Final Exam Material Flashcards
3 main causes for dizziness
- otologic
- neurologic
- general medical
Otologic
- BPPV
- vestibular neuritis
- superior canal dehiscence
- meniere’s
Neurologic
- vertibrobasilar insufficiency
- stroke
- migraine
- low CSF
General medical
- B12
- orthostatic hypotension
- hypoglycemia
Balance control
input (visual, rotation, gravity, pressure)
brain
output (ocular reflex, postural control)
A discrepancy bw any of these 3 systems can cause nausea and vertigo
Vestibular system
- semicircular canals (post, ant, lateral)
- otolith organs
- nerve
- cochlea
Semicircular Canals
- Ampulla (bulbous bony opening that houses cupula)
- Cupula (sensor/sail that houses hair cells. Directly connected to vestibular nerve)
- Canals (orthogonal…ant, post, lateral/horizontal)
Peripheral vestibular dysfunction-causes
- vestibular apparatus
- vestibular portion of CN VIII
- some cerebellopontine angle tumors
Central vestibular dysfunction-causes
- vestibular nuclei
- central pathways
- also cerebellopontine angle tumors
Peripheral vestibular dysfunction categories
- unilateral vs. bilateral
- reduced vs. absent function
- acute (BPPV) vs. Chronic (meniere’s)
Examples of peripheral diagnoses
- acoustic neuroma/other tumors
- meniere’s disease
- gentamicin otolithic ablation
- local trauma
- guillan barre or MS at CN VIII root entry
- BPPV
- infection: labyrinthitis, vestibular neuritis
- perilymphatic fistula (breakage in membrane bw middle and inner ear)
Benign Paroxysmal Positional Vertigo (BPPV)
- most common cause of vertigo
- brief episodes of vertigo precipitated by rapid change of head posture (30sec-2mins)
- ex. every time they roll over in bed it happens
BPPV epidemiology
Women>men
-spontaneous remissions common, but recurrences can occur
BPPV-cupulolithiasis
- otoconial material gets stuck in canal
- disrupts cupula’s response to gravity
BPPV-canalithiasis
- otoconial debris gets into a semicircular canal
- post/ant>lateral
- creates a suction moment which acts on the cupula and fluid can’t continue through the canal
BPPV prognosis
- tends to be self limiting (6-12 mos)
- vestibular rehab can speed up recovery SIGNIFICANTLY
- antivertiginous drugs ineffective (anavert/meclazine)
- elderly respond more slowly
- if symptoms persist, MRi indicated (acoustic neuroma, cerebellar or 4th ventricle tumor)
Vestibular Neuritis
- second most common cause of vertigo
- swelling in vestibular area
- probable viral etiology
- similar to bell’s palsy
- sometimes epidemic occurences
- upper respiratory or GI infections
- concurrent or preceded by 2 weeks
Vestibular Neuritis epidemiology and S/S
Primary ages: 30-60 (women-40, men-60)
- acute onset of prolonged severe rotational vertigo which increases with movement of the head (hits you all at once)
- associated with horizontal-rotatory nystagmus, postural imbalance, and nausea/vomiting
- could be unilateral or bilateral
Vestibular neuritis management
- vestibular suppressants (anavert/meclazine)
- bedrest x 24-72 hrs
- gradual return to functioning
- total recovery in about 6 weeks
Vestibular neuritis and vestibular rehab
- significantly speeds recovery
- slowly increases ambulation
- general conditioning
- gaze stabilization exercises
- facilitate central compensation
Meniere’s disease etiology
- hereditary or sporadic
- damage to hair cell inside cupula (changes amt of vestibular info being sent to CNS from vestibular nerve)
- distension of endolymphatic system with hair cell damage
Meniere’s disease S/S
- vestibular and AUDITORY involvement (complaints of hearing lost in 1 or both ears, sense of fullness in ears)
- vertigo usually lasts hours (preceded by ear pressure/fullness)
- change in tinnitus (ringing)
- change in hearing function
Meniere’s management
- meds for vertigo (anavert/meclazine)
- diuretics
- middle ear injections (gentamicin, steroids, surgery)
- no cure
Examples of central vestibular diagnoses
- TBI
- CVA
- MS
- Tumors
- Meningitis