Final Exam

Question 1

What is TUBS and AMBRI?

Answer 1

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Question 2

Impingement captures the mechanical reason for what two possible pathologies within the GH joint. What are the two categories of imp?

Answer 2

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Question 3

SAI vs. PII

• Structures involved + mechanism of imp
• Population and presentation
• Aggravating ROM

Answer 3

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Question 4

Describe the common theme of tendon pathology.

-Rarely tendinitis due to low blood supply but at best is tendinosis…what are some consequences?

Answer 4

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Question 5

Common challenges at the shoulder:

1) excessive ant/sup translation of numeral head
2) GIRD
3) excessive internal rotation
4) excessive protraction of girdle
5) scapular dyskinesis

Answer 5

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Question 6

4 types of shoulder dyskinesis. Describe each.

Answer 6

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Question 7

What ROM of the arm heavily depends on scapular movement? Explain in terms of scapular rotation, tilt.

Answer 7

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Question 8

Which motions would make good provocative tests? What’s the best test for type II dyskinesis

Answer 8

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Question 9

Interpreting type I, II, III SD (what is weak, what is restricted).

Answer 9

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Question 10

What test would look at GH motion? 2 versions.

Answer 10

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Question 11

List some general ROM exercises for the GH in the earlier stages of rehab.

Answer 11

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Question 12

Stretches or methods to increase extensibility of:

• posterior capsule
• pec
• lats

Answer 12

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Question 13

Resistance exercise for GH

• lateral rotators
• medial rotators
• scaption
• biceps brachii

Answer 13

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Question 14

What type of exercise do tendons usually response to?

Answer 14

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Question 15

Resistance exercise for scapula (4). A proprioceptive exercise for scapula? (1). What is the most advanced exercise for everything put together (hint: type of plank)?

Answer 15

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Question 16

Proprioceptive exercises for the shoulder.

Answer 16

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Question 17

PFS or PFPS:

• Loading. Aggravating ROM, activities, and other factors?
• Mechanism of injury/insult?
• Patellar mechanism: 3 for lateral translation, 2 for lateral tilt.
• Patellar mechanism: patellar tendinopathy - mechanics of tendinopathy?

Answer 17

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Question 18

Cruciates: ACL and PCL; vs. Collaterals: MCL/LCL

• Why are the cruciates difficult to heal/fix? (3 points); surgical method(s)?

Answer 18

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Question 19

Meniscal tearing:

• May be acute or most commonly…..
• Aggravating ROM
• Why limited healing?
• Surgical interventions (2)
• Muscle atrophy…
• Acute ROM limitations in FL and EX (why - 1 mechanism for each)
• What kinda protection for function options do we have?
• Why retrain gait?

Answer 19

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Question 20

• Differentiate osteoarthritis with rheumatoid arthritis?3 features of OA.
• Inflammatory response
• Pain patterns
• Joint stiffening
• Loss of mobility and strength in which ADLs?
• Tibiofemoral varus and valgus (stresses which compartments)
• OA has a high correlation with what 2 things?
• Total knee arthroplasty

Answer 20

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Question 21

10 structures directly stabilize the GH joint

Answer 21

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Question 22

Common challenges of knee

1) valgus knee
2) excessive internal rotation of hip
3) limited hip extension
4) limited knee flexion
5) limited knee extension

Answer 22

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Question 23

Specific ROM skills for knee (4)

Answer 23

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Question 24

Resistance exercise for knee extensors (2)

Resistance exercise considerations for:

• PFS
• Patellar instability
• ACL
• Meniscus and OA

Answer 24

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Question 25

Resistance exercise for knee flexors (hammies) - 3

Answer 25

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