ENT Flashcards

(39 cards)

1
Q

Acute epiglossitis

Px
Ix
Mx

A

Cause: Hib infection

Presentation: Adults (severe sore throat and dysphagia). Children (irritable, fever, leaning forward, drooling, muffled voice/cry, heaving breathing)

Investigation: Clinical diagnosis. Do not do throat exam.

Management: Leaning patient forwards –> Get ENT surgeon and anaesthetist –> Intubation + IV dex and ceftriaxone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Tonsillitis

A

Infection agent: >50% due to streptococcus pyogeens

Diagnosis: Clinical. No throat swab.

Scoring: Centor criteria (ant lymphadenopathy, no cough, tonsillar exudate, history of fever).

Management:

  • 1st line Antibiotics (7-10 days phenoxymethylpenicillin. If pen allergic, give erythromycin). Indications: Centor criteria met OR systemic upset OR immunocompromised OR history of rheumatic fever.
  • 2nd line Tonsillectomy. Indications: Recurrent tonsillitis (>7 eps a year), sore throat, symptoms for >1 year. Complications of haemorrhage (Primary due to inadequate haemostasis. Secondary due to infection) and pain
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

ENT abscesses

Types
Severity
Cx
Px and mx of each

A

Two type: Peritonsillar abscess and Parapharyngeal abscess

Severity: ENT emergencies!!

Cause: Complications of tonsillitis

Peritonsillar abscess

  • Px: Sore throat, uvular deviation, peritonsillar bulge, dysphagia
  • Mx: Abx and aspiration

Parapharyngeal abscess

  • Px: Diffuse swelling of the neck
  • Mx: Us to location and drainage under GA
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Laryngitis

Cx
Progression
Px
Ix
Mx
Ix to admit
A

Cause: Viral mostly
Can be secondary to bacterial infection (staph or strep) GORD (“reflux laryngitis”)
Autoimmune e.g. RA

Progression: Self-limiting

Presentation:

  • Hoarseness
  • Fever
  • Pain (hypopharyngeal, dysphagia, on phonation)

Ix: No throat swab. Clinical Dx

Management:
1st line- Conservative
2nd line- 1 week of phenoxymethylpenicillin

Indications to admit:

  • Stridor
  • Breathing difficulties
  • Life threatening emergency e.g. epiglossitis, kawasaki
  • Clinical dehydration
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Acute sinusitis

Cx
Px
Mx

A

Causes:

  • Obstruction e.g. septal deviation, polyps
  • Swimming /diving
  • Recent local infection
  • Smoking

Causative agents

  • Bacteria (Hib, strep pneomoniae)
  • Viral (rhinoviruses)

Presentation

  • Facial pain (frontal, worse on bending forwards)
  • Nasal discharge (purulent and thick)
  • Nasal obstruction

Management:
1st line: Analgesia. If symptoms >10 days, take inhaled corticosteroids
2nd line: Oral phenoxymethylpenicillin. If severe symptoms, oral co-amoxiclav

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Acute rhinosinusitis

A

What? i.e. Common cold

Presentation: Nasal discharge, nasal obstruction, facial pain/ pressure, reduced smell, endoscopic/CT signs

Duration: <12 weeks

Progression: 80% self-limiting

Management:
1st line: If <5 days, nothing
2nd line: If >5 days, corticosteroids (mometasone or fluticasone)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Chronic rhinosinusitis

A

What? RS for >12 weeks. Most commonly inflammation at the middle meatus.

Presentation: Water, anterior rhinorrhoea, purulent post-nasal drip, sneezing, snoring, nasal obstruction, mouth breathing, headaches

Investigations: Anterior rhinoscopy or nasal endoscopy. (Polyps = Pale, mobile, insensitive to touch)

Classification? With or without polyps

Management:
WITH POLYPS
1st line- Steroids (Topical beclometasone for 2 weeks then fluticasone nasap spray for 3 months)
2nd line - Add Abx (Doxy long term)
3rd line - Surgery for ESS (endoscopic sinus surgery) Risk of CSF leakage or CN II damage

WITHOUT POLYPS
1st line- Steroids or saline nasal irrigation
2nd line - If no improvement after 4 weeks, culture and start >12 weeks Abx
3rd line- CT scan and consider surgery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Septal perforation

A

Cause: Septal surgery, infections, drugs, trauma, SCC

Risk: Progressive enlargement

Management:
1st line- Symptomatic treatment (Saline nasal irrigation, petroleum jelly around edges)
2nd line- Septal prosthesis (“button surgery”)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Foreign body in nasal cavity

A

Presentation: Organic material –> Purulent unilateral dischage. Inorganic material –> Can remain unnoticed for years

Management:
1st line- Blow nose / parental kiss / removal with forceps. Batteries require URGENT removal
2nd line- ENT referral

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Nasal fractures

A

Cause: Trauma **

Presentation: Facial swelling, nasal deformity, epistaxis , black eyes.

Investigation: Nasal examaintion. Exclude head an c-spine injury. X-ray NOT required. Check for nasal haematoma (risk of complete nasal obstruction. Urgent treatment required if present)

Management:
1st line- Treat epistaxis, analgesia, ice –> Reassess in 5 days. If MUA required, perform 10-14 days after injury
2nd line- Urgent drainage and incision

manipulation under anaesthetic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

CSF leakage

Cx
Ix
Mx

A

Cause: Ethmoid bone fractures, neoplasm

Investigation
1st line: Lab testing for nasal CSF (positive for glucose)
Gold standard: Lumbar puncture (contains high B2 tau transferrin)

Management (if traumatic)
1st line- 7-10days bed with head elevation + Avoid sneezing/coughing/blowing nose + Abx + pneumococcal vaccine +/- lumbar drain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Allergic rhinitis

A

Presentation: Bilateral red itchy eyes, nasal discharge, pruritis, atopy, sneezing

Signs of auroscope: Mucosa pale, turbinates swollen, nasal polyps present

Management:
1st line- Allergen or irritant avoidance. Saline nasal irrigation
2nd line (mild-mod intermittent or mild persistent symptoms)- Loraditine OD
3rd line (mod-severe intermittent or 2nd line ineffective) - intranasal corticosteroids e.g. mometasone, fluticasone 
4th line (cover big life events) - 5-10 days course of prednisolone 

Prevention: OD SLIT (sublingual immunotherapy) or allergy vaccine Inj

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Epistaxis

A

Cause:

  • Direct trauma
  • Injury
  • Illicit drug use
  • Bleeding disorders e.g. (thrombocytopaenia, leukaemia, haemophilia, ITP, splenomegaly)
  • Cold and dry weather

Emergency management

  1. Gown up and ABCDE
  2. Pinch lower part of nose for 20 mins + lean forwards
  3. Ice of dorsum of nose
  4. Patient to blow out clots. or remove clots gently with forceps
  5. Vasoconstriction using adrenaline soaked cotton wool for 2 mins OR lidocaine
  6. Silver nitrate cautery (not of actively bleeding areas. If you cannot see bleeding origin, refer)
  7. Anterior nasal pack
  8. Postnasal pack

Management of serious posterior bleed
Options:
- EUA: If bleeding source found, the diathermy or repacking
- Arterial ligation via endoscopy e.g. of sphenopalatine artery
- Embolisation of internal maxillary or facial artery (e.g. lifesaving in stroke risk patients)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Periorbital / preseptal cellulitis

A

Cause: Infection spread from nearby structures (cellulitis, RTI, sinusitis,

Epidemiology: Children >. Winter months >

Pathogen: Staph aureus, streptocci

Presentation: Eye swelling/pain/redness + fever. *Absent orbital signs * (e.g. pain on movement, RAPD, chemosis, restricted movements, visual disturbance)

DDx: Orbital cellulitis, allergic reaction

Investigation: Bloods (raised ESR and CRP). Culture any discharge. If needing to exclude orbital cellulitis, contrast CT head required.

Management: Refer to secondary care for assessment. Microguide abx = 5 days of flucloxacillin (if pen allergic, doxy or clarithromycin)

Risk: Orbital cellulitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Labyrinthitis

A

What? Viral infection of the labyrinth (cochlear and vestibular end organs)

Duration: Acute onset for 1-2 days, then improves over week

Who? 40-70y/o

Presentation:

  • Vertigo (exacerbated by movement)
  • N/V
  • Hearing loss (can be bilateral or unilateral)
  • Tinnitus

Signs:

  • SNHL
  • Abnormal head impulse test
  • Unidirectional horizontal nystagmus

DDx:
- Hearing loss excludes vestibular neuritis

Investigation: History and exam. Check BM to exclude hypoglycaemia

Management: Vestibular suppressants e.g. Buccastem oral

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Pinna Haematoma

A

What? Bleeding into the sub-perichondrial plane typically due to blunt trauma

Management: Surgery (incision + primary closure with packing ) AND antibiotic cover with ciprofloxacin

Complications:

  • Cauliflower ear due to ischaemic necrosis and subsequent fibrosis
  • Infection and loss of cartilage
17
Q

Acute otitis media

A

What? Inflammation of the middle ear.

Organisms: Most commonly Streptococcus Pneumonia, haemophilis, moraxella

Presentation:
Symptoms: Rapid onset of pain + Fever. +/- anorexia, vomiting and irritability
Progression: Most cases resolve in 24hrs. If there is sudden relief of pain and purulent discharge, think TM perforation

Investigation:
1st line - Otoscopy (TM bulging, erythma, dilatation of the circumferential vessels)

Management:
1st line- Conservative
2nd line- Antiobiotics. Indications: Systemically unwell, immunocompromised, <3 months old, <2 years old with bilateral OM, perforation + discharge, symptoms for 4 days. What? Amoxicillin for 5 days.

Complications:
Present with continuous discharge
1. Perforation and discharge (Mx: Top Abx)
2. Recurrent AOM (Mx: Expectant treatment, long term Abx, grommet)
3. Acute mastoiditis (Mx: Urgent ENT referral for IV Abx and Surgical drainage)
4. Intracranial sepsis or meningitis (Mx: Urgent referral for IV Abx
5. Facial falsy (Mx: Urgent referral for grommet insertion)

18
Q

Chronic otitis media

A

What? Perforation of the TM likely due to recurrent or prolonged infections

Presentation: Otalgia, hearing loss, fullness, otorrhoea

Classification

  • Squamous or mucosal
  • Active or inactive.

Management:
1st line: Abx (systemic or topical), aural cleaning, water precautions
2nd line: Surgery

Complications:
- Cholesteatoma due to retraction of pars flaccida or tensa leading to epithelium build up

19
Q

Otitis Externa

A

What? Inflammation to external ear and canal

Cause: Bacterial infection e.g. Pseudomonas aeruginosa, staph aureus

Precipitating factors:

  • Excessive moisture
  • Trauma
  • Dermatitis (contact or seborrheic )

R/F

  • DM
  • Radiotherapy to head and neck

Classification:
Timing: <3 months is acute, >3 months is chronic
Severity: Diffuse, localised and malignant

Presentation:

  • Hearing loss
  • ITCH
  • Ear pain
  • Discharge

Signs: Red, swollen, eczematous ear canal / external ear

Diagnosis: By examination

Management;:
1st line: Risk factor management. Analgesia. Topical ABx
2nd line: Referral to ENT if… excessive cellulitis / extreme pain / excessive swelling / obstruction to topical Abx. URGENT referral for malignant OE as IV abx required.

Complication:

  • Abscess
  • Malignant OE
  • TM inflammation i.e. OM
20
Q

Vestibular neuronitis

A

What? Acute, spontaneous, prolonged, isolated vertigo

Cause? Viral infection leads to inflammation of the vestibular nerve

Presentation
Progression: Acute spontaneous onset. Severe Sx for 1-2 days followed by a few weeks of recovery and improvement

Symptoms:

  • Vertigo
  • N/V
  • NO hearing loss
  • NO tinnitus
  • NO focal neurology

Signs: Fine horizontal nystagmus

DDx:

  • Labyrinthitis
  • BPPV
  • Meniere’s disease
  • Central causes: Migraine, stroke, MS, cerebellar tumour

Investigation: Clinical diagnosis (HiNTs tests excludes posterior circulation stroke)

Management:
Hospital admission indications: Cannot tolerate oral intake. No improvement with 1st line medication after 1 week. Focal neurology. 6 weeks of symptoms
Acute- 1st line Symptomatic relief with antiemetics (if N/V is severe then IM or Buccal prochlorperazine or cyclizine. If moderate N/V then oral prochlorperazine, cinnarazine, cyclizine).
Chronic: Vestibular rehab exercises

21
Q

Meniere’s disease

A

What? Disease of the inner ear from an unknown case, associated with endolymph hydrops which would increase pressure

Risk factors: Autoimmunity, female gender, FH, metabolic abnormalities, vascular factor

Presentation:
Symptoms- Vertigo, fluctuating hearing loss and tinnitus in episodes lasting minutes-hrs.
Signs- Nystagmus present. Romberg’s positive

Progression: Most symptoms resolve after 5-10 years but patients left with some degree of HL.

Investigation: Clinical examination/history + audiometry to confirm SNHL

Mangement
In primary care: Refer to ENT for formal audiometry assessment
In secondary care: Audiometry assessment. Prescribe medications for acute attack of N/V and vertigo (Prochlorperazine or anti-histamine). Patient to inform DVLA and take care operating machinery
Hospital admission: Required for severe symptoms to give IV labyrinthine sedative + fluids

Prevention: Beta-histamine trial or vestibular rehab exercises

22
Q
Sensorineural hearing loss
define
causes
px
ddx
ix
mx
prognosis
A

What? Loss of hearing due to the defect in the oval window of the cochlea or the cochlear nerve

Cause:
Vascular (Cochlear vascular disease)
Inf (Meningitis, Meniere’s, measles, mumps, herpes, TB, syphilis)
Trauma
Autoimmune (MS)
Idiopathic (Presbycusis)-
Drugs (ABx e.g. streptomycin, gentamicin, vancomycin. -“Quines”

Presentation: Onset can vary depending on cause. Usually unilateral hearing loss.

DDx: CHL, OME

Investigations:
In primary care: Detailed history, esp drugs. Rinnes shows AC>BC in affected ear. Weber’s is louder in non-affected ear. Exam EAMeatus and do otoscopy
In secondary care: Formal audiology +/- audiological BS response

Management:
1st line: If inflammatory cause then 4 days oral prednisolone and taper for 8 days
2nd line: If inflammatory cause then intratympanic dexamethasone

Prognosis: Good with 30-65% getting complete/partial resolution

Presbycusis is usually a sensorineural hearing disorder. It is most commonly caused by gradual changes in the inner ear. The cumulative effects of repeated exposure to daily traffic sounds or construction work, noisy offices, equip- ment that produces noise, and loud music can cause sensorineural hearing loss

23
Q

BPPV

A

What? Benign paroxysmal positional vertigo

Who? Women> in 50-70s

Precipitating factors: Head injury, ear surgery, prolonge recumbent postition, past inner ear pathology (meniere’s, labyrinthitis, vestibular neuronitis)

Presentation: Repeated episodes of vertigo. Likely to recur and have spontaneous resolution

Diagnosis: Positive Dix-Hallpike manoeuvre

Management:
1st line in primary care: Watchful waiting or Epley manoeuvre. Follow-up in 4 weeks
2nd line: Refer balance specialist if persistent symptoms, S/S atypical, 3+ episodes, Epley manoeuvre cannot be performed or was unsuccessful
Admission to hospital: If N/V prevent oral intake.

24
Q

Acoustic Schwannoma

A

Aka acoustic neuroma

What? Indolent, benign subarachnoid tumour. Acts are SoL leading to pressure issues. Forms 90% of cerebellopontine tumours

Presentation:

  • Ipsilateral > (if bilateral, think neurofibromatosis type 2)
  • Symptoms dependent on CN involved (CN V = Absent corneal reflex. CN VII= Facial palsy. CN VIII = Vertigo, SNHL and tinnitus.

Investigation:
1st line: MRI of cerebellopontine angle + Audiometry

Management:
1st line: Urgent ENT referral. Watchful waiting initially to monitor growth.

25
Noise induced hearing loss
Cause: Long term occupation occupational exposure (8hrs+ of >85dB) or an one-off intense sound potentially fracturing ossicles or perforating the TM Presentation: Sudden or progressive bilateral symmetrical SNHL +/- tinnitus Investigation: Audiometry Treatment: Hearing aids. Prevention: Hearing protection
26
Bell's Palsy
What? Palsy of the facial nerve Cause of facial palsy - Intracranial (Strokes, MS, meningitis, BS tumour, cerebellopontine tumour e.g. acoustic schwannoma) - Intertemporal (Otitis media, Ramsay hunt syndrome) - Infratemporal (Parotid tumour, trauma) - Other (DM, Lyme's disease, *Ball's palsy*, Sarcoid) Presentation: Symptoms: Hyperacusis, altered taste, post-auricular pain, dry eyes Signs: Forehead affected, laugh line lost, lip paralysis, unilateral drooped lower lid ``` Investigations: To exclude other causes: - Glucose (DM?) - Lymes serology? - MRI (SoL? MS? Stroke?) - Parotid examination - Otoscopy (Cholesteatoma? Ramsay hunt syndrome?) - ESR (Temporal arteritis) ``` Management 1st line: 10 days prednisolone within 72 hours + EYE CARE (lubricants e.g. viscotears or lacrilube) Referral to ENT indication: Bilateral bell's palsy, recurrent bell's palsy, any doubt in diagnosis Prognosis If treated = 80% recover by 3 months In untreated = 15% permanent mod/severe weakness
27
Head and neck SCC's
Two types: Oesophageal carcinoma and paranasal sinus cancer OESOPHAGEAL CARCINOMA Associations: Smoking, alcohol, Barret's oesophagus, achalasia Px: Dysphagia, hoarseness, weight loss, cough Mx:Surgery +/- radiotherapy / chemotherapy Prognosis: Poor 5 yr survival PARANASAL SINUS CANCER Pathology: 50% SCC, 10% lymphoma Px: Blood-stained nasal discharge, nasal obstruction, cheek swelling. Suspect when chronic sinusitis presents later in life Ix: MRI or CT +/- endoscopy with biopsy Mx: Refer to ENT for radiotherapy +/- radical surgery
28
Nasopharnygeal cancer
Px: - *Cervical lymphadenopathy - CHL (due to Eustachian tube blockage) - Nasal symptoms e.g. obstruction, bleeding, discharge - CN palsy (excluding CN I, VII, VIII) Pathology: SCC Ix: 1st line Endoscopy with biopsy Staging MRI DDx: - Paranasal sinus cancer - Oesophageal carcinoma Mangement: 1st line Radiotherapy +/- chemotherapy +/- surgery (radial neck dissection)
29
Children with epiglottitis 1st line
Nebulised adrenaline and straight to theatre
30
Children with epistaxis Mx
1. First aid 2. Pinching the end of the nose 3. Naseptin in primary care Cautery with Silver nitrate in outpatient clinic
31
DIAGNOsIs AND MANAGeMeNT OF HeAD AND NeCk CANCeR - Urgent referral indications
*In general, worry is symptoms last more than 3 weeks*  with red or red and white patches of the oral mucosa which persist for more than three weeks at any particular site  ulceration of oral mucosa or oropharynx which persists for more than three weeks  oral swellings which persist for more than three weeks  unexplained tooth mobility not associated with periodontal disease  persistent, particularly unilateral, discomfort in the throat for more than four weeks  pain on swallowing persisting for three weeks that does not resolve with antibiotics  dysphagia which persists for more than three weeks  hoarseness which persists for more than three weeks  stridor (requires same day referral)  unresolved head or neck mass which persists for more than three weeks  unilateral serosanguineous nasal discharge which persists for more than three weeks, particularly with associated symptoms  facial palsy, weakness or severe facial pain or numbness  orbital masses  ear pain without evidence of local ear abnormalities.
32
Key investigation for looking in the upper airway
Fiberoptic endoscopy
33
Acute Otitis Media Complications
Present with continuous discharge 1. Perforation and discharge (Mx: Top Abx) 2. Recurrent AOM (Mx: Expectant treatment, long term Abx, grommet) 3. Acute mastoiditis (Mx: Urgent ENT referral for IV Abx and Surgical drainage) 4. Intracranial sepsis or meningitis (Mx: Urgent referral for IV Abx 5. Facial falsy (Mx: Urgent referral for grommet insertion)
34
Acute Sinusitis Presentation
1. Pain 2. Nasal discharge 3. Hyposmia 4. Nasal blockage
35
Neck lumps
BRANCHIAL CYST– uncommon cysts that typically lie between the upper and middle third of the anterior border of sternocleidomastoid, present soon after an URTI in young adults. THYROID MASSES, THYROGLOASSAL CYSTS AND DERMOID CYST: • Location: typically midline lesions. • Thyroid mass: older adults, more often in women. Move with swallowing. • Thyroglossal cysts move with swallowing and protrusion of the tongue. (remanent of thyroid descension from the tongue base during embryology) • Dermoid cysts: usually submental and tend NOT to move with swallowing or protrusion of the tongue. Cyst is lined with epidermis and may contain hair, teeth or squamous debris. Should be excised. Lymphoma – systemic features (especially weight loss, night sweats and fevers)
36
Otitis media classification
1. Acute Otitis Media (AOM) 2. Chronic Otitis Media (COM) - Mucosal: Active/ Inactive - Squamous: Active/ Inactive 3. Otitis Media with Effusion (OME)
37
exostosis
What? Bone outgrowth Cause: Cold water exposure e.g. surfers Risk: Wax collection Mx: Lift of skin and drill bone away
38
Cholesteatoma
Complication if COM Ix: Otoscopy (Finding: Chronic squamous otitis media) + Formal audiometry (BC>AC) + CT head (Inner ear and mastoid involvement?) Mx: Surgery w/Mastoid exploration Risk: - Facial nerve palsy - Balance - Hearing loss - Meningitis - Brain stem erosion and infection Prognosis 1/5 recur post operation
39
OME
Aka "glue ear" Cause Effusion after the regression of symptoms of acute OM Presentation: Conductive hearing loss. 90% resolves after 1 year (reassure parents!) Ix: 1st line Otoscopy + Formal hearing test (at initial presentation, repeated at 3 months) - Finding: BC>AC and flat tympanogram 2nd line: If recurrent and unilateral in adult, fibreoptic endoscopy Mx: 1st line: W/W for 3 months 2nd line: Grommet insertion is persistent for more than 3 months Immediate ENT referral for: • background of Down’s syndrome or cleft palate • persistent foul-smelling discharge suggestive of cholesteatoma • initial audiometry shows a severe hearing loss Concern? In adults, nasopharyngeal carcinoma blocking the out flow of fluid from Eustachian tube