FINALS Review Flashcards

(73 cards)

1
Q

pts with upper motor neuron injuries will develop this type of bladder and voiding for male (2) and female (1)

A

spastic bladder male-micturation reflex and condom cath female- in/out cath

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2
Q

lower motor neuron injury will result in this type of bladder; voiding for men/women (2)

A

flaccid bladder male and female - crede and in/out catheter

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3
Q

interventions neurogenic bladder (2)

A

1stool softener or bulk forming laxative daily 2dulcolax suppository or digital stim only as ordered.

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4
Q

how to assess an ileus (1)

A

listening bowel sounds

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5
Q

type of plegia with t1 injury

A

paraplegia

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6
Q

type of plegia with c level injury

A

quadraplegia

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7
Q

major concern with c4 level injury, interventions (6)

A

respiratory: ventilator, sp 02, oxygen, cough and deep breathing exercises, suction, cough assist

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8
Q

interventions neurogenic shock main problem(1) - plus 3 smaller resulting problems

A

tissue perfusion dt hypotension, edema and loss of temp regulation postural hypotension -attention with safe transfer safety thrombophlebitis- anticoagulant

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9
Q

muscle tone in injury above L1-L2?

A

spastic

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10
Q

muscle tone in injury below L1-L2

A

flaccid

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11
Q

interventions for muscle strength and tone (2)

A

active and passive ROM

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12
Q

areflexic bowel

A

flaccid bowel

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13
Q

reflexic bowel

A

spastic bowel

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14
Q

result of damage to the clonus medularis and cauda (3) equina -lower ext -bladder -bowel

A

flaccid lower extremities, areflexic bowel and bladder

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15
Q

nuerogenic shock is usually with injuries at this level

A

t6 and above

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16
Q

signs of spinal shock- (3)

A

loss of reflexes loss of sensation flaccid paralysis below level of injury

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17
Q

signs of neurogeic shock (6)

A

hypotension, bradycardia, decreased cardiac output, peripheral vasodiation, venous pooling, loss of ns stimulation

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18
Q

decreased stimulation of the systemic ns would result in (3) which is characterisctic of neurogenic shock

A

perifpheral vasodilation venous pooling decreased cardiac output

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19
Q

ipsilateral loss of motor function, position and vibratory sense, vasomotor paralysis

A

brown sequard

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20
Q

contralateral loss of pain and temperature

A

brown sequard

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21
Q

motor paralysis and loss of pain and temperature

A

anterior cord syndrome

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22
Q

pain and temperature and motor functions intact

A

posterior cord syndrom

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23
Q

loss of propioception

A

posterior cord syndrome

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24
Q

sensation, touch position vibration and motion intact

A

anterior cord syndrome

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25
motor weakness and sensory loss in upper and lower extremeties
central cord syndrome
26
upper extremities are more affected in this cord syndrome than lower
central
27
movement of these are intact with c8 injury
arms
28
risk for these conditions after craniotomy
DI and siadh
29
give this med for DI to retain urine, same action as \_\_
DDAVP or desmopressin same action as ADH
30
phenytoin side effects and interventions
gingival hyperplasia- oral care skin rash- notify md dysrhythmias/ hypotension- decrease infusion rate CNs effects- notify MD teratogenic cleft palate, heart defects-avoid during pregnancy
31
CNS effects of phenytoin (4)
ataxia, nystagmus, double vision, cognitive impairment
32
substances increase effects of phenytoin (5)
alcohol, diazepam, cimetidine, valproic acid, cns depressants
33
substances decreases effects of phenytoin (3)
alcohol, carbamazepine, phenobarbital
34
phytoin decreases effects of ex: (3)
hepatic drug-metabolizing enzymes; glucocorticoids, warfarin, oral contraceptives
35
pt teaching re phenytoin (4)
do not stop abruptly no driving take with meals oral hygiene dt gingival hyperplasia
36
prioritization of patient in disaster triage
based on ABCDEFGHI Airway Breathing Circulation Disability Environment F
37
hypoventilation leading to respiratory distress in spinal cord injury is due to (2) can result in (2) interventions (4)
due to decreased vital capacity and intercostal and diaphragmatic muscle dysfunction -- results in secretion pooling and ineffective cough -- suction and mechanical ventilator,02, assist coughing
38
areas to assess for respiratory function in sc injury (10), pt who is unable to do this without taking a breath needs immediate attention
breath sounds ABGs PaCO2 60 respiratory effort/ pattern subjective comments amount and color of sputum tidal volume vital capacity skin color cannot count to 10 without taking a breath needs immediate attention
39
contraindications for jeart transplant
-\>70 yo -severe pulmonary disease with likely vent dependent status post transplant -malignancy or life threatening condition with
40
indications for heart transplant
-end stage heart disease -inoperable valve disease -recurrent life threatening dysrhythmias unresponsive to interventions - function limiting cadiac abnormalities with mortality risk of \>50% at 2 year
41
transplant donation are matched based on (5)
hla typing or match, abo compatibility, geographical location, time on the wait list, and medical urgency
42
positive crossmatch vs negative crossmatch and what type of transplant is crossmatch done
crossmatch is absolutely done for kidney transplants. a negative crossmatch mean no antibodies to the donar organ and it is safe to proceed with the transplant
43
hyperactute rejection time frame
minutes to hours, rarely occurs because of hla typing just before transplant, the blood vessels are rapidly destroyed, removal of organ
44
acute rejection time frame and fix, activated by these (2)
within 6 months, lymphocyte and antibodies react to the donated organ. fixed by immunosuppressive thereapy, corticosteriods, polyclonal and monoclonal antibodies. person is at risk for infection especially early in the transplant
45
chronic rejection timeframe, caused by these antibodies. main and specific manifestations kidney, heart, lungs liver
months and years and no fix mainly supportive. caused by b and t cells. manifestations of fibrosis and scarring kidney- glomerulopathy/fibrosis heart- advance CAD lungs- loss of bile ducts liver- bronchiolitis obliterans
46
risk when taking immunosuuppressants (2)
toxicity and increased risk for infection
47
examples of immunosuppressants (7)
cyclosporin, tacrolimus, corticosteroids,mycophenolate mefetil, sirolimus, azathioprine, cyclophosphamide
48
examples of monoclonal antibodies (3)
natalizumab, daclizumab, basiliximab, muromonal-Cd3
49
se of cyclosporin (10)
neurotoxic, nephrototix, hepatotoxic, gingival hyperplasia, lymphoma, hypertension, hirsutism, leukopenia, seisures, tremors
50
loss of consciousness in this type of seisure
tonic clonic
51
definition of tonic
stiffening of the body
52
definition of clonic
jerking of the body
53
tonic clonic seisure has these three elements during a seisure and can have these (4)
loss of consciousness, tonic(stiffeining), clonic (jerking), can have excess drooling, cyanosis, incontinence, tongue or cheek biting
54
s/sx of tonic clonic seisures postictal (3)
muscle soreness, tiredness, sleepy
55
hyyperventilation and flashing lights may happen before this type of seisures, which manifest as \_\_,usually seen in children only; seen on the eeg as this -\_\_\_
absence seisure, staring or daydreamig that lasts for a few seconds. . see 3hz spike an wave pattern
56
loss of consiousness and loss of muscle tone
clonic seisure
57
sudden loss of muscle tone and drop to the ground, greatest risk for head injury, consciousness returns by the time person hits the ground
atonic siesure
58
increased tone of extensor muscles, risk for falls in this type of seisure
tonic seisure
59
types of generalized seisures (6)
tonic clonic absence atypical absence atonic tonic myoclonic
60
sudden and excessive jerking of the body
myoclonic
61
2 types of focal seisures and consciousness
simple focal- no loss of consciousness complex focal- loss of consciosness
62
simple focal seizure s/sx
aura (unusual feelings or sensations) before a seizure may feel sad, anger, joy, nausea, may see hear, smell, taste or feel things that aren't there
63
complex focal s/sx, duration
loss of consciousness then automatism or repetitive movements such as lip smacking, picking at clothing, fumbling with objects, walking away, lasts for a few seconds.
64
patient teaching re seizures about lifestyle (5)
get enough sleep, avoid alcohol and avoid fatige, proper diet, exercise
65
the most dangerous type of seisure that could result in hypoventilation, hypoxemia, dysrhythmias, hyperthermia and systemic acidosis
tonic clonc status epilepticus
66
topiramate and levetiracetam treat all types of seizures except this type
absence seisures
67
important to monitor for leviteracetam
suicide
68
cns effs of leviteracetam (6)
weakness, dizziness, agitation, anxiety, depression, suicide
69
cns side effs of topiramate (6)
cns effs (somnolence, dizziness,ataxia, nervousness, diplopia, confusion, impaired cognitive fx)
70
interventions for metab acido in topiramate monitor\_ report (3) dose
monitor bicarb report hyperventilation, fatigue, anorexia dc or reduce dose
71
meds decrease topiramate levels
phenytoin and carbamazepine
72
topiramate increases level of \_\_
phenytoin
73
interventions re se of angle closure glaucoma in topiramate
1vision check to measure ocular pressure 2pt will experience pain, redness, blurring of vision