head injury + bleeds Flashcards

1
Q

primary focal brain injuries

A

contusion or haematoma

Contusion/bruising on opposite side to primary – due to bouncing off skull
* Coup (adjacent) or contralateral (contre-coup)
o Contracoup tend to be worse

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

primary diffuse brain injuries

A

diffuse axonal injury
 Severe form of traumatic brain injury due to shearing forces
 Occur where density difference is greatest (grey/white interface)
 Excitotoxicity + apoptosis
 Inflammatory mediator release

a result of mechanical shearing following deceleration, causing disruption and tearing of axons

younger patient, reduced consciousness, difficult to manage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

causes of secondary brain injuries

A

cerebral oedema
ischaemia
infection
herniation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

cushing reflex

A

hypertension + bradycardia

-> often occurs late + is usually preterminal event

a physiological nervous system response to acute elevations of intracranial pressure (ICP),

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

extradural haematoma vs acute subdural

A

extradural
- injury with LOC
- recovery with lucid interval then rapid progression of neurological symtpoms

acute subdural = crescent shaped, white
chronic subdural = crescent shaped, dark

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

CT within 1hr criteria

A
  • GCS <13 on initial assessment
  • GCS <15 at 2hrs after injury
  • Suspected open or depressed skull fracture
  • Any sign of basal skull fracture
  • Post traumatic seizure
  • Focal neurological deficit – weak on one side
  • More than one episode of vomiting
  • Suspicion of NAI
  • Also if they experienced some loss of consciousness or amnesia since the injury –
    o >65yrs
    o Coagulopathy
    o Dangerous mechanism of injury
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

glasgow coma scale

A

Eye opening
o Open spontaneously = 4
o To speech
o To pain
o No eye opening= 1
Verbal
o Orientated = 5
o Confused
o Inappropriate
o Incomprehensible
o No verbal = 1
Motor
o Obeying= 6
o Localizing pain
o Flexing
o Abnormal flexing
o Extending
o No motor response= 1

Severity scale
o Mild = 14, 15 of brief LOC
o Moderate = 9-13
o Severe = 3-8

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

extradural haematoma

A

between dura mater + skull

lens/lemon shape on CT

causes
- acceleration-deceleration trauma
- blow to side of head

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

trauma to the pterion can cause which kind of haematoma

A

extradural
- rupture of middle meningeal artery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

ypical history of extradural haematomas

A

young patient with traumatic head injury
ongoing headache
has a period of improved neurological symptoms + consciousness

followed by RAPID decline over hours

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

rupture of bridging veins can cause what type of haematoma?

A

subdural
- bleeding into the outermost meningeal layer
- between dura + arachnoid mater

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

acute subdural presentation

A

cresent white shape on CT
- not limited to cranial sutures - can cross
commonest arounf frontal + parietal lobes

blood is more clotted in one area - big craniotomy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

chronic subdural haematoma

A

elderly falls, assoc with brain atrophy
torn cerebral vain

when becomes chronic becomes more liquified + drain - yellow tinged blood

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

intracerebral haematoma causes

A

(within brain itself)

  • spontaneous, infarct, tumour
  • aneurysm rupture
  • vasculitis
    drugs
  • hypertension, diabetes

hypertension = commonest, rupture of microaneurysms

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

intracerebral presentation + CT findings

A

presents similarily to ischaemic stroke or decreased consciousness

CT shows hyperdensity (bright lesion) within substance of brain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

subarachnoid haemorrhage

A

between pia mater + arachnoid membrane
(where CSF is)

usually by ruptured cerebral aneurysm
high mortality - low suspicion

more common in black, feale ages 45-70

17
Q

commonest cause of subarachnoid haemorrhage

A

rupture of saccular aneurysm (berry aneurysm)

-> most in territory if internal carotid artery

18
Q

conditions assoc with berry aneurysms

A

polycystic kidney disease
Ehlers-Danlos
coarctation of aorta

19
Q

presentation of subarachnoid haemorrhage

A

sudden onset occipital headache that occurs during strenuous activity - weightlifting, sex
–> thunderclap headache (“hit hard on back of head”)

neck stiffness, photophobia
vision changes

20
Q

subarachnoid haemorrhage investigations

A

CT
lumbar puncture
o If CT head is negative, at least 12hrs following onset of symptoms to develop xanthochromia
o Red cell count raised – if decreasing in number of samples probs traumatic from puncture

(Xanthochromia – yellow colour caused by bilirubin, distinguishes from trauma blood)

angiography - once confirmed to locate source

21
Q

management of subarachnoid haemorrhages

A

in accordance of causative pathology
intracranial aneurysms -
- coil or craniotomy + clipping
- nimodipine -> prevents vasospasm, CCB targeting brain vasculature

22
Q

different types of brain herniation

A

subfalcine - down under falxi cerebri
uncal - part of temporal lobe moving medially

central/coning - through foramen magnum, BAD
transcalvarial - skull fracture, pushing out of skull

23
Q

effect of uncal harniation on pupils

A

compression of oculomotor nerve leads to ipsilateral dilated pupil -> blown

24
Q

brain death

A

must be done by 2 doctors
- no pupil response
- no corneal reflex
no motor respone
no vestibuloocular reflex
no gag/cough reflex
no respiration

25
Q

cause of acute spinal cord compression

A

trauma
tumours - haemorrhage or collapse
infection

spontaneous haemorrhage - more common in anti-platelet therapy

26
Q

cord transection

A

complete lesion - all motor + sensory modalities

initially a flacced areflexic paralysis - spinal shock
upper motor neuron signs appear later

27
Q

brown-sequard syndrome

A

caused by lateral hemisection of spinal cord

28
Q

brown-sequard syndrome presentation

A
  • Ipsilateral motor level – weakness below lesion
  • Ipsilateral dorsal column sensory level – loss of proprioception + vibration sensation
  • Contralateral spinothalamic sensory level – loss of pain + temp sensation
29
Q

central cord syndrome

A

hyperflexion or extension of head injury to ALREADY STENOTIC neck or syringomyelia

predominantly bilateral upper limb weakness
cape-like spinothalamic sensory loss

30
Q

anterior cord syndrome

A

occlusion of anterior spinal artery

tracts affected
- lateral corticospinal tracts
- lateral spinothalamic tracts

presents
- bilateral paresis
- bilateral loss of pain + temp sensation

31
Q

when should excision for spinal decompression be done?

A

only if primary cancer or could walk fin an hour ago

32
Q

sudden onset thunderclap headache

A

10/10 severe – worst headache of my life
- Hit in back of the head
- Assoc with N+V

Differential – subarachnoid haemorrhage
o Immediate CT scan

***If neg – need lumbar puncture to obtain CSF sample to prove diagnosis