Exam 2 part 2 Flashcards

(48 cards)

1
Q

What are the bacteria involved in both NUG and NUP

A

P. intermedia, Spirochetes, Fusiformbacteria

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2
Q

What kind of tissue is involved in NUG

A

Involves both stratified squamous epithelium & underlying CT

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3
Q

Define NUG

A

Acute necrotizing inflammation of the gingival margin

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4
Q

Is NUG contagious?

A

No

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5
Q

What is another name for NUG and why was that made popular?

A

Trench mouth

1914 WWI, WWII

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6
Q

Age groups that NUG occurs in

A

Occurs in all ages but mainly 20-30

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7
Q

Visually what do you see in NUG

A

Forms Pseudomembrane

  • Replaces destroyed epithelium
  • Meshwork of fibrin, necrotic epithelial cells, PMN’s and various microorganisms
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8
Q

Clinical features of NUG

A
  1. Rapid onset
  2. Severe pain
  3. Gingival bleeding; may or may not be spontaneous
  4. Interdental Crater, punched put papilla, PSEUDOMEMBRANE, fetid (sulfur) breath
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9
Q

Etiology - Host Response of NUG

A
Systemic predisposing factors
Immunosuppression - AIDS
Stress
Smoking
Diet
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10
Q

Etiology - Oral Hygiene of NUG

A

Pre-existing gingivitis

Opportunistic microorganisms

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11
Q

Main Etiology of NUG

A
Microorganisms
  - P. intermedia, Spirochetes, Fusiformbacteria
Other Factors
  - Host resistance + smoking
  - Oral hygiene
  - Local irritants
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12
Q

In what zone is NUG of the Listgarten’s 4 zones

A

Zone 4: Zone of spirochetal infiltration

- Well-preserved tissue infiltrated with spirochetes

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13
Q

Describe the pseudomembrane feature of NUG

A

Replaces destroyed epithelium

Meshwork of fibrin, necrotic epithelial cells, PMN’s, and various microorganisms

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14
Q

Treatment of NUG

A
  • First visit - debridement, OHI
    • Evaluate systemic factors
  • Recall visit - 1-2 weeks, eval. OH, further tx
  • Final re-eval - 4-6 wks, consider surgery
  • If fever present give antibiotics first: Amoxicillin and Metronidazole
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15
Q

Extraoral and systemic signs and symptoms of NUG

A
  • Slight elevation in temperature
  • Local lymphadenopathy
  • Patients usually ambulatory with no systemic complications
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16
Q

What are the Listgarden’s 4 zones for NUG

A

Zone 1: Bacterial zone
- Superficial, contains bacteria
Zone 2: Neutrophil-rich Zone
- Numerous leukocytes, mostly PMN’s, bacteria, spirochetes
Zone 3: Necrotic Zone
- Dead cells, fibers, spirochetes, and other bacteria
Zone 4: Zone of spirochetal infiltration
- Well-preserved tissue infiltrated with spirochetes

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17
Q

What is NUP

A

Essentially that same as NUG, however there is bone loss involved in NUP

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18
Q

In what people can Primary herpes occur

A

In kids & Immunocompromised adults

- All races equally, both sexes

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19
Q

In what tissue does primary herpes occur?

A

Bound & Unbound tissue

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20
Q

Describe the 4 phases of clinical features of Herpes

A

Prodromal phase
Active phase
Latency
Reactivation

21
Q

Describe the clinical features of the prodromal phase of herpes

A

Fever
Irritability
Headache
Gingival Inflammation

22
Q

Describe the clinical features of the active phase of herpes

A

Vesicles rupture, yellow ulcers, red halo
Viral shedding (Ave. 12 days)
Mobile and Attached tissue
Heal in 7-14 days with no scarring

23
Q

Describe the clinical features of the latency phase of herpes

A

Virus resides in ganglia
Non-replicating state
Latency associated transcripts (LAT)

24
Q

Describe the clinical features of the reactivation of herpes

A
Recurrent herpetic stomatitis
Spontaneous or stimuli related
Virus migrates
Herpes labialis (extra-oral)
Bound down tissue - Attached gingiva (Intra-oral)
Virus overwhelms local immune response
25
What 4 things may be involved in virus activation
Sunlight Trauma Fever Stress
26
Describe herpetic whitlow
Recurrent infection Fingers and hands Latent site - dorsal root ganglion Debilitating
27
T or F, Primary Herpetic Gingivostomatitis is contagious
True
28
Diagnosis of herpes includes
History and clinical findings Tzanck smear Serum antibody titer ELISA or PCR
29
T or F, If young adults contract primary herpes it is less severe than if they were children
False, More severe
30
Treatment of primary herpes
``` Supportive treatment - Bed rest, bland mouthwashes (alcohol free) - Force fluids, Antipyretics - Lidocaine gel, CHX Systemic medication - Valcyclovir, Vibaradine, Acyclovir ```
31
Describe the periodontium in children or deciduous dentition
Pale pink Firm Either smooth or stippled (stippled in 35% of children between ages of 5-13) Interdental gingiva is broad facio-lingually, and narrow mesiodistally
32
Mean gingival sulcus depth in deciduous dentition
1mm
33
Is PDL of deciduous teeth more narrow or wider than permanent teeth
Wider than that of permanent teeth
34
Describe the trabeculae in alveolar bone of deciduous dentition
Trabeculae in alveolar bone are fewer but thicker than in the adult
35
Crests of interdental bony septa of deciduous dentition compared to permanent dentition
Deciduous dentition are flat
36
Chronic Marginal Gingivitis (deciduous dentition)
``` Most prevalent disease of childhood Looks like chronic gingivitis BOP and pocketing is less common Plaque etiology (less than in adults) - Calculus (4-6, 9%; 7-9, 18%; 10-15, 33-43%) ```
37
Malpositioned teeth in deciduous dentition:
``` Accumulate more plaque Increased gingivitis - excessive overbite - excessive overjet - nasal obstruction - mouthbreathing ```
38
Diseases altering oral mucosa including the gingiva in deciduous dentition
Varicella Rubeola (measles) Scarlatina (scarlet fever) Diphtheria
39
Describe the pre-eruption bulge
Before crown appears in oral cavity, the gingiva presents a bulge that is firm, slightly blanched and conforms to the shape of the underlying crown
40
T or F, during mixed dentition, it is normal for the marginal gingiva around the permanent teeth to be very prominent, especially in the maxillary anterior region
True
41
Suprabony pocket vs Intra/Infrabony pocket
Suprabony - Base of pocket is coronal to level of underlying bone - Horizontal bone loss - Transseptal CT fibers are horizontal Infra/Intrabony - Base of pocket is apical to the level of the adjacent bone - Vertical bone loss - Transseptal CT fibers run length of defect vertically (obliquely)
42
Why do we do pocket reduction therapy?
- Rationale for pocket reduction is based on the need to eliminate areas of plaque accumulation - Pockets create area where daily plaque removal becomes impossible
43
Define Periodontal abscess
Acute localized accumulation of viable and nonviable PMNs within the pocket wall
44
Define Gingival Abscess
Acute localized purulent infection that involves the marginal gingiva and interdental papilla due to bacteria carried into gingival tissue
45
Etiology or causes of Periodontal abscess
Extension from infected pocket: G- MO Incomplete removal of calculus Root fracture Multiple abscesses
46
Clinical features of periodontal abscess
Localized purulent inflammation in periodontal tissues Dull, constant pain, recent origin Edematous, erythematous, smooth, shiny surface Mobility Rapid pocket formation Discharge of pus with probe or pressure
47
Fenestrations vs Dehiscence
Fenestrations: Window in bone on facial or lingual Dehiscence: Loss of alveolar bone on the facial (rarely lingual) aspect of a toothat that leaves a characteristic oval, root-exposed defect from the cementoenamel junction apically
48
Normal bone morphology
1. Crest follows the CEJ and is 1-2 mm apical 2. Interproximal higher (more coronal) than facial and lingual 3. Scalloped 4. Usually thicker facial alveolar bone than lingual alveolar bone