Intracranial Neurosurgery ppt 2 Flashcards

(39 cards)

1
Q

Intracranial aneurysms are defined as:

A

focal protrusions arising from weakened arterial walls usually at major bifurcations of the arteries at the base of the brain

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2
Q

Intracranial aneurysms are most commonly treated by:

A

endovascular coiling (>50%) or microsurgical clip ligation.

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3
Q

Most important surgical considerations for intracranial aneurysms:

A

clinical presentation, aneurysm size and location, patient age, neurologic status, and medical comorbidities

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4
Q

Most common intracranial aneurysm clinical presentation:

A

Aneurysm rupture

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5
Q

System used for prognostic clinical outcome

A

Hunt Hess grading system (1 asymptomatic, 5 deep coma)

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6
Q

anterior intracranial aneurysms position:

A

supine, mayfield headrest, turned 30-45* to side away from aneurysm

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7
Q

posterior intracranial aneurysms position:

A

supine or lateral with mayfield headrest

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8
Q

EBL intracranial aneurysm:

A

250-1000 ml

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9
Q

Intracranial aneurysms considerations:

A

arterial clipping, mild hypothermia, intro angiography with access to femoral artery, electrophysiologic monitoring, brain relaxation, lumbar subarachnoid CSF drainage, decadron IV

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10
Q

intracranial aneurysm prep anesthesia considerations:

A
  • 23% have neurogenic pulmonary edema
  • PVCs, T wave inversion and ST depression are common
  • Decreased Magnesium
  • May present with increased ICP or cerebral vasospasm
  • Digital subtraction angiography is gold standard for detection
  • Pt should be Euvolemic
  • hyponatremia from SIADH
  • aline for tight BP control
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11
Q

increase in BP in intracranial aneurysm pt preop

A

rebleed, permanent neurologic deficits, or death

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12
Q

substantial decrease in BP in intracranial aneurysm prep:

A

cerebral ischemia or infarction

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13
Q

intracranial aneurysms induction:

A
  • Smooth induction
  • Decrease cerebral blood volume by inducing cerebral vasoconstriction
  • Pts on nimodine may require pressors ie phenylephrine during and after induction
  • Patients may benefit from moderate hyperventilation during induction
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14
Q

intracranial aneurysms maintenance:

A
  • Iso or Sevo ½ Mac if EP monitoring
  • Avoid N2O
  • Propofol gtt: ↓ cerebral blood volume, ↓ cerebral metabolism, and ↓ CMRO2
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15
Q

↓ PaCO2 leads to:

A

↓ cerebral vascular volume (better surgical access) + ↑ CBF to ischemic areas (“Robin Hood” effect) + ↓ anesthetic requirements + ↑ lactic acid buffering.

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16
Q

Mannitol/furosemide leads to:

A

↓ K+; monitor level and replace as necessary.

17
Q

If mannitol is administered too rapidly, what could occur?

A

↓ BP may occur, 2° peripheral vasodilation.

18
Q

Mild hypothermia (33–34°C) is used to:

A

to ↓ CMRO2and to ↓ susceptibility to ischemic injury during temporary clip application.

19
Q

At 30*C, CMRO2 decreases

20
Q

BP control: During aneurysm exposure

A

decrease MAP to ~80% baseline

21
Q

BP control: During temporary clipping

A

increase MAP to ~120% of baseline

22
Q

BP control: postclipping

A

MAP typically 70-90 mmHg

23
Q

BP control: if aneurysm ruptures

A

decrease MAP to 40–50 mm Hg

consider carotid compression

24
Q

Deep hypothermia and CPBP is used only with what aneurysms?

A

> 2.5 cm in diameter

25
intracranial hemorrhage emergence:
Hunt Hess 4 and 5- remain intubated and sedated post-op Hunt Hess 1-3- titrate BB and vasodilators as needed smooth emergence, no cough HOB elevated 30* on transport
26
Intra-op Concerns for AVM
Fiberoptic awake intubation if patient arrives with stereotactic frame. Stereotactic frame is placed in pre-op awake with local anesthesia then patient goes to CT or MRI. VAE Central line Aline Mild hypothermia Brain Relaxation (decrease PaCO2, mannitol/furosemide)
27
Emergence for AVM
Increases in BP treat with B-Blockers or vasodilators Close regulation of BP is key Suppress cough with lidocaine IV HOB elevated 20-30 degrees BP should be maintained 10% below baseline
28
Craniotomy for Tumor Pre-Op Considerations
Neurogenic pulmonary edema | Increase ICP leading to Cushings Triad (HTN, Resp Depression & Bradycardia)
29
Anesthesia for Craniotomy
Stereotactic frame are placed on the patient’s head The patient is taken to CT/MR for determination of the exact tumor site. Surgeons may visualize certain tumors by utilizing iv fluorescent dyes (e.g., fluorescein) When used in conjunction with a specialized operating microscope, these compounds allow greater definition of the tumor and its boundaries with normal brain tissue. Anaphylactic reactions can occur with the use of these agents.
30
Stereotactic Surgery Considerations
Risk of air embolus Brain stem manipulation causes BP and pulse instability Dural patch is used to expand dura at foramen magnum
31
Craniotomy Intraoperative concerns
``` Awake Fiberoptic Intubation if stereotactic frame present Minimize Increases in ICP and MAP Pre-cordial doppler BAER, SSEP, MEP CVP ```
32
Emergence Craniotomy
Increases in BP treat with B-Blockers or vasodilators Close regulation of BP is key Suppress cough with lidocaine IV HOB elevated 20-30 degrees Surgeon may request transient increase in MAP 90-100mmHg to test hemostasis after the tumor is resected
33
Awake Craniotomy
May be used for epilepsy Tumors involving motor or speech Done under MAC or (asleep- awake- asleep) technique LMA or ET
34
Awake Craniotomy- Premapping
``` Mannitol slow IV infusion Midazolam & Dexmedetomidine Propofol gtt Scalp block Aline Foley Mayfield headrest under Local anesthesia ```
35
ventricular shunt- diagnosis:
hydrocephalus
36
2 approaches to ventricular shunt:
Ventriculoperitoneal or Ventriculoatrial
37
Ventricular shunt pre-op:
Cushings triad Headache with ICP > 15mmHg No pre-op med required
38
Ventricular shunt induction:
minimize ICP | maintain normovolemia
39
Ventricular shunt maintenance:
PeCO2 35-40 mmHg Hyperventilation and hypocarbia make cannulation of vessel difficult Maintain normotension: Catheter tunneling is stimulating