Gas exchange Flashcards

1
Q

Adaptions for gas exchange surfaces?

A

-Large surface area to volume ratio
-(one layer of epithelial cells)- provides a short diffusion pathway across the gas exchange surfaces
-Maintained a concentration gradient

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2
Q

How do single celled organisms exchange gases?

A

-Absorb and release gases by diffusion through their outer surface
-Have a large surface area, thin surface and a short diffusion pathway so theres no need fore a gas exchange system

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2
Q

Describe the fish gas exchange system

A

1)Water, containing O2, enters the fish through its mouth and passes out through the gills
2)Gills are made up of lots of thin plates called gill filaments, give a big surface area for exchange gases
3)Gill filaments are covered in lamellae, increases the surface area more
4)Lamellae have lots of blood capillaries and a thin surface layer of cells to speed up diffusion.
5)Blood flows through the lamellae in one direction and water flows over in the opposite direction- called counter current system, maintains a large conc gradient between the water and the blood, conc of oxygen is higher in water than in the blood, so oxygen diffuses from the water into the blood.-Ensures the equilibrium is not reached

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2
Q

What do fish use for gas exchange

A

Counter-current system

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3
Q

What do insects use to exchange gases

A

Tracheae

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4
Q

How do gas exchange work in insects?

A

1)Air moves into the tracheae through pores on the surface called spiracles
2)Oxygen travels down the conc gradient towards the cells
3)Tracheae branch off into smaller tracheoles which have thin, permeable walls and go to individual cells- oxygen diffuses directly into the respiring cells
4)Carbon monoxide from the cells moves down its own conc gradient towards the spiracles to be released into atmosphere.
5)Use rhythmic abdominal movements to move air in and out of the spiracles.

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5
Q

Insect adaptations to prevent water loss

A

-Small surface area to volume ratio
-Waterproof exoskeleton
-Spiracles ,where gases enter and water can evaporate from, can open or close to prevent the water loss

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6
Q

Where do gas exchange occurs in dicotyledonous plants

A

-at the surface of the mesophyll cells

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7
Q

Describe the gas exchange of dicotyledonous plants

A

-plants need CO2 for photosynthesis which produces O2 as a waste gas. They need O2 for respiration, which produces CO2 gas
-Main gas exchange surface is the surface of the mesophyll cells in the leaf- have a large surface area
-Gases move in and out through special pores in the epidermis called stomata
-Stomata can open to allow gas exchange and close if the plant is losing too much water
-Guard cells control the opening and closing of stomata

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8
Q

How do plants prevent water loss

A

-Stomata usually kept open during the day to allow gaseous exchange, water enters the guards cells making them turgid which opens the stomatal pore. If the plant starts to get dehydrated, guard cells lose water and become flaccid which closes the pore.

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9
Q

What are the xerophytic adaptations

A

-Stomata sunk in pits that trap moist air, reducing the conc gradient of water between the leaf and the air. Reduces the amount of water diffusing out of the leaf and evaporating away
-A layer of hairs on the epidermis to trap the moist air around the stomata
-Reduced number of stomata so there are fewer places for water to escape
-Waxy, waterproof cuticles on leaves and stems to reduce evaporation
-Curled leaves with stomata inside, protecting them from wind

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10
Q

Gas exchange system in humans

A

-air enters the trachea
-Trachea splits into two bronchi- one bronchus leading to each lung
-Each bronchus then branches off into smaller tubes called bronchioles
-Bronchioles end in small air sacs called alveoli
-Ribcage, diaphragm and intercostal muscles work together to move in air and out

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11
Q

Describe inspiration- (beathing in)

A

-External intercostal and diaphragm muscles contract
-causes the ribcage to move upwards and outwards and the diaphragm to flatten, increasing the volume of the thoracic cavity
-As the volume of thoracic cavity increases, lung pressure decreases
-Air flow from an area of high pressure to an area of low pressure so air flows down the trachea and into the lungs
-active process requires energy

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12
Q

Describe expiration

A

-External intercostal and diaphragm relax
-Ribcage moves downwards and inwards and diaphragm becomes curved again
-Volume of the thoracic cavity decreases causing the air pressure to increase
-Air is forced down the pressure gradient and out of the lungs
-Normal expiration is a passive process-doesnt require energy

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12
Q

How the gas exchange happens in the alveoli?

A

-Alveoli made from a single layer of thin, flat cells called alveolar epithelium
-1)Huge number of alveoli in the lungs, means there is a large surface area for exchanging oxygen and carbon dioxide
2)Alveoli are surrounded by a network of capillaries
3)O2 diffuses out of the alveoli, across the alveolar epithelium and the capillary endothelium (type of epithelium that forms the capillary walls) and into haemoglobin in the blood happens down a diffusion gradient.
4)CO2 diffuses into the alveoli from the blood and is breathed out

-Steep conc gradient of O2 and CO2 between the alveoli and the capillaries which increases the rate of diffusion- maintained by the flow of blood and ventilation

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12
Q

What happens during forced expiration

A

-External intercostal muscles relax and internal intercostal muscles contract, pulling the ribcage further down and in
-Movement of two sets of intercostal muscles are said to be antagonistic

13
Q

What is tidal volume?

A

-Volume of air in each breath (usually 0.4 dm3 and 0.5 dm3 for adults)

14
Q

What is ventilation rate?

A

-number of breaths per minute (15 breaths for a healthy person)

15
Q

What is forced expiratory system volume?

A

-Max volume of air that can be breathed out in 1 second

16
Q

What is forced vital capacity?

A

-Max volume of air it is possible to breathe forcefully out of lungs

17
Q

What is pulmonary Tuberculosis (TB)

A

-Infected with tuberculosis bacteria
-Immune system cells build a wall around the bacteria in the lungs, forming a small, hard lumps known as tubercles
-Infected tissue within the tubercles dies and the gaseous exchange surface is damaged, so tidal volume is decreased
-Causes fibrosis, further reduces the tidal volume
-Reduced tidal volume means less air can be inhaled with each breath. To take enough oxygen, patients have to breath faster- ventilation rate is increased
-Common symptoms are persistent cough, coughing up blood and mucus, chest pain, shortness of breath and fatigue.

18
Q

What is fibrosis?

A

-Formation of scar tissue in the lungs- can be a result of infection or exposure to dust or asbestos
-Scar tissue is thicker and less elastic than the normal lung tissue
-Lungs are less able to expand and so can’t hold as much air as normal-Tidal volume is reduced so is FVC
-Reduction in the rate of gaseous exchange- diffusion is slower across a thicker scarred membrane
-Symptoms include shortness of breath, dry cough, chest pain, fatigue and weakness
-Patients have a high ventilation rate than normal

19
Q

What is asthma?

A

-airways become inflamed and irritated because of an allergic reaction to pollen and dust
-During an asthma attack, smooth muscle lining the bronchioles contracts and a large amount of mucus is produced
-This causes constriction of the airways, making it difficult to breathe. Air flow in and out of lungs is reduced, means that FEV is reduced
-Symptoms include wheezing, tight chest and shortness of breath
-Can be relived by drugs (inhalers) cause the muscle in the bronchioles to relax, opening up the airways

20
Q

What is emphysema?

A

-Caused by smoking or long term exposure to air pollution- foreign particles in the smoke become trapped in the alveoli
-Causes inflammation, attracts phagocytes to the area, phagocytes produce an enzyme that breaks down elastin (protein found in the walls of alveoli)
-Elastin is elastic- helps the alveoli to return to their normal shape after inhaling and exhaling air
-loss of elastin means the alveoli cant recoil to expel air
-Leads to destruction of the alveoli walls, reduces the surface area of the alveoli, rate of gaseous exchange decreases
-Symptoms include shortness of breath and wheezing,
-Increased ventilation rate