Gatrointestinal Lectures Flashcards

0
Q

Dysphagia

-4 types-

A

pain during swallowing

  • anatomical - surgery
  • physiological - radiation such as chemo damages pharynx and larynx
  • neurological - stroke, Parkinson’s Disease
  • drug induced - sedation, atxia

estimated that over 2/3 of elderly patients have dysphagia

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1
Q

Disease of Upper GI
Disease of Lower GI
Accessory Organs

A

Peptic Ulcer - upper
Crohn’s Disease - lower

liver, gallbladder, pancreas

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2
Q

Dysphagia

-4 feeding concerns-

A
  1. patient safety; aspiration and regurgitation (can enter into the airway and cause aspiration pneumonia)
  2. Individual tolerances; hot vs cold, solid vs liquid, can the pt eat alone? Can the patient feed him/herself?
    (the pt could have dementia and not away of what food is) varying degrees of tolerance
  3. Meeting nutritional requirements?
  4. Pain Control; Odynophagia - head and neck cancers will experience burning in the throat

talk to a occupational therapist, swallowing therapist, or dietitian *

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3
Q

Aspiration

-5 types of food that may increase the risk-

A
  1. sticky foods - melted cheese, peanut butter, fresh bread
  2. foods with 2 or more consistencies - stew, pies, fruit yogurt (overrides the ability to coordinate the swallow)
  3. foods with stringy fibres - celery, spinach, meats
  4. foods with small pits - cherries, grapes, citrus fruits
  5. foods that do not easily form bolus - crackers, meat, dry bread

(only some are listed, must have a pre-existing dysphagia or be a baby)

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4
Q

Dysphagia

-signs of conditions-

A
  • drooling; inability to swallow saliva
  • coughing; while eating, drinking, or following a meal
  • some people have silent symptoms; chest may make noise after eating
  • voluntary change in choosing foods ;consistencies or texture changes, solid to liquid
  • eating slowly
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5
Q

Dysphagia

-nutrition management-

A
  • complete nutrition assessment
  • monitor every week (time it takes to eat a meal, size of meal, weight)
  • depends on location of dysphagia - mouth, tongue, larynx
  • type of dysphagia - temporary (car accident) or permanent (stroke)
  • what is the cause
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6
Q

Dysphagia

-nutrition management therapies-

A
  • alteration in food texture - pureed, minced, diced
  • alteration in fluid viscosity - thickened fluids
  • high protein/high energy recommendations - malnourished, anticipated decrease in volume consumed
  • alternative feeding routes - enteral or parenteral
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7
Q

Dysphagia

-suitability of foods-

A
  • increase in salivation (sweet, spicy, sour); may help or hinder swallow
  • even textures are the easiest to swallow (mixed foods are harder); pills with water, soup
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8
Q

GastroEsophageal Reflux Disease (GER, GERD)

-description-

A
  • occurs when gastric acid refluxes into the lower esophagus through the LES
  • when this reflux is chronic, the patient is considered to have GERD
  • changes in the mucosal lining, inflammatory infiltrates, acidic damage
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9
Q

GERD

-symptoms-

A
  • chronic heartburn
  • nocturnal coughing spasms
  • regurgitation
  • chest pain
  • one or all symptoms does not diagnose the disease
  • is reversible
  • overnight coughing in babies
  • can be caused by stress
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10
Q

GERD

-Associated Factors-

A
  • increase in abdominal pressure (LES is regulated partially by pressure); the pyloris generates too much pressure, forcing food up
  • relaxed LES
  • delayed gastric emptying
  • sensitive esophageal mucosa - allergies
  • pyloric obstruction - cancers

may be due to viruses, drugs etc

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11
Q

GERD

-Predisposing conditions-

A
  1. obesity
  2. ascites - excess fluid in the abdominal cavity usually associated with liver disease
  3. scleroderma - disease that leads to hardening of tissues
  4. pregnancy - increased abdominal pressure, delayed gastric emptying
  5. hiatus hernia - part of the stomach passes above the diaphragm
  6. Incompetent LES - congenital

inflammed GI tract = higher risk
underlying cause for GERD needed

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12
Q

GERD
-treatment-

1st line is medication
2nd line is to promote gastric emptying

A

first line of defense in management of symptomology is medication*

  1. dietary modification
  2. Lifestyle modification
    - upright position for 1 hour after eating
    - not eating 2-3 hours prior to going to bed
    - avoid tight clothing in abdominal area
    - stop smoking
    - achieve a healthy body weight
    - elevate head of bed 15-20 cm when sleeping
  3. Drug Therapy - antisecretory drugs that decrease the production of stomach acid
    - proton pump inhibitors / Histamine H2 receptor antagonists —> not good for long term use, bacteria grows
    - motility agents speed up gastric emptying
  4. Surgery (last resort)
    - tightening of LES; wrap upper stomach around
  5. antacids - if issue is milk, acid is not the problem the acid is just in wrong place
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13
Q

GERD

-nutrition care suggestions-

A
  • weight loss diet
  • may require eliminating specific foods from the diet
  • eating smaller meals; decrease abdominal pressure and volume of reflux
  • high dietary fibre decreases symptoms
  • high dietary fat increases symptoms (delays gastric emptying)
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14
Q

GERD

-some foods to lessen in diet-

A
  1. fatty foods - delays gastric emptying, decreases LES pressure, may help with weight loss
  2. chocolate - decreases LES pressure
  3. caffein - decreases LES pressure
  4. spicy or acidic - may irritate esophageal mucosa
  5. alcohol - decreases LES pressure
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15
Q

GERD

-4 complications-

A
  1. esophagitis - inflammation of esophagus
  2. scar tissue - lining of esophagus comes off and forms tissue
  3. stricture - tissue sticks together and narrows the passage
  4. esophageal cancer
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16
Q

PUD

-description-

A
  • due to the h pylori bacteria - dirty water exposure
  • some drugs can increase the risk
  • stomach, lower esophagus, duodenum
  • can just be due to stress
  • break in the protective mucosa and exposure to acid
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17
Q

PUD

-PreDisposing Risk Factors-

A
  • poor nutrition (can make symptoms worse)
  • smoking
  • genetics
  • excessive alcohol intake
  • use of salicylates (aspirin) or NSAIDS (non-steroidal anti-inflammatory drugs)
  • helicobacter pylori infection
18
Q

PUD

-symptoms-

A
  • acute abdominal pain - due to food or fluid intake
  • distention
  • melena stools - black due to old blood
  • hemataemesis - vomitting blood

psychosocial factors can affect treatment and reporting - some people don’t report symptoms

19
Q

PUD

-diagnosis-

A

endoscopy

  • long tube with a camera is inserted into the GI tract
  • biopsies can be taken
20
Q

PUD

-3 areas-

A

esophagus - rare, may occur with chronic and untreated reflux

gastric - most common, defective mucosal lining, decreased blood flow, due to poor nutriton

duodenal - hyper-secretion of acid in stomach, very acidic chyme

21
Q

PUD

-treatment-

A
  1. medication
  2. diet
    - strict diet is not necessary
  3. lifestyle
  4. surgery
22
Q

PUD

-medication treatment-

A
  1. H2 histamine antagonist receptor
    - decreases stimulation of parietal cells
    - ex cimetidine
    ~ works for 60 -70% of pts
  2. antacids - neutralizes acid
  3. antibiotics - four week treatment for H pylori bacteria
23
Q

PUD

-Diet treatment-

A
  • eat balanced diet
  • avoid eating before bed time
  • limit foods that may increase acid secretion; pepper, garlic, chili powder
24
PUD | -lifestyle treatment-
- stop smoking - decrease stress - decrease alcohol intake - decrease intake of aspirin or NSAIDs
25
PUD | -surgery treatment-
- bypass or excise the pyloric sphincter - no regulation of gastric emptying - partial or complete gastrectomy
26
PUD | -Dumping Syndrome-
- simple sugars empty out of the stomach fast, glucose enters the blood fast, fluid and electrolytes follow - occurs in response to undigested food or simple carbs in the duodenum/jejunum - no pyloric sphincter - who food dumped into intestine 10-15 minutes after intake - nausea, diarrhea and cramps due to rapid movement of water into the intestine to dilute it - low blood pressure may cause dizziness, faintness, weakness (hypotension
27
PUD | -gastric surgery diet-
- NPO: ice chips 24-48 hours, clear fluids - intake match output - take 1/2 - 1 serving before or after meals - gradual progression to general diet as tolerated - limit high fat foods - monitor for dumping syndrome - avoid simple carbs - moderate temperature - small and frequent meals - avoid natural laxatives
28
PUD | -steatorrhea-
- partial gastrectomy: fat malabsorption - total gastrectomy or gastrojejunostomy: pancreatic insufficiency, defective enzymes, increase transit time - MCT oil for adequate intake - if only stomach is involved you can supplement with vitmains
29
PUD | -Gastric Surgery nutrient deficiencies-
- iron: HCl in stomach reduceds Fe3 to Fe2 which aids absorption; absorption is therefore reduced - vitamin b12: requires intrinsic factor from the parietal cells of stomach - folic acid: may have decreased intake, is secondary to low B12 intake
30
Lower GI | -6 disorders-
1. inflammatory Bowel Disease - Crohn's disease - UC 2. Celiac Disease 3. Irritable Bowel Syndrome 4. Diverticular Disease 5. Hemorroids 6. Short Gut Syndrome
31
Lower GI | -fat malabsorption-
-normal consumption is 50-100 g/day - 95% absorbed - <7g in stool - malabsorption if >7g of fat in stool (light, flats, liver disease - no bile, no fat absorption) - steatorrhea; loose, hard to flush, strong odor - fecal fat test: diet record of 75-100g of fat for 6 days, collect stool from last 3 days and measure
32
Inflammatory Bowel Disease IBD | -2 types-
- no known cause or cure - malnutrition is common in both when disease is active - bloody diarrhea is common in both - differentiate with scope testing Crohn's - can be anywhere in the lower GI - if cut out of the bowel it CAN reappear elsewhere UC - effects mucosal layer only * - no goblet cells
33
IBD | -malnutrition associates-
- not all malnutrition may be due to the disease (underweight pts due to decreased oral intake) - Crohn's causes dysphagia - increased risk for infection - increased hospital costs - increased morbidity and mortality - decreased immunocompetence - decreased enzyme function - decreased tolerance to medication - altered fluid and electrolyte balance
34
IBS | -pathophysiology-
UC - mucosal inflammation - colon and rectum - continuous lesions - removal may cure; can end up with ostomy Crohn's Disease - transmural inflammation - anywhere in GI tract (70% at terminal ilium) - skip lesions
35
IBD | -complications of UC-
- malabsorption - weight loss - colon cancer - toxic mega colon(colon crumbles, very rare) -* no fistulas, strictures, or obstructions
36
IBD | -complications of CD-
- fistulas - obstructions - strictures - malabsorption - weight loss - maybe toxic mega colon - maybe colon cancer
37
IBD | - 2 complications-
fistula - created by an abces that fills with stool preventing healing and eventually breaks blind loop syndrome - fistulas create a blind loop in the intestine
38
IBD | -inadequate intake and decreased absorbance malnutrition-
low intake - anorexia - nausea - vomiitting - dietary restriction without supplementation - restrictions due to pain (underlying disease causes pain) - pt are both very thin - UC pts don't tend to be malnourished
39
IBD | -excessive losses and increase intake needs-
excess losses - diarrhea - blood loss - trace elements - proteins losing entropathy - leaky bowel, proteins leak into bowel, decreased albumum - bile salts increase intake - inflammation - fever - surgery - infection - repletion of stores - consider other supplements being taken - stress factor due to these
40
IBD | -drug interactions malnutrition-
1. corticosteroids (prednisone) - increased requirement for protein, vitamin B6, zinc, vitaminD 2. Sulfasalzine (NSAID) - folic acid absorb inhibitor 3. Cholestyramine - reduced absorption of fat soluble vitamins rapid transit time
41
IBD | -critical spots of intestine-
Distal Jejunum - major area of nutrient absorption Distal Ileum <100 cm resection - bile wasting - diarrhea, mild steatorrhea Distal Ileum >100 cm resection - severe steatorrhea - depletion of bile - fat soluble vitamin deficiency - electrolyte and fluid imbalance - hypomagnesemia, hypocalcemia Ileocecal Valve - bacterial overgrouth --> diarrhea / steatorrhea - reduced mucosal contact time - colerrheic diarrhea - byproducts can product gases and cause distention and twisting Ascending Colon - fluid and electrolyte imbalance
42
IBD | -3 treatments-
1. Nutrition Therapy 2. Drug Therapy 3. Surgical Therapy - bowel resection with/out ileostomy
43
IBD | -medication-
1. analgesic - pain control 2. antibiotic - stop bacterial growth 3. anti-inflammatory - decrease inflammatory response