effects of aerobic and anaerobic training - week 9 Flashcards

(31 cards)

1
Q

what is training overload ?

A

when a physiological system is exercised at a level beyond its normally accustomed

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2
Q

what is specificity ? (4)

A

training being specific to …

  • muscle fibres recruited
  • energy system involved
  • velocity of contraction
  • type of contraction
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3
Q

what is reversibility ?

A

gains are lost when training ceases

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4
Q

what’s is involved in training to increase VO2 max ? (4)

A

large muscle groups, dynamic activity

20-60 mins
≥3 times a week
≥50% VO2 max

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5
Q

how much does VO2 max change with endurance training ? (2)

A

15-20% increase

if high VO2 already, increase is smaller

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6
Q

how does genetics impact VO2 max ?

A

heritability determines 50% in sedentary adults

also key role in determining training response

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7
Q

why does VO2 max change with training ?

A

due to changes in Cardiac output and stroke volume
- changes O2 consumption and VO2 max

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8
Q

how does training increase stroke volume ? (4)

A

increased preload
increased plasma volume
increased venous return

increased O2 uptake, improves systems, improves functions of heart

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9
Q

what is eccentric hypertrophy ? (2)

A

chamber size and wall thickness increase of heart

helps deal w increased volumes of heart so can pump more blood around - stronger heart

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10
Q

how does endurance training increase stroke volume ? (2)

A

increased blood, reduced resistance

increased contractility
- greater force produced

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11
Q

what factors influence stroke volume ? (3)

A

end diastolic volume (preload)

contractility

totally peripheral resistance (after load)

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12
Q

what factors influence end diastolic volume ? (3)

A

plasma volume

filling time + venous return

ventricular volume

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13
Q

what’s the equation for CO?

A

stroke volume x HR

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14
Q

what happens to max CO with training ?

A

increases !

mostly linear

increases efficiency of using O2

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15
Q

at sub-man workload HR is __ following trainmen due to increase in __ so __ can be achieved with fewer bpm

A

lower

stroke volume

CO

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16
Q

what happens due to increased muscle blood flow due to increased arteriovenous O2 difference from training?

A

decreased SNS vasoconstriction

increased diameter and compliance of arteries

17
Q

what happens to improved ability of muscle fibres to utilise O2 due to increased arteriovenous O2 difference from training?

A

increased capillary density - increased diffusion due slower blood flow through muscle

increased mitochondria

18
Q

transit time of RBCs _ due to bigger __ so they take longer to pass through

A

increase

capillary network

19
Q

during sub-max exercise blood flow in trained muscles is lower _ the A-V difference is greater

20
Q

during maximal exercise blood flow in trained muscles is higher _ the A-V difference is greater

21
Q

what are the consequences of endurance training shifting fibre types ? (2)

A

reduction of fast fibres and increase in slow

magnitude of fibre type dependent on training, duration . genetics

22
Q

what are the consequences of endurance training increasing capillaries surrounding muscle fibres ?

A

enhanced diffusion of oxygen

improved removal of waste

23
Q

how does endurance training increase mitochondrial volume + turnover in muscles?

A

increased volume subsarcolemmal + intermyofibrillar mitochondria

greater capacity for oxidative phosphorylation

increased mitochondrial volume
- reduced systolic ADP so less lactate + H+ formation

24
Q

how does endurance training change fuel utilisation ? (3)

A

increased plasma FFA transport and oxidation to muscle

transport of FFA from cytoplasm to mitochondria

mitochondria oxidation of FFA
- increased acetyl CoA formed
- slowed glycolysis

25
how does endurance training improve antioxidant capacity of muscle ? (3)
(when muscle contract they produce free radicals) increases in endogenous antioxidant enzymes - improved ability to remove radicals - protects against oxidative damage + muscle fatigue
26
how does endurance training improve acid-base balance during exercise ? (3)
(lactate production creates H+ ions which can increase fatigue) increased mitochondria - less carbs used- less pyruvate increased NADH shuttles - less NADh for lactate production change in LDH isoform - less lactate formation
27
how does training promote protein synthesis in fibres? (3)
'stress' activates gene transcription contraction activates primary and secondary messengers results in expression of genes and synthesis of new proteins
28
what are some of the primary signals for post exercise adaptations ?
mechanical stretch calcium AMP/ATP free radicals these drive proteins and enzymes that can shift/drive the adaptation
29
what is the secondary messenger AMP kinase ?
promotes glucose uptake and linked to gene expression by activation of transcriptional activating factors
30
what is the secondary messenger PGC-1a?
regulator of mitochondrial biogenesis, promotes angiogenesis and synthesis of antioxidant enzymes - activated by AMPK
31
how does detraining effect body and vo2 max? (3)
rapid decrease in VO2 max decrease in SV max decrease in maximal a-v O2 difference - less mitochondria - less oxidative capacity of muscle - type 2a fibres and increase type 2 x fibres