Intracranial Neurosurgery ppt 2 Flashcards
(39 cards)
Intracranial aneurysms are defined as:
focal protrusions arising from weakened arterial walls usually at major bifurcations of the arteries at the base of the brain
Intracranial aneurysms are most commonly treated by:
endovascular coiling (>50%) or microsurgical clip ligation.
Most important surgical considerations for intracranial aneurysms:
clinical presentation, aneurysm size and location, patient age, neurologic status, and medical comorbidities
Most common intracranial aneurysm clinical presentation:
Aneurysm rupture
System used for prognostic clinical outcome
Hunt Hess grading system (1 asymptomatic, 5 deep coma)
anterior intracranial aneurysms position:
supine, mayfield headrest, turned 30-45* to side away from aneurysm
posterior intracranial aneurysms position:
supine or lateral with mayfield headrest
EBL intracranial aneurysm:
250-1000 ml
Intracranial aneurysms considerations:
arterial clipping, mild hypothermia, intro angiography with access to femoral artery, electrophysiologic monitoring, brain relaxation, lumbar subarachnoid CSF drainage, decadron IV
intracranial aneurysm prep anesthesia considerations:
- 23% have neurogenic pulmonary edema
- PVCs, T wave inversion and ST depression are common
- Decreased Magnesium
- May present with increased ICP or cerebral vasospasm
- Digital subtraction angiography is gold standard for detection
- Pt should be Euvolemic
- hyponatremia from SIADH
- aline for tight BP control
increase in BP in intracranial aneurysm pt preop
rebleed, permanent neurologic deficits, or death
substantial decrease in BP in intracranial aneurysm prep:
cerebral ischemia or infarction
intracranial aneurysms induction:
- Smooth induction
- Decrease cerebral blood volume by inducing cerebral vasoconstriction
- Pts on nimodine may require pressors ie phenylephrine during and after induction
- Patients may benefit from moderate hyperventilation during induction
intracranial aneurysms maintenance:
- Iso or Sevo ½ Mac if EP monitoring
- Avoid N2O
- Propofol gtt: ↓ cerebral blood volume, ↓ cerebral metabolism, and ↓ CMRO2
↓ PaCO2 leads to:
↓ cerebral vascular volume (better surgical access) + ↑ CBF to ischemic areas (“Robin Hood” effect) + ↓ anesthetic requirements + ↑ lactic acid buffering.
Mannitol/furosemide leads to:
↓ K+; monitor level and replace as necessary.
If mannitol is administered too rapidly, what could occur?
↓ BP may occur, 2° peripheral vasodilation.
Mild hypothermia (33–34°C) is used to:
to ↓ CMRO2and to ↓ susceptibility to ischemic injury during temporary clip application.
At 30*C, CMRO2 decreases
~ 30%
BP control: During aneurysm exposure
decrease MAP to ~80% baseline
BP control: During temporary clipping
increase MAP to ~120% of baseline
BP control: postclipping
MAP typically 70-90 mmHg
BP control: if aneurysm ruptures
decrease MAP to 40–50 mm Hg
consider carotid compression
Deep hypothermia and CPBP is used only with what aneurysms?
> 2.5 cm in diameter