Functions and secretion of the alimentary tract: part 1 Flashcards

1
Q

food ingestion is stimulated by

A

Hunger = intrinsic for food
appetite= preferential of food

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2
Q

Mastication ( chewwing)

A

Teeth= designed for cutting (incisors) and grinding (molars) of food.
Muscles for chewing= controlled by nuclei in brain stem.

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3
Q

Stimulation of specific reticular areas (brain stem taste centers)

A

causes rhythmic chewing action.

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4
Q

what other stimulation causes chewing ?

A

hypothalamus
brain cerebral cortex
amygdala

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5
Q

mastication reflex

A

takes place in response to presence of reflex inhibition of mastication muscles (at first)→ lower
jaw drops→ initiates stretch reflex of jaw muscles→ rebound contraction→ automatically raises jaw→
closes teeth + compresses bolus against mouth linings→ inhibits jaw muscles again→ jaw drops→
rebound contraction again= process repeated again and again

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6
Q

mechanical digestion of all food

A

chewing

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7
Q

chemical digestive

A

digestive enzymes

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8
Q

initiation of the swallowing process.

A

voluntary stage

food bolus pushed and compressed by the tongue against the palate to the pharynx.

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9
Q

initiation of the swallowing process.

A

voluntary stage

food bolus pushed and compressed by the tongue against the palate to the pharynx.

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10
Q

pharyngeal stage( in the pharynx )

A

involuntary stage/ process

Bolus stimulates epithelial swallowing receptor areas in
the pharynx, and tonsillar pillars→ to send action potentials to the brain stem.
initiates a series of automatic pharyngeal muscular contractions.

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11
Q

soft palate will elavate and closes the posterior nares

A

to prevent food reflex intp the nasal cavity

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12
Q

palatopharyngeal folds

A

are pulled medially to form sagittal slit: selectively allow properly masticated food to pass.

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13
Q

function of epiglosttis

A

closes the larynx

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14
Q

elevation of the larynx ………………. and …………..

A

enlarges the oesophageal opening and upper oesophageal sphincter relaxes.

contration of the pharyngeal muscular wall= peristalsis propulsion of food into oesophagus.

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15
Q

Pharyngeal Stage of Swallowing Momentarily Interrupts Respiration:

A

last for 6 seconds
Swallowing center inhibits respiratory center of the medulla during this
time (within the 6 secs period)

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16
Q

Esophageal Stage of Swallowing involves 2 Types of Peristalsis:

A

primary and secondary perilstalisis.

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17
Q

primary peristalsis.

A

begins in the pharynx and terminates in the stomach.
last about 8-10 seconds in an upright person.

last about 6-8 secs due to gravitational force.

18
Q

secondary peristalsis.

A

when primary peristalsis fails, secondary peristalsis wave results from distention of oesophagus by retained food.

19
Q

Secondary peristalsis initiated by:

A

a) Intrinsic neural circuits in myenteric nervous system
b) Pharyngeal reflexes transmitted upward through vagal afferent fibers to medulla and back to esophagus
through glossopharyngeal & vagal efferent nerve fibers.

20
Q

Pharyngeal wall & upper 3rd esophagus.

A

has striated/ skeletal muscles= controlled by skeletal nerve impulses of
glossopharyngeal & vagal efferent nerve fibers.

21
Q

while the lower 2/3 of the esophagus

A

strongly controlled by vagal nerves associated with myenteric
nervous system.

When vagus nerve is cut→ myenteric plexus of esophagus takes over & stimulate secondary peristalsis

22
Q

when the vagus nerve is cutted what is happens?

A

→ myenteric plexus of esophagus takes over & stimulate secondary peristalsis.

23
Q

Esophageal peristaltic waves approaching stomach→

A

relaxation of the stomach through the inhibitory neurons accouring

24
Q

Function of the Lower Esophageal Sphincter (LES) (Gastroesophageal Sphincter):

A

Peristaltic wave moving down esophagus→ receptive relaxation of lower esophageal sphincter occurs→ food
enters stomach.
Tonic constriction of the LES prevents reflux of stomach contents into esophagus

25
Q

Prevention of Esophageal Reflux by Valve-like Closure of the Distal End of Esophagus:

A

a valve-like mechanism of a short portion of the oesophagus extending slightly to the stomach is called cardia prevents high intra-abdominal pressure from forcing stomach contents into oesophagus.

26
Q

storage function of the stomach

A

Food enter stomach→ stomach stretches→ vagovagal reflex
(from stomach—brain stem—back to stomach) reduce muscular
tone→ stomach stretches more (with limit of 0.8L to 1.5L).

27
Q

mixing of the food in the stomach is controlled by

A

gut wall basic electrical rhythmical waves called constrictor waves.

28
Q

when food enters the stomach

A

Gastric glands secret gastric juices→ juices mix with food
material→ mixing waves (constrictor waves) initiated by gut wall
basic electrical rhythm (“slow waves”) (move from body to
antrum)→ constrictor waves become more powerful→ food
moves towards pylorus→ pyloric muscles contracts→ impedes
emptying through pylorus→ food contents mixed (retropulsion
mixing mechanism)→ chyme formed.

29
Q

stomach emptying

A

Caused by strong peristaltic contractions in stomach
Pylorus controls the degree of chyme being emptied.

30
Q

Role of Pylorus in Controlling Stomach Emptying:

A

Pyloric circular muscles= pyloric sphincter

pyloric sphincter remains tonically contracted normally.
degree of pyloric contraction increases or decreases depending on the influence of nervous or hormonal signals.

31
Q

Gastric Factors Promoting Emptying:

A

Gastric Food Volume: increased food volume → promotes stomach emptying
Stretching of stomach wall→ stimulate local myenteric reflexes→ increase pyloric pump activity.

32
Q

hormones stimulating emptying if the stomach?

A

Stretching of the stomach wall and presence of certain foods.

G cells are stimulated to secrete gastrin then stimulate parietal cells to H ions –> Hcl to promote activity of pyloric pump

33
Q

Mixing Contractions (Segmentation Contractions):

A

Chyme in duodenum→ cause distention→ stretching
intestinal wall→ elicit local concentric contractions=
segmentation contractions→ cause mixing of chyme with
small intestine secretions.

34
Q

propulsive movement of chyme in the intestine

A

Peristalsis in Small Intestines: chyme moves through
small intestines by peristaltic contractions

35
Q

Control of Peristalsis by Nervous & Hormonal
Signals:

A

peristaltic activity increase by the gastroenteric
reflex initiated by stomach distention & conducted through
to small intestines by myenteric plexus.

Hormonal factors include gastrin, CCK, insulin, motilin,
serotonin; all enhance small intestinal motility

36
Q

Hormones that inhibits motility

A

Glucagon and secretin

37
Q

The Ileocecal Valve Prevents Backflow From The
Colon to The Small Intestine:

A

Ileocecal valve protrudes into cecum→ increased cecum
pressure→ forcefully closes it= preventing backflow into
ileum.
Ileocecal sphincter→ remain mildly constricted→ allows
slow emptying of ileal content into cecum

38
Q

main function of the colon.

A

absorption of water and electrolytes and storage of faecal matter until expelled.

39
Q

process of the large circular and longitudinal muscles contractions occurs in the large intestines. is called

A

Haustrations

which will the result-slowly dug into & roll over faecal material→ faecal
material exposed to mucosal surfaces→ absorption
occurs

40
Q

After meals, mass movement facilitated by gastrocolic
and duodenocolic reflexe due to

A

stretching of stomach and duodenum.

41
Q

peritoneointestinal reflex

A

Stimulated by irritation of the peritoneum→ Inhibits
excitatory enteric nerves→ cause intestinal paralysis
(patients with peritonitis

42
Q

Renoitestinal and vesicointestinal reflexes

A

stimulated by irritation the kiddney and gallbladde