hemodynamics Flashcards

(47 cards)

1
Q

cardiac output

A
  • best indicator of the condition of the hearts contractile (inotropic ability)
  • volume of blood ejected by the heart in 1 min into the systemic circuit
  • does not equate for differences in body size
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2
Q

normal CO

A

4-8 l/min

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3
Q

cardiac index

A

CO divided by an indiviuals body surface area

normal: 2.2-4

measured by PA cath

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4
Q

CO = ____ x ____

A

SV x HR

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5
Q

what is stroke volume

A
  • volume of blod ejected by the left ventricle during each systole
  • affected by three factors: preload, afterload, and contractility
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6
Q

normal SV

A

60-100 ml

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7
Q

what is preload

A
  • The amount of stretch placed on cardiac muscle fibers just before systole.
  • Volume of blood in the ventricle at the end of diastole
  • Amount of myocardial stretch placed on the ventricular muscle fibers prior to systole application of Frank-Starling law of the heart
  • Volume of blood creates a filling pressure
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8
Q

causes of elevated preload

A
  • volume overload
  • LV dysfunction
  • RV dysfunction
  • valvular defects
  • cardiac tamponade
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9
Q

effects of increase preload on the heart

A

Increases stroke volume
Increases ventricular work
Increases myocardial oxygen requirements

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10
Q

assessment of right sided elevated preload

A

JVD
Edema
Hepato-jugular reflux(HJR)

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11
Q

assessment of left sided elevated preload

A

Orthopnea
Dyspnea
Cough
Crackles
S3

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12
Q

ways to reduce preload

A
  • Drugs that directly reduce blood volume, e.g. diuretics
  • Drugs that promote vasodilation, e.g.
    Nitrates
  • Patient positioning (semi/high fowlers)
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13
Q

causes of decreased preload

A

Hypovolemia
Hemorrhage
Third spacing
Diuresis
Vasodilation

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14
Q

assessment findings of a decreased preload

A

Tachycardia/hypotension
Dry, cool skin
Dry mucous membranes
Poor skin turgor
Alteration in LOC
Decreased urine output/vital organ perfusion

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15
Q

right sided preload assessment

A

Right Ventricular End-Diastolic Pressure (RVEDP)
Right Atrial Pressure (RAP)
Central Venous Pressure (CVP)

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16
Q

left sided preload assessment

A

Left Ventricular End-Diastolic Pressure (LVEDP)
Pulmonary Artery Wedge Pressure (PCWP) aka
Pulmonary Artery Occlusive Pressure (PAOP)
Pulmonary Artery Diastolic Pressure (PADP

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17
Q

treatment to increase preload

A

volume administration
patient positioning - modified trendelenburg

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18
Q

what is afterload

A

Increased ventricular wall tension or stress during systolic ejection

Pressure that the ventricle has to pump against to eject blood into the circulation during systole.

Most critical factor determining afterload is vascular resistance.
- Systemic vascular resistance (SVR)

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19
Q

assessment of afterload

A

Mean arterial pressure (MAP) *not always correlated

Systemic Vascular Resistance (SVR)
- must have an invasive catheter to calculate SVR (must know cardiac output)

Pulmonary Vascular Resistance (PVR)

20
Q

what causes an elevated afterload

A

Vasoconstriction (increased SVR)

Medications
- Alpha 1 agonists

Catecholamine release (compensatory mechanisms)
- Hypovolemia
- Pain
- Hypoxia
- Hypothermia

Hypertension

Increased aortic impedance

21
Q

afterload reduction therapy

A

Vasodilator therapy
- Nitroprusside (Nipride)
- Calcium Channel Blockers (Nicardipine)
- ACE-Inhibitors (-pril)
- Angiotension Receptor Blockers (-sartan)

22
Q

what causes a decreased afterload

A

Vasodilation (decreased SVR)
Inadequate aortic valve function
Inflammatory response
Hyperthermia

23
Q

therapy to increase afterload

A

Vasopressor therapy e.g. Adrenergic stimulants
- Norepinephrine (Levophed)
- Phenylephrine (Neo-synephrine)
- Dopamine
- Vasopressin

24
Q

what is contractility

A

The inotropic action of the heart muscle during systole or the force of each ventricular contraction

25
what factors influence contractility
Intracellular calcium and ATP availability Coronary artery perfusion Cardiac oxygen supply/demand balance Heart rate Blood pressure Valve competence
26
assessment findings with low contractility
Tachycardia Cool, pale skin Decreased urine output Mental status changes Poor peripheral circulation
27
how do we measure contractility
Cardiac output (CO) Cardiac Index (CI) Ejection Fraction (EF)
28
interventions to increase contractility
increase preload positive inotropic agents
29
interventions to decrease conractility
negative inotropic agents
30
inotropic
myocardial contractility
31
chronotropic
heart rate
32
dromotropic
rate of electrical conduction
33
beta 1 receptors
Located primarily in the heart. Stimulation produces: altered ventricular function + chronotropic + inotropic
34
beta 2 receptors
Located in bronchial and vascular smooth muscle. Stimulation produces: bronchodilation
35
alpha 1 receptors
Located primarily in vascular smooth muscle Stimulation produces vasoconstriction Increased SVR
36
what is the phlebostatic axis
Reference level for placement of the transducer to ensure accuracy of measurements Point is located at the intersection of two imaginary reference lines: 1st line - 4th ICS at sternum drawn to side of body 2nd line - drawn midway between anterior and posterior chest (mid-axillary line)
37
what MAP is needed to perfuse coronary arteries
>60
38
ideal MAP
70-90
39
# arterial lines pre procedure
allens test
40
# arterial lines maintenance
Leveling & Zeroing Phlebostatic Axis Compare cuff pressure Check circulation Complications IF BP CHANGES QUICKLY: CHECK PT, CONNECTIONS & LEVEL OF TRANSDUCER
41
what does CVP measure
CVP measures the pressure in the right atrium or vena cava. Provides information regarding intravascular blood volume Indirectly reflects right ventricular end-diastolic pressure (RVEDP) Measures right ventricular preload
42
normal CVP
8
43
which port is used to measure CVP
distal closest to right atrium
44
complications to CVP monitoring
Infection Dislodgement Pneumothorax Thrombosis Air embolism
45
PA pressure monitoring
Flow Directed Pulmonary Artery Catheter “AKA” Swan-Ganz Catheter Used to obtain intra cardiac data to diagnose/evaluate heart disease, shock states and any medical or surgical condition that compromises cardiac function and output; To evaluate patient response to treatment
46
complications with PA monitoring
Pneumothorax Infection Ventricular dysrhythmias Pulmonary artery rupture or perforation Air embolus
47