Exam 2 Flashcards

(236 cards)

1
Q

multiple sclerosis

A

Myelin sheath destroyed (like rubber outside of phone charger)
Nerves not making smooth changes into muscle

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2
Q

parkinson’s disease

A

Center of balance and sensation are off
Rigidity
Can’t spontaneously put one foot in front of the other
Lots of concentration and thinking for simple tasks

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3
Q

CNS function

A

controls most body functions, including awareness, movements, sensations, thoughts, speech and memory

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4
Q

peripheral nervous system

A

broken down into somatic and autonomic

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5
Q

somatic nervous system

A

controls body movements that are under our control such as walking.

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6
Q

autonomic nervous system (and major organ)

A

further divided into sympathetic and parasympathetic
ADRENALS!

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7
Q

chain ganglia vs collateral ganglia

A

chain: spinal nerves and nerves in thoracic cavity
collateral: abdomen and pelvis

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8
Q

cerebrovascular disorder

A

functional abnormality of the CNS that occurs when blood flow to the brain is disrupted
Stroke is a major example
financial impact is profound

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9
Q

What is agnosia?
A. Failure to recognize familiar objects perceived by the senses
B. Inability to express oneself or to understand language
C. Inability to perform previously learned purposeful motor acts on a voluntary basis
D. Impaired ability to coordinate movement, often seen as a staggering gait or postural imbalance

A

A

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10
Q

nonmodifiable risk factors of cerebrovascular disorders

A

age (>55), male, black

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11
Q

manifestations of an ischemic stroke

A

Symptoms depend on the location and size of the affected area
Numbness or weakness of face, arm, or leg, especially on one side
Confusion or change in mental status
Trouble speaking or understanding speech
Difficulty in walking, dizziness, or loss of balance or coordination
Sudden, severe headache
Perceptual disturbances

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12
Q

hemiplegia vs hemiparesis

A

hemiplegia: complete paralysis
hemiparesis: partial weakness

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13
Q

dysarthria

A

difficulty speaking due to weak speech muscles

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14
Q

hemianopsia

A

only seeing on one side

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15
Q

TIA

A

Temporary neurologic deficit resulting from a temporary impairment of blood flow
“Warning of an impending stroke”

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16
Q

how to treat and prevent irreversible deficits

A

diagnostic workups

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17
Q

how to diagnose TIA

A

CT scan, cerebral angiography, lumbar puncture if CT is negative and ICP is not elevated to confirm subarachnoid hemorrhage

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18
Q

what to treat with TIA

A

vasospasm, increased ICP, hypertension, potential seizures, and prevention of further bleeding
Deliberate CALM care!!

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19
Q

cardiac endarterectomy

A

removes buildup from carotids
carotids feed brain with blood supply
hemorrhage is bad
can mess up shoulder

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20
Q

what to do for carotid stenosis and afib

A

carotid: carotid endarterectomy
afib: anticoags and antihypertensives

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21
Q

medical management in acute phase of stroke

A

prompt diagnosis and treatment
thrombolytic therapy
pt monitoring
watch for bleeding
elevate HOB unless contraindicated
maintain airway and ventilation
continuous hemodynamic monitoring and neuro assessment

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22
Q

hemorrhagic stroke caused by

A

spontaneous rupture of small vessels r/t hypertension
ruptured aneurysm
intracerebral hemorrhage r/t amyloid angiopathy
arterial venous malformations (AVMs)
intracranial aneurysms
medications such as anticoagulants
ICP increases caused by blood in subarachnoid space
Compression or secondary ischemia from perfusion & vasoconstriction causes injury to brain tissue

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23
Q

manifestations of hemorrhagic stroke

A

similar to ischemic
severe HA
early and sudden changes in LOC
vomiting
bleeding

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24
Q

assessment of pt recovering from ischemic stroke (acute phase)

A

ongoing frequent monitoring of systems esp neuro (CHECK AROUSAL LEVEL)
LOC
symptoms
speech
pupil changes
I&O
BP maintenance
bleeding
O2 sat

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25
nursing care after acute phase
Mental status Sensation/perception Motor control Swallowing ability Nutritional and hydration status Skin integrity Activity tolerance Bowel and bladder function Get men ready to pee again once they’re stable enough to stand
26
preventing joint deformities in stroke pts
turn and position in correct alignment q2h use splints passive or active ROM 2-5x/day prevention of flexion contractures prevention of shoulder abduction do not lift by flaccid shoulder quad setting and glute exercises assist patient OOB ASAP ambulation training
27
nutrition for stroke pts
Consult with speech therapy or nutritional services Have patient sit upright, preferably out of bed, to eat Chin tuck or swallowing method Use of thickened liquids or pureed diet Ice chips bad!!
28
when to perform neuro checks for pt with hemorrhagic stroke
q2-4h
29
assessment of pt with hemorrhagic stroke
Altered LOC Sluggish pupillary reaction Motor and sensory dysfunction Cranial nerve deficits Speech difficulties and visual disturbance Headache and nuchal rigidity Other neurologic deficits
30
3 complications of hemorrhagic stroke
Decreased cerebral blood flow Inadequate oxygen delivery to brain Pneumonia
31
5 complications of ischemic stroke
Vasospasm Seizures Hydrocephalus Rebleeding Hyponatremia
32
goals of hemorrhagic stroke
Improved cerebral tissue perfusion Relief of anxiety The absence of complications
33
aneurysm precautions
Provide a non-stimulating environment, prevent increases in ICP, prevent further bleeding: Absolute bed rest with HOB 30 degrees Avoid all activity that may increase ICP or BP; Valsalva maneuver, acute flexion or rotation of neck or head Stool softener and mild laxatives so they don’t bear down Non-stimulating, non-stressful environment; dim lighting, no reading, no TV, no radio Visitors are restricted
34
early identification of stroke (interventions)
Call RRT (neuro) Initiating stroke algorithm The National Institutes of Health Stroke Scale (NIHSS) labs prior to CT (CBC, BMP, coags, T&S)
35
stroke scale values
0 = no stroke 1 to 4 = minor stroke 5 to 15 = moderate stroke 16 to 20 = moderate to severe stroke 21 to 42 = severe stroke
36
Airway in pts with decreased LOC and interventions
Patients with decreased LOC have increased risk of airway compromise due to loss of protective reflexes and oral-pharyngeal reflexes Nursing Interventions: HOB > 30 degrees Suction prn O2 saturation assessment
37
S&S of dysphagia
ASPIRATION RISK Weak or absent gag reflex Drooling Excessive chewing Difficulty pushing food to back of mouth Dysarthria (difficulty speaking) Listen to the voice Gurgle, wet, weak, hoarse, strident Paresthesia of face, lips, tongue
38
what is aspiration
Aspiration is defined as the inhalation of either oropharyngeal or gastric contents into the lower airways
39
modified massey bedside swallow test
must be completed and documented w date and time for all rule out strokes and TIAs prior to any oral intake complete within 24hrs of admission or new onset TIA/CVA can't just document +/- nurses do this test when in doubt, keep pt NPO
40
aspiration PNA
Aspiration of colonized oropharyngeal material (food, secretions) Often polymicrobial Pulmonary inflammation pts look very sick
41
CXR shows what in aspiration pts
infiltration of dependent portion of lungs cloudy, can't identify outline of lobes
42
effective oral care does what 5 things
Reduces bacteria Increases appetite Increases alertness Increases salivary flow Reduces pneumonia incidence
43
diagnosis of aspiration PNA
Fever >100 F WBC > 10,000 (know normal WBC) Rales + sputum culture Productive cough PaO2 <70 mmHg CXR + for new infiltrate
44
how to manage reflux
positioning feeding/diet changes meds such as antacids, PPIs, histamine blockers, prokinetics, physical barriers
45
nursing interventions for dysphagia
NPO Swallow evaluation Ensure appropriate diet (puree, mechanical soft) Feed in upright position For pts with hemiplegia or paresis, place food on unaffected side If “pocketing” of food occurs, have patient sweep mouth with finger to remove
46
when can tPA be given
within 3hrs of stroke onset
47
5 assessment for tPA
monitor for bleeding maintain BP neuro status (for re-embolization or bleed) no SCD or BP cuff for 24h no heparin for 24h
48
what to use for non-tPA eligible pts
Early ASA therapy is recommended (150- 325mg) Pts w/ restricted mobility Prophylactic low-dose SQ Heparin or LMWH or IPC (intermittent pneumatic compression)
49
blood pressure management for stroke pts
aggressive efforts to lower blood pressure may decrease perfusion pressure and may prolong or worsen ischemia hypertension in the setting of hemorrhagic stroke should always be managed Parameters for BP management can vary depending on if patient is a candidate for t- PA
50
fever in stroke pts
mild hypothermia in the brain is neuroprotective, hyperthermia accelerates ischemic neuro injury give antipyretics and find source of fever
51
antithrombotics for day 2 post CVA/TIA
ASA Aggrenox ASA & dipyridamole Coumadin Plavix Ticlid IV Heparin LMWH – full dose
52
afib and ischemic CVA meds
long term coumadin (INR 2.5 [2-3]) ASA 75-325 mg/day if coumadin contraindicated
53
first thing we look at with people with impaired LOC
verbal response and alertness
54
lethargy
drowsy, awakens to stimulation
55
obtunded
difficult to arouse, needs constant simulation to FOLLOW SIMPLE COMMAND
56
stupor
arouses to vigorous, continuous stimulation CAN'T FOLLOW A SIMPLE COMMAND can be from increased ICP severe impairment to brain circulation immediate intervention may become comatose and exhibit abnormal motor responses if goes to irreversible coma, brainstem reflexes are absent, respirations are impaired, may be braindead)
57
akinetic mutism
Unresponsiveness to the environment, makes no movement or sound, sometimes opens eyes
58
PVS
no cognitive function but has sleep-wake cycles
59
locked in syndrome
inability to move or respond except for eye movements (up and down not side to side) lesion in the pons!
60
GCS score interpretations
9-15: mild-mod injury 3-8: major injury
61
GCS eye
4- Spontaneous 3- Loud voice 2- Pain 1- None
62
GCS verbal
5- Normal conversation 4- Disoriented conversation 3- Non coherent 2- No words, only sounds 1- None
63
GCS motor
6- Normal 5- Localized to pain 4- Withdraws to pain 3- Flexion 2- Extension 1- None
64
changes in LOC can indicate what 7 things?
Hypoxia Hypercarbia Hypotension Drug related Hypothermia Postictal state Hypoglycemia
65
complications of change in LOC
resp distress/failure PNA aspiration pressure ulcers DVT contractures
66
fixed and dilated pupils
herniation syndrome
67
posturing in relation to PVS
high potential for PVS in pts who posture and have adequate perfusion and oxygenation
68
spastic muscles
generally accompanied by rigidity, muscle is in a state of contraction, muscle spasm may be present
69
decorticate (plantar, legs, arms, hands)
plantar flexed (outward) legs internally rotated arms flexed and adducted hands flexed BETTER PROGNOSIS
70
decerebrate (plantar and arms/hands)
plantar flexed (outward) arms adducted, extended, pronated, and hands flexed outward
71
DTRs and which is superficial
triceps, biceps, brachioradialis, patellar, and achilles tendon plantar is superficial
72
in who is Babinski normal
children <2
73
cushing's triad
Increased SBP with a widening pulse pressure Bradycardia Bradypnea
74
cushing's triad caused by?
increased ICP late sign of herniation syndrome
75
normal ICP
1-15 mmHg
76
herniation syndrome
Occurs when cerebral pressure is not exerted evenly One portion of the brain herniates into another Supratentorial and infratentorial Caused by cerebral edema or mass Neuro changes can be slow or rapid Call family to establish baseline
77
maintenance of clear airway for altered LOC patient
may be orally or nasally intubated, can cause accumulation of secretions which need to be removed frequent monitoring and lung sounds positioning to accumulate secretions and prevent obstruction HOB elevated 30, lateral or semi-prone suctioning, oral hygiene, CPT
78
how to protect eyes in pt with altered LOC
clean with saline-soaked cotton balls artificial tears cautious w eye patches because cornea may contact patch
79
fluid status and body temp with altered LOC
watch fluid status, turgor, INO, labs, IV and tube feedings adjust temp and cover pt monitor temp frequently
80
diarrhea may result from what 3 things
infection meds hyperosmolar fluids (TPN)
81
mood if patient arouses from coma
may have period of agitation (low stimulation)
82
monro-kellie hypothesis
Dynamic equilibrium of intracranial pressure Limited space in skull, so increase in any components causes change in volume of others compensation by displacing or shifting CSF increasing absorption or minimizing production of CSF minimizing blood volume
83
increased ICP causes what
decreased cerebral perfusion, ischemia, cell death, and further edema may result in herniation
84
autoregulation in the brain
the brain’s ability to change the diameter of blood vessels to maintain cerebral blood flow
85
decreased and increased CO2 in relation to blood vessels
decreased: constriction increased: dilation
86
early manifestations in increased ICP
changed in LOC change in condition Restlessness, confusion, increased drowsiness, increased respiratory effort, purposeless movements (loss of spontaneous movement), hemianopsia, lost taste for sweet and salty, pulse and pulse pressure changes Pupillary changes and impaired ocular movements Weakness in one extremity or one side Headache: constant, increasing in intensity, or aggravated by movement or straining
87
late manifestations of increased ICP
Respiratory and vasomotor changes Cushing triad: bradycardia/pnea, HTN Projectile vomiting Further deterioration of LOC Going from stupor to coma Hemiplegia, decortication, decerebration, or flaccidity Respiratory pattern alterations including cheyne-stokes breathing and arrest Loss of brainstem reflexes: pupil, gag, corneal, and swallowing
88
diabetes insipidus
Decreased secretion of ADH Excessive urine output Decreased urine osmolality Serum hyperosmolality Give IV fluids, electrolyte replacement and desmopressin (synthetic vasopressin)
89
SIADH fluid restriction
<800ml/day
90
interventions for increased ICP
resp status and lung sounds head in neutral position and elevated 0-60 to promote venous drainage avoid hip flexion, valsalva, abd distention, or stimuli monitor fluid status, I&O every hr in acute phase strict asepsis
91
craniotomy
opening of the skull
92
craniectomy
excision of portion of the skull
93
cranioplasty
repair of cranial defect with metal or plastic plate
94
burr holes
circular openings for exploration or diagnosis to provide access to ventricles or shunting procedures, aspirate a hematoma or abscess, or make a bone flap
95
preop meds for increased ICP
corticosteroids, fluid restriction, hyperosmotic (mannitol), diuretics antibiotics diazepam for anxiety
96
care of pt undergoing intracranial surgery
Assess dressing and for evidence of bleeding or CSF drainage from nares or intracranial drain, may look orange or yellow monitor fluid status and labs
97
how often to check VS and neuro for intracranial patients
every 15 mins to an hour
98
when does cerebral edema peak
24-36 hours
99
HOB to maintain cerebral perfusion
0-30
100
turning and repositioning/breathing intracranial patients
q2h use incentive spirometer humidify oxygen
101
interventions for intracranial pts
Monitor I&O, weight, blood glucose, serum and urine electrolyte levels, and osmolality, and urine specific gravity prevent infection by assessing incision site, CSF leak, don't do anything to increase ICP, asepsis!
102
causes of seizures
Cerebrovascular disease Hypoxemia Fever (childhood) Head injury HTN CNS infections Metabolic and toxic conditions Brain tumor Drug and alcohol withdrawal Allergies
103
assessment of HA
Include medication history and use Persons undergoing a headache evaluation require a detailed history and physical assessment with neurologic exam to rule out various physical and psychological causes Diagnostic testing may be used to evaluate underlying cause if there are abnormalities on the neurologic exam Find out about predecessors, new meds being used, history of drug abuse
104
meds for migraines and cluster headaches
abortive medications instituted as soon as possible with onset
105
heat and cold for what type of headache
heat for tension cold for migraine
106
CN I
olfactory (smell) offer patient something to smell
107
CN II
optic (vision) snellen chart with and without aids ask when patient sees you wiggling your fingers at side of their head ishihara plates for color blindness ask which finger is moving in each quadrant PERRLA
108
CN III
oculomotor (eyelid movement, pupil reflex, coordinated movement of eyes) 6 cardinal points in H pattern without moving head PERRLA look for failure to move, nystagmus, drooping, double vision
109
CN IV
trochlear (eyelid movement, pupil reflex, coordinated movement of eyes) 6 cardinal points in H pattern without moving head PERRLA look for failure to move, nystagmus, drooping, double vision
110
CN V
trigeminal (face sensation, corneal reflexes, chewing) sensory: cotton and pin near jawline, cheek, and forehead; cotton on cornea should make pt blink motor: open mouth against resistance or pretend to chew, left index finger on pt chin and strike with tendon hammer, slight protrusion of jaw
111
CN VI
abducens (eyelid movement, pupil reflex, coordinated movement of eyes) 6 cardinal points in H pattern without moving head PERRLA look for failure to move, nystagmus, drooping, double vision
112
CN VII
facial (face movement, taste, salivation) raise eyebrows, close eyes, keep closed against resistance puff cheeks and show teeth taste
113
CN VIII
vestibulocochlear/acoustic (hearing and balance) whisper test rinne test (tuning fork on mastoid and next to ear, ear should be louder) weber test: fork on center of forehead, should sound same in each ear
114
CN IX
glossopharyngeal (throat sensation, taste, movement of tongue for swallowing/gag reflex, phonation) swallow or elicit gag reflex phonation by listening to vocal sounds assess taste
115
CN X
vagus (throat sensation, taste, movement of tongue for swallowing/gag reflex, phonation) swallow or elicit gag reflex phonation by listening to vocal sounds assess taste
116
CN XI
spinal accessory (some neck movement and shoulder) shrug shoulders and turn head against resistance
117
CN XII
hypoglossal (tongue movement) stick out tongue and check for deviations
118
right sided CVA
left paralysis spacial difficulties impulsive behavior poor judgment time blindness
119
left sided CVA
right paralysis difficulty knowing left and right slow cautious movements impaired cognition dep and anxiety
120
warning signs of chemo toxicity
loss of appetite fatigue SOB
121
what med to give before chemo
benadryl corticosteroids
122
platinum based chemo
neuropathy!!
123
chemo and thrombocytopenia
noooo!
124
supportive care for palliative
Insertion of gastric feeding tube Placement of central venous access device Prophylactic surgical fixation of bones at risk for pathologic fracture
125
intrathecal chemo
injected in spinal column
126
Central vascular access device (VAD) administration
Placement in large blood vessels Frequent, continuous, or intermittent administration Can be used to administer other fluids (blood, electrolytes, etc.) Minimize discomfort and emotional distress! Avoid continuous punctures through peripheral lines
127
intraarterial regional chemo
Delivers drug through arteries supplying tumor
128
intraperitoneal regional chemo
Delivers drug to peritoneal cavity for treatment of peritoneal metastases with 1-2L of fluids for 1-4 hours then drained. Heated intraperitoneal chemotherapy (HIPEC), done for liver or colon cancer that metastasized into peritoneal cavity
129
intrathecal/ventricular regional chemo
Involves lumbar puncture & injection of chemo into subarachnoid space
130
intravesical bladder regional chemo
Agent added to bladder by urinary catheter and retained for 1 to 3 hours
131
acute chemo toxicity
Anaphylaxis, hypersensitivity, extravasation (comes out of the blood vessel and goes into tissue), anticipatory nausea, vomiting, dysrhythmias
132
delayed effects of chemo toxicity
Nausea and vomiting, mucositis (mucus membranes break down), alopecia, skin rashes, bone marrow depression, altered bowel function, neurotoxicities (especially with platinum-based chemo)
133
chronic chemo toxicity
Damage to heart, liver, kidneys, and lungs Can immediately develop after treatment and manifest months later SE can be in more than one category, long lasting ones can have effects on patient’s survival
134
platinum based chemo for what types
bladder, lung, testicular, and ovarian
135
topoisomerase inhibitors for what type of cancer
ovarian, colon, small cell lung
136
vinca alkaloids for what type of cancer
solid tumors leukemia, lymphoma, hodgkin lymphoma
137
antifolates for what type of cancer
sarcomas, carcinoma, ALL lymphoma, non-neoplastic like immunosuppression
138
treatment induced effects of chemo (bone marrow suppression)
monitor CBC, neutro, PLT, RBC, nadir in 7-10 days so space out chemo q2w, neutropenia, anemia, fatigue, thrombocyto, infection, sepsis, hemorrhage
139
treatment induced effects of chemo (mucosal lining disturbances)
stomatitis, mucositis, esophagitis, N/V/D/C, dysgeusia, hepatotoxicity)
140
treatment induced effects of chemo (skin changes)
erythema, flushing, hyperpigmentation acral erythema, alopecia rashes, dryness, finger and toenail discoloration peeling (macerated) skin caused by normal RBCs being destroyed Especially the ones rapidly proliferating like bone marrow, GI lining, bone, skin, and nails
141
when is nutritional counseling indicated after chemo
when 5% weight loss noted
142
diet for chemo pts
Soft, non irritating, high-protein & high-calorie foods should be eaten throughout the day. avoid extreme temps, tobacco, alcohol, spicy/rough foods nutritional supplements biweekly weights 10lb weight loss=hard to maintain nutritional status monitor albumin and prealbumin
143
causes for fatigue in chemo pts
anemia accumulation of toxic substances after cells are killed need for extra energy to repair and heal body tissue lack of sleep from drugs
144
pulmonary effects of chemo
immediate effects can be alarming because they mimic symptoms that precipitated the cancer diagnosis Pneumonitis, pulmonary fibrosis, pulmonary edema, hypersensitivity pneumonitis, interstitial fibrosis & pneumonitis produced by an inflammatory reaction or destruction of alveolar-capillary endothelium are all possible Cough, dyspnea, pneumonitis, pulmonary edema
145
treatment of pulmonary effects of chemo
bronchodilators expectorants/cough suppressants bed rest oxygen
146
primary cause of death of chemo
infection in lungs, GU, mouth, rectum, peritoneal, blood occurs from ulceration, compression of vital organs, and neutropenia
147
3 obstructive emergencies of chemo
SVC syndrome spinal cord compression third space syndrome
148
SVC syndrome and S&S
obstruction by tumor or thrombus by lung cancer, non-hodgkins lymphoma, and breast cx Facial and periorbital edema Distention of veins of head, neck, and chest Seizures Headache Confusion and disorientation mediastinal mass seen on chest xray
149
what increases risk of SVC syndrome and treatment
presence of a central venous catheter & previous radiation therapy to the mediastinum radiation therapy or chemo to site of obstruction
150
spinal cord compression and S&S
tumor in epidural space of spinal cord Intense, localized, persistent back pain Motor weakness Sensory paresthesia and loss Change in bladder or bowel function
151
third space syndrome, S&S, and treatment
Shifting of fluid from vascular to interstitial space Signs of hypovolemia including hypotension, tachycardia, low central venous pressure, and decreased urine output Treatment: replacement of fluids, electrolytes, and plasma protein hypervolemia can occur from treatment
152
5 metabolic emergencies by production of ectopic hormones
SIADH hypercalcemia tumor lysis syndrome septic shock DIC
153
SIADH
abnormal or sustained production of ADH cancer cells manufacture, store, and release it chemo agents stimulate release
154
SIADH S&S
Weight gain without edema, weakness, anorexia, N/V, personality changes, seizures, oliguria, decreased reflexes, coma, hyponatremia
155
SIADH treatment
Treat underlying malignancy & correct the sodium- water imbalance (fluid restriction, oral salt tablets or isotonic [0.9]) saline and IV administration of 3% sodium chloride solution. Furosemide (Lasix) may also be a helpful treatment in the initial phases. Demeclocycline (Declomycin) may be needed on an ongoing basis Monitor sodium level because sometimes SIADH…? I’m assuming SIADH causes hyponatremia
156
what 2 things contribute to or exacerbate hypercalcemia
immobility and dehydration
157
S&S of hypercalcemia
Apathy, depression, fatigue, muscle weakness ECG changes, polyuria, nocturia, anorexia, N/V
158
tumor lysis syndrome
rapid destruction of large numbers of cells increased serum phosphate causes decreased calcium can cause biochem changes resulting in renal failure can be fatal prevent renal failure and serious electrolyte imbalance usually 24-48h after chemo and lasts a week
159
hallmark signs of tumor lysis syndrome
hyperuricemia hyperphosphatemia hyperkalemia hypocalcemia
160
infiltrative emergencies in chemo pts
cardiac tamponade and cardiac artery rupture sometimes in head and neck cancer, risk
161
S&S of cardiac tamponade
heavy feeling in chest, SOB, tachycardia, coughing, difficulty swallowing, hoarse, perspiring a lot, uncomfy and anxious
162
cardiac artery rupture
Invasion of artery wall by tumor Erosion following surgery or radiation Bleeding can manifest as minor oozing or spurting of blood in case of a blowout pressure should be applied
163
surgical management of carotid artery rupture
ligation of the carotid artery above and below the rupture site and reduction of local tumor
164
what hormones do the renal system release (what do the hormones help with)
RBC production bone metabolism BP control
165
physical structure of ureters
wide then thinner, where stones get stuck
166
gerontological considerations of renal and urinary
GFR decreases (causing more effects of meds bc they can't excrete them as well) diminished osmotic stimulation (hypernatremia and FVD) structural (tumor) or functional changes to bladder (incontinence or BPH) FEWER NEPHRONS
167
lining of bladder is?
VERY VASCULAR
168
hallmark sign of upper UTIs like pyelonephritis
fever or chills
169
when would a patient be on a long term catheter
paralyzed, BPH, PVS or coma, MS, any neuro problem that interferes with message telling brain “I have to void”
170
how do drugs affect urine output
tobacco/nicotine vasoconstricts, alcohol suppresses ADH
171
nephrotic syndrome is what kind of defect
structural
172
gout and urinary
buildup of uric acid crystals, very painful when they lodge in a joint, can get it anywhere
173
kidney pain physical exam and clini manifestations
CVA tenderness (dull, severe, sharp, colicky) N/V, diaphoresis, shock -> obstruction, stone, blood clot, pyelonephritis, trauma
174
bladder pain physical exam and clini manifestations
suprapubic (intense with voiding and full bladder) urgency, pain at the end of void, straining ->infection, tumor, interstitial cystitis, overdistended bladder
175
uteral pain physical exam and clini manifestations
CVA, flank, lower abd, labium (severe, sharp, stabbing, colicky) N/V, paralytic ileus -> ureteral stone, stricture, blood clot
176
prostate pain physical exam and clini manifestations
perineum, rectum (vague discomfort, feeling of fullness, back pain) suprapubic tenderness, obstruction, hesitancy, frequency, urgency, nocturia -> prostate ca, prostitis
177
urethral pain physical exam and clini manifestations
MALES: from penis to meatus FEMALES: urethra to meatus frequency, urgency, dysuria, nocturia, urethral discharge -> infection, irritation of bladder neck, foreign body
178
interstitial cystitis
not a bladder infection, inflammation of lining of bladder that comes and goes, resembles infection but cultures are (-). Bleeding and pain when urinating, looks like pyelonephritis. Treated with steroids.
179
where is the labium
right above the hip
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frequency
urinating > q3h
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hesitancy
difficulty initiating voiding
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enuresis
involuntary voiding during sleep
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oliguria
urine output <500 ml/day
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anuria
urine output < 50ml/day
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encopresis
stool at night
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urine specific gravity range
1.010-1.025
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normal serum creatinine
0.6-1.2
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normal BUN
7-18 8-20 in pts >60
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cystoscopic examination
metal rod with clip at the end goes in penis for biopsy leads to hematuria
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interventions before urinary tract testing
use correct terminology in a way pt will understand encourage fluid intake unless contra reduce discomfort like sitz baths analgesics and antispasmodics assess voiding and hygiene
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pyridium
antispasmodic turns urine orange/red very potent same as uristat but higher concentration
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postop management of kidney surgery
Potential hemorrhage and shock Widening pulse pressure HR increases then decreases Pallor Nausea potential abd distention from bleeding and paralytic ileus (use blakemore sengsten tube [black and weighted]) potential infection and thrombus that can turn into PE
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complications of kidney surgery
bleeding , pneumonia, infection, and DVT
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drains after kidney surgery
pt goes home with them hand suction on JP drain, should first be blood, then serosang, then sero tubes sutured in
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causes of uro and nephrolithiasis
hyperparathyroidism renal tubular acidosis cancers granulomatous (sarcoid, TB) increase Vit D Excessive Milk and dairy myeloproliferative disease
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diagnosis of uro an nephrolithiasis
x-ray, blood chemistries, and stone analysis; strain all urine and save stones
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possible reasons for stone development
-deficiency of citrate, Mg, nephrocalcin, and uropontin (prevents crystallization) -dehydration (binging alcohol) -certain conditions like infection, urinary stasis, periods of immobility -increased calcium concentrations in blood and urine
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Causes of Hypercalcemia and Hypercalciuria
hyperparathyroidism renal tubular acidosis cancers granulomatous (sarcoid, TB) increase Vit D Excessive Milk and dairy same as causes of uro and nephrolithiasis
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med management of kidney stones
allow stone to pass pain management hot bath or moist heat to flank ten 8oz glasses of water a day urine output 2L/day strain urine NO OPIOIDS, use toridol (antiinflammatory)
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gerontologic consideration for kidney stones (temperature)
normal looking temperature like 99 is bad normally they’re a bit colder like 97 so 99 can be sepsis not good outcome
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dietary teaching for uric stones
low purine diet, no shellfish, mushrooms
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dietary teaching for cystine stones
low protein
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dietary teaching for oxalate stones
no spinach, strawberries, rhubarb, chocolate, tea, peanuts, and wheat bran
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bladder cancer risk factors
>55 male smoking (tobacco) environmental carcinogens recurrent or chronic UTIs high urinary pH bladder stones high cholesterol pelvic radiation therapy cancers of nearby areas
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S&S of bladder cancer
Visible painless hematuria (most common symptom) Frequency and urgency secondary to infection Any alteration in voiding pattern Pelvic or back pain with metastatic
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surgery for bladder cancer
transurethral resection or fulguration, cystectomy (simple or radical) Conduit to bypass bladder (connects kidneys to skin)
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urinary diversion reasons
Bladder cancer or other pelvic malignancies Birth defects, trauma, strictures, neurogenic bladder, chronic infection or intractable cystitis Used as a last resort for incontinence
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types of urinary diversion
Cutaneous urinary diversion: ileal conduit, cutaneous ureterostomy, vesicostomy, nephrostomy Continent urinary diversion: Indiana pouch, Kock pouch, ureterosigmoidostomy
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pt teaching of urinary ostomy
changing appliance, controlling odor, managing the ostomy appliance, cleaning and deodorizing the appliance, continuous care
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BPH manifestations
those of urinary obstruction, urinary retention, and urinary tract infections (due to urinary stasis)
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treatment of BPH and SE
alpha-adrenergic blockers, alpha- adrenergic antagonists, antiandrogen agents (-zosins) Finasteride Catheterization if unable to void Prostate surgery antiadrogen causes gynecomastia and facial feminization
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risk factors of BPH
Estrogen and testosterone play a role smoking, Etoh, obesity, sedentary lifestyle, HTN, CVD, DM, Western diet; increase fat, animal protein, refined carb
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S&S of BPH
Frequency, urgency, nocturia, hesitancy, decreased force of stream, incomplete void, straining, dribbling, urinary retention, recurrent UTI fatigue, anorexia, nausea, vomiting, pelvic discomfort
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diagnosis of BPH
voiding diary health/family hx DRE: large, rubbery, non-tender, gloved hand goes in to feel for prostate PSA: prostatic specific antigen American urological association scoring to help grade severity of symptoms UA, culture Ultrasound
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management of BPH
Meds: alpha-adrenergic blockers alfuzosin, terazosin 5-alpha-reductase inhibitor finasteride saw palmetto (can mask serious problem) little data to support but often taken by pt. Minimally invasive: transurethral microwave heat treatment (TUMT) transurethral needle ablation (TUNA)
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surgery for BPH
Transurethral resection of prostate (TURP) watch catheter lots of bleeding, should look pinkish but not red red if clots r there, should be small enough to pass
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three way system for bladder irrigation
irrigating bladder wall, VERY vascular so try to minimize bleeding
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risk factors of prostate cancer
increasing age, familial predisposition, and African-American race
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manifestations of prostate cx
Early disease has few/no symptoms Symptoms of urinary obstruction, blood in urine or semen, painful ejaculation Symptoms of metastasis may be the first manifestations Back pain usually brings person in to hospital EARLY SCREENINGS VITAL
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prostate cx treatment
Watchful waiting Radiation Therapy Brachytherapy: putting radioactive seeds into the site Hormonal strategies Chemotherapy Regional: radioactive liquid in bladder, must hold it for 1.5-2 hours. Very uncomfortable because they want to urinate Surgical- Prostatectomy Radical prostatectomy Cryosurgery TURP
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S&S of prostate cx
signs of obstruction: Difficulty and frequency of urination Urinary retention Decrease force of stream Painful ejaculation (prostate, not urinary!) Blood in urine or semen Hematuria if cancer of urethral or bladder signs of metastasis: back or hip pain, anemia, weight loss, spontaneous fractures
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assessment of prostate cx
DRE: hard, fixed (not mobile) stony prostate Elevated PSA or velocity of PSA Usually presents with LUTS (lower urinary s/s) Ultrasound, needle biopsy Bone scan, MRI to look for mets
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complications of prostate cx
Hemorrhage and shock Infection DVT Catheter obstruction Sexual dysfunction
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what to watch w prostate cx surgery
fluid balance!
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what do bladder spasms cause in prostate surgery pts
feelings of pressure and fullness, urgency to void, and bleeding from the urethra around the catheter use meds and warm compresses/sitz baths analgesics walk dont sit! prevent constipation irrigate catheter
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pt teaching after prostate surgery
Sometimes clamp off bladder so urine builds up, helps them regain sensation Information that regaining control is a gradual process (dribbling may continue for up to 1 year depending upon type of surgery) perineal exercises avoid straining, heavy lifting, and long car rides for 6-8w fluids! no coffee, alcohol or spice urologist for sex questions
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testicular cancer risk factors
undescended testicles, positive family history, cancer of one testicle, Caucasian-American race
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manifestations of testicular cancer
painless lump or mass in the testes (painful is infection)
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treatment of testicular cancer
orchidectomy, retroperitoneal lymph node dissection (open or laparoscopic), radiation therapy, chemotherapy
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Nitrates in urine
E.coli in pyelonephritis
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How long should u fast before cerebral angio
8-10h
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Hyperuricemia value
>7
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Hyperphosphate level
>4.5
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3 meds to dry secretions when dying
Scopolamine Glycopyrrolate Hyoscyamine
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How to treat acute hypercalcemia
Hydration (3 L/day) and bisphosphonate therapy
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Tumor lysis syndrome med
use allopurinol/zyloprim to decrease uric acid