Hunger Flashcards
Steps in Digestion
- Chewing
- Swallowing
- Absorption (Duodenum)
- Emulsification (Bile)
- Ejection
Energy storage
- Fats
- Glycogen
- Proteins
Metabolism
Chemical changes by which energy is made available for an organisms use
basal metabolism
Consumption of Energy by the basic sustaining functions
Phases of Energy metabolism
Cephalic Phase, Absorptive Phase and Fasting Phase
Cephalic Phase
Sight, Smell,Thought of Food. Insulin is released into bloodstream.
Absorptive Phase
Energy is absorbed to meet bodies immediate energy needs
Fasting Phase
All unstored energy from previous meal is used. Withdrawal of energy from reserves to meet energy requirements. (High levels of glucagon, low levels of insulin in blood )
- Release of free fatty acids from adipose tissue and conversion to ketones
Insulin
Pancreatic Hormone,
facilitates entry of glucose and amino acids into cell, conversion of glucose into glycogen,
transport of fat into adipose tissue
Glucagon
Pancreatic Hormone that promotes conversion of liver glycogen into glucose
Lesion in Ventromedial hypothalamus (VMH)
Chronic hunger, exessive eating ( hyperphagia), massive weight gain
Lesion in lateral hypothalamus (LH)
Chronic satiety, refusal to eat
hypothalamic appetite network
-POMS neurons
-NPY neurons
Arcuate nucleus of hypothalamus
Leptin
Production in fat cells into bloodstream. Regulates bodys energy reserves in form of fat
Ghrelin
Released from Stomach and Duodenum into bloodstream. Appetite stimulant
PYY
In cells of Ileum and colon. Suppresses appetite
CCK
In cells of duodenum. Suppresses appetite. direct action on vagus nerve
Melanin Concentrating hormone
Production in lateral hypothalamus. Stimulates appetite
Orexin
Production in lateral hypothalamus. Appetite stimulant
Factors influencing WHAT we eat
- Learned taste preferences
- pattern of taste preferance is adaptive
- Learning to eat vitamins and minerals
Factors influencing how much we eat
- Satiety signals
- Sham eating
- Appetizer effect
- Serving size
- Social influences
- Sensory specific satiety
Set Point Assumption (glucostatic and Lipostatic)
hunger implies energy deficit Three mechanisms 1.Set point mechanism 2. Detector mechanism 3. Effector mechanism
Positive-incentive value
Anticipated pleasure of a behaviour–>
Not driven by internal energy deficit but anticipated pleasure of eating
Settling points model of body weight
Weight drifts around natural settling point