exam 1 - lecture 4 Flashcards

(47 cards)

1
Q

horses are ____ grazers

A

continuous

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2
Q

which bacteria in the colon is not a pathogen

A

Helicobacter equinus

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3
Q

what hormones control intake regulation

A

leptin
ghrelin - stimulate appetite

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4
Q

what is trituration

A

process ingesta to optimize digestion and absorption upon entry into the intestine

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5
Q

what allows liquids to depart the stomach without need for propulsive peristaltic contractions

A

thick circular musculature of pylorus

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6
Q

average gastric half emptying time for solid food

A

1.56 hr

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7
Q

half emptying time for liquid

A

10-74 min

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8
Q

half emptying time for liquid for foals

A

4-14 min

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9
Q

stomach will be empty of food in adult horse in

A

12 hr

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10
Q

where is the pH highest in the stomach

A

near the squamous portion

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11
Q

how does HCl secretion work

A

continuous

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12
Q

what stimulates HCl production

A

histamine
gastrin - cortisol
ACh
prostaglandins

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13
Q

what inhibits HCl production

A

somatostatin

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14
Q

2 forms of equine gastric ulcer syndrome

A

EGSD, EGGD
squamous and glandular disease
both can be in one patient

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15
Q

why do squamous ulcers develop

A

minimally protected from acid
normally covered with ingesta - unless not

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16
Q

what in humans is like EGSD

A

gerd

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17
Q

risk factors for EGSD

A

empty stomach, exercise on empty stomach, pyloric outflow obstruction

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18
Q

location for EGSD

A

lesser curvature at margo plicatus

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19
Q

why does EGGD happen

A

failure of normal protective mechanisms
causes unknown but H. pylori, NSAIDs and idiopathic are suspected

20
Q

treatment for EGUS

A

PPI - omeprazole

21
Q

is acid damage the cause of glandular disease

A

no - but is an aggravating factor

22
Q

CS for EGUS

A

asymptomatic, anxious, colicky, food aggressive, wont perform, grumpy, bruxism

23
Q

risk factors for EGGD

A

warmblood, specific trainer, multiple people, lots of exercise,

24
Q

diagnosis of EGUS

A

endoscopy, biopsy, US

25
how to do gastric endoscopy
NPO 16 hrs, restraint, pass like NG tube, inflate and deflate, confirm healing with follow up
26
gastric US
left flank, cranioventral not caudal to IC14
27
EGUS treatments and prevention
small frequent forage meals supplements mirrors water corn oil omeprazole - expensive, poor oral bioavailability with full forage, more effective for EGSD
28
are grades used for EGSD or EGGD
EGSD
29
complications of EGUS
uncommon recurrence peritonitis gerd polypus pyloris intussusception
30
primary gastric distention disorders
ingestion of fermentable food/grain overload food impaction
31
secondary distention disorders
small intestinal obstruction aklaline fluid buildup
32
what is most common form of gastric distention disorder
secondary
33
gastric outflow obstruction causes
ulceration, delayed gastric emptying syndrome, neoplasia, phytobezoar
34
what gets retained in delayed gastric emptying
solids
35
signs of delayed gastric emptying
asymptomatic, inappetence, mild recurrent colic, cutaneous sensitivity food bolus in stomach after 16 hr of fasting
36
Dx of delayed gastric emptying
CS, NG tube, enlargement of stomach via US, retention of food after fasting
37
gastric impaction predisposing factors
dental, gastritis, ulcers
38
clinical presentation of gastric impaction
colic, cant pass NG tube
39
Dx gastric emptying
contrast rads - no aboral movement into stomach by 30-90 min
40
common fall MO obstruction object
persimmons - surgery
41
what is gastrorrhexis
gastric rupture
42
why should you employ NG tube during Dx and management of colic
look for distension which can lead to rupture
43
is gastric rupture fatal
invariably
44
is gastric cancer common
no
45
gastric cancers
gastric SCC lymphosarcoma leiomyoma
46
who gets gastric SCC
old horses
47
Dx gastric SCC
endoscopy, US, analysis peritoneal fluid