Heart failure Flashcards

(44 cards)

1
Q

Causes of cardiac muscle dysfunction

A
  • HTN
  • CAD
  • Cardiac dysrhythmias
  • renal insufficiency
  • cardiomyopathy
  • heart valve abnormality
  • pericardial effusion
  • pulmonary embolism
  • pulmonary HTN
  • spinal cord injury
  • age-related changes
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2
Q

Heart failure

A
  • chronic progressive condition
  • heart muscle is unable to pump enough blood to meet needs for blood and oxygen
  • heart failure usually results in an enlarged heart
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3
Q

Heart failure typical path

A
  • normal: ventricles fill with blood and then pump out about 70% of the blood
  • diastolic dysfunction: the stiff ventricles fill with less blood and then pump out 70% but this is less than normal
  • systolic dysfunction: enlarged ventricles fill with blood and then pump out 40-50% of the blood
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4
Q

What does dysfunction in the heart eventually cause

What does it impact first and then what happens from there?

A
  • first impacts SV and EF
  • chronic elevation of pressures will be transmitted up the atria into the vascular system
  • the increase pressure leads to transudate of fluid from the vessels to the interstitial spaces of the lungs or peripheral tissue
  • leads to edema
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5
Q

Classifications of heart failure

A
  • right heart failure = peripheral edema
  • left heart failure = pulmonary edema
  • biventricular
  • systolic vs diastolic dysfunction
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6
Q

heart failure with reduced ejection fracture

A
  • EF<40
  • systolic dysfunction
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7
Q

heart failure with preserved EF >50%

A
  • still denotes lower than normal SV
  • diastolic dysfunction
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8
Q

Right sided heart failure typical presentation

A
  • congestion of peripheral tissues
  • dependent edema and ascites (fluid in abdomen)
  • GI tract congestion = anorexia, GI distress, weight loss
  • liver congestion: signs related to impaired liver function
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9
Q

left sided heart failure presentation

A
  • decreased CO: activity intolerance and signs of decreased tissue perfusion
  • pulmonary congestions: impaired gas exchange (cyanosis and hypoxia) and pulmonary edema (cough with frothy sputum, orthopnea, PND)
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10
Q

What does pulmonary edema sound like

A
  • crackles
  • wet = gurgling
  • dry = fire burning
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11
Q

what sound is expected with CHF

A
  • S3
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12
Q

pulse pressure related to CHF

A
  • normal = 40 (120-80)
  • CHF: systolic decreases and diastolic increases leading to a lower pulse pressure
  • indication heart is not functioning
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13
Q

Look at Left and right -sided heart failure diagram

A
  • look
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14
Q

Clinical manifestations of left sided heart failure

A
  • fatigue and weakness
  • poor tolerance to activity
  • progressive dyspnea
  • orthopnea and PND
  • tachypnea, pallor, cyanosis
  • crackles or rales
  • adventitious heart sounds (s3)
  • hypoxia/hypercapnia
  • voice sounds consistent with consolidation/infiltrates
  • decrease pulse pressure
  • weak pulse
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15
Q

clinical manifestations of right sided heart failure

A
  • fatigue and weakness/poor tolerance to activity
  • dependent peripheral edema/ascites
  • weight gain (fluid)
  • cold, pale and cyanotic extremities
  • JVD
  • decreased peripheral and ventilatory muscle strength and endurance
  • poor activity/exercise tolerance or physical work capacity
  • decreased quality of life/disability
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16
Q

Clinical diagnosis of CHF

A
  • echocardiography: left ventricular EF, structure of LV, other structural abnormalities
  • lab findings: elevated BNP, BUN, hematologic changes (increase RBCs), hypoxia/hypercapnia, changes in liver and electrolyte levels
  • radiologic findings: Evaluation of size and shape of cardiac silhouette; presence of interstitial, perivascular, alveolar edema (fluid in lungs)
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17
Q

NYHA classification of CHF

A
  • class 1: no limitation of physical activity
  • class 2: slight limitation of physical activity, comfortable at rest
  • class 3: marked limitation of physical activity, comfortable at rest (may have dyspnea at rest)
  • class 4: inability to carry any physical activity with discomfort, symptoms present even at rest
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18
Q

ACC/AHA stages of CHF

A
  • Stage A: high risk for developing CHF, no structural disorder to heart
  • stage B: structural disorder or heart, never developed symptoms of CHF
  • Stage C; past or current symptoms of CHF, symptoms associated with underlying heart disease
  • stage D: end-stage disease, requires specialized treatment strategies
19
Q

describe the CHF cycle

A
  • initially may have impaired cardiac function but have CO sufficient for activity

THEN

  • As HF progresses may decompensated with less tolerance to activity
  • this can become a long term cycle of compensated then uncompensated
20
Q

Ability of pump or accept blood depends on

A
  • total blood volume
  • body position and gravity
  • skeletal muscle pump
  • intrathoracic pressure
  • atrial contribution to ventricular filling
  • venous tone
  • intrapericardial pressures
21
Q

associated effects of CHF

On differnet systems

A
  • pulmonary: back up of blood and pressures
  • biochemical/nutritional: anorexia
  • renal: sodium and fluid retention
  • MSK: decrease blood flow, muscle atrophy
  • hematological: abnormal blood cell function and increase blood volume
  • pancreatic: impaired insulin and glucose imbalance
  • hepatic: hypo fusion and/or venous congestion leading to cirrhosis
  • neurochemical: increase sympathetic stimulation
22
Q

HF impact on the heart

A
  • stretching of the myocardium with increase ventricular EDV
  • contractile state of myocardium declines
23
Q

How can the kidneys contribute to the progression of CHF

A
  • sense low arterial blood flow
  • intiate a process to retain fluid
  • this contributes to CHF
24
Q

Pulmonary edema and its relation heart failure

A
  • left ventricle cannot handle overload of blood volume
  • pressure increases in the pulmonary vasculature
  • fluid moves out of the pulmonary capillaries into the interstitial space of lungs and alveoli
25
Stage 1 of pulmonary edema in relation to left sided heart failure
- excessive fluid is present but managed by lymphatic system - pressures are starting to increase
26
Stage 2 of pulmonary edema in relation to left sided heart failure
- characterized by the presence of interstitial edema - increases in pressure causes the fluid to move into the interstitial space - dry crackles with lung sounds
27
Stage 3 pulmonary edema in relation to left sided heart failure
- characterized by the presence of alveolar edema - invades the parenchyma - fluid overload - compromised gas exchange - wet crackles with lung sounds - blood twinged sputum
28
Cardiogenic causes of pulmonary edema
- left ventricular failure - HTN - cardiomyopathy
29
non-cardiogenic causes of pulmonary edema
- smoke or toxin inhalation - idiopathic pulmonary HTN - sepsis/pneumonia (increase permeability) - near drowning (inhaling water)
30
intrapulmonary shunt
- hypoxic blood - O2 goes out of the alveoli normally - with a fluid filled alveoli there is not as much gas exchange and therefore the it is perfused by not ventilated
31
Medical treatment for pulmonary edema
- manage HTN and CAD - diet, nutrition, fluid management - sometimes given a set amount of water to drink so they do not increase fluid too mcc - proper prescription of activity (too high can cause fluid overload) - medications - mechanical/surgical intervention - education and disease self-management is key
32
Management of heart failure
- directed at underlying causes or cause - HTN, atherosclerosis, ischemia, valve dysfunction, arrhythmia etc.
33
What are the goals when managing heart failure
- improve heart pump - reduce the workload for the heart - control sodium intake and water retention - supplement vitamins, minerals and amino acids - decrease sodium intake - fluid restrictions - eating heart healthy foods with low cholesterol and fat
34
Medication that patients with heart failure may be on
- diuretics: lassie, thiazides to eliminate fluids - vasodilators: nitrates to decrease PVR - ACE inhibitors: prevent vasoconstriction - beta blockers: decreases sympathetic stimulation - positive inotropic drugs: increase contractility
35
Look over management diagrams in lecture slides and understand where each comes into play
36
Mechanical/surgical management for CHF
- cardiac assistive devices (pacemakers/defibrillators) - cardiac transplant - assisted circulation (mechanical circulatory derives - MCD's)
37
examples of assisted circulation devices
- intra-aortic ballon pump (can go home with this) - extra corporal membrane oxygenation - ventricular assist device
38
Life vest
- external cardiac defibrillator - these people are waiting for other treatment such as transplant
39
transplantation
- if you work with these patients you are working in a specific center
40
Intra-aortic ballon pump
- systole: deflation to decrease after load, decrease cardiac work, decrease myocardial oxygen consumption and increase CO - diastole: inflation: augmentation of diastolic pressure, increase coronary perfusion, allows more blood flow to the heart - inserted through femoral artery
41
Extra corporal membrane oxygenation (ECMO)
- not going home with this - waiting to get LVAD or transplant - gives heart and lungs a break - ECMH manages all the blood - femoral access - try to avoid this as it limits mobility - commonly used with premature babies since they don't have surfactant
42
Ventricular assist devices
- can be left or right or biventrcualr - heart can rest - blood gets sucked or pumped out of the ventricle - LVAD = left ventricular assistive device
43
Bridge vs destination therapy with HF
- bridge is that the LVAD or ECMO is bridging them to the next step - destination therapy is the last thing they are doing
44
Education and self management techniques for patients with CHF
- assist with patient with taking responsibility for health (monitoring weight and symptoms) - optimization of medical therapy/consistency with meds - vigilant management and follow up - exercise monitoring during and next day/next treatment - early attention to fluid overload