exam 5- gi Flashcards

(167 cards)

1
Q

GI System organs

A

mouth,

pharynx,

esophagus,

stomach,

small intestine,

large intestine,

liver,

gallbladder,

pancreas

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2
Q

Subjective data for gi system

A

focused or part of total health assessment –

nutrition, diet screening.

Dietary habits and history of gi symptoms,

normal weight,

any weight loss,

meds,

current health history,

socioeconomic status

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3
Q

Objective data-

A

physical assessment –

ht
weight,
bmi,
oral, abdominal assessment,
bowel sounds and last bowel movement, passing flatus

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4
Q

Barium Swallow/upper GI Series

look for
drink
observing
diet
cannot take
eliminate by
stool

Diagnostic Tests- Esophagus & Stomach

A

-looks for inflammation, ulcerations, hernia and polyps.

Pts drink 16-20 ounces of barium-

observing movement of barium contrast by bronchoscope.

diet may be altered a few days before.

Pts cannot take narcotics or anticholinergics 24 hrs before /

/ after study- eliminate barium by increasing fluids for 48 hrs,

stool may be light in color

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5
Q

Esophageal Acidity –
diagnoses
inserted
normal

Diagnostic Tests- Esophagus & Stomach

A

diagnoses lower esophageal sphincter issues and chronic reflux.

A catheter with a ph probe is inserted

normal is between 5-6

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6
Q

Gastric Emptying Studies

evaluates

Diagnostic Tests- Esophagus & Stomach

A

–evaluates ability of stomach to empty liquids or solid

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7
Q

MRI
identify
no
need

Diagnostic Tests- Esophagus & Stomach

A

–identify sources of bleeding,

no metal implants,

need to lay flat and still

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8
Q

EGD- esophagogastroduodenoscopy –

direct
looking
pts need
monitored
local
check

Diagnostic Tests- Esophagus & Stomach

A

direct visualization of esophagus, stomach, duodenum-

looking for polyps or ulcers,

pts need to be npo,

monitored for anesthetic care/

/ local anesthetic is used in the throat

check gag reflex prior to giving them anything

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9
Q

Barium Enema –

used to identify
contrast
colon must be
liquids prior
give what after
increase

Diagnostic Tests- Intestines

A

used to identify abnormalities of the colon or rectum,

contrast medium and rectal area is looked through scope,

colon must be clear of fecal content,

clear liquids prior ,

given laxatives and enemas after,

increase fluids

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10
Q

Colonoscopy -,

entire/looking
pts
liquids
taking/tolerating
if coming in
after education

Diagnostic Tests- Intestines

A

entire colon, looks for polyps, tumors, bleeding , strictures,

pts are npo

are clear liquids,

taking oral bowel prep, must be able to tolerate oral prep,

if coming in for n/v or gi complaints bowel prep may be challenging/

/ after procedure educate on abdominal cramping and flatus for a few hrs

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11
Q

Guaiac Fecal Occult Blood

checking

Diagnostic Tests- Intestines

A

– checking fecal matter for hidden blood

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12
Q

Stool Culture –

what looking at

Diagnostic Tests- Intestines

A

looking at form, consistency, color and odor

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13
Q

Lower GI Series –

drink
film
must tolerate
increase
change

Diagnostic Tests- Intestines

A

drink contrast medium,

films are taken and can be used with upper gi series or barium swallow,

must be able to tolerate bowel prep to get rid of fecal matter,

increase fluids

changes in stool color

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14
Q

Ultrasound –

what is it
checks

Diagnostic Tests- Gallbladder, Pancreas, Liver

A

high frequency sound waves that pass through the body structures

to check for abnormalities

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15
Q

Cholangiogram

contrast
evaluates

Diagnostic Tests- Gallbladder, Pancreas, Liver

A

–contrast medium is injected into the common bile duct

to evaluate filling

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16
Q

CT -

what is it
might need

Diagnostic Tests- Gallbladder, Pancreas, Liver

A

360 view of body structures,

might need oral contrast,

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17
Q

Endoscopic Retrograde Cholangiopancreatography (ERCP)-

visualizes
retrieves

Diagnostic Tests- Gallbladder, Pancreas, Liver

A

visualizes gi structures

, retrieves gallstones from bile duct and dilate structures

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18
Q

Magnetic Resonance Cholangiopancreatography (MRCP)-

non
evalautes
no
lie

Diagnostic Tests- Gallbladder, Pancreas, Liver

A

non invasive mri

evaluate biliary and pancreatic ducts

, no metal implants,

lie flat and still

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19
Q

Liver Biopsy –

rules out
monitor/no
pts
preop
check
needle
after biopsy

Diagnostic Tests- Gallbladder, Pancreas, Liver

A

used to rule out metastatic cancer, liver cirrhosis

, monitor anticoagulants prior, no aspirin and ibuprofen a week prior to biopsy,

pts are npo,

vitals preop,

check bleeding times,

needle is inserted into upper right quadrant,

after biopsy pts are laying on right side to maintain pressure

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20
Q

Stomatitis-

common
oral mucosa
mucosal lining

A

Common disorder of mouth

Inflammation, ulcers of the oral mucosa

Thin, fragile mucosal lining is damaged leading to significant pain and discomfort

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21
Q

Stomatitis-

causes

infections
t
i
nd
ca

A

Viral, bacterial or fungal infections,

trauma,

irritants,

nutritional deficiencies,

chemo agents

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22
Q

who’s at risk for Stomatitis-

A

immune compromised patients,

chemo pts,

frail elderly pts,

HIV,

corticosteroids

dentures

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23
Q

Stomatitis-

diagnosis
DV
C
sm
les

A

direct visualization and physical exam ,

cultures,

smears for systemic illness,

lesions spreading down into esophagus

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24
Q

Stomatitis-

manifestations-pain/inability

leads to

A

oral pain,

inability to eat, drink or swallow,

leads to complications of malnutrition and fluid and electrolyte imbalances ,

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25
treatment Oral hygiene
–soft toothbrush, avoid alchhol based mouthwashe
26
medications Stomatitis- Coating agent contain dont
-coat the mouth Often contain lidocaine – don’t want to swallow,
27
- Fungal meds stomatitis treats what med+considerations oral antifungl meds-
treat thrush or candidas Nystatin- oral suspension//swish and swallow Oral antifungals- fluconazole, ketoconazole
28
Medications- Viral meds stomatitis
Topical/oral antiviral – acyclovir, valacyclovir
29
Stomatitis- type--Cold sore, Fever Blister causes manifestation treatment
Causes--Herpes Simplex Virus manifestations--Burning, vesicular lesion treatment--Resolves on own Antiviral - “cyclovir”
30
stomatitis type--Aphthous ulcer- Canker Sore causes manifestations treatment
causes--unknown; herpes virus manifestations- Shallow white/yellow erosions with red ring treatement- Topical steroid, numbing agents
31
Candidiasis (thrush)- stomatitis causes manifestasions treatments
causes--Candida Albicans manifestations-White, curd-like patches treatment-antifungals- nystatin, fluconazole, ketoconazole
32
Stomatitis- health promotions identify education(eliminate/if chemo/ )
Identify risk factors –oral care, poor denture use, Educate –eliminate spicy/hot foods, if on chemo, avoid irritants like alcohol and tobacco
33
Priorities of Care hygiene maintain
Oral hygiene, prevention, treatment Maintain adequate nutrition- hydration
34
GERD- what is it what results from
Backward flowing of gastric contents Results from  relaxation of esophageal sphincter and increased pressure from stomach
35
GERD contributing factors increased p hh environmental
increased gastric volume and pressure positioning hiatal hernia environmental/diet-smoking, alcohol, coffee, chocolate
36
GERD- Manifestation other manifestations
Heartburn –usually after meals, when bending over Atypical chest pain, Sore throat , Hoarseness
37
gerd complications of manifestations
Esophageal strictures, leading to dysphagia Barrett’s esophagus –cell wall changes leading to cancer risk
38
GERD- Diagnosis
Barium Swallow; evaluate GI track Upper endoscopy; direct visualization, Bx Bernstein Test; Saline & acid solution mimicking symptoms 24-hour pH monitoring Esophageal manometry; pressure measurement of sphincters & peristalsis
39
PPIs drug how work before do not meds GERD- Treatment
–drug of choice, reduce acid secretion, before meals, do not crush, - omeprazole and pantoprazole
40
H-2 Receptor Blockers – how work what meds GERD- Treatment
reduce acidity of gastric juices. Famotidine, rimantadine,
41
Anti-ulcer Agent – how work when take GERD- Treatment
react with gastric acid to fomr thick coating or paste, coats damaged gastric tissues and promotes healing, 1hr before meals and bedtime,
42
antacids how work what med when take GERD- Treatment
buffer/neutralize gastric acids, relieve pain and prevent damage, tums, sperate 2 hrs from other meds
43
motility Agent- how work what med GERD- Treatment
stimulate gi tract motility and gastric emptying, metoclopramide/reglan
44
nutrition and lifestyle GERD- Treatment
Ideal body weight & diet selection
45
surgery how work GERD- Treatment
Lap/Nissen Fundoplication Increases pressure in lower esophagus , inhibiting reflux
46
health history priority education GERD- Nursing Care
Heath history, symptoms- manifestations, what foods are irritants, positioning issues, Priority- symptom relief Educate; irritants to avoid –stay away from meds that irritate gi tract like aspirin and nsaids, naproxin
47
Hiatal Hernia-
portion of stomach that protrudes through diaphragm, increases with age
48
Hiatal Hernia- manifestations
Reflux, fullness, chest pain, dysphagia, bleeding, belching, indigestion
49
Hiatal Hernia- diagnosis
barium swallow upper scope
50
Hiatal Hernia- treatment-similar surgery why keys to prevention-(ideal, remain, no)
Similar to GERD Surgery- if conservative treatment is unsuccessful Keys to prevention 1-ideal body weight 2-remain upright for 2 hrs after eating 3- no eating before bedtime
51
Peptic Ulcer Disease- what is gi tract-where results
Break in mucous lining of GI tract where is comes in contact with gastric juices GI Tract- duodenum most common Results in peptic ulcers
52
Peptic Ulcer Disease- risk factors
asprin & NSAID use –inc risk of bleeding Age, history of ulcers Smoking H. Pylori infection
53
Peptic ulcer disease manifestations classis symptoms
pain (indigestion), epigastric, below sternum area Pain-food-relief pattern- pain 2-3 hrs after meals and in middle of night which is relieved by eating
54
Peptic ulcer disease manifestations vague symptoms
hemorrhage obstruction perforation
55
Peptic ulcer disease manifestations Zollinger-Ellison Syndrome what is it what does/leads to
Gastrin secreting tumor- hypersecretion of gastric acid leading to ulceration
56
Peptic Ulcer Disease- diagnosis upper gi endoscopy- fecal analysis gastic analysis
Upper GI Endoscopy & biopsy- rule out ulcer and provides direct visualization of ulcers and surrounding tissues Fecal Analysis-stool analysis and looking for h pylori Gastric Analysis- for suspected seilinger Ellison syndrome,
57
Peptic Ulcer Disease- Treatment therapy discontinue receptor-promote __protectant +meds
Antibiotic Therapy- 14 days & PPI Discontinuation of NSAIDs PPIs & H2 receptor PPI’s faster healing relief Mucosa protectant Sucralfate, bismuth compounds, antacids
58
Peptic Ulcer Disease- Treatment Cont nutriton surgery
Nutrition Balanced meals, regular intervals No smoking -slow healing Surgery Needed due to complications of PUD –if hemorrhage , perforation or obstruction
59
Hemorrhage goal what does nurse do Peptic Ulcer Disease- Complication Management
Goal restore circulation NPO, PPI’s via IV, iv fluids, blood transfusion, ng tube
60
obstruction due to gastric decompression Peptic Ulcer Disease- Complication Management
Due to repeated inflammation, healing, scarring, edema Gastric decompression- ng tube, iv
61
Perforation-Peritonitis risk positioning needs Peptic Ulcer Disease- Complication Management
Risk contamination, treat with ng tube and antibiotics, Positioning  semi fowlers so contaminates pool in lower abdomen/ pelvis Surgery
62
Peptic Ulcer Disease- health promotion-avoid priorities in care
_avoid nsaids, aspirin Priorities in care reducing discomfort, nutritional status, identifying and preventing compliacations
63
Gastritis- what is most often from 3 kinds
Inflammation of stomach lining Most often from gastric irritants such as : aspirin, alcohol, caffeine or contaminated foods Acute, Chronic, Erosive
64
acute gastritis local why is it what causes
*Local irritant *Acid and pepsin comes in contact with gastric tissues- inflammation, irrigation, superficial erosion
65
acute gastritis manifestations
*Anorexia, N&V, melena, pain, hematemesis, shock
66
chronic gastritis overview progressive h autoimmune
Progressive disorder, atrophy of gastric tissues H. Pylori gastritis Autoimmune gastritis- intrinsic factors affected, unable to absorb B 12- pernicious anemia
67
chronic gastritis manifestations
Vague, asymptomatic Fatigue & Anemia
68
erosive gastritis overview induced ischemia maintain
Erosive, stressed induced- trauma, shock, burns, head injury, major surgery Ischemia of gastric tissue from massive vasoconstriction Maintain pH with medications to prevent acid secretion
69
erosive gastritis manifestations
Painless gastric bleeding couple days after stressor
70
Gastritis- Diagnosis -why h pylori gastric analysis hh rbc b12 upper endoscope
H. Pylori Testing-source of inflammation Gastric Analysis-gastric content H&H, RBC-anemia B 12 levels-perniscous anemia-unable to absorb b 12 Upper Endoscope-gastric mucosa
71
medications Gastritis- Treatment
PPIs, H2 receptor blockers, sucralfate, antibiotics
72
NPO status slow intriduction keep hydrated Gastritis- Treatment
Slow introduction clears-full liquids-general keep hydrated- iv fluids
73
gastric lavage why used ___via lavage nursing considerations-do not Gastritis- Treatment
Used with poisonous or corrosive ingestion Dilution and removal via lavage Nursing Considerations  do not induce vomiting, further damage could occur
74
complementary therapy Gastritis- Treatment
Herbal & aromatherapy- chamomile tea, garlic, ginger, mint
75
health promotion assessment priorities in care gastritis nursing care
Health Promotion Prevention, safe food preparation Assessment Health and history, symptoms, risk factors Priorities in care Pain management, healing of tissue, nutritional status
76
Dumping Syndrome- common complication following what patho- quick/stimulated/ increased/ leading to
Common complication following gastrectomy or gastric bypass patho Quick food bolus, drawing fluid into duodenum Peristalsis is stimulated Intestinal motility is increased Leading to dumping and Systemic symptoms
77
systemic symptoms dumping syndrome
tachycardia, hypotension, flushing, , diaphoresis dizziness
78
Dumping Syndrome- management meals separate rest in after
Small, frequent meals Liquids and solids separate Rest in recumbent/semi-recumbent 30-60 minutes after meals
79
Pyloric Stenosis- what is what does it cause
Pyloric sphincter muscle doesn't empty properly and is unable to pass fluid causes projectile vomiting
80
pyloric stenosis assessment(4-6 weeks/ watch for) treatment(/ww/s)
Assessment 4-6 weeks of age, vomiting after feedings, projectile vomiting Watch for s/s of dehydration Treatment Watch & wait Surgery
81
Irritable Bowel Syndrome aka what looks like manifestations
aka spastic bowels Abdominal pain, bloating, constipation and/or diarrhea Manifestations: change in bowel habits, frequency, form, passage of mucous
82
Irritable Bowel Syndrome- Diagnosis ss gs bs be b
Stools samples , GI scope, bowel series, barium enema, biopsy
83
Irritable Bowel Syndrome- Treatment meds
Bulk forming laxative- Anticholinergic- reduce spastic bowel and decrease bowel motility Anti-diarrhea- Antidepressants- ssri or tricyclics
84
Education for irritable bowel syndrome additional avoid
; dietary habits- additional fiber , avoid trigger foods like dairy, caffine and soda
85
Appendicitis- inflammation obstruction pressure leading to perforation =
Inflammation of the appendix obstruction of proximal lumen Pressure builds, leading to inflammation, edema, infection Perforation= peritonitis
86
mcbunreys point Appendicitis- tenderness relief/release
Localized and rebound tenderness at “McBurney’s point” relief of pain when direct palpitation followed by pain on release
87
Appendicitis- Diagnosis treatment surgery
Ultrasound, CT scan, WBC Hydration, antibiotic therapy, pain medications PRN surgery- appendectomy
88
Gastroenteritis- what is it culprit how do infectious organisms enter body
What is it? Inflammation of the stomach and small intestine Culprit  bacteria, viruses, parasites or toxins Infectious organisms enter body in contaminated food or water- food poisoning
89
manifestations Gastroenteritis-
Anorexia, n/v, abdominal pain, cramping, diarrhea
90
complication-due to how treat gastroenteritis
Electrolyte imbalances due to vomiting IV fluid - hydration stabilization
91
Gastroenteritis- diagnosis
Labs, stool samples, lower GI Scope
92
Gastroenteritis- treatment meds- what types nutrition-what give gastric lavage-what does
Medications - Antibiotics, antidiarrheal Nutrition  fluid supplements, oral electrolytes, Gastric Lavage  washes out stomach and lining
93
Inflammatory Bowel Disease- two conditions
ulcerative colitis Crohns
94
Ulcerative Colitis what is it
Chronic inflammatory bowel disorder of the mucosa and submucosa of distal colon and rectum
95
Ulcerative Colitis diarrhea-> ranges cramping/releif manifestations
Diarrhea  blood in mucus with abdominal pain Ranges from mild-severe Left lower quad cramping- relieved by defecation
96
ulcerative colitis complications TM P MH CC
Toxic megacolon, perforation, massive hemorrhage, colorectal cancer
97
Chrons what is it
Chronic, relapsing inflammatory process affecting GI tract (Ileum & Ascending colon)
98
chrons manifestations persistant pain
persistent diarrhea, no blood or mucus Right lower quad pain, periumbilical
99
chrons complications O F AF M CC
Obstruction, fistulization, abscess formation, malabsorption, colon cancer
100
Ulcerative Colitis & Crohn’s- inspection stools C__/H_ elevated low diagnosis
Inspection of bowel (Scope, barium x ray) Stools examination  blood and mucus CBC, H&H Elevated Sedimentation rate & C-Reactive proteins Low Albumin
101
Ulcerative Colitis & Crohn’s- meds nutrition surgery
Medications- steroids Nutrition- Fiber moderation- fiber contraindicated with intestinal strictures, inflammation and scaring Surgery- colectomy, ostomy or illesotomy
102
Ulcerative Colitis & Crohn’s- Health Promotion- educate need monitor monitor
educate on need for supplemental vitamins need to be on steroids for exacerbations monitor weight and nutrition monitor daily bowel movements
103
Diverticular Disease what is it 2 kinds
Outpouching of colon, occurring in rows- anywhere in intestinal tract 2 kinds- Diverticulosis Diverticulitis
104
Diverticulosis presence complications
Presence of diverticula, often asymptomatic Complications- Hemorrhage, diverticulitis
105
Diverticulitis inflammation what settle complications
Inflammation in and around the diverticula Undigested food and bacteria settle Complications: Perforation-fever and pain complaints
105
Diverticular Disease-Manifestations pain con/inc other manifestations
Pain- left side, ranges from mild to severe Constipation or increased stools N/V, fever
106
Diverticular Disease complications obs form hem narrow
Bowel obstruction, fistula formation, hemorrhage , bowel narrowing
107
Diverticular Disease- diagnosis enema ray oscopy scan labs
Barium Enema, x rays, sigmoidoscopy/colonoscopy , CT scan Labs- H&H, WBC’s, guaiac stool
108
Diverticular Disease- Treatment what meds inc what nutrition-high, avoid,bowel rest
Antibiotics Hydration Nutrition- high fiber, avoiding small seeds, shells, popcorn bowel rest-slow gradual feeding
109
surgery diverticular disease treats 2 stage procedure
To treat peritonitis, hemorrhage, resection ( 2- stage procedure) –temporary colosotmy
110
Diverticular Disease- education assessment priority
Education- diet assessment- subjective/ objective- pain, history, bowel sounds, tenderness, blood stool Priority preventing complications- pain and anxiety
111
Malabsorption Syndromes- condition diseases of intestines
Condition where intestinal mucosa ineffectively absorbs nutrients Diseases of intestines- Crohn’s, gastric bypass, celiac, lactose intolerant
112
manifestations Malabsorption Syndromes-
Anorexia, bloating, weight loss, weakness, fatigue, difficulty concentrating
113
Polyps- what is it most/some asymptomatic-how found
What is it?? –mass of tissue that arise from bowel wall Most benign, some malignant Asymptomatic found on routine screens or painless rectal bleeding
114
complications with large polyps o p sc
Obstruction, pain stool changes
115
Colorectal Cancer- what improves survival rate what type of growth no symptoms until when
Early diagnosis and treatment improved survival rate No symptoms until advanced Growth slow
116
risk factors of colorectal cancer
Age, polyps , family history, IBD, radiation, diet, obesity, smoking, alcohol use
117
manifestations advanced disease manifestations complications colorectal cancer
manifestations-Rectal bleeding, change in bowel habits advanced disease manifestations-pain, anorexia, weight loss complications-bowel obstruction and perofration
118
Colorectal Cancer- Prevention lifestyle habits(inc/dec/ healthy/ do what) age 50
Lifestyle habits- inc vegetables, decrease red meat, healthy weight, exercise At age 50; start screening measures
119
colorectal cancer diagnosis use what use if suspected metastasis what labs
gi Scope; tissue biopsy Ct/ MRI- if suspected metastasis labs; CBC, guaiac, Tumor marker
120
Surgical Resection-> Laser photocoagulation-> colostomy placement reason Colorectal Cancer- Surgery
Surgical Resection; lymph node is removed Laser photocoagulation-light beam, to destroy tumor Colostomy placement- diversion
121
colostomy naming line going left junction of line to small intestine upwards side down by anus Colorectal Cancer- Surgery
Colostomies take name of the portion of colon from which they come from line going left- illeostomy ceconstomy - junction of line to small intestion ascending -upwards transverse-side descending-down sigmoid-by anus
122
Colostomy- assessing L/t S-surrounding O-consitency
Assess location and type of colostomy Assess stoma, surrounding skin Assess output- consistency of drainage depends on stoma location
123
Colorectal Cancer- Radiation used with reduces shrink
Used with surgical resection for tumors Reduces recurrence of pelvic tumors Shrink to allow for surgery
123
Consistency changes –look up Ascending Transverse Descending Sigmoid
Ascending Transverse Descending Sigmoid
124
Colorectal Cancer- chemo reduces
Reduces rate of tumor reoccurrence/ Metastasis
125
Intestinal Obstructions- what is it mechanical obstructions (1/2/3) Gas and fluid accumulation, distending the bowel- does what
what is it-Failure of intestinal contents to move through mechanical obstructions 1-scars 2. adhesions 3. tumors or inflammatory obstruction Gas and fluid accumulation, distending the bowel- compromises blood flow leads to necrosis
126
Small Bowel Obstruction caused by
1. adhesions 1. scar tissue 1. hernia
127
small bowel obstructions manifestations early /late on
Cramping, n/v fecal matter , early on high pitched bowel sounds, later on silent bowels
128
Small Bowel Obstruction- Complications Hypo pvent nec perf
Hypovolemia(/ic shock) renal insufficency Pulmonary ventilation Necrosis from Strangulation- no blood supply Perforation - can lead to septic shock
129
Large Bowel Obstruction occurance area
Less frequent occurrence; sigmoid area- cancer of bowel
130
manifestations of large bowel obstructions normal manifestations late signs-not common what types of sounds
constipation , colicky abdominal pain Deep, cramping; vomiting late sign- not all that common High pitched, tinkling bowel sounds with rushes/gurgles
131
complications of large bowel obstructions colon dialation ->
Colon Dilation increasing pressure which impairs circulation and can lead to Gangrene, perforation, peritonitis, diaphragm involvement
132
Bowel Obstruction- Diagnosis
X ray, CT Labs (WBC/ Electrolytes/ ABGs )
133
Bowel Obstructions- Treatment conservative treatment decompresion npo status pain meds ng type of meds dvt
Decompression- NG tube, IV fluids NPO status  watch fluid status, urine output Pain medications, nausea medications NG- Collects unwanted fluid & gas while allowing bowels to rest Antibiotics DVT prevention -Ambulation
134
Bowel Obstructions- Treatment surgery required if does what to treat
Required if conservative treatment fails, or complete obstruction, strangulation Remove obstructing cause- treat the problem
135
preop bowel surgery marking tube perform
Marking of stoma site NG tube placement and management Perform bowel prep as ordered
136
postop bowel surgery monitor assess support managing complications
Monitor bowel sounds Assess surgical site dressing and drainage-CHECK bleeding Emotional Support  depression, loss of interst in actiivtes Managing post-op problems- pain, gas pain, nausea Complications- peritonitis
137
Bowel Obstructions- Priorities imbalances treating observe managing problems discharge education
Fluid and electrolyte imbalances Treating infection Observe dressing and drainage Managing post-op problems- pain, gas pain, nausea  encourage ambulation Discharge education- normal bowel movements
138
Peritonitis- inflammation sterile caused by
Inflammation of peritoneum Sterile peritoneal cavity becomes contaminated Caused by bacteria
139
Abdomen/GI Symptoms peritonitis
Pain Tenderness with rebound Board-like rigid abdomen Diminished/absent bowel sounds Distention n/v
140
Systemic Symptoms peritonitis f t t r c o
Fever Tachycardia Tachypnea Restlessness Confusion Oliguria
141
Peritonitis- Treatment meds surgery nutrition decompression
Medications - (Antibiotics Pain control) Surgery -(Identify cause of contamination and fix/ Peritoneal lavage ) Nutrition (IV fluids, parenteral nutrition) Decompression Ng tube- Relieve distention, promote bowel rest
142
Hernias- what is it how classified/classifications
What is it?? Defect in abdominal wall that allows abdominal contents to protrude outwards Classified by location Inguinal, umbilical, incisional/ventral
143
Inguinal hernia males-> indirect-> direct->
Males; bulge/lump with lifting/strain, sometimes dull ache Indirect- improper closure of the tract as the testes descend at birth Direct- acquired from weakness of posterior inguinal wall
144
umbilical hernia women -> enlarge/pain->
Women; Pregnancy and obesity Enlarge steadily, sharp pain with coughing/straining
145
incisional hernia where at what looks like often
At previous surgical sites, or abd muscle tear Bulge; often asymptomatic
146
hernia manifestations abdominal contents
abdominal contents protrude through abdominal wall and form sac
147
Reducible hernia: abdominal contents returns
abdominal contents move through sac with increased abdominal pressure returns to abdominal cavity when pressure returns to normal
148
complications of hernias low risk w incarcerated obs/stra
Risk is low with reducible Incarcerated- if contents can not be returned to abdominal cavity Obstruction, strangulation (severe pain, distention, n/v,)
149
Hernias- Treatment exam how treated what looking for for nursing care
Physical examination Treated with surgery Suturing opening, put mesh inside Nursing Care History of symptoms, looking for bulge
150
Hemorrhoids- manifestations diagnosis meds do what
Manifestations pain, rectal bleeding Diagnosis- Patient history, physical exam Medications- to improve constipation, reduce straining
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Hemorrhoids- Treatment Sclerotherapy Hemorrhoidectomy
Sclerotherapy –chemical irritant injected to cause scarring Hemorrhoidectomy –surgical removal
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Anorectal Surgery- why sensitive used for what
Sensitive- sensory nerves, painful procedure Hemorrhoids, Anal Fissure, Anorectal Abscess, Anorectal Fistula
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nursing care anorectal surgery what after stools no what observing what returnof gi function Dietary support/teaching Limit pressure and positioning on surgical site
Sitz bath after stools No rectal medications Observe dressing/drainage Return of GI function-start fluid intake Dietary support/teaching-high fiber diet, and fluid intake Limit pressure and positioning on surgical site -low head of bed, supine position, don’t elevate hob
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Cholecystitis- what is it acute obstruction difference between Cholecystitis /Cholelithiasis
Inflammation of gallbladder Acute obstruction of cystic duct by stone Cholecystitis inflamttion of actual gall bladder Cholelithiasis presence of stone
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Cholecystitis Manifestations onset temp pain where
Abrupt onset, low grade temp, Right quad pain radiates to back, right shoulder/scapula
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chronic manifestations of Cholecystitis-
repeated bouts or gallbladder irritation
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Cholecystitis- diagnosis
CBC, bilirubin, amylase & lipase Ultrasound X-ray Gallbladder Scans
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Cholecystitis- meds does what meds antibiotics usage
Reduce cholesterol content of stones; gradual dissolution Ursodiol, chenodiol Antibiotics- infection, reduce edema, inflammation
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Cholecystitis- surgical Treatment nutrition dietary avoid
Laparoscopic Cholecystectomy Nutrition- Reduce food intake during attack Dietary low texture diet that’s bland Avoid obesity, hyperlipidemia, high cholesterol, high fat foods
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Hep a trasmission what else
Fecal-Oral- contaminated food/ water/ shellfish/ direct contact with and infected person-sex
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Hep b trasmission what else
Blood/body fluids Perinatal High risk group, iv drug users, multiple sex partners, exposed to blood products-oral contraceptives do not provide protection
161
Hep c trasmission what else
Blood/body fluids Injection drug users is primary risk-asymptomatic long after exposure//hand hygiene and ppe for healthcare providers
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Hep d trasmission what else
Blood/body perinatal Only causes infection in pts with hep b
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Hep e trasmission what else
Fecal Oral Fecal contamination of water supply, oral contraceptives do not provde protection
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Hepatitis- Diagnosis assessment increase in what labs presence of what what kinda biopsy
Assessment –inflammation of liver Labs; liver function tests ALT, AST, ALP, Bilirubin -increase Presence of antigens and antibodies Liver biopsy