Exam 3 Flashcards

(46 cards)

1
Q

what is afterload

A
  • pressure the heart must pump against to eject blood
  • pressure of aorta
  • mean arterial pressure
  • aortic mean pressure
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2
Q

what is cardiovascular drift

A

increased HR that occurs with no change in workload during endurance exercise

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3
Q

what are the different breathing irregularities that could affect exercise? how would they affect exercise?

A
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4
Q

how is maximum heart rate estimated

A

Tanaka equation
- HRmax = 208 - (0.7 x age)

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5
Q

how is max HR affected by training

A
  • HR increases as exercise intensity increases up to maximal heart rate
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6
Q

how is max HR affected by age

A

max HR decreases as age increases

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7
Q

Karvonen formula

A
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8
Q

how is the Karvonen formula used to calculate target heart rate

A
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9
Q

how and when is HR used to monitor exercise intenstity

A

Derived from LT or VT or RPE scale

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10
Q

what is the percentage of cardiac output that is distribute to skeletal muscles during rest

A

15-20%

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11
Q

what is the percentage of cardiac output that is distributed to skeletal muscles during exercise

A

80-85%

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12
Q

how does the body increase blood flow to certain areas of the body while decreasing blood flow to others

A

Decreased blood flow to less active organs, redistribution depends on metabolic rate and exercise intensity

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13
Q

how does SV increase with exercise

A
  • during exercise, more blood is pumped back to heart so there is more blood to be pumped, which increases SV
  • frank starling method, increased contractility via sympathetic nerve stimulation or circulating catecholamine (EPI & NORE release from adrenal medulla), reduced afterload (LV can eject greater volume because semilunar valve remains open longer)
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14
Q

how is SV altered with posture (laying down, standing)

A

SV upright exercise: SV doubles from resting to maximal values
- SV supine exercises: SV increases about 20-40%
- This happens because blood does not pool in legs, blood returns easier to heart→ resting SV are higher in supine position vs. upright position

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15
Q

how are SV of elite athletes different from SV of trained and untrained individuals

A
  • increases up to 40-60% VO2max in untrained & up to max levels in trained
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16
Q

explain the integration of the cardiovascular system’s response to exercise

A

Increased LV wall thickness and increased LV chamber size, LV mass is highly correlated with VO2 max
- Decreased ESV facilitated by decrease in peripheral resistance
- Increased a-VO2 difference accounts for 50% of increased VO2 max

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17
Q

how do we buffer against acidosis in the blood

A

First line: cellular buffers- proteins, bicarbonate and phosphate groups - Blood buffers- bicarbonate, hemoglobin, and proteins
- Second line: respiratory compensation- increased ventilation in response to increased H+ concentration

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18
Q

how do we buffer against acidosis in the skeletal muscle

A
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19
Q

what is normal blood pH

20
Q

how is VE increased with exercise

A

VE increased in direct proportion to metabolic needs of exercising muscle

21
Q

how is VE calculated

A

VE = Tidal volume (TV) x breathing frequency (f)

22
Q

why is ventilation closely regulated by exercise intensity

A

body needs to match oxygen delivery and carbon dioxide removal to the metabolic demands of working muscles

23
Q

explain how VT is related to exercise performance & how it relates to acidosis (bicarbonate buffer system)

A

VT is used to characterize endurance athletes, predict endurance performance, describes the ability to sustain high intensity exercise and allows for the correlation of muscle fiber composition and biochemical properties of skeletal muscle

24
Q

what is the Fick equation

A

VO2=Q x a-VO2diff

25
what is the main limiting factor of VO2max
Q (cardiac output)
26
what is mean arterial pressure? what factors affect MAP?
MAP = 2/3 DBP + 1/3 SBP - MAP = Q x TPR --> vasodilation radius of vessels shrink --> TPR will lessen
27
what are cardiovascular & metabolic adaptations to moderate and high intensity endurance training
- improved ability of the muscle to extract oxygen from the blood which is achieved by: increased heart size, stroke volume, Q, muscle blood flow and total blood volume - anaerobic- both type 2a and 2b cross sectional area increases, greatest change in 2a
28
what are the training principles
- individuality: everyone is unique - specificity: doing drills unique or specific for a sport - reversibility: use it or lose it - progressive overload: getting stronger by doing heavier weights - variation: changing up workouts - diminishing returns: as you get stronger, need to work harder for gains or improvement; as you are weaker its easier to get gains and improvement
29
what is muscular power
rate at which work is performed - product of force & velocity - power = force x distance/time - explosive strength & speed of movement
30
compare muscular endurance and strength
31
contrast muscular endurance and strength
32
what is concurrent training
training for endurance & strength on the same day - strength first then endurance
33
how is anaerobic power/capacity measured
wingate test
34
what is AOD
accumulated oxygen deficit - difference between actual VO2 and theoretical VO2 during exercise - can be used to quantify non-mitochondrial energy release during one bout of high intensity exercise - measures anaerobic metabolism
35
what is the difference between hyperplasia & hypertrophy
- hyperplasia: increased number in muscle fibers - hypertrophy: an increase in muscle mass & cross sectional area
36
what are the neuromotor adaptations to strength training
- increased recruitment of muscles - increased synchronization of recruitment - decreased autogenic inhibition - decreased co-activation of antagonists
37
what are the muscular adaptations to endurance training
develop larger cross sectional area, increases up to 25% have been reported, magnitude depends on intensity, duration and length of training program
38
what stimulates protein synthesis
Individual muscle fibers from resistance training appears to result from net increase in muscle protein synthesis - Protein is always being synthesized and degraded - During exercise protein synthesis decreased while protein degradation increases - A single bout of resistance exercise can elevate net protein synthesis for up to 24 hours
39
what are the RDA for protein intake
0.8 g per kg of body weight/day
40
outline resistance training recommendations for children & older populations
41
what is sarcopenia
- loss of muscle mass - loss of both type I and II fibers - atrophy of type II fibers - loss of intramuscular fat & connective tissue
42
what is OTS
43
how does OTS compare with overreaching
Overarching is pushing the athlete to just the limit that they need to succeed at what they need to without hitting OTS
44
what methods can be used to detect OTS
Running different tests Hormones Psychological Biochemical markers Heart rate variability
45
what is muscle atrophy
Loss of muscle size and fibers and number of fibers
46
what are some physical & cellular changes that occur with detraining
Loss of the recruitment and synchronization of muscles Gets weaker Loses size of muscles Neuron not firing may cause this