Exam 3 - Cindy's Flashcards

(48 cards)

1
Q

Concussion

A

Mild brief loss of neurological function

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2
Q

DAI

A

Prolonged coma caused by coup/countercoup injury

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3
Q

Epidural hematoma (EDH)

A

ARTERIAL bleeding, bleeding between dura and skull; middle meningeal artery rupture, Brief loss of consciousness, followed, by a lucid period and then deep coma

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4
Q

Craniotomy Care

A

Post-Op: neuro checks, monitor for IICP, check airway, pain, nutritional status and any CSF leaks

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5
Q

Complications of SAH (subarachnoid hemorrhage)

A

Vasospasms
Rebleeds
Hydrocephalus

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6
Q

C-1 thru C-4 Cervical spine injury

A

Assess airway, lose diaphragm use, ventilator dependent

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7
Q

Major complications of spinal cord injuries

A

DVT / PE

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8
Q

Autonomic dysreflexia

A

Exaggerated autonomic response t visceral stimulation occurring with injuries above T6.
MEDICAL EMERGENCY

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9
Q

TX/Interventions for Autonomic dysreflexia

A

Elevate HOB, check for bowel impaction, bladder distenstion, and kinks in foley, evaluate skin for pressure areas. Medicate for HTN using HYPERSTAT, Resperpine or Atropine

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10
Q

Plasmaphresis

A

Infection, hypovolemia, clotting abnormalities, hypokalemia, hypocalcemia, hypotension, myasthenic and cholinergic crisis

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11
Q

Contusion

A

Bruising of the Brain

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12
Q

Subdural hematoma (SDH)

A

VENOUS bleeding, between dura and arachnoid layers; see progressive neuro changes over 2-4 weeks

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13
Q

SIADH

A

Complication post head injury, see coma, may have lung crackles decreased urine output

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14
Q

Subarachnoid hemorrhage (SAH)

A

Bleeding into subarachnoid space between pia and achachnoid layers, usually from rupture of Berry aneurysms.

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15
Q

Nimotop (nimodipine)

A

Prevents vasospasms, give for 21 days

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16
Q

Spinal shock

A

May last weeks to months, see flaccid paralysis below the level of lesion, lose temp control, see hypotension

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17
Q

Use of steroids in spinal cord injury

A

Treats secondary injuries, decreases swelling/inflammation of cord

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18
Q

S/S of Autonomic Dysreflexia

A
Severe HTN
HA
Bradycardia
Flushing of the face/neck
pupil dilation
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19
Q

Myasthenia Gravis

A

Affects neuromuscular transmission of voluntary muscles. See excessive weakness and fatigue of voluntary muscles. Any age, more common in women ages 14-35 and men over 40. Tensilon test will confirm diagnosis

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20
Q

Guillian-Barre syndrome

A

Rapidly ASCENDING peripheral and cranial nerve dysfunction, leading to paralysis. Respiratory arrest is most common cause of death. check vital capacity and ABGs every shift in assessing respiratory function.

21
Q

Monroe-Kellie Doctrine

A

An increase in blood, CSF, or brain tissue is accompanied by a reciprocal change in the volume in one of the others

22
Q

How to estimate cerebral perfusion pressure. What is normal CPP?

A

CPP = MAP - ICP

Normal CPP is 60-100 mmHg

23
Q

Factors that increase ICP

A

Valsalva, coughing, sneezing, body positions, neck flexion

24
Q

Cushing’s Triad

A

Increased systolic pressure with decreased diastolic pressure (widened pulse pressure) and bradycardia

25
ICP Monitoring
Zero and maintain at Foramen of Monroe (corner of eye) never flush if connected to hemodynamic system
26
Pharmacological management of ICP
Mannitol, lasix, steroids, barbituates, and anticonvulsants
27
Assessment of neuro status
Restlessness
28
Basal skull Fracture s/s
Raccoon's eyes, battles sign
29
Kernig's sign
Cannot extend leg when thigh flexed on abdomen (menningeal irritation)
30
Posturing
Decorticate: flexion of arms to center Decerebrate: extension of arms
31
DKA
Glucose 200-800 Positive serum and urine ketones Insulin dependent diabetics (TYPE 1) Acetone smell to breath
32
HHNS / HHNK
``` Glucose 800-2000 No ketones Serum osmolarity high Elderly Higher mortality rate ```
33
DIC
Overstimulation of normal coagulation mechanisms, leading to microvascular thrombi, consumes all coagulation factors which leads to hemorrhage
34
Labs in DIC
Platelets decreased Fibrinogen level decreased PT/PTT prolonged Fibrin degradation products (FDP) increased
35
Shock
hypo perfusion of body tissues
36
Types of shock
Volume (hemorrhage) Pump (cardiac) Vessel (sepsis and anaphylaxis)
37
MAST
Military anti-shock trousers, used to shunt blood from legs to abdomen and vital organs for perfusion
38
Burn rule of 9s
``` head & neck = 9 arm = 9 ea trunk =18 front/ 18 back legs = 18 ea perineum = 1 ```
39
Calculation of fluid resuscitation for burns
``` parkland formula 4mL/kg X %BSA burned = fluids for first 24hrs -1st 8hrs = 1/2 total Amt -2nd 8hrs = 1/4 total Amt -3rd 8hrs = 1/4 total Amt ``` LR is solution of choice
40
Shock stage of burns
Hypovolemic: fluids shift from vascular to interstitial space, see hemoconcentration and edema. Fluids and electrolytes are lost. HYPERKALEMIA, HYPONATREMIA Diuretic phase: fluids shift back to vascular space, see hemodilution and diuresis.
41
Priorities in burn care
-Airway management first, then fluid resuscitation
42
Cause of myoglobinuria
Skeletal muscle breakdown, increased Ca+ in and destroys muscle fibers. urin is dark rusty brown to black
43
Extremity management in burns
If circumferential, monitor pulses and venous return, monitor for numbness and pain. An escharotomy may be necessary.
44
Wound care in burns
``` Cleansing (hubbard tank) debridement Silvadene (painless) Sulfamylon (painful) Silver nitrate (stains black) ```
45
Primary Assessment for Trauma
Airway breathing circulation, control and external hemorrhage, IV, MAST, immobilize potential fractures Disability, Exposure Fahrenheit, Get full set of vitals, History
46
Secondary assessment for trauma
More thorough and complete assessment Intubate, Place on 100% O2, Stabilize fractures Additional labs and x-rays, possible peritoneal lavage or abdominal scan, OR, ICU, ongoing assessments
47
Factors that have contributed to success of organ transplantation
Cyclosporine Surgical techniques Organ and tissue preservation Legal definition of brain death
48
Drugs used to prevent rejection
Cyclosporine, steroids, prograf