exam 2 Flashcards

(276 cards)

1
Q

hypoxemia assessment findings

A

Tachypnea
Pallor
cyanosis
Respiratory distress signs
weak peripherial pulses

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2
Q

Resp distress symptoms

A

retractions, nasal flaring, grunting, head bobbing, restlessness, stridor, wheezing, rales

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3
Q

First sign of resp illness?

A

Tachypnea

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4
Q

hypoxemia management

A

-Provide O2 at the lowest liter flow that corrects the hypoxemia
-Less than 91% requires nursing intervention
-Less than 86% is a life-threatening emergency
-Chest physiotherapy–> promotes mucus clearance by mobilizing secretions
-Suctioning–> always suction the mouth before the nose in kids–> do not want to increase risk of aspiration pneumonia

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5
Q

Hypoxemia priorities of care

A

o2 therapy
pulse ox
CPT
suctioning

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6
Q

What is Aerosol therapy?

A
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7
Q

Physical findings of CF (GI)

A

-decrease pancreatic enzymes=abd distention and thick mucous
-meconium ileus at birth –>difficulty passing stool –> vomiting
-steatorrhea
-FTT
-Vitamin A, D, E, K deficiency
-PMH of respiratory infections

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8
Q

diagnostic for CF

A

Sweat chloride test
Sodium higher than 90

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9
Q

SCT indication of CF?

A

> 40 in infants (less than 3mo)
->60 for all ages

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10
Q

CF management (pulmonary)

A

-airway clearance therapy
-CPT
-aersol therapy

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11
Q

How does dornase alfa help alleviate sx of cystic fibrosis?

A

-decreases viscosity of mucus and bronchodilators

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11
Q

What medication is given with cystic fibrosis to treat pulmonary sx?

A

dornase alfa

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12
Q

Croup assessment findings

A

-barking cough
-inspiratory stridor
-tachypnea
-sudden onset at night
-self limiting (resolves on its own)
-lasts 3-5 days

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13
Q

priorities of croup

A

-educate families on sx
hospitalization for significant stridor at rest or severe retractions
-cool mist/steamy bathroom
O2—>ox continuous
-hydration

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14
Q

Racemic epinephrine

A

decrease edema, effects last up to 2 hours and sx may worsen requiring another tx

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15
Q

Chronic Asthma medications

A

Long-acting bronchodilators/B2-adrenergic Agonist (formoterol) Inhalation corticosteroid (Fluticosone) Mast-cell Stabilizer (Cromolyn) Leukotriene Receptor Antagonists (Montelukast

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16
Q

s/s of asthma

A

hacking, nonproductive cough, dyspnea, chest tightness, wheezing or crackles
A SILENT CHEST IS A OMNIOUS SIGN (no airmovement)

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17
Q

Status asthmaticus

A

a prolonged severe asthma attack uncontrolled by typical regimen. LIFE THREATENING

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18
Q

Status asthmaticus symptoms

A

wheezing or lack of air movement in lungs
labored breathing
accessory muscles
hypoxia
diaphoresis

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19
Q

Status asthmaticus Nursing actions/ Priority of care

A

Cardioresp monitoring, O2, ABG’s
-admin bronchodilators and anti-inflammtory meds
-prepare for intubation

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20
Q

Status asthmaticus medication management

A

Theophylline
Mg sulfate Iv
Heliox
Ketamine

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21
Q

Theophylline

A

anti inflammatory and reverses corticosteroid resistance

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22
Q

Theophylline has a risk for toxicity so we have to frequently monitor?

A

Blood levels

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23
Q

Mg sulfate

A

relaxes bronchial muscles, expanding airways

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24
Heliox
mix of Helium and O2---> decreases airway resistance
25
Ketamine
smooth muscle relaxant
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Epiglottis assessment findings
rapid onset (within hours): High fever and toxic appearance unable to whisper or speak Lateral neck x-ray +Epiglottis -Tripod position neck thrust forward. anxiety -drooling and dysphagia !!! NO COUGH
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Management of Epiglottis
Focuses on airway maintaince, icu admission iv abx -100% NRB mask.
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Priorties of Epiglottis
-Never attempt to visualize the throat -No supine position -need HOB elevated.
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Foreign Body Aspiration assessment findings
sudden onset of cough, wheezing, stridor: upper airway Unilateral BS (wheezing & decreased in R/clear in L)
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Foreign Body Aspiration Risk factors
common in ages 6 months-3 years, upper and lower resp.
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Foreign Body Aspiration parent education
keep coins, small batteries, latex balloons out of reach -no popcorn, peanuts until age 3, chop all foods into pieces
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Where are most objects aspirated?
right mainstream bronchus because it is at a less acute angle than the left mainstream bronchus.
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Dehydration assessment findings
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Dehydration Management: oral rehydration
sunken fontanells decreased LOC sunken eyes no tears dry mm sudden weight loss decreased UOP delayed cap refill
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dehydration findings vitals
-electrolyte imbalances -increased hr -increased rr -decreased bp
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Rehydration Therapy Oral
attempt 1st. Pedialyte mild: 50ml/kg in 4 hours moderate: 100ml/kg in 4 hours Diarrhea losses: 10ml/kg for each stool
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Rehydration IV
20ml/kg of Ns or LR bolus (in addition to maintenance)
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Maintenance fluid
100ml/kg--1st 50ml/kg-2nd 20ml/kg for remaining. 24 hours--->24/24
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G.E.R.D. managements
Conservative: small frequent feedings, thickened feedings, meds Surgical: nissan Meds: PPI/ h2 receptor antagonist
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G.E.R.D. pt education
sit up after meals
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G.E.R.D s/s
arching of head an neck during feeding, frequent vomitting, irritability during feeding, wet burps, apnea, ALTE
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When should patients with G.E.R.D take their medications?
30 minutes before feeding
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PPI's include
esomprazole, lansoprazole, omeprazole, pantoprazole
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H2-receptor-antagonist include
ranitidine, cimetidine, famotidine
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What will labs show if patient has GERD?
CBC may show anemia Hemoccult: positive for blood if chronic esophagitis
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complications of GERD
laryngitis, recurrent PNA, asthma, apnea or ALTE
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What is ALTE
apparent life threatening event
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apparent life threatening event is?
a sudden event where the infant exhibits apnea, change in color, change in muscle tone and choking
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physical cues in children
heartburn, abd pain, diff swallowing, chronic cough, non cardiac Cp emesis with blood or bile
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physical cues in infants
arching of head and neck during feeding, irrtiability frequent spitting up or vomitting. Resp issues, FTT, apnea or ALTE
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what is HPS and when does it occur?
Hypertrophy of the pylorus muscle causing gastric outlet obstruction. usually in 1st 3-6 weeks of life
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s/s of HPS
foreceful projectile vomitting hunger soon after vomitting weight loss olive shaped movable mass in RUQ
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Labs for HPS
Hypochloremia hypokalemia metabolic alkalosis
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Management for HPS
Laproscopic surgery IVF NGT
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Priority care of HPS
Iv fluids--> correct electrolyte and dehydration NGT---? decompression NPO, strict I/O -Wound care Resume PO feedings in 1-2 days
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Hirschsprung's expected findings newborn
failure to pass meconium, bilious emesis abd distention
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Hirschsprung's expected findings infant/child
FTT, chronic constipation
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Treatment of Hirschsprungs
4 phase surgery with colostomy
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Management of Hirschusprungs
high protein high cal diet accurate I/O iv fluids and abx post op teach about stoma care
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What should we observe for in Hirschusprungs?
entercolitis
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S/s of entercolitis
fever, vomitting, abd distention, explosive diarrhea or rectal bleeding. NOTIFY IMMED
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tx of entercolitis
Broad spectrum abx, IVF resuscitation and rectal washout
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Name of surgery for Hirshusprungs
resection and re-anastamosis: done in stages depening on degree of colitis/bowel dilation
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What is intussusception
proximal segment of bowel telescopes into a distal portion of bowel. Results in lymphatic and venous obstruction
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s/s Intussusception
flare then resolves spontaneous reduction sudden onset of crampy or severe adb pain, draws knees to chest. v/d
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Hallmark sign of Inutssusception
sausage shaped mass in upper mid abdomen. Red current jelly stools
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Treatment of Intussusception
IVF/NGT air enema to correct or surgery if recurrent
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What do stools look like in Intussusception?
blood and mucus (red current jelly)
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What is important with intussusception?
keep patient NPO
70
Cleft lip and palet pre op
priority: nutrition and infant parent bonding -burp infant to expel excess air -encourage therapeutic techniques
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cleft lip and palet post op
Priority: preventing injury to suture line position on side of supine immediately post op with arm restraints -avoid straw, spoon anything in mouth -prevent crying
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Risk factors for Cleft
smoking prenatal infection folate deficiency antidepressants and steroid use
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Complications of Cleft
Feeding difficulties Regurgitation Altered Dentition Delayed/altered speech Otitis media/effusion
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What is effusion?
fluid build up in the middle ear.
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What is heart failure
inability of heart to adequately pump blood to meet metabolic and physical demand of the body
83
L sided heart failure symptoms
increased work of breathing, tachypnea, wheezing, rales, cough, DOE, feeding difficulties
84
Right sided heart failure symptoms
hepatomegaly, edema, JVD, periorbital edeam, wt gain
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Pathophysiology of coarctation of the aorta (CoA)
-narrowing of the aorta that occurs most often near or beyond the PDA (patent ductus arteriosus)
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During CoA, pressure increases near the defect and distal to it. What are assessment findings because of this?
Increased BP in upper extremities and decreased BP in lower extremities
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What is most important to assess when suspecting a patient has CoA
all pulses
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Key assessment findings for CoA
-full bounding pulses in upper extremities -weak or absent pulses in lower extremities -soft or moderately loud systolic murmur at base of left axilla -frequent epistaxis ; leg pain with activity (older child)
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Diagnostics for CoA
-echo: assess extent of narrowing and collateral circulation -CXR: left sided cardiomegaly, rib notching -CT, MRI, ECG: PRN
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Treatments for CoA
Infants and children: balloon angioplasty Adolescents: stents Surgical: repair of defect in children <6 months
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What is ventricular septal defect (SVD)
-most common congenital heart defect -Acyanotic heart failure
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Pathophysiology of VSD
-opening between the ventricles causing left to right shunt -incrased blood flow to RV --> increased blood flow to lungs --> pulmonary artery HTN, right ventricular hypertrophy
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complications of VSD
-aortic valve regurgitation and endocarditis
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Physical assessment findings for VSD
-most children asymptomatic -palpable thrill in chest -CHF symptoms -HOLOSYSTOLIC HARSH MURMUR (big one) heard alone the left sternal border
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What type of CHD is Patent Ductus Arteriosus (PDA)
cyanotic (increased pulmonary flow)
96
Assessment findings of PDA?
-depends on size of opening -tachycardia -diastolic BP typically low due to shunting -bounding peripheral pulses (from increased CO) -widened pulse pressure (> 30 mmHg) -hypoxia/ resp distress (due to pulmonary edema) -harsh, continuous, machine-like murmur (loudest under left clavicle at 1st/2nd ICS)
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Treatment for PDA
Non surgical: admin of indomethacin, insertion of coils to occlude the PED, diuretics, extra calories for infants Surgical: thoracoscopic repair (ligate vessels)
98
What is the 'fatal four' found in Tertralogy of Fallot
1. ventricular septal defect (VSD) 2. pulmonary stenosis (R to L shunt) 3. hypertrophy of right ventricle 4. overriding aorta (hypoxemia)
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Clinical features of tetralogy of fallot
-fainting, difficulty breathing, easy fatigue, color changes w/ feeding, crying, activity -loud, harsh systolic murmur -polycythemia -TET spells (blue baby) especially in AM
100
Specific symptoms of TET spells found in Tetralogy of Fallot
-cyanosis -hypoxemia -dyspnea -agitation -all the signs lead to anoxia and unresponsiveness
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treatment of tetralogy of fallot
-prostaglandins (to keep PDA open to increase pulmonary blood flow) -surgical repair of R ventricular outflow obstruction and VSD closure
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Nursing interventions for tetralogy of fallot
-place infant or child in knee-to-chest position or squatting -supplemental oxygen -administer morphine sulfate -supply IV fluids -administer propranolol
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Is Kawasaki's disease acquired or congenital
acquired (caused by unknown infectious organism)
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Assessment findings for Kawaski's disease
-High fever 103 F for at least 5 days unresponsive to ABX -bilateral conjunctivitis (without exudate) -dry mouth and throat, fissured lips, strawberry tongue, pharyngeal/ oral mucosa edema -cervical lymphadenopathy -desquamation (peeling) of fingers, toes and perineal areas; rash over body -CV: tachycardia, gallop, murmur; note hyperdynamic precordium
105
Nursing management of Kawaski disease
-administer IVF, IVIG, and PO fluids as ordered -Acetaminophen (fever) , cool cloths -DW , strict I&O -rest and quiet, family support -lip lubricants and mouth care -clear liquids and soft foods
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Medication management for Kawasaki
-Immunoglobulin (IVIG) -high dose IV (2g/kg) over 8-12 h -High Dose aspirin -80-100 Mg/Kg/day q 6 h -follow w/ low dose after fever breaks -indefinite use if aneurysms develop -additional anti-coag if large aneurysm
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What medications are given for heart failure?
Metoprolol Lasix: used to manage edema Captopril/ Enalopril Digoxin
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Nursing considerations and side effects of metoprolol
-Monitor HR and BP b4 administering -SE: dizziness, hypotension, HA
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Nursing actions and side effects of Lasix
-Monitor for hypokalemia, monitor BP, monitor I & O, monitor DW -SE: hypokalemia, NV, dizziness, ototoxicity
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Nursing actions for ace-inhibitors
-monitor BP before and after administration
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Nursing actions for digoxin?
-count apical pulse 1 full minute -HOLD IF: <90 infant, <70 child, <60 adolescent -monitor digoxin levels (0.8-2)
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What are signs of digoxin toxicity?
-N/V, anorexia, bradycardia, dysrhythmias -green yellow halos
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Digoxin antidote
-digoxin immune fab
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Indications of metoprolol
-decrease HR and BP and promote vasodilation
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Indications of Lasix
manage excess fluid and sodium
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Indication for captropril
reduce afterload by causing vasodilation --> decreased pulmonary and systemic vascular resistance
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Indication of digoxin
decrease contractility of heart muscle
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What is the pediatric assessment triangle?
addresses Childs appearance, work of breathing, and circulation to skin
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Examples of appearance the nurse should assess for in the pediatric assessment triangle
-abnormal tone -decreased interactiveness -decreased consolability -abnormal look/ gaze -abnormal speech / cry
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Examples of work of breathing the nurse should assess for in the pediatric assessment triangle
-abnormal sounds -abnormal position -retractions -flaring -apnea/gasping
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What is the purpose of Aspirin to treat Kawasaki disease?
prevent clots
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Why are clients wit kawaskis disease at risk for dehydration
-painful mouth makes them less likely to drink -already losing fluids from fever
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What is acute rheumatic fever?
-Group A beta hemolytic strep (GAS) ; strep throat
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clients with acute rheumatic fever may report a history of?
-sore throat and pharyngitis within past 2-3 weeks -recent URI -recurrent skin infection -fever -joint pain
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What children are likely to acquire acute rheumatic fever
-children who have less access to healthcare
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Minor manifestations of acute rheumatic fever
Fever, arthralgia, increased ESR, prolonged PR interval
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Major manifestations of acute rheumatic fever
-carditis (w/ valvulitis) -polyarthritis (multiple joints inflamed/ migratory) -subcutaneous nodules (firm and painless nodules over knees, wrists, elbows) -chorea (jerking muscle movements in face hands and feet) -erythema marginatum (rash on trunk and extremities)
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Diagnostic criteria for acute rheumatic fever includes?
-presence of 2 major manifestations OR -presence of 1 major and 2 minor
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Sinus tachycardia characteristics
fever, pain, fear, fluid loss or hypoxia.
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sinus tachycardia in infant characteristics
rate is <220(160-220)
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sinus tachycardia management
Focus on underlying cause -INAPPROPRIATE and dangerous to treat with medications or defib aimed at decreasing heart rate
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Sinus tachycardia characteristics in children
<180 (130-180)bpm with a beat-to beat variability, p wave present and normal QRS normal
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SVT characteristics
infants: >220bpm Children >180 with onset of termination, p wave is flattened, and QRS is normal
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SVT management Compensated
Vagal maneuvuers such as ice to face of blowing through a straw that is obstructed -adeosine if vagal maneuver fails.
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Sinus bradycardia characteristics
No cardiac nodal abnormality p-wave and qrs remain normal on ecg
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What causes SVT?
re-entry problem in the cardiac conduction system or genetic abnormalities like WOLFF-Parkinson, WHITE syndrome, or with meds like caffeine or theophylline
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Sinus bradycardia <60
life threatening altered perfusion hypoxia shock respiratory compromise
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Sustained bradycardia is commonly associated with ___ and is a ___
arrest, ominous sign
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SVT management uncompensated
adenosine or synchronized cardioversion
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post-streptococcal glomerulonephritis history
current or past history of recent pharyngitis/strep throat or skin infection
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Compensated SVT s/s
tachycardia, Heart rate >220 Abnormal p waves alert well perfused child ha, dizzy in older child
137
post-streptococcal glomerulonephritis physical findings
fever, lethargy, HA, decreased urine output, abd pain, vomitting, and anorexia Htn edema, urine for gross hematuria
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Uncompensated SVT s/s
Tachy, hr>220 abnormal p-waves signs of shock: aloc, poor perfusion, weak pulse.
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post-streptococcal glomerulonephritis labs and dx
urine dip stick: hematuria and proteinuria Bun/ Creatinine: increase ESR: increased ASO Titer: increased
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post-streptococcal glomerulonephritis priority of care
-Monitoring fluid status and managing hypertension -Maintain sodium and fluid restriction during acute phase -Daily weights -Urine output improvement of color -skin break down -bed rest
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meds for post-streptococcal glomerulonephritis
antihypertensives: nifedipine, labetalol diuretics
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What should we educate our clients and family to do for post-streptococcal glomerulonephritis?
monitor OPand BP and follow diet restrictions
141
What is post-streptococcal glomerulonephritis?
inflammation resulting in altered GF function (decreased GFR )
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Hemolytic uremic syndrome history
Recent episode of acute gastroenteritis watery diarrhea accompanied by cramping that becomes bloody over several days. Possible vomitting
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Hemolytic uremic syndrome assessment findings
pallor and toxic appearance edema and oliguria or anuria Neuro: irritability, altered LOC, seizures, posturing or coma.
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UA of post-streptococcal glomerulonephritis
proteinuria, hematuria, leukocytes, casts
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What does the urine look like in post-streptococcal glomerulonephritis?
tea color, cola color or dirty green color
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CBC of HUS
CBC: mod-sev anemia mild-sev thrombocytopenia,
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What kind of edema do they have in post-streptococcal glomerulonephritis?
general or periorbital, signs of fluid overload or CHF.
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Chem panel of HUS
decreased sodium, increased potassium, increased PO4 Hyponatremia/ Hyperkalemia
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ABG of HUS
metabolic acidosis
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Patient education of hus
prevention measures: handwashing after everything bahahha cook to 155
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Priority of care HUS
maintaining fluid balance, managing hypertension, acidosis, electrolyte abnormalities
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Continued priorities of care for HUS
PRBC and platelets (only for active bleeding or severe thrombocytopenia. IVIG may be considered REPORT all abnormal findings
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What are the three features of hemolytic uremic syndrome?
hemolytic anemia, thrombocytopenia, acute renal failure
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Hydrocele
fluid in scrotal sac: usually resolves by 12 months of age: transilluminate
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Varicocele
venous varicosity along the spermatic cord
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What are other assessments from the history we should pay attention to for HUS?
ingestion of ground beef visits to water park public pool petting zoo all prior to developing diarrhea
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What does the scrotal look like with varicocele?
worm like
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What can varicocele cause?
low sperm count and infertility
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Management for Compensated SVT
-vagal maneuvers: ice on face / blow through obstructed straw -Adenosine if maneuvers fail
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Uncompensated SVT management
-adenosine or synchronized cardioversion
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If we notice our client has sudden weight gain, what is our priority nursing action?
-assess height and weight and compare -take daily weights
158
If our client has difficulty eating, what questions should we ask the parent?
Onset of sx, how much they eat, if they tire easily, if there are color changes present
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Nursing actions with difficulty feeding?
1. limit feedings to 20 min --> remainder via OG / NG 2. cut cross in nipple 3. semi-upright position
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Interventions if our client has a decreased activity level?
-cluster care, frequent rest periods, bathe PRN
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If our client has decreased # of wet diapers, what nursing assessments should we perform?
1. I and O 2. edema 3. pulses 4. organomegaly
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Nursing actions for decreased number of wet diapers
I and o's, medications
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What current/ past Hx will be seen with glomerulonephritis?
-recent pharyngitis/ strep throat or skin infection
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What are physical assessment findings of glomerulonephritis
-fever, lethargy, HA, decreased UOP, abd pain, vomiting, anorexia
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Will blood pressure be high or low in glomerulonephritis
High --> have HTN
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Treatment of hydrocele and varicocele?
surgery to correct spontaneous resolution
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Glomerulonephritis causes what kind of edema?
-general or periorbital -other sx of fluid overload or CHF
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Management of hydrocele?
watchful waiting, to observe for spontaneous resolution
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What color is urine in glomerulonephritis
-tea color -cola color -dirty green color
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Management of Varicocele
if no resolution or decreased testicular volume, urologist referral for possible surger
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What will be seen on a urine dipstick that indicate glomerulonephritis
-hematuria, proteinuria
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Important teaching for Hydrocele and Varicocele
Hydrocele does not interfere with fertility but VARICOCELE can if left untreated.
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BUN, creatinine, and ESR will be elevated or decreased in glomerulonephritis?
elevated
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Hydrocele physical cues
enlarged painless scrotum decreases in size when lying down. Transilluminate with light source. Fluid renders testes impalpable
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What is the priority of care for glomerulonephritis?
-monitoring fluid status and managing HTN
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Varicocele physical cues
mass on one or both sides, blueish in color, worm-like spermatic veins and pain with palpitation
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What should be restricted in glomerulonephritis during the acute phase
sodium and fluids
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What is nephrotic syndrome?
kidney filtration disorder where too much protein (albumin) is filtered out of the blood due to damaged basement membrane of the renal glomerulus
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Nursing interventions during glomerulonephritis also include?
-DW -monitor urine output and color -monitor skin breakdown -monitor for renal/ neuropathy changes -maintain bed rest and cluster care
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Nephrotic syndrome most common in?
children (minimal change nephrotic syndrome with onset of age by 6.
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What precautions should children with glomerulonephritis be placed on
seizure precautions
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Nephrotic syndrome history
Periorbital edema upon waking that progresses to generalized edema throughout the day FROTHY URINE
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What medications are used to treat glomerulonephritis
-ABX for strep -nifedipine and labetalol -diuretics
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Nephrotic syndrome physical cues
irritability or fussiness skin stretched, tight appearance, pallor or breakdown heart and lung sounds related to fluid overload abd distention and note ascities
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What are the three features of hemolytic uremic syndrome (HUS)
-hemolytic anemia, thrombocytopenia, acute renal failure
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Nephrotic syndrome nursing management
I/O daily weights, urine protein, sodium restriction when edematous. Prevent infection: can cause relapse.
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What bacteria causes hemolytic uremic syndrome?
e-coli
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Nephrotic syndrome labs
Proteinuria hyperlipidemia edema
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what happens in the kidneys causing hemolytic uremic syndrome?
RBC are destroyed and fibrin clots are formed clogging the kidneys
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Medications for Nephrotic syndrome
Diuretics (furosemide), may require K replacement Corticosteroids (prednisone) take with meals for 4-6 weeks then taper over 2-5 months
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What may our clients say in their recent hx that will pinpoint hemolytic uremic syndrome?
-recent acute gastroenteritis -watery diarrhea -cramping -becomes bloody -possible vomiting -ingestion of ground beef, visits to water park/public pool/ petting zoo prior to developing bacteria
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Side effects of Prednisone
increased appetitie, weight gain, cushings features, mood swings.
179
Physical assessment findings of hemolytic uremic syndrome?
-pallor and toxic appearance -edema, HTN, oliguria, anuria -jaundice and weakness -dark colored urine -petechiae, ecchymoses, hematuria, hematoemesis, splenomegaly
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Neuro status with hemolytic uremic syndrome will be
-irritable -altered LOC -seizures -posturing and coma
180
What is Enuresis ?
Bedwetting: continued incontience beyond the age of toilet training
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What is hydrocele?
-fluid in scrotal sac -benign, common in newborns -usually resolves by 12 months
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Primary enuresis
never achieved extended dry periods
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What is varicocele?
-venous varicosity along the spermatic cord -most common in adolescence -can cause low sperm count or reduced motility --> infertility
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Secondary enuresis
Onset after a period of urinary continence
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Physical cues of hydrocele?
-enlarged painless scrotum -size decreases when lying down -testes impalpable -transilluminate with light source
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Enuresis nursing assessment
daytime or nighttime incontience urine holding behaviors (squatting dancing staring, rushing to bathroom)
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Physical cues of varicocele?
-mass on one or both sides -blueish in color -worm like veins -pain with palpation
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Enuresis history
note history of constipation toilet training history family disruption and stress and excessive family demands regarding TT. Large amounts of fluid before HS, caffiene? night time routine
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Management of hydrocele
-'watchful waiting' to observe for spontaneous resolution -surgical correction if no resolving on own
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Enuresis interventions
Assist in management of incontinence and promote wellness and prevent complications
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Management of varicocele?
-if no resolution or decreased testicular volume urologist referral -possible varicocelectomy
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Enuresis parent education
restrict fluids 2 hours before bedtime void before going to bed include child in bed linen changes. avoid pull ups.
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What is nephrotic syndrome?
too much protein (mainly albumin) is filtered out of the blood due to damaged membrane of glomerulus
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Medications for Enuresis
Antiduretic (desmopressin)
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Nephrotic syndrome is usually idiopathic which means?
it occurs on its own ; usually occurs by age 6
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Desmopressin for Enuresis
pill or melt away at bedtime hold for diarrhea or vomiting adverse: HA nausea
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Children with nephrotic syndrome will show a history of
periorbital edema upon waking that progresses to generalized edema thorughout the day
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Phimosis
Foreskin can not be retracted, normal in newborn, pathological if persists
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Physical cues of nephrotic syndrome include
-recent, sudden weight gain -frothy urine -N/V -pallor and skin breakdown -stretched skin -irritability -sx of fluid overload (have severe edema)
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Paraphimosis
restrictive band behind the glans penis---->incarceration and necrosis pain or swollen penis BOTH: irritation, erythmea, edema, or discharge
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Do children with nephrotic syndrome have edema, proteinuria, and hyperlipidemia?
yes
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Phimosis s/s
uti, irritation, balantis (inflammation of the foreskin) bleeding in the prepuce on dysuria
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Serum albumin will be ___ with nephrotic syndrome, but urine albumin will be ?
deceased ; increased (have proteinuria)
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Tx of Phimosis
topical steroid cream BID x1
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will cholesterol and lipids be elevated in nephrotic syndrome?
yes
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Treatment of Paraphimosis
surgical reduction
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will BUN and creatinine be elevated in nephrotic syndrome?
yes IF renal failure is occurring
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Priorities of care for Phimosis and paraphimosis
Apply topical steroid medications as prescribed for phimosis with genital retraction of foreskin.
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Priority nursing care for nephrotic syndrome include?
-monitoring I and O's -monitoring urine protein -DW -sodium restrictions -prevent infection -- can cause relapse ; vaccination status
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Priority care of paraphimosis
Routine post-op care and pain management of surgical site teach families appropriate hygiene
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What medications are given with nephrotic syndrome>
Prednisone (60 mg/m2/day for 4-6 wks then tapered over next 2-5 months) Furosemide (diuretics)
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Hypospadias
an abnormal urethral opening on ventral surface of penis (below glans penis)
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Epispadias
an opening on the dorsal penile surface (above glans penis)
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Nursing considerations with prednisone
-take with meals -can cause increased appetite, weight gain, Cushing's features, and mood swings
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Hypospadias physical findings
urethral opening off center/ not a tip of penis presence or absence of testicles in scrotal sac (cryptorchidism) or hydrocele or inguinal hernia.
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Nursing consideration with diuretics
may require K+ replacement
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Hypospadias/ Epispadias main s/s
cannot direct urine stream in standing position Circumcision should be delayed until repaired.
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Children with nephrotic syndrome taking corticosteroids may experience
altered body image
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hypospadias treatment
surgically repaired
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What education is important to tell families with children that have nephrotic syndrome
-may return to school but avoid sick children -monitor temp and urine dipsticks to prevent relapse -how to properly use urine dipstick to monitor for protein
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Post op Hypospadias/ Epispadias care
secure urethral stent/drainage tubing. Compression dressing, Double diapering.
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What is enuresis?
continued incontinence beyond the age of toilet training
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What is primary and secondary enuresis
primary: never achieved extended dry periods Secondary: onset after a period of urinary continence
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Fluid requirements and children with enuresis?
encourage fluids during the day, restrict at least 2 h before bed
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Caffeine and chocolate with enuresis
-limit caffeine and chocolate after dinner
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What type of toileting schedule should be used for children with enuresis?
ensure child voids before bedtime ; wake up at scheduled intervals to void
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Should pull-ups be used in enuresis?
no pull-ups, use a reward system
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Children with enuresis that wet the bed should be included in?
bed linen changes in a non-punitive manner
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Nursing interventions for enuresis?
-conditioning therapy - alarm sensor -Kegal/pelvic exercises
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What medication is given for enuresis?
antidiuretic (desmopressin)
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Nursing considerations for giving desmopressin to treat enuresis?
-give at bedtime -hold for diarrhea and vomiting -restrict fluid intake after dinner -only allow sips of water 1 h b4 taking and 8 h after
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Adverse effects of desmopressin?
headache and nausea
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What is phimosis?
when the foreskin cannot be retracted, which is normal in the newborn, but pathological later
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What is paraphimosis?
serious medical condition causing a restrictive band behind the glans penis, which can cause an incarceration and necrosis of the penile head
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What should be applied BID for one month to treat phimosis?
topical steroid cream
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If urine is retained in foreskin after voiding during phimosis, what can occur?
irritation, UTI, and balanitis
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What is required to correct paraphimosis?
surgical reduction
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What can be done to cure both phimosis and paraphimosis?
circumcision
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Physical cues for phimosis?
-irritation or bleeding in the prepuce -dysuria
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Physical cures for paraphimosis?
-pain and swollen penis
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Irritation, erythema, edema, or discharge from penis are physical cues for both
phimosis and paraphimosis
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What is hypospadias?
-when urethra opens on ventral surface of penis
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What may happen if hypospadias is left uncorrected
-may cause erectile dysfunction
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Physical findings of hypospadias?
-hx of unusual urine stream -urethral opening off center/ not at tip of penis -chordee (fibrous band that causes penis to bend downward) -presence of absence of testicles in scrotal sac (cryptorchidism) -hydrocele -inguinal hernia
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What is the treatment for hypospasias
surgery
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What should be delayed in infants to help treatment for hypospadias
circumcision
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Management of GI sx in cystic fibrosis include
-pancreatic enzymes -high protein and calorie diet -fluids -fat-soluble vitamins
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