Exercise Flashcards

1
Q

What is Acute?

A

Single, isolated session that elicits transient physiological response

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2
Q

What is Chronic?

A

Repetition of single bouts
produce permanent physiological adaptations
Exercise training response

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3
Q

What are general responses to acute exercise?

A

Increased metabolism due to increased muscle contraction
Cardiovascular system increases to meet metabolic demand
Adjusting Q, blood flow and pressure

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4
Q

Acute

What is Resting Heart Rate?

A
Average HR = 70 bpm
highly variable (40-100bpm)
Affected by 
-environment and stress
Decreases with age and increase cardiovascular fitness
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5
Q

Acute

What is the HR response to exercise?

A

HR increases at exercise onset
-Levels off during submax
-increases during graded exercise
Plateau at HR max

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6
Q

Acute

What is max HR?

A
Highest HR is acheived during max
215-220 bpm
Declines with age
Sympathetic increases
Parasympathetic decreases
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7
Q
Formula
HR
SV
Q
MABP
A

220-age
EDV-ESV
HR x SV
Q x TPR

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8
Q

Acute

Stroke Volume at rest

A

Blood pumped by one ventricle in a single contraction
Average is 70ml/beat
25% lower in women

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9
Q

Acute

Stroke Response to exercise

A

SV increases at exercise onset up to 40-60% max intensity
May increase up until max (depends on body position)
Major determinant of max cardiovascular work capacity

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10
Q

Three Mechanisms responsible for increasing SV

A

1) Frank-Starling Mechanism
2) Increased contractility due to sympathetic stimulation
3) Reduced TPR

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11
Q

1) Frank - Starling Mechanism

A

Increased venous return = increase blood prior to contraction
Increased EDV stretches (Preload)
Greater stretch = greater force
Increased SV

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12
Q

2) Increased contractility due to sympathetic stim

A

Independent of changes in EDV

Greater stretch - optimal length - greater contractility

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13
Q

3) Reduced TPR (After load)

A

Vasodilation in working muscles

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14
Q

Acute

What is Cardiac Output?

A

Volume of blood pumped/ventricle during a given period of time
Average 5ml/min
Indicator of cardiovascular systems capacity to meet exercise demands

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15
Q

Acute

Cardiac output during exercise

A

Increase during exercise with increase intensity
Amount of increase depend on trained state
Sedentary = 20L/min
Trained = 40L/min
When intensity exceed 40-60% max, Q increase is due to increased HR

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16
Q

Acute

Blood Flow

A

At rest muscles receive 20-25% of Q

During exercise muscles receive up to 90% of Q

17
Q

Acute

Blood Flow during exercise

A

Increase tissue metabolism
Increase release of metabolic vasodilators into ECF
Arterioles dilate
Decrease resistance = increase blood flow
O2 and nutrients supply to tissue increases as long as metabolism increases

18
Q

What is blood flow determined by?

Ateriole diameter is controlled by what?

A

Vessels resistance to flow
Tonic release of nor-epinephrine
(increased signal rate, blood vessels constrict, decrease signal = vessels dilate)

19
Q

Acute

Blood Pressure during exercise

A

Mean arterial BP rises slightly despite decrease resistance

Depends on type of exercise

20
Q

Acute

What are theories for increase in MAP without activating baroreceptors reflex?

A

Receptors threshold is reset to higher pressure
Central inhibition of afferent signal
Chemoreceptors sensitive to metabolites override barorecpetor reflex

21
Q

Chronic cardiovascular adaptations to training

Aerobic exercise training

A

Improve endurance

Important measure to assess endurance capacity is VO2 max

22
Q

What is VO2 max and what systems are involved?

A

The max intake, transportation and utilization of oxygen.

Integration of cardiovascular/respiratory and neuromuscular system

23
Q

What changes occur with increased VO2 max?

A
HR
SV
Q
Heart size
BP
Blood flow
Blood volume
24
Q

Chronic

Hear rate

A

Decrease with endurance training
Highly trained have bradycardia
Increase in parasympathetic activity
Decrease in SA node firing

25
Chronic | Submax HR
Decreases with training | Heart does not beat as fast for same amount of work
26
Chronic | Maximal HR
Remains unchanged or slightly decreased
27
Chronic | Stoke volume
Increased at rest, submax and max
28
Chronic | What causes the increase in SV
``` Greater EDV (increased volume and filling time) Increase in ventricle size ```
29
Chronic | Cardiac Output
Remains unchanged at rest and during submax exercise | Increases at max due to increased SV (HR max remains changed)
30
More filling time Larger ventricles More blood volume
More time between beats Greater stretch/ preload Greater stretch= greater force Over all the heart is better at getting blood from the heart to the body
31
What happen to heart size?
Cardiac hypertrophy is a result from training -Increase wall thickness -Increase in cavity size Depends on overload stimulus
32
What is pressure overload?
pressure due to afterload as a result of strength training Heart contracts harder against resistance Concentric hypertrophy
33
What is volume overload?
Volume due to high preload and amount of blood passing through Increased stretch = increased size Eccentric hypertrophy
34
What cavity of the heart changes the most?
Left ventricle because of increased filling. Mostly cavity size increase in endurance but there is some wall size increase
35
Chronic | Blood Pressure
Both systolic and diastolic decrease at rest Most pronounced in people with hypertension Reduces sympathetic hormones May decrease during submax
36
Chronic | Blood Flow
Increases with training - increased max Q - increase blood volume - more effective blood distribution - increased capillarization
37
Chronic | Blood Volume
``` Increase with training due to increased plasma RBC increase (decrease hematocrit though b/c plasma increases more) ```
38
What does increased blood volume do?
Enhances thermoregulation and circulation and facilitates in O2 delivery contributes to eccentric hypertophy and increases SV