Increased ICP Flashcards

1
Q

What is ICP

A

The amount of pressure that blood, brain, and CSF exert on the skull

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2
Q

What is the normal ICP range

A

5-15mmhg

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3
Q

Causes of increased ICP

A

Space occupying lesions; bleed, tumor etc
Cerebral edema; trauma, stroke, ischemia
Metabolic disorders; uremia, hepatic encephalopathy
Increased CSF production from tumor
Decreased CSF drainage; lesion, Chiari
Increased venous pressure; clot, stenosis, HF
Idiopathic intracranial hypertension

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4
Q

What opthalmic findings suggest increased ICP

A

Fixed dilated pupils
Papiledema

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5
Q

What neuroimaging is suggested for increased ICP

A

CT: edema, loss of sulci, loss of white/grey matter, enlarged ventricles
MRI: enlarged ventricles, herniation, mass effect, tumors

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6
Q

When should an LP be considered in r/o increased ICP

A

If neuro imaging is negative

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7
Q

What unit should a patient with increased ICP be admitted to

A

ICU

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8
Q

What are three devices that can measure ICP

A

Fiber optic catheter: inserted into brain parenchyma
EVD: connects directly into the ventricles
Optic nerve sheath diameter: measures pressure behind optic nerve

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9
Q

What is CPP and what level should it be maintained at

A

CPP= MAP - ICP
~60-7pm HMG

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10
Q

What vent changes can be made to decrease ICP transiently

A

Hyperventilation

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11
Q

Pharmacological therapy for increased ICP

A

Mannitol: creates osmotic gradient
Steroids: if there is a tumor present
3% hypertonic saline
Acetazolamide: decreases CSF production
Glyburide
Barbituates

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12
Q

What risk do you run providing an LP to decrease ICP

A

Rapid decrease in CSF with reflex increase in ICP producing herniation

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13
Q

Surgical options for increased ICP

A

Shunt
Optic fenestrations
Crainiotomy

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14
Q

How is Cushing’s connected to increased ICP

A

Reflex to produce better blood flow to brain
Late stage of increased ICP
Imminent herniation

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15
Q

What are the s/s of Cushing’s triad

A

Widened pulse pressure, bradycardia, irregular respirations

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16
Q

Describe the difference between first, second, and third stage Cushing’s

A

First: Hypertension and tachycardia until MAP supersedes ICP
Second: hypertension and bradycardia
Third: increased ICP, compression of brain stem, irregular respirations