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micro unit 6 > enteric bacteria 2/3 > Flashcards

enteric bacteria 2/3 Flashcards

(52 cards)

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1
Q

salmonella stain and morphology

A

gram negative and rod

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2
Q

is salmonella motile

A

yes

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3
Q

is salmonella lactose fermenting

A

can be, seldomly

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4
Q

salmonella oxidase?

A

negative

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5
Q

salmonella urease

A

negative

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6
Q

salmonella sulfur

A

positive

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7
Q

what diseases does salmonella cause

A

enterocolitis, enteric fevers and septicemia

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8
Q

salmonella enterocolitis caused by which organisms and how

A

typhimuriam, enteritidis, heidelberg invade the gut wall. there is immune containment and bacteremia is rare

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9
Q

salmonella enteric fever (typhoid) organisms

A

typhi and paratyphi

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10
Q

salmonella enteric fever (typhoid) infectious dose

A

high

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11
Q

salmonella enteric fever (typhoid) pathogenesis

A

invasion of gut wall via M cells leads to transport by macrophages and spreading via lymphatics top major organs. there is macrophage apoptosis, bacteremia, toxemia.

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12
Q

salmonella septicemia who gets it?

A

rare in healthy adults maybe a complication of sickle cell

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13
Q

what is the progression of salmonella septicemia

A

osteomyelitis, pneumonia, meningitis. metastatic abscesses form in previously damaged tissues like aneurysms

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14
Q

how to diagnose salmonella enterocolitis

A

culture from stool. there will be nonbloody diarrhea, fever, dehydration.

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15
Q

how to diagnose salmonella enteric fevers

A

high fever, HA, tender abdomen, anorexia, lethargy and rose spots. culture from stool, blood, bone marrow.

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16
Q

how to diagnose salmonella septicemia

A

focal symptoms at affected organ.

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17
Q

how to prevent salmonella

A

notify authorities about possible sources of outbreak. hand-washing and food prep.

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18
Q

how to treat salmonella enterocolitis

A

rehydrate and test for sensitivity of ABs

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19
Q

how to treat salmonella enteric fever

A

ceftriaxone or ciprofloxacin. there is a vaccine, but rarely used

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20
Q

how to treat salmonella septicemia/ focal diseease

A

drain abscesses. ceftriaxone or ciprofloxacin

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21
Q

Yersinia enterolitica and pseudotuberculosis gram and morphology

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A

negative oval rods

22
Q

Yersinia enterolitica and pseudotuberculosis lactose

23
Q

Yersinia enterolitica and pseudotuberculosis urease

24
Q

Yersinia enterolitica and pseudotuberculosis motility

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A

motile at 25, nonmotile at 37

25
Yersinia enterolitica disease?
enterocolitis. false appendicitis when spreads to lymph nodes. can predispose to reactive arthritis
26
Yersinia enterolitica infectious dose
high
27
Yersinia enterolitica virulence factors
on chromosome and plasmid
28
who most common gets Yersinia enterolitica
young children
29
Yersinia enterolitica bacteremia
rare but deadly
30
pseudotuberculosis who gets this?
rare infection. immunocompromised or liver disease.
31
what does the pediatric infection of pseudotuberculosis produce
izumi fever
32
how do we diagnose Yersinia enterolitica and pseudotuberculosis
diarrhea, dehydration, false appendicitis, on exam. culture from blood, stool, cold-enrich and plate on CIN
33
treatment of Yersinia enterolitica and pseudotuberculosis enterocolitis or mesenteric adenitis
replace fluids and electrolytes
34
treatment for Yersinia enterolitica and pseudotuberculosis bacteremia, abscess, severe enterocolitis
sulfa-trimethylprim or cipro
35
prevention of Yersinia enterolitica and pseudotuberculosis
food prep and hand washing
36
listeria monocytogenes stain and morphology
positive rod
37
listeria monocytogenes motility
tumbling.
38
listeria monocytogenes hemolysis
beta hemolytic
39
listeria monocytogenes growth?
grows well in the cold, found on animals, plants and soil
40
how do we contract listeria monocytogenes
unpasteurized dairy products, undercooked meats, raw vegetables, contact with livestock and manure
41
who does listeria monocytogenes primarily infet
fetus across placenta, newborns during delivery, pregnant women, immunocompromised
42
what virulence factors does listeria monocytogenes have
internalin -binds to host E-cadherin, mediates invasion of cells in GI tract, placenta and meninges. listeriolysin -opens the phagosome and bacteria escapes into the cytoplasm ActA virulence -actin rockets for infectivity
43
how does listeria monocytogenes grow in the host
intracellularly
44
how does listeria monocytogenes spread throughout the host
cell-cell spread using actin-rockets by ActA virulence
45
diagnosis of listeria monocytogenes
gram stain diptheroids, culture: small gray beta hemolytic colonies further distinguished by sugar metabolism and motility.
46
what happens when pregnant women is infected by listeria monocytogenes
abortion, premature delivery, postpartum sepsis
47
what happens when newborn is infected with listeria monocytogenes
meningitis
48
what happens when immunocompromised is infected with listeria monocytogenes listeria monocytogenes
sepsis, meningitis
49
what happens when a previously healthy person is infected qith listeria monocytogenes
watery diarrhea, fever, HA, myalgias, abdominal cramps, little vomit. outbreaks from food
50
treatment for listeria monocytogenes gastrenteritis
self-limited
51
treatment for listeria monocytogenes sepsis/meningitis
sulfa-tri. or ampicillin + gentamycin
52
prevention of listeria monocytogenes
counsel pregnant women and immunocompromised to avoid farm animals, unpasteurized dairy, deli meats, unwashed raw vegetables

micro unit 6 (7 decks)

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