Intro, degenerative part 1 Flashcards

1
Q

General description of inflammatory joint disease

A

Soft tissue swelling, edema, uniform loss of joint space, erosions juxta-articular osteoporosis
Monoarticular or para-articular
Systemic pattern
Greater predisposition to ankylosis

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2
Q

General description of degenerative joint disease

A

Nonuniform loss of joint space, osteophytes, subchondral sclerosis, cysts
Asymmetric

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3
Q

Describe T1 MR

A

Short time repitition, short time echo

Black CSF, white bone marrow

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4
Q

Describe T2 MR

A

Long time repitition, long time echo

White CSF, black bone marrow

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5
Q

General description of metabolic joint disease

A

Noteable soft tissue masses within periarticular soft tissues
Well marginated bone lesions
Relative preservation of joint space
Overlapping degenerative and inflammatory changes is common
AKA lumpy bumpy arthritis

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6
Q

General description of infectious/septic joint disease

A

Known cause of grossly destroyed and disintegrated joints
Greatest incidence below 30
Monoarticular moat common
Bloodborne pathogens and direct implantation
Staphylococcus aureus most common organism
Radiographic findings lag behimd clinical

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7
Q

ABCDs pf joint doseases

A
Alignment
Bone
Cartilage
Distribution
Soft tissue
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8
Q

Generalized age onset of each joint disease

A

0-20 JRA
20-40 seronegative, seropositive
>40 degwnerative, DISH, gout, CPPD

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9
Q

What joints are usually affected in degnerative arthritis?

A

Weight bearing articulations of the spine, hips and knees
1st MTP, 1st MC trapezium
DIP

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10
Q

How do you get degenerative arthritis? What are some symptoms?

A

Insidious onset, intermittent exacerbations, aching pain, stiffness, environmental
Changes such as cold and lowered barometric pressure as it may aggravate symptoms

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11
Q

underlying etiology of primary osteoarthritis

A

no evidence of underlying etiology

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12
Q

underlying etiology of secondary osteoarthritis

A

abnormal forces including obesity, trauma, and joint deformity
pre-existing cartilage pathology such as RA, fractures and meniscal damage
collapse of subchondral bone, such as avascular necrosis and osteoporosis

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13
Q

risk factors of DJD

A
age (increased DJD with age)
gender (females)
obesity
trauma (most significant local factor)
physical activity (higher impact)
inactivity or excessive activity
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14
Q

clinical features of DJD

A
poor radiographic-clinical correlation
stiffness, especially with rest
normal blood work
spinal stenosis
vertebrobasilar ischemia
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15
Q

osteophytes have a _____ cortex and internal structure continuous with the parent bone and capped with____.

A

distinct

cartilage

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16
Q

general radiology features of DJD

A
asymmetric distribution
asymmetric loss of joint space
osteophytes
subchondral sclerosis
subchondral cysts (geodes)
subluxation (midalignments)
intra-articular loose bodies
entheseopathy
ankylosis (rare)
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17
Q

osteophyte definition

A

bony outgrowths in the locality of capsular insertion. bony excrescences with internal trabeculation and cortex, with a cartilage cap pathologically from carilage metaplasia and ligamentous traction and infrequently will create ankylosis

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18
Q

subchondral sclerosis definition

A

increased mechanical stresses cause thickened existing trabeculation and formation of new ones, localized compensatory increase in bone mass

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19
Q

subchondral cysts definition

A

focal regions of bone loss within the subchondral bone. they are ovoid or rounded cysts often with a sclerotic border due to intraosseous synovial fluid intrusion through exposed articular plate

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20
Q

how are intraarticular loose bodies made?

A

from cartilage degradation, flaking, and fragmentation and occasionally include subchondral bone

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21
Q

articular deformity results form what?

A

time and continued repetitive stress deforms secondary to trabecular remodeling, fracture and collapse

22
Q

joint subluxations from (1,2,3,4) render the joint prone to displacement

A
  1. progressive loss of joint space
  2. capsular laxity
  3. ligamentous laxity
    4 joint deformation
23
Q

entheseopathy definition

A

pathological change of an enthesis

24
Q

degeneration of hands is common among what?

A

middle-aged postmenopausal women

25
DIP soft tissue nodes
Heberden's nodes
26
PIP soft tissue nodes
Bouchard's nodes
27
gull wing sign
bilateral DJD with central joint erosions
28
target locations on hands for DJD
DIP PIP 1st MCP osteophytes, sclerosis, cystic changes, loss of joint space, misalignment
29
radiographic changes in DJD
``` osteophytes decreased joint space sclerosis lateral displacement cystic changes ```
30
describe erosive osteoarthritis
variant of DJD appears unusually inflammatory not to be confused with RA
31
symptoms/signs of erosive osteoarthritis
gull wing sign pain, redness, swelling and decreased motion middle aged females
32
describe degeneration of feet
common in first MTP joint known as hallux rigidus when present with symptoms of pain and stiffness metatarsal varus, hallux valgus joint misalignment is common bunion
33
where are heel spurs specifically?
on the inferior part of the calcaneous on the posterior side of it
34
spurs are an _______change
enthesopathic
35
which joint of the shoulder is most commonly associated with DJD?
AC joint
36
how does DJD affect the AC joint?
osteophytes extend inferiorly and may impinge on rotator cuff tendons leading to tendon calcification and superior migration of the humerus
37
how does CPPD affect the glenohumeral joint?
osteophytes and joint misalignment and arouse suspicion of significant previous trauma or CPPD
38
what is shoulder impingement syndrome?
elevation of the humeral head degenerative enthesopathic changes of the humeral head spurring of the AC joint rotator cuff degeneration
39
where does the supraspinatus tendon ususally insert? where does it break and why?
inserts on the greater tuberosity of the humeral head | usually tears about an inch from the tuberosity because that's where there is less blood flow
40
how does a supraspinatus tnedon tear show up on MR?
``` increased whiteness (the tendon is usually black) ```
41
what is HADD?
deposition of calcium within tendons and bursa
42
wherre is HADD commonly seen?
shoulder, elbow, rist, hip, knee, ankle, spine
43
where is HADD most common?
supraspinatus tendon
44
what age range usually experiences HADD?
40-70 years old
45
what are the signs and symptoms of HADD?
pain, tenderness, localized swelling, reduced ROM | calcification of soft tissue on radiograph
46
what are the 3 parts of the knee?
medial tibiofemoral lateral tibiofemoral retropatellar
47
what are radiographic signs of DJD of the knee?
asymmetric loss of joint space subchondral sclerosis articular deformity and iregularity hypertrophic changes of the intercondylar spines enthesopathy of the anterior non-articular surface of the patella
48
what part of the knee most commonly experiences DJD?
medial compartment causing genu varus
49
pelligrini-steida calcifications
calcification of medial tibial collageral ligament (HADD) | post-traumatic dystrophic changes
50
what do you need to differentiate for pelligrini-steida?
fabella
51
what are intraarticular fragments?
fragments of cartilage, meniscus or synovium within the joint heterogeneously dense, popcorn ball looking
52
what is synovial osteochondrometaplasia?
laminated, stippled, concentric calcific densities