Exam 1 - 1 Flashcards

(336 cards)

1
Q

Sports medicine - 2

A
  • broad field of medical practice related to PA and sport
  • multidisciplinary including the physiological, biomechanical, psychological, and pathological phenomena associated with exercise and sports
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2
Q

ACSM

A

american college of sports medicine

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3
Q

4 things that the coach is responsible for?

A
  • prevention of injuries
  • provide/direct appropriate health care to the injured athlete
  • thorough understanding of the skills, techniques, and environmental factors that may adversely affect the athlete
  • work closely with medical staff
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4
Q

umbrella - 2 sides of sports medicine

A

performance enhancement

injury care and management

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5
Q

8 professions of performance enhancement

A
exercise physiology 
biomechanics 
sports psychology 
sports nutrition 
strength and conditioning 
personal fitness training 
coaching 
physical ed
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6
Q

8 professions of injury care and management

A
practice of med - physicians and assistants 
AT
sports PT 
sports massage therapy 
sports dentistry 
osteopathic med 
orthotists/prosthetists
sports chiropractic
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7
Q

triangle sports med model

A

coach, treatment, performance, bottom is prevention surrounding the athlete

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8
Q

sports med team 1-9

A

treatment

  • sports med, physician (canadian academy of sports and exercise
  • orthopedic surgeon
  • AT
  • Sports PT
  • Massage Therapist
  • nutritionist
  • dentist
  • podiatrist
  • chiropractor
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9
Q

Sports sci 1-7

A
biomechanist 
exercise physiologist 
sports psychologist 
strength and conditioning coach 
biochemist 
anatomist 
bioengineer
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10
Q

When collaborating with other personels on the team, what’s important

A

know the roles and responsbilities of each medical professonal and stay in your lane

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11
Q

What kind of treatments do we aspire to provide - 4

A

knowledge, competency, effective and evidence based

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12
Q

what kind of treatments can sports professionals give - 7

A
  • injury prevention and health promotion
  • clinical examination and diagnosis
  • acute care of injury and illness
  • therapeutic interventions
  • psychosocial strategies and referral
  • health care admin
  • PD and responsibility
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13
Q

sports med physician (CASEM) and 4 responsibilities

A

absolute authority in determining health status of an athlete who wishes to participate in a sports program

  • compile medical histories
  • diagnosing injury
  • deciding on a disqualification and return to play
  • attending practices and games - on call
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14
Q

AT and 3 responsibilities

A

start to finish - most time - prevention, immediate care and management of athletic injuries

  • prevention focus on MSK assessment, equipment, prophylactic support
  • immediate care includes injury assessment and basic emergency life support
  • management - contemporary rehab techniques and modalities to facilitate the healing of an injury
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15
Q

CAT - how to become one (3)

A

certified athletic therapist - devoted to the health care of the physically active ind

  • Bachelors’s, athletic therapy program at 1/7 CATA accredited institutions
  • valid first responder certificate and 1200 hrs of onfield and inclinic practical training
  • national certification exam - written and practical
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16
Q

5 competencies in AT

A
prevention 
assessment 
intervention 
practice management 
professional responsibility
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17
Q

6 things ATs do

A

onfield urgent emerge care, assessment and management
onfield nonurgent assessment, management and transportation
sideline return to play decision
prophylactic support techniques
orthopedic physical assessment (advanced, course, spinal and peripheral)
rehab (advanced cource)

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18
Q

kinesiologists - 3

A

independently with client to develop training programs - rehab clinics, primary care networks and with other health care professionals such as chiros, PTs, OTs, ATs, and dieticians

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19
Q

CSEP - CEP

A

canadian society for exercise physiology - certified exercise physiologist

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20
Q

CSEP - CPT

A

canadian society for exercise physiology - certified personal trainer

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21
Q

exercise physiologist/therapist -2

A
  • csep- cep - assessments, prescribe conditioning exercise, exercise supervision, counseling and healthy lifestyle ed with healthy or pop. with med conditions, functional limitations or disabilities associated with MSK, cardiopulmonary, metabolic, neuromuscular and aging conditions
  • csep - cpt
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22
Q

strength and conditioning specialist - 3

A

certified strength and conditioning specialist -improve performance - sports specific testing sessions to design and implement safe and effective programs, also nutrition and injury prevention
tactical strength and conditioning facilitator
CPT/CEP
consult with and refer athletes to other professionals when appropriate

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23
Q

To participate in a sport you need - 5

A

medical clearance - communicable disease
EAP - site specific - emergency action plan - address, call person, access of facility and phone numbers
facility safety
personal equipment and readiness
observation - event, MOI throughout, previously injured athlete, high risk athletes, atheletes with pre-existing medical conditions

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24
Q

when an injury happens - 6

A

enter field when SAFE to do so - no glass
witness - info from bystander
number of athletes involved - triage
C spine mechanisms and control - stabilize head and neck
posture of athlete - decerabrate vs decordecate - call 911
Enact EAP

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25
decerabrate vs decordacate
yardsale and limp | neurological trauma where everything is moving towards in and seizure
26
Level of consciousness - 6
posture of athlete obvious LOC - talking/walking - conscious/unconsious AVPU - alert/verbal/painful/unresponsive C-spine control Position/location of athlete Unresponsive EMS activation
27
air way - 4
obvious open airway - talking, heavy breathing removal of potential obstructions airway management - practice based on your level of training - head tilt chin lift, manual maneuvers, OPA/MPA, other EMS?
28
breathing - 4
obvious breathing - talking breathing heavy hypo/hyperventilation management - practice based on your training - talk to, calm - O2 administration AR (CPR standard) EMS activation
29
circulation - 3
obvious circulation - CCBMP = coughing, colour&skin temp, moving, breathing, pulse - always check for neck CPR EMS
30
deadly bleeding - 3
obvious arterial bleeding femoral and carotid artery first aid standard management - direct pressure, elevate, dressing, pressure points
31
After injury - what do you look for - 6
``` level of consciousness airway breathing circulation deadly bleeding CNS ```
32
CNS check - 3
Rule out spinal injury - spine not involved, no MOI, chief complaint elsewhere - if no, stabilization and through exam required with urgent paradigm rule out head injury - in not, thorough exam required, use SCAT or urgent paradigm directly to peripheral jts/area
33
urgent vs non-urgent
stop and load - graduated model | non-urgent - staying and playing
34
focused secondary survey - 3
initial orthopedic scan - jt/area defined assessment management
35
assessment - 4
what prudent skill sets and or qualifications of professional peers SAM - skeletal - bony integrity, articular - movement (Active/passive) - luxated, dislocated, motor-control of the) MSC - muscular- isometric/passive (dont let me move my finger), sensory - temp, 2 pt discrimination, sharp/dull, altered sensation, what am i touching? circulation - color and temp, pulse clear joint above and below as warranted (MOI, CC)
36
management of focused secondary survey - 3
transport, immobilize/stabilize - where - sideline/med facility - how? - walk with aid, stretcher, 2 man lift, ems
37
Immob/transport - 5
``` Where stablize - method - hand above and below immob - method transport - destination and method EMS? ```
38
assess/referral - 6
through clinical sideline assessment referral - whom, when, timely follow up (regarless of RTP status communication - player, coaches, parents follow up documnet consideration for immediate RTP
39
graduated return to play/prescription - 4
preparation - stabilize, protect, therapy/rehab functional test sport specific - load decision about RTP - clearance - doc/supervisor monitor and document - half time, post event, next workday
40
3 actions following focused secondary survey
immob/transport assess/referral graduated RTP/RX
41
liability
legally responsible for the harm one causes another
42
AT main responsibilities - 2
prevention of injury and reducing further injury or harm
43
Why do we care about liability - 2
accidents happen and legal lawsuits against authority - know your legal limitations and provide health care responsibilities as dictated by law
44
what is legal action tried under? - 3
TORT law - civil wrong done to an ind - act of omission/nonfeasance - ind fails to perform their legal duty - act of commision/malfeasance - ind commits an act that is not legally theirs to perform
45
standard of care
measured by what another minimally competent ind educated and practicing in that profession would have done in the same or similar circumstances to protext an ind from harm or further harm - what a resonable and prudent ind would do within your knowledge and training
46
standard care is dictated by
professionals duty/scope of practice - roles and responsibilities of an ind in that profession and delineates what should be learned in the professional prep of that ind
47
Why are we behind in treatment
conservative and the principle of not hurting anyone
48
duty of care
AT to their participants | failure to provide that results in liability or negligence
49
negligent torts - misfeasance
commiting an act that is ones responsibility to uses the wrong procedure or right procedure in an improper manner
50
negligent torts - malpractice
commits a negligent act while providing care
51
negligent tort - gross negligence
total disregard for safety of others
52
Participants assume what risks?
the ones inherent with PA but not the risk that a professional will breach the duty of care
53
to find an ind liable you must prove 4 things
there was a duty of care there was a breach of duty there was harm the resulting harm was a direct cause from that breach of duty
54
good samaritan law
limited protection to someone who choses to provide first aid who voluntarily chooses to do so should sth go wrong but we have a duty of care
55
you will be judged on your
performance so know the expected competency
56
statue of limitations
length of time to sue for damages from negligence - 1-3 yr sometimes 3 yrs after they turn 18
57
AT and coaches can take 5 steps to limit the risk of litigation
1. inform the participant about inherent risks of participation - assumption of risk by waivers - minors are your responsibilities 2. foreseeing the potential for injury and correcting the situation before harm occurs 3. obtaining informed consent from ind/guardian before participation and treatment - duty of care must help 4. quality products and equipment 5. strict confidentiality of all med records
58
16 axns that can result in litigation
failing to warn about risks treating without consent failing to provide med info concerning alt treatment or risks with needed treatment failing to provide safe facilities, fields and equipment aware of potentially danger but didnt do anything about it failing to provide a adequate injury prevention program allowing an injured or unfit player to play and resulting in further injury or harm failing to provide quality training, instruction, supervision unsafe equipment moving injured before properly immobilizing failing to employ qualified med personnel failing to have a written EAP failing to properly recognize injury or illness failing to immediately refer an injured to proper physician failing to keep adequate records treating an injury that did not occur within your facility
59
23 things you can do to manage athletic injuries and decrease risks of litigation
warn of danger supervise constantly and attentively properly prepare and condition properly instruct skills proper and safe equipment and facilities good personal relationships specific policies and guidelines for operation of athletic health care program develop and follow EAP familiar with health status and med history of athletes for additional care/caution records of injuries and rehab document efforts to create a safe playing environment detailed job description in writing written consent when providing health care - esp minors cofidentiality of records dont dispense any drugs certify in CPR/AED and first aid no use/precense of faulty/hazardous equipment work with team doc and AT and use protective equipment no injured player unless cleared esp head always follow orders of AT and doc liability insurance to protect against litigation and know the limits know limits of expertise and applicable state regulations common sense - health and safety of athlete
60
4 - why is communication important
personal relationships with athletes parents and coworkers good record of injuries and rehab dont give drugs common sense in decisions of safety and health
61
assumption of risk - 3
legal liability waiver to express/imply agreement that they assume the risk involved in the B take a risk when they play but should be made aware of potential risks AT's responsibility that they are aware
62
Sample form for treatment
med authorization/consent for med treatment of | - agree to pay all fees and costs arising
63
general liability insurance
slip and fall - injury at school/work property
64
professional liability insurance - 4
registered kins - pro athletes with a contract - their contract is on your insurance when they are under your care - covers claims of negligence on ind part - know the limits of your coverage - may not cover criminal complaint
65
professionalism and dating
just date and tell people
66
professionalism - 6
``` look and act like one appropriate touching dont act in a sexually inappropriate way professional and social context texting and social media code of ethics ```
67
mission of CCES - 2
foster ethical sport for all canadians - we will not cheat | you are a educated role model now
68
CCES
canadian centre for ethics in sports
69
3 purposes of CCES
Canada's anti doping program - govt give money to amateur sports but they are subjected to random drug tests serve public interest and protect the rights of athletes to fair and ethical competition by promoting and striving for fair and doping free sport in canada meet mandatory requirements of the world anti doping program
70
russians and doping
no anti doping officers that are wata approved
71
3 fundamentals of CCES
authority through govt's canadian policy against doping in sport - when you sign as a member of the sport you promise not to cheat sport adopt CADP into by laws for govt funding sign on to SADP through signing as members of their sport
72
top 6 threats to good sport
``` doping - most significant violence - no game or sports related bad parental B weak sport governance neg pro sport values lack of access and inclusion ```
73
doping
performance enhancing substances or methods
74
what constitutes as doping - 2/3
sci evidence that substances or methods have the potential to enhave sport performance - fairness sci ecidence that use of substances/methods rep a potential health risk - harm use of it violates the spirit of the sport - integrity
75
doping control officer
official trianed and authorized by CCES with delegated responsibility for onsite management of sample collection session
76
type of testing - 2
incompetition - game day/event out of competition - whereabouts program types of athletes tested - registered testing pool domestic athletes - random - highly ranked
77
motto of CCEP
anytime - anywhere
78
6 ways to be considered as doping
presence, use, refusing, failure, tampering, possesion, traficking - just take it, admin
79
5 consequences of doping
presense, possesion, use = up to 4 y ban refusal and tampering - up to 4 yr trafficking and admin - 4+ yrs all made public to media by CCES must establish - how it entered the body - was not performance enhancing, did not mask - evidence
80
4 ways to decide who to test
random selection targeted testing intelligence based testing - good athletes biological passport program
81
what % of canadians were tested before socci
100
82
liability of doping
you - responsible for what you consume and what is found in your urine/blood
83
top drug seizure in ab, bc, on
marijuana
84
top drug seized in quebec
steroids and its also the number 2 seized drug in quebec
85
whats on the prohibited list - 9
1. Anabolic Agents 2. Hormones and related substances (EPO, Insulin) 3. Beta-2 agonists (asthma) 4. Agents with anti-estrogenic activity 5. Diuretics or other masking agents 6. Stimulants 7. Narcotics 8. Cannabinoids 9. Glucocorticosteroids
86
drug prohibited at all times
Drugs prohibited at ALL times! 1. Anabolic Agents 2. Hormones a. EPO, hGH 3. Beta-2 Agonists a. Enhance flow of oxygen 4. Agents with Anti-Oestrogenic Activity 5. Diuretics and Other Masking Agents 6. Methods enhancing Oxygen transfer a. Blood doping / packing 7. Pharmacological, Chemical, and Physical Manipulation a. Urine tampering 8. Gene Doping
87
Prohibited doping during comp only - 6
1. Stimulants 2. Narcotics 3. Cannabinoids 4. Glucocorticosteroids a. Anti-inflammatories 5. Other Drugs a. Alcohol, beta blockers etc. 6. Specified Substances a. Generally available but may be abused for performance enhancement purposes. i.e. ephedrine, inhaled beta-2 agonists etc.
88
3 prohibited doping methods
1. Enhancement of Oxygen transfer 2. Chemical and Physical manipulation (urine and blood samples) 3. Gene Doping - hyperplase muscles - Hypoxic devices are ok.
89
gene doping
genetic enhancement that cannot be detected nor shut off - hyperplasia doenst stop so you die - allele with myostatin knocked out
90
how do i know if sth is ok - 3
drug info number - every sport fed has own rules | drug ref online - globaldro and CCES
91
how many global dro inquiries last yr
108000
92
suppliments
CCES media release supplement kaizen HMB and pos test
93
declaration of doping
declare all prescription and /or non meds and or supplements taken in the past 7 days
94
why does the drug program exist - 2
sport and society cannot exist w/o rules or agreed upon codes of conduct - protect the rights of clean athletes but not to catch dirty or cheating ones
95
8 reasons why athletes dope
* Money drives the athlete to win? * Edge over competition * Peer Pressure * Physical appearance * Status, recognition * Coaches are paid to win * Parents * Pharmaceutical companies?
96
highest use of anti doping lab in QC - 3
MLB CCES NBA
97
anti doping lab in QC
second largest in the world
98
designer steroid
on the rise
99
stimulant s6
nightmare
100
stimulant s6
nightmare
101
new weight loss stim
methylhexanamine/dimethylhexanamine - bad
102
where do designer steroid/stim come from?
china
103
anabolic steroids in 1988 vs 2006
20 vs more than 200
104
top 3 steroids
testosterone nandrolone stanozolol
105
the clear
* THG or Tetrahydrogestrinone (often referred to as THG or The Clear) is an anabolic steroid. * THG is a Designer steroids which means it was designed to keep the T:E ratio low. * Kelli White – modafinil and THG
106
top 3 sports with AAs
cycling - pedal harder baseball hockey - power endurance
107
HGH - 8
* Somatrem or Somatropin * Anabolic hormone that affects all body systems and is important in muscle growth * One of the most highly sought after drugs among athletes. Why? * Anabolic properties and difficult to detect * hGH causes hyperplasia vs. hypertrophy with AAS * Affects of hGH persist after cessation of use vs. AAS * Beware! * Acromegaly, stimulation of ALL tissues including internal organs, nonreversible effects, injection administration
108
EPO
metabolic agent erythropoietin - increases the oxygen carrying capacity of the blood, decreases exercise heart rate, and lowered post-exercise lactate levels • Normally taken with AAS such as nandrolone to potentiate the effects of EPO! • BEWARE – Increased systolic blood pressure, increased blood viscosity, seizures, thrombosis, as you dehydrate, your hematocrit % increases or your blood thickens! 50% to 60%
109
the way you play
how you live, behave and who you are
110
etiology
cause of injury/disease - mechanism
111
pathology
structural or functional changes that result from the injury process
112
symptom
perceptible change that indicates injury or disease - (what they feel and or describes)
113
sign
objective, definitive and obvious factor for a specific condition
114
diagnosis 1 -3
name of a specific condition - physician legal implications differential Dx - ATs index of suspicion
115
prognosis - 4
prediction of the course and outcome of the condition - spectrum what is to be suspeccted as it heals (when can i play) how long will you be experiencing pain/disability - permanent? time frame/expected outcome
116
sequela
condition resulting from disease or injury, development of a additional condition as a complication of an existing injury
117
syndrome
group of S&S that together, indicate a particular injury or disease
118
anatomical planes - 3
med profes refer to sections of the body in terms of anatomical planes/flat surfaces - imaginary lines drawn through and upright body -used to describe specific body part
119
axial
transverse anatomical direction
120
what are anatomical planes in references to?
anatomical position
121
midline
sagittal plane
122
ventral
ant
123
cephalad/cranial
head
124
caudal/caudad
tail/tail end
125
What do we do with abdominopelvic quadrants
palpate for tenderness and gas
126
right upper quadrant - 6
liver, rt, kidney, colon, pancreas, gall bladder
127
right lower quadrant - 4
appendix, ascending colon, right ureter, major vessels - artery and vein
128
left upper quadrant - 5
stomach , spleen, left kidney, transverse and descending colon, pancreas
129
left lower quadrant - 4
descending colon, small intestine, left ureter, major vessels - aetery and vein
130
eversion
turning outward
131
inversion
turning inward
132
pronation to the foot vs hand - 3 -1
combo of eversion, abduction of forefoot and dorsi flexion | hand turned down
133
supination to the foot vs hand - 3 - 1
inversion, plantarflexion and adduction of the forefoot | hand turned up
134
valgus
deviation of part of extremity distal to jt towards midline - knocked knees
135
varus
deviation of part of extremity distal to jt away from midline - bow legged
136
bones of the foot
26 14 phalangeal 5 metatarsal 7 tarsal
137
foot anatomy compared to hand anatomy
wider base for balance and propelling the body forward
138
metatarsals
5 bones articulate with the tarsals and phalanges
139
tarsals
between lower leg and metatarsal
140
medial longitudinal arch
medial border of calc to distal head of 1st metatarsal
141
lat longitudinal arch
lat aspect of calc, cuboid, and 5th metatarsal
142
ant metatarsal arch
distal heads of metatarsals
143
transverse arch
transverse tarsal bones - cuboid and middle cuneiform
144
interphalangeal jt - 1- 2
bw proximal and distal phalanx - flexion and extension
145
metatarsophalangeal jt 1-4
MT and proximal phalanx - flexion, ext, add, abd
146
tarsometatarsal jt 1-4
cuboid and all 3 cuneiforms and MT | flex, ext, add, abd
147
subtalar jt - 4
talus and calc - pronation, sup, in/e
148
talocrural jt 2-2
ankle mortis talus and distal tib fib ankel PF and DF
149
why are you unstable on your toes?
talus is wide in the front and narrow in the back
150
2 phases of gait
stance - move from pos lat to big toe heel strike, midstance and toe off swing - after toe off and initiate contact for heel strike
151
greater trochanter bursitis
too much swing in hips
152
why is pronation important for gait | 4 prolonged pronation injuries
shock absorption | stress fracures, tendonopathies, medial tibial stress syndrome, knee pain
153
why is supination important for gait? | 3 prolonged supination injuries
forceful propulsion of body forward | stress fractures, tendonopathies, lower back pain
154
high arch
supinated - tight musculature on the inside of your calf
155
ATFL injury from
Supination
156
Footware for pronation - 4
rigid heel counter, less flexible shoe, board lasted | hypermobility problems
157
footwear for supination - 4
flexible shoe, increased shock absorption, stitch lasted | hypomobility problems
158
lower leg
portion of lower extremity that lies between the knee and ankle tibia, fibula, talus and calcaneous
159
tibia - 2
2nd longest bone in body | 3 surfaces - pos, med, lat
160
fibula - 2
attachment for muscles | 6 wks you can play with plates
161
talus
link between lower leg and foot
162
calcaneous - 2
heel and site of attachment for achilles
163
sup and inf tibiofibular jt
sup - reinforced by stabilizing ligs ant and post | inf - reinforced by ankle ligs
164
interosseous membrane - 2
cover entire length of both bones - ant and pos tibfib lig is the distal of the interosseous mem
165
medial lig - 5
ant talofibular, pos talofibular and calc tabolibular deltoid - triangle later mal is longer so eversion less than inversion med surface of talus, calc, and navicular resistance to eversion - eversion test
166
3 lateral lig
ant talofib - refrain ant displacement of talus calcfib - restrain inversion of calc pos talofib - refrains pos displacement oftalus
167
3 muscles of the ant compartment and function
tibialis ant, extensor hallicus longus - in front of med mal, extensor digitorum longus - dorsiflex
168
3 muscles of lat compartment and functions
peroneous longus and brevis - evert foot | peroneous tertius - dorsiflex ankle
169
2 muscles of sup pos compartment and function
gastroc and soleus - achilles tendon - plantarflex
170
3 muslces of deep pos compartment and function
tibialis pos, flexor digitorum longus and flexor hallicus longus - behind medial malleous
171
3 articulations in your foot
proximal and distal interphalangeal jt | metatarsalphalangeal jt
172
intrinsic muscle in the foot
extensor digitorum brevis
173
``` Ant compartment 3 muscles pulse muslce action ```
``` Tom's hairy dog - med to lat on ant surface TA - most prominent and medial extensor hallicus longus - DF first toe extensor digitorum longus - extends toe dorsalis pedisis artery pulse peroneous tertius DF ankle and extend toes ```
174
lateral compartment - 2
peroneous longus and brevis - evert and DF foot
175
``` Deep pos compartment 3 muscles nerve muslce function ```
Tom dick and harry - ant to pos tib pos - invert and PF foot flexor digitorum longus - flx toes pos tibial artery - main blood supply to foot tibial nerve - main nerve supply to sole of foot (tarsal tunnel flexor hallicus longus - cannot be palpated invert the ankle
176
claw toes - 2
hyperflexion of MTP and flexion of proximal and distal IP jt associated with pes cavus and painful calluses
177
hammer toes
similar to claw
178
morton's foot - 2
second toe is longer than first - stress to 2nd toe and hypomobility to 1st toe difficulty putting on tight shoes
179
accessory navicular - 2
double ankle jt | prominence on the navicular tubercle
180
pes cavus - 2 signs and symptoms - 3 management - 3
high arch excessive supination general foot pain, metatarsalgia abnormal shortening of achilles tendon asymptomatic dont correct orthotic, stretching achilles tendon and plantar facsia
181
pes planus -2 cause - 2 signs and symptoms - 2 management - 3
flat foot excessive foot pronation weak musculature invertors or stretched plantar lig pain and feeling of weakness in the med longitudinal arch calcaneal eversion, bulging of navicular bone, flattening of the medial longitudinal arch asymptomatic dont treat orthotics strengthening of the foot invertors, toe curls
182
assessment of the foot - 7
``` weight bearing walking - stance, swing, push off running talocrural - dorsiflex - active 20, plantarflex - active 50 subtalar supination pronation ```
183
mechanical injury - 2
injuries occur when force applied to any part of the body results in a harmful disturbance in function and or to the structure - external force directly on the body or occur internally within the body
184
tissue properties - 2
tissues have the ability to resist a particular load - stronger the tissue, greater the magnitude of load it can with stand
185
stress strain curve
toe - elastic - (ultimate strength) - plastic (necking) -failure range (strain deformation
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toe
1.5-4% of total fibre lengthening that is possible
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necking
point at which tissue strength noticeably decreases so that less stress is needed to cuase a change in the tissues length - partially rips
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plastic range
doesnt completely recover
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stretching and tissue properties - 2
toe is where everyone goes has to go through plastic for permanent deformation - PNF - passive to barrier (passive restraint) then you keep going and get past it - compensation from the body and scream and push back but hold for 10 seconds and repeat till no slack - you have to hurt a little
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does static stretching increase flexibility?
no
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slow vs fast load
fast much more dangerous and forceful
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load
outside forces acting on tissue
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stress
internal reaction or resistance to an external load
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strain
extent of deformation of tissue under loading
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viscoelastic
material whose mechanical properties vary depending on rate of load
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yield point
elastic limit of tissue
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mechanical failure
elastic limit of tissue exceeded, causing tissue to break
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5 primary tissue stresses leading to injuries
tension, stretching, compression, shearing, bending
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tension
force that pulls or stretches tissue
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stretching
when go beyond yield pt leads to rupturing of soft tissue or fracturing of bone
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compression
large force that crushes tissue
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shearing
force that moves across in a parallel fashion across tissue - muscle is okay but bone will break
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bending
force on a horizontal axis that places enough stress to cause the structure to bend
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acute injuries and 6 examples
within 3 days- some sort of healing and it was an event - sudden and traumatic contusions, sprains, strains, dislocations, subluxations, fractures
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contusions - 6 MOI What happens
bruise external force causes soft tissues to be compressed against hard bone capillaries may break and cause bleeding into the tissues which accumulates under the connective tissue deep contusions can bruise the bone pain improves within a few days and discoloration decreases with a few weeks muscle pain with active contraction
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myositis ossificans MOI What happens
dont take care of the inflammation - you need to pad and let it heal occurs when a muscle or soft tissue is bruised repetitively ca begins to deposit in muslce fibre - blood pooling and ph changes may impact movement
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Treatment of myositis ossificans
usually surgical intervention is needed
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prevention of myositis ossificans
protect from repeated contusions - use padding
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common 2 places of myositis ossificans
quads and biceps
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muscle strain
tearing or stretching of the muslce fiber when its forced to contract against increased resistance
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degree of strain is related to
number of muscle fibres torn
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grade 1 of muscle strain
some fibres stretched or torn - minimal swelling, tenderness and pain with AROM, rest doesnt hurt and full ROM
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grade 2 muscle strain
more fibres torn, increased pain and greater loss of muslce function with AROM - divot or depression - may be swelling
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grade 3 muscle strain
complete rupture | no pain, palpable defect, loss of muscle function, swelling and bruising
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what is the usual MOI for a grade 3 muslce strain
fast eccentric force
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3 common grade 3 muscle strains
biceps, achilles, hamstrings
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charlie horse RTP rule
if you cant actively get 90 degrees
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ligaments
tough inelastic bands connecting bone to bone
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lig sprain
when jt is forced to move beyond normal limits, damage to lig or jt capsule that provides support to a jt - crushing and tearing - disrupting the integrity of a joint
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can you play on a lig injury?
yes if your muslces are still good
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how are lig sprains graded?
severity of damage
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grade 1 sprain
minor stretching /tearing of fibers - pain, localized swelling, minimal instability
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grade 2 sprain
more tearing, increased pain, swelling, more instability
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grade 3 sprain
complete rupture of fibres, little to no pain, groww swelling, joint stiffness, complete instability
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dislocation/luxation - 2
one bone in a jt forced completely out of normal alignment - deformity can result in other soft tissue injuries - rupture of lig and tendons, avulsion fractures, chronic joint stability
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how to reduce a dislocation
manually or surgically
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common dislocation - 3
shoulders, elber, fingers
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subluxation - 3
partial dislocation bone forced out but goes back into alighment can cause lig stretching/tearing, capsular distention - increase likeliood of reoccurrence
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common subluxation - 3
patella, shoulder jt, fingers
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fractures | signs and symptoms
extreme stress/strain on bones deformity, point tenderness, swelling, pain with AROM/PROM, crepitus X-ray or other dianostics to rule out
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open/closed fracture
protrude/breaks through skin | little or no displacement between bone ends
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our tests for fractures - 4
tuning for, vibration, indirect pressure, axial loading
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chronic injuries - 2 | 6 ex
``` repetitive microtraumas and overuse inflammation is the process for acute injuries - when the source of irritation is not removed, inflammatory process becomes chronic tendinopathy tendinitis tenosynovitis bursitis osteoarthritis stress fractures ```
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``` tendinitis MOI signs and symptoms treatment ex - 4 ```
inflammation of tendon repetitive movements - when it moves or slides on other things its irritated and inflamed pain with movement, swelling, increased temp, crepitus rest, cross training to allow tendon to rest/decrease inflammation tennis elbow, bicep tendinitis, achilles, ITB
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cross training
2 or more aerobic classes
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ankle - 6 bones
``` tib fib talus calc navicular metatarsals ```
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tibia - 3 parts
ant border posteromedial border med mal
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fibula - 2
head of fibula in sup ribiofibular jt | lat mal
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important part of talus
head
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calcaneous - 3
sustentaculum tali - supports talus and attachment site for spring (deltoid lig) peroneal tubercle - distal to la mal, separate peroneus longus and brevis medial tubercle med tubercle - med plantar surface - attachment of plantar aponeurosis - weight bearing
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part of navicular
navicular tuberosity
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metatarsals - 2 things we need to know
1st and 5th metatarsal | styloid of 5th
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medial lig
deltoid
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dorsum lig
ant inf tibiofibular lig
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lat lig - 3
ant talofibular lig cfl pos talofibular lig
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4 special tests
eversion inversion ant drawer thompson
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eversion test - 2
stabilize tib and evert calc | assess deltoid lig stability
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inverstion test - 2
assess ATFL and CFL stability | talus gaps in ankle mortise
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Ant drawer test - 2
ATFL is the only structure preventing forward subluxation of the talus stablize tib, grip calc and draw calc (and talus) forward while pushing the tib pos
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thompson test - 3
tests continuity of the achilles tendon patient lies prone on table, squeeze muslce belly of the gastroc - should have resultant PF of foot may also present - pain, swelling, TOP, inability to PF foot strongly, able to walk but absense of push off, toe off, and flat foot gait
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6 common injuries to the ankle and lower leg
``` ankle sprains achilles tendonitis, rupture shin splints plantar fasciitis (Arch) mortons neuroma turf toe ```
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ankle sprains MOI S&S - 4 management
most common athletic injury esp inversion sprains inversion of the ankle with PF or DF (inversion or lateral sprains), eversion with generally DF pain and disability, unable to weight bear, swelling, bruising PIER, xray to rule out fracture, strenthening and balance exercises
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grade one ankle sprain
tearing of some fibres
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grade 2 ankle sprain
more tearing of fibres with noticable laxity in the jt
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grade 3 ankle sprain
complete rupture of fibres
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what might lead to ankle injury
poor balance
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after an ankle injury
balance may be affected due to subject's inability to determine position of ankle therefore gait will be affected as well
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achilles tendon rupture MOI S&S - 5 management - 2
``` quick motion from PF to DF sudden snap in achilles area pain may be present swelling over tendon point tenderness inability to raise up onto toes non-operative - PIER, Nonsteroidal anti-inflammatory drugs, casting, gentle stretching and strengthening operative - surgical attachment of ends of achilles, then PIER, ROM, strengthening and balance exercises ```
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achilles tendonitis MOI - 2 S&S - 3 management - 5
``` overloaded due to excessive stress - decreased flexibility is also a contributing factor pain and stiffness in area decrease in strength of G/S complex crepitus with movement need to decrease stress on the tendon PIER and correct faulty mechanics Heel lift to put the foot into slight PF Stretching GS complex casting if athlete is non-compliant ```
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shin splints - 4
medial tibial stress syndrome stress reacion inflammation of the periosteal and musculotendonis fascial junctions distance runners rule out stress fracture and ant compartment syndrome
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Shin splint MOI - 4 S&S - 3 Management - 6
training errors, improper or poor footwear, tight achilles repetitive microtrauma pain in middle or distal 1/3 of lower leg varies in intensity from with activity only to pain with ADL 4 grades of pain referral to physician to rule out stress fracture modify activity correct abnormal pronation, local treatment, stretch GS, taping
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plantar fasciitis - 2
pain under proximal arch and heel | plantar fascia supports the arch of the foot
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5 causes of plantar faciitis pain
``` stretching of facia as in toe extension fascia shortens as a result of improper footwear or gait mechanics leg length discrepancy excessive pronation tightness of GS ```
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Plantar fasciitis S&S - 2 | management - 6
``` pain on anteromedial aspect of heel with weight bearing and toe extension burning pain with weight bearing esp morning orthotics night split PIER arch taping stretching GS proper footwear ```
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Metatarsalgia/morton's neuroma | 5 causes
pain in forefoot tight GS increased pressure on forefoot due to gait alterations causes nerves to be impinged between matatarsals aggravated with collapse of transverse arch pronated foot
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2 S&S for metatarsalgia/morton's neuroma | management -3
pain in forefoot tingling and/or numbness in forefoot orthotics, stretching GS strenthening foot intrinsic muslces with toe curls
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Turf toe causes - 2
hyperextension results in sprain of MTP jt of 1st toe | occur b/c shoes are very flexible of turf (surface) is very sticky and allows more flexion of the 1st MTP jt
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2 S&S of turf toe | 4 management
pain and swelling in and around 1st MTP jt pain with 1st toe flexion when pushing off stiffer shoes in the forefoot taping to prevent toe extension PIER
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Retrocalcaneous bursitis cause - 3
inflammation of the bursa that lies bw the achilles and calcaneous - pressure and rubbing - exostosis - bony outgrowth - pump bump
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2 S&S of retrocalcaneal bursitis | 4 management
``` pain with palpation swelling on both sides of insertion PIER NSAIDS heal lift to decrease irritation on tendon find irritating cause ```
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clonic spasm vs tonic spasm
intermittent contraction and relaxation vs constant muscle contraction
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MOI of leg cramps
unknown - thought to occur from lack of fluid
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S&S of Leg cramps
considerable pain and cramping
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management of leg cramps - 3
stretching with pressure applied over muscle cramp ice rehydrate
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acute leg fractures MOI | S&S - 2
direct/indirect trauma pain and disability immediate swelling over fracture site splint and refer
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stress fracture MOI - 3
repetitive trauma pronated - fibular stress fractures supinated - tibial stress fractures
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S&S of stress fractures -3
along bone with palpation positive fracture test at times bone scan to rule out fracture
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management of stress fractures
correct mechanical causes, NWb for rest | stretch tight structures
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Type of tape for prophylactic taping principles
white, non-elastic, adhesive 1 1/2 inch width
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what to consider while buying tape - 3
grade of backing fibres - thread count/quality quality of adhesive winding tension
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Tape prep for prophylactic taping - 4
skin surface, nicks and cuts, shaving, allergies
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9 things for a tape job
``` razor soap tape remover - dehesive tuff skin prowrap heel and lace skin lube tape and stretch tape scissors/shark ```
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3 phases of healing process
inflammatory (hemostasis) response - 0-4d fibroblastic repair 1-inf maturation - remodeling 2-inf continuum - overlap and have no set/end pt
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goal of you during healing process
create an environment that is conducive to the healing process
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When does inflammatory begin what does it result in? duration?
once tissue is injured - direct injury to cells of soft tissue Release of materials needed for the inflammatory response 2-4 d - ice in 72 hrs
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purpose of inflammation - 2
protect/splint, localize and rid body of some injurious agent in prep of healing associated with vascular, cellular and chemical responses
287
Why is the inflammation phase critical?
if it does not accomplish what is needed/does not subside, normal healing cannot take place
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Symptoms of inflammatory response
``` Swelling Heat Altered function Redness Pain ```
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2 events of inflammatory phase
vascular reaction | formation of a clot
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vascular reaction - 2
vascular spasm - vasoconstriction of vascular walls to reduce blood flow (5-10min - ice, tensor, immob), followed by vasodilation formation of a platelet plug, blood coagulation, and growth of fibrous tissue chemical mediators released - histamine, leukotaxin, necrosin to limit amt of exudate and swelling, swelling depends on the extent of tissue damage
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formation of a clot - 3
damage to BV exposes the endothelium and collagen fibers platelets and leukocytes adhere to the exposed fibers, eventually forming a plug localize the injury process
292
Fibroplasia
Period of scar formation
293
Fibroblastic phase time frame S&S- 2
formation of scar first few hours and up to 4-6 weeks inflammatory S&S subside tender to touch and painful with certain movements - depends on the tissues diff peak points
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What is happening in fibroblastic phase - 2
new capillaries bud into wound to deliver oxygen - increase blood flow and nutrients to site - initially a granulation tissue to fill in gaps as healing continues fibroblast accumulate at the would site and synthesize new extracellular matrix that contains collagen, elastin, ground substance and additional proteins
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day 6-7 of fibroblastic - repair phase - 3
collagen fibers deposited in a random fashion, forming a scar tensile strength increases in proportion to the rate of collagen synthesis increase in tensile strength decreases fibroblast production to begin next phase
296
Maturation-remodeling phase - 5
long term - up to several yrs realignment or redeling of collagen fibers that make up the scar tissue and ongoing breakdown and synthesis of collagen increased stress and strain causes the collagen fibers to realign along the lines of tension tissues gradually assume normal appearance and function by the end of 3 weeks - firm, strong, contracted, non-vascular scar forms
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Chronic inflammation - 4
acute inflammatory phase does not eliminate the agents and restore tissue to normal state can last for m to y repeated microtraumas and overuse of a particular structure typically resistant to both physical and pharmacological treatments
298
Cartilage healing capability
dependent on whether the damage is to the cartilage or subchrondral bone - bone progresses normally
299
Lig healing capability - 3
normal fibrin clot helps to bridge the torn ends of the lig over m scar continues to mature in response to stress and strain - up to 12 m
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why is lig healing variable - 2
scar tissue is insufficient | lig not reattached to the proper location on the bone - lig failure
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3 stages of healing in muscles
in large and force producing muslces - hemorrage, edema, phagocytosis to clear debris scar formation, myiblastic activity to regenerate myofibrils maturation - collagen fibers arrange themselves along lines of tensile force - needed for active muscle contraction
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active muscle contraction is needed for
normal tensile strength
303
Length of rehab in muscles depend on
degree of strain = can be longer than lig sprain
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Muscle strain and too soon for return to play
reinjury and start healing process again
305
Nerve healing | process - 3
cells dont regenerate when they die but nerve fibers do peripheral nerve regeneration at 3-4mm/d damaged nerves in CNS regenerate very poorly bc they lack CT sheaths and schwann cells dont proliferate surgical intervention in severed nerve - increases healing potential
306
Bone healing - 3
callus formation needs to occur at the fracture site remoddling process - ongoing where osteoblasts lay down new bone and osteoclasts remove bone according to the forces placed on the healing bone - wolff's law - bone adapts to the mechanical stress and strain by changing size, shape and structure osteoblast/clast activity may cont for 2-3 years
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3 types of trauma to bone
contusion of periosteum closed nondisplaced fractures displaced open fractures that involve soft tissue
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factors that impede healing - 7
``` extent of injury - edema hemorrhage poor vascular supply infection separation of tissue muscle spasm health, age and nutrition ```
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extent of injury and healing -2
greater the damage the more inflammation present - microtears vs macrotears
310
edema and healing
increased pressure causes tissue separation, inhibits neuromuscular control and impedes nutrition to the injured structure
311
hemorrage and healing - 3
increased bleeding causes increased pressure, additional tissue damage if its bleeding its not healing
312
poor vascular supply and healing
poorly and slow - tendons and ligs
313
infection and healing - 2
delays healing and increases granulation tissue
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separation of tissue and healing - 2
need more scare tissue to fill the gap | smooth edges vs jagged/separated edges
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muscle spasm
cause traction of torn tissue, separating the 2 ends, affecting approximation
316
health, age and nutrition - 2
``` older age has less elastic properties of tissue vit c(collagen synthesis), vit k(clotting), vit A(immune system) ```
317
PIER
pressure - pressure on blood vessels to make it harder for fluid to leak out ice - vasocontriction therefore reducing swelling elevation - uses gravity to assist in fluid drainage in area rest - ceases damage to the area by not placing stress on injured structures
318
Pain - 2
subjective sensation | unpleasant sensory and emotional experience associated with actual or potential tissue damage
319
Goal with pain
control acute pain by encouraging the body to heal through exercise designed to progressively increase functional capacity and return the patient to full activity as quick and safe as possible
320
acute vs chronic pain
less than 6m vs greater than 6m and cont beyond usual normal healing time
321
referred pain
away from the actual site of irritation
322
How do you treat pain -2
therapeutic modalities and manual therapy techniques ex: heat, cold, electrical stim, ultrasound, manipulation, ART, massage etc medication - Physician - NSAIDs or analgesics
323
4 ways to treat pain with therapeutic exercises
exercises/stretches to ROM; helps enhance realignment of scar tissue exercises to help strengthen injured tissues or surrounding areas include proprioception advance to functional exercise
324
infection in inflammatory stage
cant get out of it
325
what colors are healed scares
white, if its pink small roots of capillaries are still feeding it
326
what to make sure with scars
mobile and flexible to ensure consistency matches skin
327
healing by primary intention
stitches
328
how to figure out when to use stitches
open it to see deepness and if it goes back together its okay steri strip, if not - get stitches
329
In what environment do cuts heal better in?
moist environment
330
how do you heal chronic inflammation
reset it to square 1
331
stability for a jt after a lig injury
generally pretty good, some are within the jt capsule and gets less blood
332
3 treatments during skeletal inflammatory
milk massage, ice, anti inflammatory
333
2 treatments of skeletal during fibroblastic phase
gentle ROM, take gravity out and test
334
2 treatments of skeletal during fibroblastic phase
gentle ROM, take gravity out and test
335
smoking and healing
worse - 256x for CO to bind to RBC than O2
336
1-10 scale for pain
5 you are stopping2, for a break, 7 youre crying, 9/10 hospital