Exam 4 Flashcards
(50 cards)
what is WBGT
- wet bulb globe temp
- accounts for conduction, convection, evaporation, radiation
- better tool to evaluate level of environmental stress during exercise
how is a sling psychrometer used to measure wet bulb globe temp (WBGT)
- wbgt = 0.1Tdb + 0.7Twb + 0.2*Tg
- wet bulb, dry bulb, globe
- use sling psychometer to measure Tdb & Twb (Tg = Tdb)
what are the different heat illnesses
- heat cramps: caused by loss of sodium & dehydration (due to hugh sweat rates) or accumulation of fluid in muscle cells (lactate draws water into muscle) or both; most common in heavy sweaters who are poor sodium conservers; proper hydration, liberal salt intake with food & in bevs consumed during exercise
- heat exhaustion: diziness, fatigue, nausea, vomiting, fainting, weak & rapid pulse, pale & cool skin, Tcore < 39 degrees c, cant dissipate heat quick enough because insufficient blood volume is available to allow blood flow to skin, unfit or unacclimated people are most susceptible, Tx: rest in cooler environment with feet elevated, salt water ingestion, iv admin of saline solution
- heat stroke: life threatening, Tcore > 40 deg c, altered mental status, cessation of active sweating, whole body immersion, teperate water immersion, wrapping entire body in cold, wet sheets, vigorous fanning
what heat illnesses are life threatening
heat stroke
what heat illnesses are the least severe
heat cramps
what are the ways body heat is transferred to/from the body
- conduction: direct molecular contact with object
- convection: motion of gas or liquid across heated surface
- radiation: infrared rays (at rest 60% of excess heat is lost through radiation, exercise heat loss via radiation 5%)
- evaporation: primary avenue for heat dissipation during exercise, when air temp is close to skin temp evap is only way of cooling, accounts for ~80% total heat loss during evap, 10-20% heat loss at rest, heat loss not gain
what is the main avenue for heat dissipation at rest
radiation
what is the main avenue for heat dissipation at exercise
evaporation
what is the most effective way to cool a person who has a heat stroke
whole body immersion
what is the difference between hypohydration & dehydration
- hypohydration: decrease in PV only, state of insufficient body water, body is below normal hydration level
- dehydration: decrease in PV & increase in osmolality, losing water leading to a water deficit
what are ways humans adapt/acclimate to the heat? which adaptation occurs the soonest?
plasma volume increases, heart rate decreases, earlier sweat onset, decrease in perceived exertion, decrease NaCl sweat, increasing sweat rate, decrease T core
- earliest: early onset of sweating which leads to increased PV & reduced HR
what is pokilothermic
- 30 C (86 F): shivering ceases & body becomes poikilothermic (passively cools to Ta)
- homeostasis body reg of internal temp
how does age affect our ability to thermoregulate
- reduces simulated body fluid (SBF) & degree of vasoconstriction in response to cold air exposure
- possible that blood vessels cant maintain constrictor tone effectively
as humans, how do we adapt to the cold? what is the difference between the way we adapt vs the way Ama divers adapt to the cold?
- habituation: blunted, less vigorous shivering in response to Ta
- metabolic pattern: less observed than habituation, greater heat production, more pronounced shivering & non shivering thermogenesis in cold
- insulation pattern: less observed than habituation, augmentation of VC responses in skin & superficial muscles, may involve increased fat thickness in subcutaneous tissues
- conserve E rather than conserve heat
- ama:
> insulative: better better VC
> metabolic (increased BMR)
> habituation: increased tolerance to cold (less shivering, later onset of shivering)
what is the main substrate we rely on in cold environments? why?
fats because we rely on thermogenesis which burns fat to create heat
what effect does ascent to altitude have on the hemoglobin-O2 dissociation curve
leftward shift causes hemoglobin binds oxygen at a partial pressure less oxygen delivery at tissue level
what are the different altitude illnesses
- acute altitude mountain sickness: low ventilatory response to altitude, co2 accumulates, acidosis, mostly experienced >3600 m, continous & throbbing, hypoxia > cerebral vasodilation > stretch pain receptors
- altitude sickness insomnia: interruption of sleep stages due to hypoxia
what are the renal, respiratory, CV, and metabolic responses to altitude
- kidneys excrete more bicarbonate to increase pH
- pulmonary ventilation increases immediately
- ventilation at altitude = hyperventilation
- respiratory alkalosis = high blood pH
what are the differences between the physiology of highlands & lowlanders when residing at altitude
- larger heart dimensions
- greater cardiac muscle mass
- larger lungs
- more capillaries
- smaller body size with larger chest (larger lung capacity for smaller body)
- more coronary artery circulation = greater capacity for o2 diffusion across lungs capillaries
what is lactate paradox`
- inc in anaerobic metabolism = inc in lactic acid
- lactic acid production decreases over time
- no explanation
what is hypocapnia
condition with a reduced amount of carbon dioxide in blood (usually below 35 mmHg)
what effect does ascent to high altitude have on vo2max
- decreases as altitude increases past 1500 m
- lowers because HR decreases and less o2 to breathe
how does altitude affect aerobic performance
- declines
- low alt (may be decreased, restored by acclimation)
- moderate alt: performance and aerobic capacity decline, may or may not be restored by acclimation
- 1500 m difference produced 7% impairment in repeat spring performances
- high alt: performance declines, not restored by acclimation
- few if any physiological effects <1500 m
how does altitude affect anaerobic performance
- unaffected
- minimal o2 requirements
- ATP-PCr & anaerobic glycolytic metabolism