lower flos and PaO2 is not as high with VV (more mixing of native blood)
  • When oxygenated blood from the outflow cannula mixes with the blood going into the inflow cannula can cause recirculation circle
  • inflow and outflow lines same color, increased ECMO SvO2 with decreased patient oxygenation
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13
Q

Goals of VV ECMO

A
  • to support the patients oxygenation/ventilation needs and allow the lungs to heal/recover or to be transplanted
  • VV ECMO does not replace the lungs, it adds to them -
  • if the FI02 is too low, the patients sats drop, if the ECMO flow is too low, the sats drop.. higher flows to better oxygenate
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14
Q

VV ECMO and Cardiac output

A
  • VV ECMO flow is a portion of the patients total CO
  • VV ECMO flow is fixed, CO is variable
  • think of it as a percentage (2/3 of CO is going through the ECMO circuit, 1/3 is not)
  • As CO increases that percentage of non ECMO blood gets larger, “awake” patients generally have high heart rate and BP— and thus will not oxygenate as well
  • sedation/ analgesia decrease WOB but also decrease CO
  • Beta blockers may be utilized- continuous infusions may work better than IVP– esmolol gtt
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15
Q

What can compromise ECMO flow

A
  • inadequate proload- bleeding 3rd space shift
  • intra-thoracic/ intra-abdominal HTN ( Tamponade/domain issues, coughing, breathing)
  • large patient, small cannula
  • HTN (systemic and pulmonary) Afterload sensitive
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16
Q

How to maximize flow

A
  • increase RPMs
  • volume resuscitate
  • sedation/paralyze (decrease intra-thoracic pressure)
  • Open chest- relieves domain/ tamponade physiology
  • add another inflow cannula
  • lower goals..
17
Q

“Suckdown events”

A
  • acute drops in flow: flow below 1-2L/min
  • may result in hemolysis or damage to the vessel wall or pump malfunction
  • turn down RPMs until flow stabilizes
  • fluid bolus -250ml
  • sedation/paralytic if awake/agitated
  • increase RPMs to original rate when possible
  • if events are frequent, discuss alternatives with team
18
Q

NIRS monitoring

A
  • NOT a POX
  • it is reflective of venous and arterial oxygen
  • rSO2 levels calculated by the NIRS will not be as high as POX reading
  • watch for trend not numbers
  • changes to report (NIRS valve below 40, decrease in NIRS valve more than 25% from baseline, >25% difference in NIRS from one extremity to the other
19
Q

Vent settings with VA EMCO- little to no cardiac function

A
  • ECMO is completely supporting heart/lungs
  • PaC02 on the ABG will be 400-500 if on 100% Fio2
  • “The vent isnt needed”
  • Turn vent settings to minimal
  • goal is to prevent atelectasis
20
Q

Vent with VA ECMO with return of cardiac ejection

A
  • watch for north south syndrome

- adjust ventilator based on radial ABG

21
Q

Vent with VV ECMO

A
  • “rest” settings on ventilator initially

- increase ventilator support with weaning or if ECMO is not meeting patients needs