HEART FAILURE Flashcards
(21 cards)
What is the reasons of heart failure
Htn and cad
Systolic dysfunction due to
Reduce contractillity and reduce mass due to mi
Diastolic dysfunction due to
Restriction ventecular fill lead to stiffness and hypertrophy
What is the compenasantary response to maintain circulation
1- tachycardia ,increase contractility
2- increase crease pre load by the water retention and sodium increase stroke volume
3- vasoconstriction
4- hypertrophy and remodelling
What is the primary symptoms of hf
Dyspnea , excersie intolerance other symptoms pulmonary
PND , tachypnea , periphrlk edema , Orthoptera
المريض راح يملكة صعوبة بالتنفس من ينام بس تروح لما يكعد
How to demonstrate the hypertrophy and edema
By radiograph and ecg
How to demonstrate the systolic or diastolic fdysfunction
By echocardialgraph
What is the functional class limitations
FC I : PATIENT HAVE NO ACTIVITY LIMITATION
FCII : PATIENT HAVE SLIGHT LIMITAYION
FCIII PATIENT HAVE MARKED LIMITAYTION
FC IV PATIENT HAVE NO CARRY ON ON ACTIVITY
Even though there is renal impairment it will still work which one
Loop diuretics
The cough 15% - 20% is common to discontinue ACEi , what is the alterninatve
Bradykinine limited breakdown , might be change with sacubitril \ valsartan
What is the recommended ARBS
Candesartan , valsartan , losartan
Best arni is ?
Sacubtril/valsartan
Most common side effect of arni
Cough , hyperkalemia, angioedema {sacubtril/valsartan }, dizziness , renal impairment
Diuressis and Naturesis is enhanced by
The ing of neprilysin enzyme , which is responsible of degrade of bradykinie and vasodilation and Naturesis and diuresis
Arni is contraindicated in ?
Patient with angioedema, renal impairment , due to ACEi and ARBS
Pattering with absence of the cardio genie shock
Held the beta blocker medication
Patient with pulmonary distress
Due to pulmonary edema restrict the fluid intake , <2g
Irreversible advanced HF treated with
Cardiac transplantation
Patient with ADHF which drugs is the first line
Diuretics are the first line of
For patient with diuressis resistant
Can be treated with iv bolus transition to iv diuresis , then with diuretic with different mechanism
What are vasopressin receptors
They are v1a and V2 v1 are located in epithelial and myocardium causes vasoconstriction and positive ionotrop and v2 are located in the renal tubular to control the water abosorption