Exam 3 - Abomasal Disorders Flashcards
(29 cards)
what is the ‘classical’ abomasal impaction animal?
beef cattle on poor forage diets
what is the pathogenesis of classical abomasal impaction?
firm viscus in right anterioventral abdomen - hypochloremic metabolic alkalosis
how is classical abomasal impaction treated?
abomasotomy - poor success rate
T/F: it is more common to see abomasal impaction secondary to TRP than the ‘classical’ presentation
true
what is the most common presentation of abomasal impacttion?
post partum dairy cattle in the first 4 weeks after calving attributed to dietary changes that occur around calving
how is abomasal impaction secondary to TRP treated?
lavage the abomasum through rumenotomy, use mineral oil or psyllium - poor success rate
what are the clinical signs of abomasal impaction in post partum dairy cattle?
poor appetite, ketosis, floating LDA suspects
what is the surgical & medical treatment for abomasal impaction in post partum dairy cattle?
surgical - manual breakdown
medical - fluids & mineral oil/psyllium
what are the 3 main causes of abomasal impaction?
- classical - beef cattle on poor forage diets
- secondary to TRP
- post partum dairy cattle
what sheep breed is associated with having an abomasal emptying defect?
suffolk breed
where are the common ulcer locations for calves (beef/veal/dairy/feedlot)?
beef - mid fundus on greater curvature
veal - pyloric antrum
feedlot - pyloric region
dairy calves - fundic region
if an animal has only an ulcer (no blood loss), what clinical signs may be seen?
decreased appetite/milk production, bruxism, salivation, mild abdominal pain
if an animal has only an ulcer with blood loss, what clinical signs may be seen?
weakness, hemorrhagic shock, melena
decreased appetite/milk production, bruxism, salivation, mild abdominal pain
what are some causes of abomasal ulcers?
not entirely known - low abomasal pH (off feed - not influenced by trichobezoars), & decreased abomasal blood flow
what clinical signs are seen in an animal with an ulcer & peritonitis?
increased WBC in peritoneal fluid, local peritonitis, diffuse peritonitis, & recumbent with expiratory groan
if an animal has a gi ulcer that is causing symptoms, what treatment should be used?
get the animal to eat!!!!! saliva production/buffer
if an animal has a gi ulcer that is bleeding, what treatment should be used?
antibiotics, oral antacids, blood transfusions, get animal to eat
if an animal has a gi ulcer that has perforated, what treatment should be used?
antibiotics
what is the suggested etiology of C3 ulcers in camelids?
unclear - stress, concurrent disease, heavy concentrate feeding, NSAIDS
what is the pathogenesis of C3 ulcers in camelids?
speculative - poor blood flow & localized trauma
**camelid C3 anatomy
what treatment has proven efficacy for C3 ulcers in camelids?
pantoprazole - 1mg/kg IV SID; 1-2 mg/kg SQ SID
what treatment has been shown not to work for C3 ulcers in camelids?
cimetidine, ranitidine, & oral omeprazole
how can C3 ulcers be diagnosed in camelids?
ultrasound, endoscopy, bloodwork (anemia/hypoproteinemia), fecal occult blood, & serum pepsinogen
