Exam 2 Flashcards

(142 cards)

1
Q

Anxiety versus Fear

A

FEAR is a state of alarm in response to a specific threat
-Focused on a specific object, person, or circumstance (e.g., afraid of the dark)

ANXIETY is a state of alarm in response to a vague sense of danger
-Source might not be so clearly identifiable
-Not necessarily an immediate danger but rather a perception of threat
-Same physiological features:
Breathing fast, sweating, tensing up

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2
Q

Can Anxiety Be Adaptive?

A

Anxieties and fears can be adaptive if or when they:
-Prepare us for fight / flight when threatened

There are normal fears and anxieties for each phase of development. For example:

  • For a child to fear being separated from parents
  • For college students to have a fear of not reaching goals or of being lonely or isolated

For some people, anxiety is too severe, too frequent, or too easily triggered
-This results in anxiety or anxiety-related disorders

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3
Q

Anxiety Disorders - Prevalence

A

Most common disorder in the US

18% of adults suffer from an anxiety disorder in any given year

29% develop a disorder throughout their lifetime

Only 1/5 of individuals seek treatment

Societal impact
-$42 billion in health care, lost wages and lost productivity

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4
Q

Anxiety Disorders and Comorbidity

A
7 disorders:
Generalized Anxiety Disorder (GAD)
Phobia
Panic Disorder
Obsessive-Compulsive Disorder (OCD)
Acute Stress Disorder
Posttraumatic Stress Disorder (PTSD)
Separation Anxiety Disorder (SAD)

Comorbidities
-Another anxiety disorder (see
graph at right)
-Depression

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5
Q

Generalized Anxiety Disorder

A

Symptoms

  • Intense anxiety
  • Excessive worries
  • Overly sensitive, irritable, restless
  • Physical / muscle tension
  • Sleep difficulties
  • Easily tired

Prevalence

  • 6% lifetime prevalence; often begins in adolescence
  • Women outnumber men: ~ 2:1
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6
Q

Generalized Anxiety Disorder con’td.

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Psychodynamic Formulations

Freud: Everyone experiences anxiety and uses defense mechanism to help control it

  • Realistic: results from actual danger
  • Neurotic: results from fears of expressing conflicting or unconscious impulses
  • Moral: results from conflicts between underlying impulses and the conscience

Psychoanalysts trace anxiety back to the parent-child relationship

Other contemporary psychodynamic theories broaden the source and content of conflicts, but emphasize their mostly unconscious nature (E.g., psychosocial approach; interpersonal role theory)

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7
Q

Generalized Anxiety Disorder - Psychodynamic Perspective

A

Research:

  • People with GAD do use defense mechanisms
  • Children who were severely punished for expression of sexual or aggressive impulses do develop more anxiety later in life

Therapy:
-Same general techniques: free association, interpretations
-Specific treatment for GAD:
Focus less on fear and more on regulation of impulses
Object-relations: help identify and resolve early relationship conflicts
-Controlled research doesn’t show traditional psychoanalysis to be all that helpful
-Short-term dynamic therapy may be more useful

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8
Q

Generalized Anxiety Disorder - Cognitive Formulations

A

Basic maladaptive assumptions lead people to behave in inappropriate ways. Examples:
-It is a necessity for humans to be loved by everyone.
-It is catastrophic when things are not as I want them.
-If something is fearful, I should be terribly concerned and dwell on the possibility of its occurrence.
-I should be competent in all domains to be a worthwhile person.
(When these are applied to daily life, GAD develops)

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9
Q

Generalized Anxiety Disorder - Cognitive Perspectives

A

Aaron Beck: people with generalized anxiety disorder constantly hold silent assumptions

  • “A situation or a person is unsafe until proven to be safe”
  • “It is always best to assume the worst”

Research:
-People with generalized anxiety disorder do indeed hold maladaptive assumptions, particularly about dangerousness

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10
Q

Treating GAD

A
Challenge maladaptive assumptions
-Rational emotive therapy (RET)
    Point out irrational assumptions
    Suggest more appropriate assumptions
    Assign homework to test assumptions

Challenge cognitions and responses

  • Psychoeducation about worrying and GAD
  • Assign self-monitoring of bodily arousal and cognitive responses
  • Clients become skilled at identifying worry and misguided attempts at control by worrying
  • Address realistic bases of anxiety
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11
Q

Generalized Anxiety Disorder - Biological Formations

A

Generalized anxiety is caused by biological factors
-Family pedigree studies:
15% of relatives vs. 6% in general population
-GABA (gama-aminobutyric acid – primary inhibitory neurotransmitter in CNS)
*GABA causes a neuron to stop firing messages
*In normal fear response:
~Key neurons fire rapidly to create a state of excitability (fear/anxiety)
~Continuous firing triggers a feedback system and the brain and body try to reduce the excitability
~Some neurons release GABA to inhibit fear/anxiety
~Problems in the feedback system cause GAD

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12
Q

Generalized Anxiety Disorder - Treatment

A
  • Antianxiety drugs: Benzodiazepines
    • Modest, temporary relief, but can cause rebound anxiety, withdrawal, physical dependence, side effects, or multiply the effects of other drugs

-Some antidepressants and antipsychotics can be effective

  • Relaxation training: Physical relaxation leads to psychological relaxation
    • Used with biofeedback: Use feedback from electrical signals from the body to train people to control physiological processes
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13
Q

Phobias

A

-Persistent and unreasonable fears of particular objects, activities, or situations

  • Avoidance of the object or situation as well as thoughts about it
    • More intense and persistent
    • Greater avoidance
    • Distress that interferes with functioning
  • Although most phobias are categorized as “specific” (e.g., “acrophobia” or fear of heights) there are also two broader kinds:
    • Social phobia and agoraphobia
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14
Q

Specific Phobias

A

-How common is it?
9 % of US in any year
12% at some point in their lives

  • Who gets it?
    Women outnumber men 2:1
  • Most people do not seek
    treatment
  • Comorbidities:
    More than 1 phobia at a time
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15
Q

What Causes Specific Phobias?

A

Behavioral explanations:
-Phobias develop through modeling Observation and imitation

-Phobias are maintained through avoidance

  • Phobias may develop into GAD when a person acquires a large number of them
    • Process of stimulus generalization: Responses to one stimulus are also elicited by similar stimuli
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16
Q

Treatment of Specific Phobias

A

Behavioral treatment is the most widely used
-Systematic desensitization:
*Teach relaxation skills
*Create fear hierarchy
*Pair relaxation with feared object or situation
~In vivo: live
~Covert: imagined
-Flooding
*Forced nongradual exposure to the feared object or situation, with response prevention (extinction)
-Modeling
*Therapist confronts the feared object while patient watches and then imitates

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17
Q

Agoraphobia

A

People with agoraphobia are afraid of being in situations where escape might be difficult, should they experience panic or become incapacitated

In any given year, about 2% of adults experience this problem, women twice as frequently as men

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18
Q

Social Anxiety Disorder

A
  • Severe, persistent, and irrational fears of social or performance situations in which scrutiny by others and embarrassment may occur
    • May be narrow – talking, performing, eating, or writing in public
    • May be broad – general fear of functioning poorly in front of others
    • In both forms, people judge themselves as performing less competently than they actually do

-This disorder was called social phobia in past editions of the DSM

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19
Q

Explanations for Agoraphobia

A

Although broader than specific phobias, agoraphobia is often explained in ways similar to specific phobias

Many also are prone to experience extreme and sudden explosions of fear – called “panic attacks” – and may receive a second diagnosis of panic disorder

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20
Q

How is Agoraphobia Treated?

A

Behavioral therapy with an exposure approach is the most common and effective treatment for agoraphobia

  • Therapists help clients venture farther and farther from their homes to confront the outside world
  • Therapists use exposure techniques similar to those used for treating specific phobia but, in addition, use support groups and home-based self-help programs
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21
Q

Panic Disorder

A

Panic is an extreme anxiety reaction that can affect anyone when a real threat emerges

  • “Panic attacks” are short episodes of panic that occur suddenly, reach a peak, and then pass
  • The person fears he/she will die, go crazy, lose control, in the presence of no real threat

More than ¼ of all people will experience a panic attack in their lives but other people have them repeatedly and unexpectedly

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22
Q

Panic Disorder - Prevalence

A

How common is it?
-2.8% each year; 5% over a lifetime

Who get’s it?
-Women 2:1

Develops in late adolescence and early adulthood

  • Low SES 50% more likely
  • Only about 35% of people seek treatment
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23
Q

Panic Disorder - Biological Approach

A
  • Family pedigree studies show greater likelihood in relatives
  • Helped more by anti-depressants than anti-anxiety medicine
  • Related to changes of norepinephrine in the locus ceruleus
  • Different biological circuit than GAD
    • Amygdala
    • Central gray matter
    • Locus ceruleus
    • Ventromedial nucleus of hypothalamus
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24
Q

Panic Disorder - Drug Therapies

A

Antidepressant drugs restore proper norepinephrine activity in locus ceruleus and help reduce panic

Improvement seen in 80% of patients

Benzodiazepines also effective (e.g., Xanax)

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Panic Disorder - Cognitive Approach
Individual misinterprets bodily sensations - Panic-prone individuals are very sensitive to certain sensations and interpret them as a medical catastrophe * “Overbreathe”- hyperventilate - Biological Challenge Tests * Make people hyperventilate and see how they react * Participants with panic disorder experience greater distress - Anxiety sensitivity: * Focus on bodily sensations, unable to assess logically, interpret as potentially harmful
26
Panic Disorder - Cognitive Therapy
- Psychoeducation - Teach about accurate interpretations of bodily sensations - Coping skills: relaxation and breathing techniques - Can combine with behavioral therapy * Exposure: Induce panic sensations in the client so that their new skills can be tested in vivo - Effectiveness: * 80% of clients who receive cognitive treatment become panic-free * Useful for panic disorder with agoraphobia (more expos
27
Obsessive-Compulsive Disorder
Made up of two components: Obsessions Persistent thoughts, ideas, impulses, or images that seem to invade a person’s consciousness Compulsions Repetitive and rigid behaviors (or mental acts) that people feel they must perform to prevent or reduce anxiety
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Features of Obsessions
``` Take various forms: Wishes Impulses Images Ideas Doubts ``` ``` Have common themes: Dirt/contamination Violence and aggression Orderliness Religion Sexuality ```
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Examples of Obsessions
-Intrusive image: Stabbing a loved one -Thought: I will get HIV if I touch the chair where the infected person sat. -Impulse: I am going to jump off a high place.
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Features of Compulsions
Compulsions - “Voluntary” behaviors or “mental acts” * Feel mandatory / unstoppable - Most persons recognize that such behaviors are unreasonable * Believe, though, that something terrible will occur if they do not perform the compulsive acts - Performing behaviors reduces anxiety * But only for a short time - Behaviors often develop into rituals
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Features of Compulsions - Common Forms/ Themes
- Cleaning (e.g., hand washing, showering and grooming in ritualized manner) - Checking (e.g., locks, lights, stove) - Order or balance (e.g., need for symmetry) - Touching, verbal, and / or counting (tap or touch or rub object or body part) - Can be observable (cleaning) or silent (like counting) - Rituals can be secretive
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Obsessions and Compulsions?
Most people with OCD experience both Compulsive acts often occur in response to obsessive thoughts - Compulsions seem to represent a yielding to obsessions - Compulsions also sometimes serve to help control obsessions
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Obsessive-Compulsive Disorder / Diagnosis
Diagnosis is called for when symptoms: - Feel excessive or unreasonable - Cause great distress - Take up much time - Interfere with daily functions Between 1% and 2% of U.S. population suffer from OCD in a given year; as many as 3% over a lifetime Disorder ‘waxes and wanes’
34
OCD: Psychodynamic Perspective
Anxiety disorders develop when children come to fear their impulses and use ego defense mechanisms to lessen their anxiety OCD differs from other anxiety disorders in that the “battle” is not unconscious; it is played out in overt thoughts and actions - Id impulses = obsessive thoughts (e.g., reaction formation) - Ego defenses = counter-thoughts or compulsive actions (e.g., undoing)
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OCD: Psychodynamic Therapies
Goals are to uncover and overcome underlying conflicts and defenses Main techniques are free association and interpretation Research has offered little evidence -Some therapists now prefer to treat these patients with short-term psychodynamic therapies
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OCD: Behavioral Perspective
Behaviorists have concentrated on explaining and treating compulsions rather than obsessions - Argue that compulsions begin as random acts… * In a fearful situation, they happen to perform a particular act (washing hands) * When the threat lifts, they associate the improvement with the random act - After repeated associations, they believe the compulsion is changing the situation * Bringing luck, warding away evil, etc. -The act becomes a key method to avoiding or reducing anxiety
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OCD: Behavioral Therapy
Exposure and response prevention (ERP) - Clients are repeatedly exposed to anxiety-provoking stimuli and are told to resist performing the compulsions * Many behavior therapists now use this technique in individual and group therapy formats * Homework is an important component - Between 55-85% of clients have been found to improve considerably with ERP, and improvements often continue indefinitely * But: As many as 25% fail to improve at all * And the approach is of limited help to those with obsessions but no compulsions
38
OCD: Cognitive Perspective
Everyone has repetitive, unwanted, and intrusive thoughts -People with OCD blame themselves for normal (although repetitive and intrusive) thoughts and expect that terrible things will happen as a result - To avoid such negative outcomes, they attempt to “neutralize” their thoughts with actions (or other thoughts) * Seeking reassurance * Thinking “good” thoughts * Washing * Checking - When a neutralizing action reduces anxiety, it is reinforced
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OCD: Cognitive Therapy
Cognitive therapists focus on the cognitive processes that help to produce and maintain obsessive thoughts and compulsive acts - May include: * Psychoeducation * Guiding the client to identify, challenge, and change distorted cognitions - Research suggests that a combination of the cognitive and behavioral models is often more effective than either intervention alone - These treatments typically include psychoeducation as well as exposure and response
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OCD: Biological Perspective
Family pedigree studies suggest that OCD may be linked in part to biological factors -Studies of twins find a 53% concordance rate in identical twins, versus 23% in fraternal twins More direct evidence: - Abnormal serotonin activity * Serotonin-based antidepressants reduce OCD symptoms * Abnormal brain structure and functioning * OCD linked to orbitofrontal cortex and caudate nuclei brain circuit that converts sensory information into thoughts and actions
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OCD: Biological Treatments
Biological therapies - Serotonin-based antidepressants * Clomipramine (Anafranil), fluoxetine (Prozac), fluvoxamine (Luvox) * Bring improvement to 50–80% of those with OCD * Relapse occurs if medication is stopped - Research suggests that combination therapy (medication plus cognitive behavioral therapy approaches) may be most effective
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Obsessive-Compulsive Related Disorders
In recent years, a growing number of clinical researchers have linked some excessive behavior patterns (e.g., hoarding, hair pulling, shopping, sex) to Obsessive Compulsive Disorder DSM-5 has created the group name “Obsessive-Compulsive-Related Disorders” and assigned four patterns to that group: hoarding disorder, hair-pulling disorder, excoriation (skin-picking) disorder, and body dysmorphic disorder Theorists typically account for OCD-related disorders by using the same kinds of explanations that have been applied to OCD Similarly, clinicians treat clients with these disorders using the kinds of treatment used with OCD, particularly antidepressant drugs, exposure and response prevention (ERP), and cognitive therapy
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Epidemiology (Rates) – TTM
Subclinical: 7-22% Adult Clinical Rate: 1.5-3.4% Child Clinical Rate: ≤ 9.25% Onset during early childhood OR adolescence
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Gender & Culture - TTM
Much more common in women (9:1 ratio) Possible reasons: treatment bias, culture norms Similar across cultures
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Impact of TTM
Moderate impact across social, occupational (or academic), and psychological domains. - Up to 78% lifetime comorbidity, 39% current comorbidity (Houghton et al., unpublished manuscript) - Can have physical impact (scarring alopecia, trichobezoars)
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Subtypes of TTM
Automatic Pulling Focused Pulling
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Causal Pathways - Etiology
- Support for genetic vulnerability * Shared genetic etiology with OCD - Abnormalities in brain structures involved in motor control and learning, affect regulation, and reward learning * Cortico-striatal-thalamic pathways - Monoaminergic and glutamate systems * Decreases with serotonin reuptake inhibitors * Increases with dopamine agonists * Responds to glutamate modulators in adults (but not children)
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Cognitive-Behavioral Etiology
Cognitive system – inhibitory control Behavior – operant learning model Can be thought of as a pathological habit!
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Treatment - TTM
Moderate to little response to SSRIs Some response to N-acetylcysteine (glutamate) Large response to behavior therapy (and cognitive behavior therapy) (McGuire et al., 2014) - Habit reversal therapy (HRT) - HRT + Cognitive Behavior Therapy, Acceptance and Commitment Therapy, and Dialectical Behavior Therapy
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Habit Reversal Training
Awareness Training Competing Response Training Social Support Stimulus Control (not a core component)
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Excoriation Disorder (ExD)
Repetitive picking or scratching at the skin resulting in skin damage
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Epidemiology, Gender, Culture - ExD
High comorbidity with TTM (around 15.5-20.8%) Lifetime prevalence of 7.3-11.8% in clinical samples Onset during early adolescence Chronic but fluctuating course Gender ratio biased toward females (7-9:1) Not enough cultural research Similar sub-patterns (automatic vs. focused)
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Causal Pathways - Etiology
- Possible genetic link to TTM and OCD (Sapap3 region of chromosome 1p35) * TTM and ExD likely have common and unique genetic associations - Imaging: reduced blood-oxygen flow in regions involved in motor inhibition (anterior cingulate cortex), increased activation of ventral striatum (reward learning) in ExD compared to TTM - Possible involvement of serotonin and GABA
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Cognitive-Behavioral Etiology
Cognitive System – Possible motor inhibition deficits Behavior – Problems with emotion regulation (operant learning model) -Harm avoidance vs. reward dependence
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Evidence-Based Treatment
- Gelinas & Gagnon (2013) – meta-analysis * Both pharmacological and behavioral approaches are effective (significantly larger effect for behavioral approaches: ES .78 vs 1.52) * Effective medications: SSRIs and anticonvulsants * Effective psychotherapy: Cognitive-Behavior Therapy, Habit Reversal Training, Acceptance and Commitment Therapy
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Treatment Resources for TTM and ExD
Tourette and OCD Clinic at TAMU (touretteclinic.tamu.edu; 979-458-4218) Director: Dr. Doug Woods Trichotillomania Learning Center trich.org StopPulling.com
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Future (My) Research -Guest Speaker
Neuropsychological traits across body focused repetitive behavior disorders (inhibitory control, executive functioning, memory, attention) Sensory processing dysfunction across OC-spectrum (sensory phenomena, sensory thresholds, sensorimotor integration)
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Stress, Coping, and Anxiety
The state of stress has two components: - Stressor: event that creates demands - Stress response: person’s reactions to the demands * Influenced by how we judge both the events and our capacity to react to them effectively * People who sense that they have the ability and resources to cope are more likely to take stressors in stride and respond well
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Stress and Arousal: The Fight-or-Flight Response
The features of arousal and fear are set in motion by the hypothalamus -Two important systems are activated: *Autonomic nervous system (ANS) An extensive network of nerve fibers that connect the central nervous system (the brain and spinal cord) to all other organs of the body *Endocrine system A network of glands throughout the body that release hormones There are two pathways through which arousal and fear are produced: Sympathetic Nervous System Pathway Hypothalamic-Pituitary-Adrenal (HPA) Pathway
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Stress and Arousal: The Fight-or-Flight Response - Pathway 1
Pathway 1: Sympathetic Nervous System Stressor  Sympathetic nervous system  Key organs are stimulated directly (e.g., heart) or indirectly (e.g., adrenal glands) -When the perceived danger passes, the parasympathetic nervous system helps return body processes to normal
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Stress and Arousal: The Fight-or- Flight Response - Pathway 2
Pathway 2: Hypothalamic-Pituitary-Adrenal (HPA) Stressor  Hypothalamus  Pituitary gland releases adrenocorticotropic hormone (ACTH; “major stress hormone”)  Adrenal cortex releases corticosteroids (stress hormones) into bloodstream
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Stress and Arousal: The Fight-or-Flight Response - Reaction
The reactions on display in these two pathways are collectively referred to as the fight-or-flight response Each person has a particular pattern of autonomic and endocrine functioning - Trait anxiety: person’s general level of arousal and anxiety. - State or situation anxiety: person’s sense of which situations are threatening
63
Stress, Coping, and Anxiety - Physical Disorders
The physical disorders of stress are typically called psychophysiological disorders - These disorders are listed in DSM-5 under “psychological factors affecting medical condition” * Significant stressors trigger an interaction of biological and psychological factors to help produce or worsen a physical illness or ailment
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The Physical Stress Disorders: Psychophysiological Disorders
It is important to recognize that these psychophysiological disorders bring about actual physical damage -They are different from “apparent” physical illnesses like factitious disorders or somatic symptom disorders (discussed in Chapter 7)
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Traditional Psychophysiological Disorders
Before the 1970s, the best known and most common of the psychophysiological disorders were ulcers, asthma, insomnia, chronic headaches, high blood pressure, and coronary heart disease Recent research has shown that many other physical illnesses may be caused by an interaction of psychosocial and physical factors
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Traditional Psychophysiological Disorders - Chronic Headaches
Chronic headaches - Frequent intense aches of the head or neck that are not caused by another physical disorder * Tension headaches affect 45 million Americans each year * Migraine headaches affect 23 million Americans each year - Causal psychosocial factors: * Environmental pressures; general feelings of helplessness, anger, anxiety, depression - Causal physiological factors: * Abnormal serotonin activity, vascular problems, muscle weakness
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Traditional Psychophysiological Disorders - Hypertension
Hypertension - Chronic high blood pressure, usually producing few outward symptoms - Affects 75 million Americans each year - Causal psychosocial factors: * Constant stress, general feelings of anger or depression - Causal physiological factors: * 10% caused by physiological factors alone * Obesity, smoking, poor kidney function, collagen (rather than elastic) tissue in an individual’s blood vessels
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Traditional Psychophysiological Disorders - Contributions
A number of variables contribute to the development of psychophysiological disorders, including: - Biological factors - Psychological factors - Sociocultural factors
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Traditional Psychophysiological Disorders - Biological Factors
- Defects in the autonomic nervous system (ANS) are believed to contribute to the development of psychophysiological disorders - Other more specific biological problems may also contribute * For example, a weak gastrointestinal system may create a predisposition to developing ulcers
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Traditional Psychophysiological Disorders - Psychological Factors
- According to many theorists, certain needs, attitudes, emotions, or coping styles may cause people to overreact repeatedly to stressors – increasing their chances of developing psychophysiological disorders * Examples: a repressive coping style, a Type A personality style – particularly hostility and time urgency
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Traditional Psychophysiological Disorders - Sociocultural Factors
Sociocultural factors | Adverse social conditions (e.g., poverty, unemployment, quality of medical care).
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New Psychophysiological Disorders
Since the 1960s, researchers have found many links between psychosocial stress and a wide range of physical illnesses Researchers have increasingly looked to the body’s immune system as the key to the relationship between stress and infection This area of study is called psychoneuroimmunology
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Psychoneuroimmunology
Researchers now believe that stress can interfere with the activity of lymphocytes, slowing them down and increasing a person’s susceptibility to viral and bacterial infections Several factors influence whether stress will result in a slowdown of the system, including biochemical activity, behavioral changes, personality style, and degree of social support
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Psychoneuroimmunology - Biochemical
Biochemical activity - Stress leads to increased activity by the sympathetic nervous system, including a release of norepinephrine * In addition to supporting nervous system activity, this chemical also appears to slow down the functioning of the immune system - Similarly, the body’s endocrine glands reduce immune system functioning during periods of prolonged stress through the release of corticosteroids * In addition, corticosteroids also trigger increased cytokines, which lead to chronic inflammation
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Psychoneuroimmunology - Behavior/Personality
Behavioral changes -Stress may set in motion a series of behavioral changes poor sleep patterns, poor eating, lack of exercise, increase in smoking and/or drinking – that indirectly affect the immune system Personality style -An individual’s personality style (including their level of optimism, constructive coping strategies, and resilience) experience better immune system functioning and are better prepared to fight off illness
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Psychoneuroimmunology - Social Support
People who have few social supports and feel lonely seem to display poorer immune functioning in the face of stress than people who do not feel lonely Social support and affiliation with others may protect people from stress, poor immune system functioning, and subsequent illness, and speed up recovery from illness or surgery
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Psychological Treatments for Physical Disorders
Clinicians have applied psychological treatment to more and more medical problems - The most common of these interventions are relaxation training, biofeedback training, meditation, hypnosis, cognitive interventions, support groups, and therapies designed to increase awareness and expression of emotion - The field of treatment that combines psychological and physical interventions to treat or prevent medical problems is known as behavioral medicine
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Psychological Treatments for Physical Disorders - Combination
Combination approaches - Studies have found that the various psychological interventions for physical problems tend to be equal in effectiveness * Psychological treatments are often of greatest help when they are combined and used with medical treatment * With these combined approaches, today’s practitioners are moving away from the mind–body dualism of centuries past
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Factitious Disorder
- Known popularly as Munchausen syndrome, people with a factitious disorder often go to extremes to create the appearance of illness * Many secretly give themselves medications to produce symptoms * Patients often research their supposed ailments and are impressively knowledgeable about medicine - In a related pattern, factitious disorder imposed on another, known popularly as Munchausen syndrome by proxy, parents make up or produce physical illnesses in their children
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Conversion Disorder
People with this disorder display physical symptoms that affect voluntary motor or sensory functioning, but the symptoms are inconsistent with known medical diseases -In short, the individuals experience neurological-like symptoms – blindness, paralysis, or loss of feeling – that have no neurological basis
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Conversion Disorder - Distinguish
- Conversion disorder often is hard to distinguish from genuine medical problems * It is always possible that a diagnosis of conversion disorder is a mistake and the patient’s problem has an undetected medical cause - Physicians sometimes rely on oddities in the patient’s medical picture to help distinguish the two * For example, conversion symptoms may be at odds with the known functioning of the nervous system, as in cases of glove anesthesia
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Somatic Symptom Disorder
People with somatic symptom disorder become excessively distressed, concerned, and anxious about bodily symptoms that they are experiencing Two patterns of somatic symptom disorder have received particular attention: - Somatization pattern - Predominant pain pattern People with a somatization pattern experience many long-lasting physical ailments that have little or no organic basis *Also known as Briquet’s syndrome Patients with this pattern often describe their symptoms in dramatic and exaggerated terms * Most also feel anxious and depressed * Patients usually go from doctor to doctor in search of relief
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Somatic Symptom Disorder - Predominant Pain
- If the primary feature of somatic symptom disorder is pain, the individual is said to have a predominant pain pattern - Although the precise prevalence has not been determined, this pattern appears to be fairly common * The pattern often develops after an accident or illness that has caused genuine pain - The pattern may begin at any age, and more women than men seem to experience it
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What Causes Conversion and Somatic Symptom Disorders? - Psychodynamic View
The psychodynamic view - Psychodynamic theorists propose that two mechanisms are at work in hysterical disorders: * Primary gain: bodily symptoms keep internal conflicts out of conscious awareness * Secondary gain: bodily symptoms further enable people to avoid unpleasant activities or receive sympathy from others
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What Causes Conversion and Somatic Symptom Disorders? - Behavioral View
The behavioral view - Behavioral theorists propose that the physical symptoms of hysterical disorders bring rewards to sufferers * May remove individual from an unpleasant situation * May bring attention from other people
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What Causes Conversion and Somatic Symptom Disorders?
In response to such rewards, people learn to display symptoms more and more This focus on rewards is similar to the psychodynamic idea of secondary gain, but behaviorists view the gains as the primary cause of the development of the disorder Like the psychodynamic explanation, the behavioral view of these disorders has received little research support
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What Causes Conversion and Somatic Symptom Disorders? - Biological Role
A possible role for biology -The impact of biological processes on somatoform disorders can be understood through research on placebos and the placebo effect *Placebos: substances with no known medicinal value *Treatment with placebos has been shown to bring improvement to many – possibly through the power of suggestion, or because positive expectations trigger the release of endogenous chemicals
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How Are Conversion and Somatic Symptom Disorders Treated? - Focusing on Causes
Many therapists focus on the causes of the disorders and apply techniques including: - Insight – often psychodynamically oriented - Exposure with response prevention – client thinks about traumatic event(s) that triggered the physical symptoms - Drug therapy – especially antidepressant medication
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How Are Conversion and Somatic Symptom Disorders Treated? - Addressing Physical Symptoms
Other therapists try to address the physical symptoms of these disorders, applying techniques such as: - Suggestion – usually an offering of emotional support that may include hypnosis - Reinforcement – a behavioral attempt to change reward structures - Confrontation – an overt attempt to force patients out of the sick role
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Illness Anxiety Disorder
People with illness anxiety disorder, previously known as hypochondriasis, experience chronic anxiety about their health and are concerned that they are developing a serious medical illness, despite the absence of somatic symptoms They repeatedly check their bodies for signs of illness and misinterpret bodily symptoms as signs of a serious illness - Often their symptoms are merely normal bodily changes, such as occasional coughing, sores, or sweating - Although some patients recognize that their concerns are excessive, many do not
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Illness Anxiety Disorder - Explanation and Treatment
Theorists explain this disorder much as they explain various anxiety disorders: --Behaviorists: classical conditioning or modeling Cognitive theorists: oversensitivity to bodily cues Individuals with illness anxiety disorder typically receive the kinds of treatments applied to OCD: - Antidepressant medication - Exposure and response prevention (ERP) - Cognitive-behavioral therapies
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Body Dysmorphic Disorder
People with this disorder, also known as dysmorphobia, become deeply concerned about some imagined or minor defect in their appearance -Most often they focus on wrinkles, spots, facial hair, swelling, or misshapen facial features (nose, jaw, or eyebrows)
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Body Dysmorphic Disorder - Explanations and Treatment
Theorists typically account for BDD by using the same kinds of explanations – both physical and psychological – that have been applied to anxiety disorders and OCD Similarly, clinicians typically treat clients with this disorder by applying the kinds of treatment used with OCD, particularly: - Anti-depressant drugs, exposure and response prevention, and cognitive therapy
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What Is Trauma?
“A very difficult or unpleasant experience that causes someone to have mental or emotional problems for a long time …..” “A mechanism or event that causes trauma” APA – “An emotional response to a terrible event….” - An event, or a response? - Defined by its consequences? Only traumatic if you experience it as such?
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Examples of Traumatic Events
Exposure to combat 20% of veterans report symptoms of PTSD Exposure to natural disasters (Hurricane Katrina, Aug 2005) Involvement in accidents (Bonfire collapse Nov 19, 1999) Civilian traumas 10 times more frequent than combat-related traumas Sexual assault and other victimization Exposure to terrorism (September 11, 2001)
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What Is Post-Traumatic Stress? Criteria "A"
Criterion A – Exposure to trauma -Direct exposure to death, threatened death, actual or threatened serious injury or violence -May be indirect exposure, for example: *By learning that a close relative was exposed to trauma *Repeated or extreme indirect exposure to aversive details ~As in first responders collecting body parts from disaster scene ~Professionals repeatedly exposed to details of child abuse
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What Is PTSD? Criteria "B"
Criterion B – Intrusion symptoms such as: - Recurrent, involuntary, intrusive memories - Traumatic nightmares - Dissociative reactions (e.g., flashbacks) occurring on a continuum from brief episodes to complete loss of consciousness - Intense or prolonged distress – which may be emotional or physiological – after exposure to traumatic reminders
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What Is PTSD? Criteria "C"
Criterion C – Persistent avoidance of stimuli associated with the trauma - Avoidance of trauma-related thoughts or feelings - Avoidance of trauma-related reminders (e.g., people, places, activities, objects)
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What Is PTSD? Criteria "D"
Criterion D – Negative alterations in cognitions and mood associated with the traumatic event - Dissociative amnesia - Persistent (often distorted) negative beliefs – e.g., self-blame or global negative expectancies of others - Persistent negative emotions – e.g., fear, horror, anger, shame - Emotional numbing * Diminished interest in pre-traumatic significant activities * Alienation (detachment or estrangement) from others * Constricted affect – inability to experience positive emotions
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What Is PTSD? Criteria "E" and "F"
``` Criterion E – Alterations in arousal and reactivity that began or worsened after the traumatic event Irritable or aggressive behavior -Self-destructive or reckless behavior -Hypervigilance -Exaggerated startle response -Problems in concentration -Sleep disturbance ``` Criterion F – Significant impairment in functioning, with persistence of symptoms for more than 1 month
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How Do We Treat PTSD?
- Pharmacotherapy – Management of most acute anxiety (or comorbid depressive) symptoms - Prolonged exposure – Aimed at extinction of emotional and physiological reactivity (EMDR) - Cognitive processing therapy * Provides information about causes, symptoms of PTSD * Encourages labeling of impact: thoughts and feelings * Promotes changes in beliefs, attributions regarding the traumatic event * Promotes self-regulation and coping skills
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Relationships and PTSD
Combat exposure is a risk factor for posttraumatic stress disorder (PTSD) Veterans with higher levels of support report fewer PTSD symptoms Spouses and intimate partners often serve as a primary source of social support Research question: Are effects of partner support on PTSD “mediated” by communication and self-disclosure?
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Mediational Model
Partner Support ---> Disclosure and PTSD Symptoms Disclosure ---> PTSD Symptoms Triangle
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Implications of Findings
Service members and veterans may benefit from disclosure of traumatic experiences to an intimate, supportive partner Disclosure may facilitate the cognitive and emotional processing of combat and deployment related experiences Initial support for the encouragement of disclosure in post-deployment couple interventions
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Interpersonal Trauma
Can various “interpersonal” or “intimate relationship” injuries be traumatic? The case for sexual infidelity: - High lifetime prevalence (21% of men, 11% of women) - One of leading causes of divorce - Rated as among most difficult marital problems to treat - High rates of negative impact * Depression, suicidality, PTSD symptoms * Physical aggression, alcohol/substance abuse
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Three Stage Model of Recovery
Stage 1 - Absorbing the blow Stage 2 - Giving meaning, establishing new assumptions Stage 3 - Moving forward
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Final Comments …. PTSD
Your own risk for exposure to PTSD - About 50% of individuals will experience a traumatic event during their lifetime - Many (but not all) of those will develop symptoms of PTSD - In general (civilian) population, lifetime rates of PTSD: * Clinical levels: 8-10% * Subclinical levels: 15-20% Most individuals will have close, indirect experience with someone suffering from PTSD For your neighbor, friend, or loved one … - Offer understanding - Provide information to normalize symptoms - Offer encouragement regarding treatment resources For yourself ... - Offer self-acceptance and patience - Engage in physical, emotional, spiritual self-care - Share with intimate others - Seek out qualified professional help - Retain hope
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Symptoms of Dissociative Disorders
Dissociative disorders are each characterized by significant memory loss or identity disruption -Dissociative amnesia (and dissociative fugue) -Dissociative identity disorder (DID) (multiple personality disorder) -Depersonalization - derealization disorder
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Dissociative Amnesia
People with dissociative amnesia are unable to recall important information, usually of an upsetting nature, about their lives - The loss of memory is much more extensive than normal forgetting and is not caused by physical factors - Often an episode of amnesia is directly triggered by a specific upsetting event Dissociative amnesia may be: - Localized – most common type; loss of all memory of events occurring within a limited period - Selective – loss of memory for some, but not all, events occurring within a period - Generalized – loss of memory beginning with an event, but extending back in time; may lose sense of identity; may fail to recognize family and friends - Continuous – forgetting continues into the future; quite rare in cases of dissociative amnesia In all types, memory for abstract or encyclopedic information – usually remains intact
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Dissociative Fugue
An extreme version of dissociative amnesia is dissociative fugue - People with dissociative fugue not only forget their personal identities and details of their past, but also flee to an entirely different location - For some, the fugue is brief – a matter of hours or days – and ends suddenly - For others, the fugue is more severe: people may travel far from home, take a new name and establish new relationships, and even a new line of work; some display new personality characteristics Dissociative fugue disorders are rare: ~0.2% of the population -It usually follows a severely stressful event Fugues tend to end abruptly - When people are found before their fugue has ended, therapists may find it necessary to continually remind them of their own identity - The majority of people regain most or all of their memories and never have a recurrence
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Dissociative Identity Disorder (DID)
A person with dissociative identity disorder develops two or more distinct personalities (subpersonalities) each with a unique set of memories, behaviors, thoughts, and emotions At any given time, one of the subpersonalities dominates the person’s functioning - Usually one subpersonality –the primary or “host” personality – appears more often than the others - The transition from one subpersonality to the next (“switching”) is usually sudden and may be dramatic
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Dissociative Identity Disorder - Prevalence
Most cases are first diagnosed in late adolescence or early adulthood - Symptoms generally begin in childhood after episodes of abuse * Typical onset is before age 5 Women receive the diagnosis three times as often as men
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Dissociative Identity Disorder - Subpersonalities Interaction
The relationship among subpersonalities varies from case to case Generally there are three kinds of relationships: - Mutually amnesic relationships – subpersonalities have no awareness of one another - Mutually cognizant patterns – each subpersonality is well aware of the rest - One-way amnesic relationships – most common pattern; some personalities are aware of others, but the awareness is not mutual * Those subpersonalities that are aware (“co-conscious subpersonalities”) are “quiet observers
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Dissociative Identity Disorder - Subpersonalities Differ
Subpersonalities often display dramatically different characteristics, including: - Identifying features (age, sex, race, family history) - Abilities and preferences (ability to drive, speak a foreign language, play a musical instrument) - Physiological features (autonomic nervous system activity, blood pressure levels
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Dissociative Identity Disorder - How Common?
The number of people diagnosed with the disorder has been increasing Although the disorder is still uncommon, thousands of cases have been documented in the U.S. and Canada alone - Two factors may account for this increase: * A growing number of clinicians believe that the disorder does exist and are willing to diagnose it * Diagnostic procedures have become more accurate
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Possible Etiologies of DID - Psychodynamic View
Psychodynamic theorists believe that dissociative disorders are caused by repression, the most basic ego defense mechanism -People fight off anxiety by unconsciously preventing painful memories, thoughts, or impulses from reaching awareness Most of the support for this model is drawn from case histories, which report brutal childhood experiences, yet: -Some individuals with DID do not seem to have these experiences of abuse
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Possible Etiologies of DID - Behavioral View
Behaviorists believe that dissociation grows from normal memory processes and is a response learned through operant conditioning: - Momentary forgetting of trauma leads to a drop in anxiety, which increases the likelihood of future forgetting - Like psychodynamic theorists, behaviorists see dissociation as escape behavior Also like psychodynamic theorists, behaviorists rely largely on case histories to support their view of dissociative disorders
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Possible Etiologies of DID - State Dependent Learning
People who are prone to develop dissociative disorders may have state-to-memory links that are unusually rigid and narrow; Each thought, memory, and skill is tied exclusively to a particular state of arousal Hence, they recall a given event only when they experience an arousal state almost identical to the state in which the memory was first acquired
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Possible Etiologies of DID - Self Hypnosis
Although hypnosis can help people remember events that occurred and were forgotten years ago, it can also help people forget facts, events, and their personal identity -Called “hypnotic amnesia,” this phenomenon has been demonstrated in research studies with word lists
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Treatments for Dissociative Disorders
People with dissociative amnesia often recover on their own -Only sometimes do their memory problems linger and require treatment In contrast, people with DID usually require treatment to regain their lost memories and develop an integrated personality -Treatment for DID tends to be less successful than treatment for dissociative amnesia
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How do therapists help individuals with DID?
Therapists usually try to help the client by: -Recognizing the disorder -Recovering memories -Integrating the subpersonalities *Integration into a single identity *Integration is a continuous process *Following integration, further therapy is typically needed to ~Maintain the complete personality, and ~Teach social and coping skills to prevent later dissociation
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Depersonalization - Derealization Disorder
Depersonalization: Individual feels as though they have become separated from their body and are observing themselves from outside In contrast to depersonalization, derealization is characterized by the feeling that the external world is unreal and strange Depersonalization and derealization experiences by themselves do not indicate a disorder - Transient depersonalization or derealization reactions are fairly common - The symptoms of a depersonalization - derealization disorder are persistent or recurrent, cause considerable distress, and interfere with social relationships and job performance The disorder occurs most frequently in adolescents and young adults, hardly ever in people older than 40
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Eating Disorders
Western society today often equates thinness with health and beauty -Thinness has become a national obsession There has been a rise in eating disorders in the past three decades -The core issue is an irrational fear of weight gain Two main diagnoses: - Anorexia nervosa - Bulimia nervosa
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Incidence Patterns - Eating Disorders
About 90%–95% of anorexia and bulimia nervosa cases occur in females The peak age of onset is between 15 and 21 years Symptoms may last for several years with periodic letup
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Symptoms of Anorexia Nervosa
Despite their dietary restrictions, people with anorexia nervosa are preoccupied with food -Excessive thinking about food and planning for meals Persons with anorexia also think in distorted ways: - Usually have a low opinion of their body shape - Tend to overestimate their actual size / proportions - Hold maladaptive attitudes and misperceptions * “I must be perfect in every way” * “I will feel better if I deprive myself” * “I can avoid guilt by not eating”
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Anorexia Nervosa: Comorbid Disorders
``` Psychological Depression Anxiety Low self-esteem Insomnia or other sleep disturbances Substance abuse Obsessive-compulsive patterns Perfectionism ``` ``` Medical Amenorrhea Low blood pressure Reduced bone density Metabolic and electrolyte imbalances Poor circulation Body swelling ```
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Bulimia Nervosa
Bulimia nervosa, also known as “binge-purge syndrome,” is characterized by binges: -Repeated bouts of uncontrolled overeating during a limited period of time Also characterized by inappropriate compensatory behaviors, including: - Forced vomiting - Misusing laxatives, diuretics, or enemas - Fasting - Exercising excessively
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Bulimia Nervosa: Binges
People with bulimia nervosa may have between 1 and 30 binge episodes per week Binges are often carried out in secret - Binges involve eating massive amounts of food very rapidly with little chewing * Usually sweet, high-calorie foods with soft texture Although the binge itself may be pleasurable, it is usually followed by extreme self-blame, guilt, depression, and fears of being discovered
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Bulimia: Compensatory Behaviors
After a binge, people with bulimia nervosa try to compensate for and “undo” the caloric effects - Many resort to vomiting * Fails to prevent the absorption of half the calories consumed during a binge * Repeated vomiting affects the ability to feel satiated  greater hunger and bingeing Compensatory behaviors may temporarily relieve negative feelings attached to binge eating -Over time, however, a cycle develops in which purging  bingeing  purging…
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Bulimia vs. Anorexia Nervosa
Similarities: Begin after a period of dieting Fear of becoming obese; drive to become thin Preoccupation with food, weight, appearance Feelings of anxiety, depression, perfectionism Distorted body perception Heighted risk of suicide attempts Differences: People with bulimia nervosa tend to be more sexually experienced and active People with bulimia nervosa are more likely to have histories of mood swings, low frustration tolerance, and poor coping
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Binge Eating Disorder
Like those with bulimia, individuals with binge eating disorder engage in repeated eating binges during which they feel no control -These individuals do not perform inappropriate compensatory behaviors As a result of their binges, two-thirds of people with this disorder become overweight or obese -It is important to recognize, however, that most overweight people do not engage in repeated binges
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What Causes Eating Disorders? - Psychodynamic Theories
Parents respond to their children ineffectively, failing to attend to biological and emotional needs -Deficiencies in parenting contribute to a broad cognitive distortion underlying disordered eating (e.g., negative self-judgment based on body shape and weight) Anorexia stems from an effort to delay or interrupt sexual maturation Anorexia reflects a passive-aggressive response to conflicts around control and autonomy Bulimia reflects self-nurturing with food in the absence of adequate parental nurturing
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What Causes Eating Disorders? - Mood Disorders
There is also evidence that mood disorders set the stage for eating disorders: - Persons with an eating disorder have higher rates of major depressive disorder - Close relatives of those with eating disorders have higher rates of depressive disorders - People with eating disorders, especially those with bulimia nervosa, have serotonin abnormalities - Symptoms of eating disorders are helped by antidepressant medications
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What Causes Eating Disorders? - Biological Disorders
Biological factors: The hypothalamus and related brain areas may be responsible for weight “set point” or “weight thermostat” -Set by genetic inheritance and early eating practices, this mechanism is responsible for keeping an individual at a particular weight level *If weight falls below set point:  hunger,  metabolic rate  binges *If weight rises above set point:  hunger,  metabolic rate -Dieters end up in a battle against themselves to lose weight
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What Causes Eating Disorders? - Societal Pressures
Societal pressures: Western standards of female attractiveness may contribute to the emergence of eating disorders - Western standards have changed throughout history toward a thinner ideal (evidence in Miss American contestants, Playboy centerfolds) - Models, actors, dancers, and certain athletes are at higher risk for eating disorders * 9% of college athletes meet full criteria for an eating disorder while another 50% have one or more symptoms * 20% of gymnasts appear to have an eating disorder
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What Causes Eating Disorders?
The socially accepted prejudice against overweight people may also add to the “fear” and preoccupation about weight -About 50% of elementary and 61% of middle school girls are currently dieting Families may play an important role in the development of eating disorders -As many as half of the families of those with eating disorders have a long history of emphasizing thinness, appearance, and dieting
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Gender Differences in Eating Disorders
Some men develop eating disorders as linked to the requirements and pressures of a job or sport -For example: Jockeys, wrestlers, distance runners, body builders, swimmers Some men exhibit a new kind of eating disorder – reverse anorexia nervosa or muscle dysmorphobia
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How Are Eating Disorders Treated?
Eating disorder treatments have two main goals: - Correct dangerous eating patterns - Address broader psychological and situational factors that led to, and maintain, the eating problem With anorexia, treatment goals are to: -Promote normal eating behavior, regain lost weight, and recover from malnourishment With bulimia, treatment goals are to: -Eliminate binge-purge patterns, and promote normal eating behavior
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Treatment for Anorexia Nervosa
The most popular weight-restoration technique has been the combination of supportive nursing care, nutritional counseling, and high-calorie diets In life-threatening cases, clinicians may need to force tube and intravenous feedings on the patient People with anorexia nervosa must overcome their underlying psychological problems to achieve lasting improvement In most treatment programs, a combination of behavioral and cognitive interventions are included - Clients monitor feelings, hunger levels, and food intake and the ties among those variables - Patients are encouraged to recognize their underlying feelings - Clients are helped to change their attitudes about eating and weight
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Anorexia Nervosa Treatment Outcomes
Positive outcomes: - Weight gain is often quickly restored, with many patients maintaining improvements after several years - Menstruation resumes after return to normal weight Negative outcomes: - As many as 25% of patients fail to improve - Initial recovery is often not permanent * Anorexic behavior recurs in at least one-third of recovered patients, usually triggered by new stresses * Many patients still express concerns about their weight and appearance - Lingering emotional problems are common
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Treatment for Bulimia Nervosa
Cognitive-behavioral therapy: - Behavioral techniques * Eating and purging diaries * Exposure and response prevention (ERP) is used to break the binge-purge cycle - Cognitive techniques * Help clients recognize and change their maladaptive attitudes toward food, eating, weight, and shape * Teach individuals to identify and challenge the negative thoughts that precede the urge to binge Other interventions: - Group formats provide an opportunity for patients to express their thoughts, concerns, and experiences with one another - Antidepressant medications – may help as many as 40% of patients
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Treatment Outcomes for Bulimia
Treatment provides immediate, significant improvement in about 40% of cases -An additional 40% show moderate response Relapse can be a significant problem, even among those who respond successfully to treatment - Relapses are usually triggered by stress - Relapses are more likely among persons who: * Had a longer history of symptoms * Vomited frequently * Had histories of substance use * Have lingering interpersonal problems