Intro lecture Flashcards
articular vs extraarticular process
articular: pain & limitation of AROM + PROM
extraarticular: pain & limitation of AROM mostly
if someone has raynaud’s what should you check for?
possible autoimmune disease– if they have an underlying disorder
raynaud’s phenomenon vs disease
phenomenon is just what happens. the physiology
disease is when secondary causes have been ruled out
tx for raynauds (1st, 2nd line & if severe)
1st line: prevention + smoking cessation
2nd: DHP CCB
if severe: sildenafil
tx associated complications
3 questions to consider when assessing joint pattern of involvement
1) is there inflammation?
2) how many joints?
3) what joints?
4 cardinal signs of inflammation
erythema
swelling
warmth
pain
other than ESR/CRP, what are 3 other lab evidence of inflammation
hypoalbuminemia
anemia of chronic dz
thrombocytosis
inflammatory or noninflammatory
- WORSE w/ rest
- AM stiffness over 60 mins
inflammatory pain
what is this? what are the common sites?
common degeneative d/o of articular cartilage associated w/ hypertrophic changes in the bone
RF: genetics, past trauma, advancing age, obesity
joint narrowing
OA
sites: DIP, CMC
differentiate heberden’s & bouchard’s nodes. what condition are they both associated with?
OA– they are bony growth on joints.
if on DIP– heberDens
if on PIP– bouchards
- crepitus
- osteophytes
- decreased ROM
- malalignment
- tender to palpate +/- joint effusion
exam findings for OA
3 XR views for assessing OA
AP, lateral & sunrise or merchant
4 pharmacologic tx for OA
Acetaminophen 1 gm TID
NSAIDs
Topicals– capsaicin, diclofenac
injections– steroids or hyaluronate
for these conditions, state which are poly- or monoarthritic
septic arthritis
erosive OA
SLE
RA
spondyloarthropathy
OA
mono– septic & erosive
the rest are poly
what is the condition? what are the two primary joints?
- morning stiffness over 1 hr
- symmetric, polyarticular arthritis
- (+) RF & anti CCP
RA– MCP, PIP
