Integumentary Flashcards

(46 cards)

1
Q

role of lymphatic system

A

drainage/sanitation
one way system to transport lymph w proteins/water/fatty/acids/cellular components through lymph nodes, thymus, bone marrow, spleen, tonsils, Small intestine
- remove waste, excess fluid
- alert immune system
- return fluid and plasma proteins to blood

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2
Q

lymphatic system anatomy small to large

A

lymph capillary
pre collectors
collectors
lymph nodes
lymphatic trunks
R lymphatic duct and thoracic duct

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3
Q

lymph formation

A

precollectors fill with low interstitial pressure
interstitial pressure increases with filling, inlet valves open w Pressure
fill lymph vessel w fluid, lowering pressure and close valves
valves open to precollector and fluid flows onward

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4
Q

transport capacity

A

amount of fluid the system can move at maximum intensity
normally works at 10% capacity

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5
Q

functional reserve

A

difference between transport capacity and amount of fluid being transported at rest

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6
Q

lymphatic load

A

amount of fluid being transported at rest

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7
Q

dynamic insufficiency

A

caused by immobility, CHF, sprained ankle, increased LL

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8
Q

mechanical insufficiency

A

caused by damage to lymphatic system making it unable to handle increased LL
eg lymphedema
also surgery, infection, trauma causing reduced TC

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9
Q

causes of lymphedema

mechanical cause within lymphatic system

A

TC dropping below LL
causes accumulation of fluid in subcutaneous tissue
risk factors include: excess weight, arm infection/injury

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10
Q

diagnosis of lymphedema

primary/secondary

A

primary: from abnormally developed lymphatic system
secondary: results from known injury

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11
Q

clinical presentation of lymphedema

A

slow progression
mild warmth
no color change
painless
full/heavy feeling
pitting edema
asymmetrical limbs

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12
Q

differences between lymphedema and general edema

A

risk factors
other diagnoses
Stemmer sign: + for lymphedema
acute injury is general
general chronic edema comes with skin changes, achy pain, progressing through day

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13
Q

stages of lymphedema

A

0: no edema, - stemmer
1: soft pitting edema, reversible w elevation, increase w activity, - stemmer
2: spontaneously irreversible, edema progressing to nonpitting brawny edema, not reversed w elevation, + stemmer, fibrosclerotic tissue, frequent infection/skin changes
3: lymphostatic elephantiasis, severe brawny edema not reversed w elevation, + stemmer, fibrosclerotic tissue, frequent infection, skin changes

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14
Q

Stemmer sign

A

+ if skin cannot be pinched and lifted at base of fingers or toes

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15
Q

lymphedema treatment

A

depends on cause, which should be addressed
medications

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16
Q

cellulitis

A

bacterial skin infection with open wound

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17
Q

cellulitis s/s

A

red, edema, tender, pain, warm, blister, fever, headache, chills, weakness, red streaks

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18
Q

cellulitis diagnosis

A

blood tests, skin culture

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19
Q

cellulitis treatment

A

antibiotics (oral/topical/IV)
wound dressing
pain meds
surgery

20
Q

complications of cellulitis

A

extensive tissue damage, gangrene, blood infection causing sepsis, amputation, deat

21
Q

prevention of cellulitis

A

hygiene, protect dry skin, protective footwear, wound prevention

22
Q

function of the epidermis

A

skin integrity
physical barrier to pathogens
protect against cellular fluid loss

23
Q

function of the dermis

A

tensile strengh/support
retain moisture, blood O2

24
Q

normal wound healing timeline

A

1: inflammatory 1-10 days
2: proliferative 3-21 days
3: maturation 7 days - 2 years

25
factors affecting wound healing
age comorbidities edema bad wound care infectionlifestyle stress medications
26
what happens when healing phases are interrupted?
1: chronic inflammation cycle can occur 2: delay healing resulting in chronic wound 3: scar tissue remodeling, only 80% strength of normal tissue
27
ideal wound healing environment
moist breathable barrier controlled exudate for peri wound integrity change bandage when leakage
28
wound characteristics to take note off
location size shape edges tunneling base compared to sides periwound area pain
29
serous drainage
clear/watery inflammatory/proliferative phases
30
sanguineous drainage
red/watery inflammatory/proliferative phases
31
serosanguineous drainage
clear/pink/watery inflammatory/proliferative phases
32
seropurulent drainage
cloudly/opaque/yellow/waterly early warning sign of infection, abn finding
33
purulent drainage
yellow/green/thick wound infection, abn finding
34
necrotic tissue types
slough: easily removed moist/stringy white or yellow tissue attached to wound eschar: hard dehydrated tissue adhered to healthy tissue gangrene: death of tissue due to interrupted blood dlow hyperkeratosis: white callus, firm/soggy texture
35
primary intention wound closure
surgeon closes edges by approximating with flue/stitches, etc opening called dehiscence
36
secondary intention wound closure
wound left open to heal on its own new tissue laying down in wound bed closes eventually heal deepest to superficial
37
tertiary intention wound closure
delayed primary secondary intention fails and it is closed surgically may be purposefully delayed due to infection, closed once healed
38
abn findings in wound healing
changing: color, odor persistent edema necrotic tissue tunneling infection ridge at wound edge hypertrophic scarring
39
venous ulcer
edema, pain in dependent position, cyanotic, pigmentation of skin, exudate
40
arterial ulcer
poor pulse, severe pain, cool to touch, trophic changes to skin, deep ulcers, black gangrenous skin
41
diabetic ulcer
diminished pulse sensory loss ulcer present may develop gangrene
42
pressure ulcer
pain if sensation intact color change warm if infection/fever necrotic tissue over bony prominences can be gangrene
43
Braden Scale
predict pressure sore risk use sensory perception, moisture, activity, mobility, nutrition, friction/shear
44
stages of pressure ulcers
1: intact skin, nonblanchable redness 2: partial thickness tissue loss, no slough 3: full thickness tissue loss, visible structures underneath 4: full thickness w exposed bone, slough or eschar unstageable: full thickness, covered in slough deep tissue injury: purple area of discolored skin due to soft tissue damage
45
risk factors for diabetic ulcers
poor footwear/deformity/foot trauma non compliance w dx management lack of protective sensation skin changes Hx of amputation alc/tobacco immobile advanced age weakened immune vascular disease
46
wagner diabetic ulcer grades
0: no open lesion, may be preulcerative 1: superficial ulcer, not subcutaneous 2: deep ulcer into subcutaneous, may expose structures 3: deep ulcer w abscess/bone infection 4: gangrene --> maybe amputation 5: gangrene w deep tissue injury --> definitely amputation